Interactions between prolactin and kisspeptin to control reproduction (2016)
- Authors:
- USP affiliated authors: DONATO JÚNIOR, JOSÉ - ICB ; FRAZÃO, RENATA - ICB
- Unidade: ICB
- DOI: 10.1590/2359-3997000000230
- Subjects: ANATOMIA; FISIOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Archives of Endocrinology and Metabolism
- ISSN: 2359-4292
- Volume/Número/Paginação/Ano: v. 60, n. 6, p. 587-595, 2016
- Status:
- Artigo publicado em periódico de acesso aberto (Gold Open Access)
- Versão do Documento:
- Versão publicada (Published version)
- Acessar versão aberta:
-
ABNT
DONATO JUNIOR, José e FRAZÃO, Renata. Interactions between prolactin and kisspeptin to control reproduction. Archives of Endocrinology and Metabolism, v. 60, n. 6, p. 587-595, 2016Tradução . . Disponível em: https://doi.org/10.1590/2359-3997000000230. Acesso em: 01 abr. 2026. -
APA
Donato Junior, J., & Frazão, R. (2016). Interactions between prolactin and kisspeptin to control reproduction. Archives of Endocrinology and Metabolism, 60( 6), 587-595. doi:10.1590/2359-3997000000230 -
NLM
Donato Junior J, Frazão R. Interactions between prolactin and kisspeptin to control reproduction [Internet]. Archives of Endocrinology and Metabolism. 2016 ; 60( 6): 587-595.[citado 2026 abr. 01 ] Available from: https://doi.org/10.1590/2359-3997000000230 -
Vancouver
Donato Junior J, Frazão R. Interactions between prolactin and kisspeptin to control reproduction [Internet]. Archives of Endocrinology and Metabolism. 2016 ; 60( 6): 587-595.[citado 2026 abr. 01 ] Available from: https://doi.org/10.1590/2359-3997000000230 - Neuronal STAT5 signaling is required for maintaining lactation but not for postpartum maternal behaviors in mice
- Changes in Leptin Signaling by SOCS3 Modulate Fasting-Induced Hyperphagia and Weight Regain in Mice
- Fatness rather than leptin sensitivity determines the timing of puberty in female mice
- Brain STAT5 signaling and behavioral control
- Leptin resistance is not the primary cause of weight gain associated with reduced sex hormone levels in female mice
- A short-day photoperiod delays the timing of puberty in female mice via changes in the kisspeptin system
- Conspecific odor exposure predominantly activates non-kisspeptin cells in the medial nucleus of the amygdala
- STAT5 signaling in kisspeptin cells regulates the timing of puberty
- Effects of growth hormone in the central nervous system
- Central regulation of metabolism by growth hormone
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