The potential role of suppressors of cytokine signaling in the attenuation of inflammatory reaction and alveolar bone loss associated with apical periodontitis (2008)
- Authors:
- USP affiliated authors: GARLET, GUSTAVO POMPERMAIER - FOB ; CAMPANELLI, ANA PAULA - FOB
- Unidade: FOB
- Subjects: CITOCINAS; GRANULOMA PERIAPICAL; REABSORÇÃO ÓSSEA ALVEOLAR
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal Endodontics
- ISSN: 0099-2399
- Volume/Número/Paginação/Ano: v. 34, n. 12, p. 1480-1484, Dec. 2008
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ABNT
MENEZES, Renato et al. The potential role of suppressors of cytokine signaling in the attenuation of inflammatory reaction and alveolar bone loss associated with apical periodontitis. Journal Endodontics, v. 34, n. 12, p. 1480-1484, 2008Tradução . . Disponível em: http://www.sciencedirect.com/science?_ob=ArticleURL&_udi=B82X7-4TPND8N-6&_user=5674931&_rdoc=1&_fmt=&_orig=search&_sort=d&view=c&_acct=C000049650&_version=1&_urlVersion=0&_userid=5674931&md5=5980119d5c71ccda3a338973ca1c6a63. Acesso em: 18 nov. 2024. -
APA
Menezes, R., Garlet, T. P., Trombone, A. P. F., Repeke, C. E., Letra, A., Granjeiro, J. M., et al. (2008). The potential role of suppressors of cytokine signaling in the attenuation of inflammatory reaction and alveolar bone loss associated with apical periodontitis. Journal Endodontics, 34( 12), 1480-1484. Recuperado de http://www.sciencedirect.com/science?_ob=ArticleURL&_udi=B82X7-4TPND8N-6&_user=5674931&_rdoc=1&_fmt=&_orig=search&_sort=d&view=c&_acct=C000049650&_version=1&_urlVersion=0&_userid=5674931&md5=5980119d5c71ccda3a338973ca1c6a63 -
NLM
Menezes R, Garlet TP, Trombone APF, Repeke CE, Letra A, Granjeiro JM, Campanelli AP, Garlet GP. The potential role of suppressors of cytokine signaling in the attenuation of inflammatory reaction and alveolar bone loss associated with apical periodontitis [Internet]. Journal Endodontics. 2008 ; 34( 12): 1480-1484.[citado 2024 nov. 18 ] Available from: http://www.sciencedirect.com/science?_ob=ArticleURL&_udi=B82X7-4TPND8N-6&_user=5674931&_rdoc=1&_fmt=&_orig=search&_sort=d&view=c&_acct=C000049650&_version=1&_urlVersion=0&_userid=5674931&md5=5980119d5c71ccda3a338973ca1c6a63 -
Vancouver
Menezes R, Garlet TP, Trombone APF, Repeke CE, Letra A, Granjeiro JM, Campanelli AP, Garlet GP. The potential role of suppressors of cytokine signaling in the attenuation of inflammatory reaction and alveolar bone loss associated with apical periodontitis [Internet]. Journal Endodontics. 2008 ; 34( 12): 1480-1484.[citado 2024 nov. 18 ] Available from: http://www.sciencedirect.com/science?_ob=ArticleURL&_udi=B82X7-4TPND8N-6&_user=5674931&_rdoc=1&_fmt=&_orig=search&_sort=d&view=c&_acct=C000049650&_version=1&_urlVersion=0&_userid=5674931&md5=5980119d5c71ccda3a338973ca1c6a63 - CCR5+ cells modulate RANKL+ cell migration, inflammatory reaction development and experimental periodontitis outcome in mice
- Involvement of CCR5 and CXCR3 in RANKL+ cell migration and bone resorption throughout experimental periodontitis in mouse
- Associação diferencial de fatores genéticos, microbianos e inflamatórios com os níveis de MMP-1 no periodonto saudável e com periodontite
- IL-4/CCL22/CCR4 axis controls regulatory T-cell migration that suppresses inflammatory bone loss in murine experimental periodontitis
- Distinct roles for genetic (MMP1-1607 and -519 SNPs) factors and periodontopathogens in the modulation of MMP-1 levels in healthy and diseased periodontal tissues
- The broad effects of the functional IL-10 promoter -592 polymorphism: modulation of IL-10, TIMP-3 and OPG expression and its association with periodontal disease outcome
- Evidence supporting a protective role for Th9 and Th22 cytokines in human and experimental periapical lesions
- IL-10 and TNF-α differentially modulate the outcome of alveolar bone repair upon homeostatic and infectious conditions
- Characterization of tumor-infiltrating regulatory T-cell subpopulation in oral squamous cell carcinoma
- Strong and persistent microbial and inflammatory stimuli overcome the genetic predisposition to higher matrix metalloproteinase-1 (MMP-1) expression: a mechanistic explanation for the lack of association of MMP1-1607 single-nucleotide polymorphism genotypes with MMP-1 expression in chronic periodontitis lesions
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