Effect of FGF2 on human keratinocytes expressing H-RasV12 (2010)
- Authors:
- Autor USP: ARMELIN, HUGO AGUIRRE - IQ
- Unidade: IQ
- Subjects: FIBROBLASTOS; GENES SUPRESSORES DE TUMOR
- Language: Inglês
- Imprenta:
- Source:
- Título: Memórias do Instituto Butantan
- ISSN: 1982-3045
- Volume/Número/Paginação/Ano: v. 67, res. 2.12, 2010
- Conference titles: Annual Scientific Meeting
-
ABNT
ZEIDLER, Juliana Dias e ARMELIN, Hugo Aguirre. Effect of FGF2 on human keratinocytes expressing H-RasV12. Memórias do Instituto Butantan. São Paulo: Instituto de Química, Universidade de São Paulo. . Acesso em: 06 nov. 2024. , 2010 -
APA
Zeidler, J. D., & Armelin, H. A. (2010). Effect of FGF2 on human keratinocytes expressing H-RasV12. Memórias do Instituto Butantan. São Paulo: Instituto de Química, Universidade de São Paulo. -
NLM
Zeidler JD, Armelin HA. Effect of FGF2 on human keratinocytes expressing H-RasV12. Memórias do Instituto Butantan. 2010 ; 67[citado 2024 nov. 06 ] -
Vancouver
Zeidler JD, Armelin HA. Effect of FGF2 on human keratinocytes expressing H-RasV12. Memórias do Instituto Butantan. 2010 ; 67[citado 2024 nov. 06 ] - Selection of normal revertants from tumorigenic balb 3t3 mouse cell lines carrying the human ej-ras oncogene
- Regulation of cyclin D1 and CDKs in mouse adrenocortical cells by the adrenocorticotropic hormone (ACTH)
- Effects of ACTH balb 3T3 fibroblasts and AR-1 cells transfected with the ACTH receptor
- Synthesis of FGF and PDGF by the mouse Y-1 adrenocortical cell line
- Divergence between insulin and FGF signals in Ras-3T3 fibroblasts
- Revertants of tumorigenic mouse cell lines transformed by the human ej-ras oncogene
- Pma induction of tumorigenic potential in balb 3t3 mouse cells carrying the human ej-ras oncogene
- Effects of interactions between FGF2 and PMA in cell cycle arrested BALB 3T3 fibroblasts
- Regulation of expression of fos, jun an myc protooncogenes and activity of ap.1 transcription factor in y-1 adrenocortical cell effects of acth dcamp and phorbol ester
- Role of c-Ki-Ras protein overexpression in adrenocortical Y-1 cells: a study of signal transduction
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