Acetylcholine induces relaxation dependent on nitric oxide synthase and cyclooxigenase during sepsis (2008)
- Authors:
- USP affiliated authors: VERCESI, JULIANA APARECIDA - FCFRP ; BENDHACK, LUSIANE MARIA - FCFRP
- Unidade: FCFRP
- Subjects: SEPSE; ÓXIDO NÍTRICO; FÁRMACOS (SISTEMA CARDIOVASCULAR)
- Language: Inglês
- Imprenta:
- Publisher place: Águas de Lindóia
- Date published: 2008
- Source:
- Volume/Número/Paginação/Ano: 2008
- Conference titles: Congresso Brasileiro de Farmacologia e Terapêutica Experimental
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ABNT
ARAÚJO, Alice V. et al. Acetylcholine induces relaxation dependent on nitric oxide synthase and cyclooxigenase during sepsis. 2008, Anais.. Águas de Lindóia: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2008. . Acesso em: 23 abr. 2024. -
APA
Araújo, A. V., Ferezin, C. Z., Bonaventura, D., Lunardi, C. N., Rodrigues, G. J., Vercesi, J. A., & Bendhack, L. M. (2008). Acetylcholine induces relaxation dependent on nitric oxide synthase and cyclooxigenase during sepsis. In . Águas de Lindóia: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Araújo AV, Ferezin CZ, Bonaventura D, Lunardi CN, Rodrigues GJ, Vercesi JA, Bendhack LM. Acetylcholine induces relaxation dependent on nitric oxide synthase and cyclooxigenase during sepsis. 2008 ;[citado 2024 abr. 23 ] -
Vancouver
Araújo AV, Ferezin CZ, Bonaventura D, Lunardi CN, Rodrigues GJ, Vercesi JA, Bendhack LM. Acetylcholine induces relaxation dependent on nitric oxide synthase and cyclooxigenase during sepsis. 2008 ;[citado 2024 abr. 23 ] - Acetylcholine induces relaxation dependent on nitric oxide synthase (NOS) and an alternative pathway during sepsis
- Vascular smooth muscle relaxation induced by nitric oxide(NO) donor
- Endothelium negatively modulates the vascular relaxation induced by nitric oxide donor, due to uncoupling NO synthase
- The contribution of guanylyl cyclase and potassium channels to the relaxation induced by the NO donors [Ru(terpy)(bdq)NO+]3+ (Terpy) and sodium nitroprusside (SNP) in rat mesenteric resistance arteries
- Nitric oxide (NO) released form NO donors has hypotensive effect and induces rat aorta relaxation due to the activation of K+ channels, guanylyl-cyclase and SERCA
- The nitric oxide (NO) donors [Ru(terpy)(bdq)NO+]3+(TERPY) and sodium nitroprusside (SNP) have different cellular effects in rat basilar arteries relaxation
- Cellular mechanisms involved in the venodilation induced by nitric oxide donors
- Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP)
- Molecular mechanisms involved in the dual blockade of the renin angiotensin system (RAS) on the left ventricular remodeling in renal hypertensive rats (2K-1C)
- Espécies reativas de oxigênio podem modular negativamente o relaxamento induzido pelo nitroprussiato de sódio em aortas de ratos hipertensos
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