Molecular mechanisms involved in the dual blockade of the renin angiotensin system (RAS) on the left ventricular remodeling in renal hypertensive rats (2K-1C) (2010)
- Authors:
- USP affiliated authors: VERCESI, JULIANA APARECIDA - FCFRP ; RIUL, MARIA ELENA - FMRP ; ROSSI, MARCOS ANTONIO - FMRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidades: FCFRP; FMRP
- Subjects: HIPERTROFIA VENTRICULAR ESQUERDA; SISTEMA RENINA-ANGIOTENSINA
- Language: Inglês
- Imprenta:
- Publisher place: Ribeirão Preto
- Date published: 2010
- Source:
- Título do periódico: Programa
- Conference titles: Congresso Brasileiro de Farmacologia e Terapêutica Experimental
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ABNT
CORRÊA, J. W. N et al. Molecular mechanisms involved in the dual blockade of the renin angiotensin system (RAS) on the left ventricular remodeling in renal hypertensive rats (2K-1C). 2010, Anais.. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2010. . Acesso em: 28 mar. 2024. -
APA
Corrêa, J. W. N., Callera, G. E., Yogi, A., He, Y., Araújo, A. V., Vercesi, J. A., et al. (2010). Molecular mechanisms involved in the dual blockade of the renin angiotensin system (RAS) on the left ventricular remodeling in renal hypertensive rats (2K-1C). In Programa. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Corrêa JWN, Callera GE, Yogi A, He Y, Araújo AV, Vercesi JA, Riul ME, Prado CM, Rossi MA, Touyz RM, Bendhack LM. Molecular mechanisms involved in the dual blockade of the renin angiotensin system (RAS) on the left ventricular remodeling in renal hypertensive rats (2K-1C). Programa. 2010 ;[citado 2024 mar. 28 ] -
Vancouver
Corrêa JWN, Callera GE, Yogi A, He Y, Araújo AV, Vercesi JA, Riul ME, Prado CM, Rossi MA, Touyz RM, Bendhack LM. Molecular mechanisms involved in the dual blockade of the renin angiotensin system (RAS) on the left ventricular remodeling in renal hypertensive rats (2K-1C). Programa. 2010 ;[citado 2024 mar. 28 ] - Acetylcholine induces relaxation dependent on nitric oxide synthase (NOS) and an alternative pathway during sepsis
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- Impaired cardiac left ventricular hypertrophy and neointimal hiperplasy are not related to an additional blockade of the RAS and reduction in arterial pressure of 2 kidney-1 clip hypertensive rats
- Endothelium negatively modulates the vascular relaxation induced by nitric oxide donor, due to uncoupling NO synthase
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- The nitric oxide (NO) donors [Ru(terpy)(bdq)NO+]3+(TERPY) and sodium nitroprusside (SNP) have different cellular effects in rat basilar arteries relaxation
- Cellular mechanisms involved in the venodilation induced by nitric oxide donors
- Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP)
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