Endothelium negatively modulates the vascular relaxation induced by nitric oxide donor, due to uncoupling NO synthase (2009)
- Authors:
- USP affiliated authors: VERCESI, JULIANA APARECIDA - FCFRP ; SILVA, ROBERTO SANTANA DA - FCFRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidade: FCFRP
- DOI: 10.1016/j.jinorgbio.2009.07.015
- Subjects: ÓXIDO NÍTRICO; ENDOTÉLIO VASCULAR; TROMBOXANOS; FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Inorganic Biochemistry
- ISSN: 0162-0134
- Volume/Número/Paginação/Ano: v. 103, n. 10, p. 1366-1374, 2009
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
BONAVENTURA, Daniella et al. Endothelium negatively modulates the vascular relaxation induced by nitric oxide donor, due to uncoupling NO synthase. Journal of Inorganic Biochemistry, v. 103, n. 10, p. 1366-1374, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.jinorgbio.2009.07.015. Acesso em: 16 fev. 2026. -
APA
Bonaventura, D., Lunardi, C. N., Rodrigues, G. J., Neto, M. A., Vercesi, J. A., Lima, R. G. de, et al. (2009). Endothelium negatively modulates the vascular relaxation induced by nitric oxide donor, due to uncoupling NO synthase. Journal of Inorganic Biochemistry, 103( 10), 1366-1374. doi:10.1016/j.jinorgbio.2009.07.015 -
NLM
Bonaventura D, Lunardi CN, Rodrigues GJ, Neto MA, Vercesi JA, Lima RG de, Silva RS da, Bendhack LM. Endothelium negatively modulates the vascular relaxation induced by nitric oxide donor, due to uncoupling NO synthase [Internet]. Journal of Inorganic Biochemistry. 2009 ; 103( 10): 1366-1374.[citado 2026 fev. 16 ] Available from: https://doi.org/10.1016/j.jinorgbio.2009.07.015 -
Vancouver
Bonaventura D, Lunardi CN, Rodrigues GJ, Neto MA, Vercesi JA, Lima RG de, Silva RS da, Bendhack LM. Endothelium negatively modulates the vascular relaxation induced by nitric oxide donor, due to uncoupling NO synthase [Internet]. Journal of Inorganic Biochemistry. 2009 ; 103( 10): 1366-1374.[citado 2026 fev. 16 ] Available from: https://doi.org/10.1016/j.jinorgbio.2009.07.015 - The contribution of guanylyl cyclase and potassium channels to the relaxation induced by the NO donors [Ru(terpy)(bdq)NO+]3+ (Terpy) and sodium nitroprusside (SNP) in rat mesenteric resistance arteries
- Cellular mechanisms involved in the venodilation induced by nitric oxide donors
- Nitric oxide (NO) released form NO donors has hypotensive effect and induces rat aorta relaxation due to the activation of K+ channels, guanylyl-cyclase and SERCA
- The nitric oxide (NO) donors [Ru(terpy)(bdq)NO+]3+(TERPY) and sodium nitroprusside (SNP) have different cellular effects in rat basilar arteries relaxation
- Vascular smooth muscle relaxation induced by nitric oxide(NO) donor
- Acetylcholine induces relaxation dependent on nitric oxide synthase and cyclooxigenase during sepsis
- Acetylcholine induces relaxation dependent on nitric oxide synthase (NOS) and an alternative pathway during sepsis
- New NO donor induces relaxation of mesenteric resistance arteries and reduces resistance of mesenteric bed of normotensive and 2K-1C hypertensive rats
- Vascular relaxation induced by nitric oxide donor in mesenteric artery from hypertensive (2K-1C) rats
- Reactive oxygen species are involved in the vascular relaxation induced by nitric oxide donor in mesenteric resistance artery from 2K-1C rats
Informações sobre o DOI: 10.1016/j.jinorgbio.2009.07.015 (Fonte: oaDOI API)
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