Pkc role in acth mechanism of action (1995)
- Authors:
- Autor USP: ARMELIN, HUGO AGUIRRE - IQ
- Unidade: IQ
- Assunto: BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Bioquimica e Biologia Molecular
- Publisher place: São Paulo
- Date published: 1995
- Source:
- Título: Programa e Resumos
- Conference titles: Reuniao Anual da Sociedade Brasileira de Bioquimica e Biologia Molecular
-
ABNT
FRIGERI, C K e ARMELIN, Hugo Aguirre. Pkc role in acth mechanism of action. 1995, Anais.. São Paulo: Sociedade Brasileira de Bioquimica e Biologia Molecular, 1995. . Acesso em: 06 fev. 2026. -
APA
Frigeri, C. K., & Armelin, H. A. (1995). Pkc role in acth mechanism of action. In Programa e Resumos. São Paulo: Sociedade Brasileira de Bioquimica e Biologia Molecular. -
NLM
Frigeri CK, Armelin HA. Pkc role in acth mechanism of action. Programa e Resumos. 1995 ;[citado 2026 fev. 06 ] -
Vancouver
Frigeri CK, Armelin HA. Pkc role in acth mechanism of action. Programa e Resumos. 1995 ;[citado 2026 fev. 06 ] - Selection of normal revertants from tumorigenic balb 3t3 mouse cell lines carrying the human ej-ras oncogene
- Selection of FGF2-resistant cell sublines led to separation between classical mitogenic signaling and novel cytostatic mechanisms of FGF2 in K-Ras-driven mouse Y1 adrenocortical tumor cells
- Regulation of 'G IND.0'-> 'G IND.1'-> s transition a genetic approach
- Antiproliferative effects of FGF2 on human embryonic kidney cell lines (HEK293) displaying a ras-dependent malignant phenotype
- Steroidogenesis regulation by phorbol esther in mouse y-1 adrenocortical cells
- Pattern of c-jun expression in malignant transformation by the human ej-ras oncogene
- Acth inhibits a ras-dependent anti-apoptotic and mitogenic pathway in mouse Y1 adrenocortical cells
- Analysis of FGF2-signaling by RNAi in the mouse Y adrenocortical cell line
- Role of growth factor inducible early response genes (c-fos, c-iin, c-myc, je, kc) in balb-3t3 cell's growth control and viral transformation
- Ras family in FGF2 induced senescence in K-Ras-driven mouse adrenocortical malignant cells
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