IL-4/CCL22/CCR4 axis controls regulatory T-cell migration that suppresses inflammatory bone loss in murine experimental periodontitis (2015)
- Authors:
- USP affiliated authors: CAMPANELLI, ANA PAULA - FOB ; GARLET, GUSTAVO POMPERMAIER - FOB
- Unidade: FOB
- DOI: 10.1002/jbmr.2376
- Subjects: CITOCINAS; LINFÓCITOS T; PERIODONTITE; REABSORÇÃO ÓSSEA ALVEOLAR
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Bone and Mineral Research
- ISSN: 0884-0431
- Volume/Número/Paginação/Ano: v. 30, n. 3, p. 412-422, Mar. 2015
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
-
ABNT
ARAUJO‐PIRES, Ana Claudia et al. IL-4/CCL22/CCR4 axis controls regulatory T-cell migration that suppresses inflammatory bone loss in murine experimental periodontitis. Journal of Bone and Mineral Research, v. 30, n. 3, p. 412-422, 2015Tradução . . Disponível em: https://doi.org/10.1002/jbmr.2376. Acesso em: 12 ago. 2024. -
APA
Araujo‐Pires, A. C., Vieira, A. E., Francisconi, C. F., Biguetti, C. C., Glowacki, A., Yoshizawa, S., et al. (2015). IL-4/CCL22/CCR4 axis controls regulatory T-cell migration that suppresses inflammatory bone loss in murine experimental periodontitis. Journal of Bone and Mineral Research, 30( 3), 412-422. doi:10.1002/jbmr.2376 -
NLM
Araujo‐Pires AC, Vieira AE, Francisconi CF, Biguetti CC, Glowacki A, Yoshizawa S, Campanelli AP, Trombone APF, Sfeir CS, Little SR, Garlet GP. IL-4/CCL22/CCR4 axis controls regulatory T-cell migration that suppresses inflammatory bone loss in murine experimental periodontitis [Internet]. Journal of Bone and Mineral Research. 2015 ; 30( 3): 412-422.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1002/jbmr.2376 -
Vancouver
Araujo‐Pires AC, Vieira AE, Francisconi CF, Biguetti CC, Glowacki A, Yoshizawa S, Campanelli AP, Trombone APF, Sfeir CS, Little SR, Garlet GP. IL-4/CCL22/CCR4 axis controls regulatory T-cell migration that suppresses inflammatory bone loss in murine experimental periodontitis [Internet]. Journal of Bone and Mineral Research. 2015 ; 30( 3): 412-422.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1002/jbmr.2376 - CCR5+ cells modulate RANKL+ cell migration, inflammatory reaction development and experimental periodontitis outcome in mice
- The potential role of suppressors of cytokine signaling in the attenuation of inflammatory reaction and alveolar bone loss associated with apical periodontitis
- Involvement of CCR5 and CXCR3 in RANKL+ cell migration and bone resorption throughout experimental periodontitis in mouse
- Associação diferencial de fatores genéticos, microbianos e inflamatórios com os níveis de MMP-1 no periodonto saudável e com periodontite
- Distinct roles for genetic (MMP1-1607 and -519 SNPs) factors and periodontopathogens in the modulation of MMP-1 levels in healthy and diseased periodontal tissues
- The broad effects of the functional IL-10 promoter -592 polymorphism: modulation of IL-10, TIMP-3 and OPG expression and its association with periodontal disease outcome
- Evidence supporting a protective role for Th9 and Th22 cytokines in human and experimental periapical lesions
- IL-10 and TNF-α differentially modulate the outcome of alveolar bone repair upon homeostatic and infectious conditions
- Characterization of tumor-infiltrating regulatory T-cell subpopulation in oral squamous cell carcinoma
- Strong and persistent microbial and inflammatory stimuli overcome the genetic predisposition to higher matrix metalloproteinase-1 (MMP-1) expression: a mechanistic explanation for the lack of association of MMP1-1607 single-nucleotide polymorphism genotypes with MMP-1 expression in chronic periodontitis lesions
Informações sobre o DOI: 10.1002/jbmr.2376 (Fonte: oaDOI API)
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