Effect of FGF2 on human keratinocytes expressing H-RasV12 (2010)
- Authors:
- Autor USP: ARMELIN, HUGO AGUIRRE - IQ
- Unidade: IQ
- Subjects: FIBROBLASTOS; GENES SUPRESSORES DE TUMOR
- Language: Inglês
- Imprenta:
- Source:
- Título: Memórias do Instituto Butantan
- ISSN: 1982-3045
- Volume/Número/Paginação/Ano: v. 67, res. 2.12, 2010
- Conference titles: Annual Scientific Meeting
-
ABNT
ZEIDLER, Juliana Dias e ARMELIN, Hugo Aguirre. Effect of FGF2 on human keratinocytes expressing H-RasV12. Memórias do Instituto Butantan. São Paulo: Instituto de Química, Universidade de São Paulo. . Acesso em: 19 fev. 2026. , 2010 -
APA
Zeidler, J. D., & Armelin, H. A. (2010). Effect of FGF2 on human keratinocytes expressing H-RasV12. Memórias do Instituto Butantan. São Paulo: Instituto de Química, Universidade de São Paulo. -
NLM
Zeidler JD, Armelin HA. Effect of FGF2 on human keratinocytes expressing H-RasV12. Memórias do Instituto Butantan. 2010 ; 67[citado 2026 fev. 19 ] -
Vancouver
Zeidler JD, Armelin HA. Effect of FGF2 on human keratinocytes expressing H-RasV12. Memórias do Instituto Butantan. 2010 ; 67[citado 2026 fev. 19 ] - Selection of normal revertants from tumorigenic balb 3t3 mouse cell lines carrying the human ej-ras oncogene
- Selection of FGF2-resistant cell sublines led to separation between classical mitogenic signaling and novel cytostatic mechanisms of FGF2 in K-Ras-driven mouse Y1 adrenocortical tumor cells
- Regulation of 'G IND.0'-> 'G IND.1'-> s transition a genetic approach
- Antiproliferative effects of FGF2 on human embryonic kidney cell lines (HEK293) displaying a ras-dependent malignant phenotype
- Pkc role in acth mechanism of action
- Steroidogenesis regulation by phorbol esther in mouse y-1 adrenocortical cells
- Pattern of c-jun expression in malignant transformation by the human ej-ras oncogene
- Acth inhibits a ras-dependent anti-apoptotic and mitogenic pathway in mouse Y1 adrenocortical cells
- Analysis of FGF2-signaling by RNAi in the mouse Y adrenocortical cell line
- Role of growth factor inducible early response genes (c-fos, c-iin, c-myc, je, kc) in balb-3t3 cell's growth control and viral transformation
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