Strong and persistent microbial and inflammatory stimuli overcome the genetic predisposition to higher matrix metalloproteinase-1 (MMP-1) expression: a mechanistic explanation for the lack of association of MMP1-1607 single-nucleotide polymorphism genotypes with MMP-1 expression in chronic periodontitis lesions (2009)
- Authors:
- USP affiliated authors: GARLET, GUSTAVO POMPERMAIER - FOB ; CAMPANELLI, ANA PAULA - FOB
- Unidade: FOB
- DOI: 10.1111/j.1600-051X.2009.01447.x
- Subjects: POLIMORFISMO; PERIODONTITE; METALOPROTEINASES; CITOCINAS
- Language: Inglês
- Imprenta:
- Publisher place: Copenhagen
- Date published: 2009
- Source:
- Título: Journal of Clinical Periodontology
- ISSN: 0303-6979
- Volume/Número/Paginação/Ano: v. 36, n. 9, p. 726-738, Sept. 2009
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
REPEKE, Carlos Eduardo et al. Strong and persistent microbial and inflammatory stimuli overcome the genetic predisposition to higher matrix metalloproteinase-1 (MMP-1) expression: a mechanistic explanation for the lack of association of MMP1-1607 single-nucleotide polymorphism genotypes with MMP-1 expression in chronic periodontitis lesions. Journal of Clinical Periodontology, v. 36, n. 9, p. 726-738, 2009Tradução . . Disponível em: https://doi.org/10.1111/j.1600-051X.2009.01447.x. Acesso em: 06 out. 2024. -
APA
Repeke, C. E., Trombone, A. P. F., Ferreira Júnior, S. B., Cardoso, C. R., Silveira, E. M., Martins Junior, W., et al. (2009). Strong and persistent microbial and inflammatory stimuli overcome the genetic predisposition to higher matrix metalloproteinase-1 (MMP-1) expression: a mechanistic explanation for the lack of association of MMP1-1607 single-nucleotide polymorphism genotypes with MMP-1 expression in chronic periodontitis lesions. Journal of Clinical Periodontology, 36( 9), 726-738. doi:10.1111/j.1600-051X.2009.01447.x -
NLM
Repeke CE, Trombone APF, Ferreira Júnior SB, Cardoso CR, Silveira EM, Martins Junior W, Trevilatto PC, Silva JS, Campanelli AP, Garlet GP. Strong and persistent microbial and inflammatory stimuli overcome the genetic predisposition to higher matrix metalloproteinase-1 (MMP-1) expression: a mechanistic explanation for the lack of association of MMP1-1607 single-nucleotide polymorphism genotypes with MMP-1 expression in chronic periodontitis lesions [Internet]. Journal of Clinical Periodontology. 2009 ; 36( 9): 726-738.[citado 2024 out. 06 ] Available from: https://doi.org/10.1111/j.1600-051X.2009.01447.x -
Vancouver
Repeke CE, Trombone APF, Ferreira Júnior SB, Cardoso CR, Silveira EM, Martins Junior W, Trevilatto PC, Silva JS, Campanelli AP, Garlet GP. Strong and persistent microbial and inflammatory stimuli overcome the genetic predisposition to higher matrix metalloproteinase-1 (MMP-1) expression: a mechanistic explanation for the lack of association of MMP1-1607 single-nucleotide polymorphism genotypes with MMP-1 expression in chronic periodontitis lesions [Internet]. Journal of Clinical Periodontology. 2009 ; 36( 9): 726-738.[citado 2024 out. 06 ] Available from: https://doi.org/10.1111/j.1600-051X.2009.01447.x - CCR5+ cells modulate RANKL+ cell migration, inflammatory reaction development and experimental periodontitis outcome in mice
- The potential role of suppressors of cytokine signaling in the attenuation of inflammatory reaction and alveolar bone loss associated with apical periodontitis
- Involvement of CCR5 and CXCR3 in RANKL+ cell migration and bone resorption throughout experimental periodontitis in mouse
- Associação diferencial de fatores genéticos, microbianos e inflamatórios com os níveis de MMP-1 no periodonto saudável e com periodontite
- IL-4/CCL22/CCR4 axis controls regulatory T-cell migration that suppresses inflammatory bone loss in murine experimental periodontitis
- Distinct roles for genetic (MMP1-1607 and -519 SNPs) factors and periodontopathogens in the modulation of MMP-1 levels in healthy and diseased periodontal tissues
- The broad effects of the functional IL-10 promoter -592 polymorphism: modulation of IL-10, TIMP-3 and OPG expression and its association with periodontal disease outcome
- Evidence supporting a protective role for Th9 and Th22 cytokines in human and experimental periapical lesions
- IL-10 and TNF-α differentially modulate the outcome of alveolar bone repair upon homeostatic and infectious conditions
- Células T regulatórias (Tregs) atenuam a severidade da periodontite experimental induzida por A. actinomycetemcomitans via IL-10 e CTLA-4
Informações sobre o DOI: 10.1111/j.1600-051X.2009.01447.x (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas