Selection of FGF2-resistant cell sublines led to separation between classical mitogenic signaling and novel cytostatic mechanisms of FGF2 in K-Ras-driven mouse Y1 adrenocortical tumor cells (2014)
- Authors:
- Autor USP: ARMELIN, HUGO AGUIRRE - IQ
- Unidade: IQ
- Subjects: CICLO CELULAR; CÉLULAS CULTIVADAS DE TUMOR
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq)
- Publisher place: São Paulo
- Date published: 2014
- Source:
- Título: Program and Index
- Conference titles: Annual Meeting of the Brazilian Biochemistry and Molecular Biology Society (SBBq)
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ABNT
FONSECA, C. S et al. Selection of FGF2-resistant cell sublines led to separation between classical mitogenic signaling and novel cytostatic mechanisms of FGF2 in K-Ras-driven mouse Y1 adrenocortical tumor cells. 2014, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2014. Disponível em: http://www.sigeventos.com.br/sbbq/cd/2014/resumos/R08528-1.pdf. Acesso em: 08 jun. 2025. -
APA
Fonseca, C. S., Matos, T. G., Dias, M. H. S., Stocco, R. de C., & Armelin, H. A. (2014). Selection of FGF2-resistant cell sublines led to separation between classical mitogenic signaling and novel cytostatic mechanisms of FGF2 in K-Ras-driven mouse Y1 adrenocortical tumor cells. In Program and Index. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq). Recuperado de http://www.sigeventos.com.br/sbbq/cd/2014/resumos/R08528-1.pdf -
NLM
Fonseca CS, Matos TG, Dias MHS, Stocco R de C, Armelin HA. Selection of FGF2-resistant cell sublines led to separation between classical mitogenic signaling and novel cytostatic mechanisms of FGF2 in K-Ras-driven mouse Y1 adrenocortical tumor cells [Internet]. Program and Index. 2014 ;[citado 2025 jun. 08 ] Available from: http://www.sigeventos.com.br/sbbq/cd/2014/resumos/R08528-1.pdf -
Vancouver
Fonseca CS, Matos TG, Dias MHS, Stocco R de C, Armelin HA. Selection of FGF2-resistant cell sublines led to separation between classical mitogenic signaling and novel cytostatic mechanisms of FGF2 in K-Ras-driven mouse Y1 adrenocortical tumor cells [Internet]. Program and Index. 2014 ;[citado 2025 jun. 08 ] Available from: http://www.sigeventos.com.br/sbbq/cd/2014/resumos/R08528-1.pdf - Selection of normal revertants from tumorigenic balb 3t3 mouse cell lines carrying the human ej-ras oncogene
- Alteration in 'G IND.1' phase organization caused by the oncogenese
- Como oncogenes modificam a resposta celular a fatores de crescimento ?
- C-fos / c-myc induction pattern in normal revertants of ej-ras transformed 3t3 cells
- Role of growth factor inducible early response genes (c-fos, c-iin, c-myc, je, kc) in balb-3t3 cell's growth control and viral transformation
- Acth responses in mouse adrenocortical cells lacking PKA
- Acth effects on fibroblasts expressing the acth receptor
- Expression of acth receptor from mouse y-1 adrenocortical cell line on 3T3 fibroblasts and ar-1 cells
- Acth inhibits a ras-dependent anti-apoptotic and mitogenic pathway in mouse Y1 adrenocortical cells
- Molecular and cell biology of acth action in adrenocortical cells
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