Acth inhibits a ras-dependent anti-apoptotic and mitogenic pathway in mouse Y1 adrenocortical cells (2000)
- Authors:
- Autor USP: ARMELIN, HUGO AGUIRRE - IQ
- Unidade: IQ
- Assunto: BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Source:
- Título: Endocrine Research
- ISSN: 0743-5800
- Volume/Número/Paginação/Ano: v. 26, n. 4, p. 911-914, 2000
-
ABNT
FORTI, Fabio Luis e ARMELIN, Hugo Aguirre. Acth inhibits a ras-dependent anti-apoptotic and mitogenic pathway in mouse Y1 adrenocortical cells. Endocrine Research, v. 26, n. 4, p. 911-914, 2000Tradução . . Acesso em: 22 jan. 2026. -
APA
Forti, F. L., & Armelin, H. A. (2000). Acth inhibits a ras-dependent anti-apoptotic and mitogenic pathway in mouse Y1 adrenocortical cells. Endocrine Research, 26( 4), 911-914. -
NLM
Forti FL, Armelin HA. Acth inhibits a ras-dependent anti-apoptotic and mitogenic pathway in mouse Y1 adrenocortical cells. Endocrine Research. 2000 ; 26( 4): 911-914.[citado 2026 jan. 22 ] -
Vancouver
Forti FL, Armelin HA. Acth inhibits a ras-dependent anti-apoptotic and mitogenic pathway in mouse Y1 adrenocortical cells. Endocrine Research. 2000 ; 26( 4): 911-914.[citado 2026 jan. 22 ] - Selection of normal revertants from tumorigenic balb 3t3 mouse cell lines carrying the human ej-ras oncogene
- Ras family in FGF2 induced senescence in K-Ras-driven mouse adrenocortical malignant cells
- Acth causes rapid change in higher-order chromatin structure of adrenocortical cells
- Caracterização do papel de FGFs e FGFRs em queratinócitos humanos
- Ectopic expression of FGF2 protects ras-transformed cells from senescence induced by FGF2
- HMW-FGF2 did not induced the senescence caused by LMW-FGF2 in ras-transformed cells
- FGF2 targets an "Achilles' hell" of Ras-driven mouse malignant cells
- Characterization of keratinocyte responces to FGF1, FGF2 and FGF7
- Anomalias na fase 'G IND.0' / 'G IND.1' do ciclo celular de celulas 3t3 transformadas neoplasticamente pelo encogene humano ej-ras
- Vasopressin triggers senescence in K-ras transformed cells via RhoA-dependent downregulation of cyclin D1
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