Stable SET knockdown in head and neck squamous cell carcinoma promotes cell invasion and the mesenchymal-like phenotype in vitro, as well as necrosis, cisplatin sensitivity and lymph node metastasis in xenograft tumor models (2014)
- Authors:
- USP affiliated authors: SOUSA, LUCAS OLIVEIRA - FCFRP ; CABRAL, HAMILTON - FCFRP ; GREENE, LEWIS JOEL - FMRP ; CURTI, CARLOS - FCFRP ; LEOPOLDINO, ANDRÉIA MACHADO - FCFRP
- Unidades: FCFRP; FMRP
- DOI: 10.1186/1476-4598-13-32
- Subjects: NEOPLASIAS DE CABEÇA E PESCOÇO; CARCINOMA DE CÉLULAS ESCAMOSAS; PROTEÍNAS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Molecular Cancer
- ISSN: 1476-4598
- Volume/Número/Paginação/Ano: v. 13, art. 32, 2014
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
SOBRAL, Lays M. et al. Stable SET knockdown in head and neck squamous cell carcinoma promotes cell invasion and the mesenchymal-like phenotype in vitro, as well as necrosis, cisplatin sensitivity and lymph node metastasis in xenograft tumor models. Molecular Cancer, v. 13, 2014Tradução . . Disponível em: https://doi.org/10.1186/1476-4598-13-32. Acesso em: 29 mar. 2024. -
APA
Sobral, L. M., Sousa, L. O., Coletta, R. D., Cabral, H., Greene, L. J., Tajara, E. H., et al. (2014). Stable SET knockdown in head and neck squamous cell carcinoma promotes cell invasion and the mesenchymal-like phenotype in vitro, as well as necrosis, cisplatin sensitivity and lymph node metastasis in xenograft tumor models. Molecular Cancer, 13. doi:10.1186/1476-4598-13-32 -
NLM
Sobral LM, Sousa LO, Coletta RD, Cabral H, Greene LJ, Tajara EH, Gutkind JS, Curti C, Leopoldino AM. Stable SET knockdown in head and neck squamous cell carcinoma promotes cell invasion and the mesenchymal-like phenotype in vitro, as well as necrosis, cisplatin sensitivity and lymph node metastasis in xenograft tumor models [Internet]. Molecular Cancer. 2014 ; 13[citado 2024 mar. 29 ] Available from: https://doi.org/10.1186/1476-4598-13-32 -
Vancouver
Sobral LM, Sousa LO, Coletta RD, Cabral H, Greene LJ, Tajara EH, Gutkind JS, Curti C, Leopoldino AM. Stable SET knockdown in head and neck squamous cell carcinoma promotes cell invasion and the mesenchymal-like phenotype in vitro, as well as necrosis, cisplatin sensitivity and lymph node metastasis in xenograft tumor models [Internet]. Molecular Cancer. 2014 ; 13[citado 2024 mar. 29 ] Available from: https://doi.org/10.1186/1476-4598-13-32 - SET oncoprotein accumulation regulates transcription through DNA demethylation and histone hypoacetylation
- SET overexpression in HEK293 cells regulates mitochondrial uncoupling proteins levels within a mitochondrial fission/reduced autophagic flux scenario
- SET/I2PP2A is an essential protein for cell migration in head and neck squamous cell carcinoma
- SET protects mitochondria from apoptosis signaling and favors autophagy in HNSCC cells exposed to a pro-oxidant
- Epigenetic regulation by SET protein in HNSCC: histone acetylation versus DNA methylation
- SET protects mitochondrial function and induces autophagy in response to redox unbalance
- SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis
- Neuroprotective effects of C-Phycocyanin in neuronal cell lines and transient ischemic rat retina
- Potential modulation of PTEN phosphatase by PP2A and oxidative stress
- SET regulates levels of HnRNP K and its binding to nucleic acid in HNSCC
Informações sobre o DOI: 10.1186/1476-4598-13-32 (Fonte: oaDOI API)
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