The contribution of guanylyl cyclase and potassium channels to the relaxation induced by the NO donors [Ru(terpy)(bdq)NO+]3+ (Terpy) and sodium nitroprusside (SNP) in rat mesenteric resistance arteries (2010)
- Authors:
- USP affiliated authors: BENDHACK, LUSIANE MARIA - FCFRP ; VERCESI, JULIANA APARECIDA - FCFRP ; SILVA, ROBERTO SANTANA DA - FCFRP
- Unidade: FCFRP
- Subjects: VASODILATAÇÃO; ÓXIDO NÍTRICO; FARMACOLOGIA; HIPERTENSÃO
- Language: Inglês
- Imprenta:
- Source:
- Título: The Faseb Journal
- ISSN: 0892-6638
- Volume/Número/Paginação/Ano: v. 24, res. 984.5, 2010
- Conference titles: Experimental Biology
-
ABNT
ARAÚJO, Alice Valença et al. The contribution of guanylyl cyclase and potassium channels to the relaxation induced by the NO donors [Ru(terpy)(bdq)NO+]3+ (Terpy) and sodium nitroprusside (SNP) in rat mesenteric resistance arteries. The Faseb Journal. Bethesda: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 10 fev. 2026. , 2010 -
APA
Araújo, A. V., Vercesi, J. A., Biazzotto, J. C., Silva, R. S. da, & Bendhack, L. M. (2010). The contribution of guanylyl cyclase and potassium channels to the relaxation induced by the NO donors [Ru(terpy)(bdq)NO+]3+ (Terpy) and sodium nitroprusside (SNP) in rat mesenteric resistance arteries. The Faseb Journal. Bethesda: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. -
NLM
Araújo AV, Vercesi JA, Biazzotto JC, Silva RS da, Bendhack LM. The contribution of guanylyl cyclase and potassium channels to the relaxation induced by the NO donors [Ru(terpy)(bdq)NO+]3+ (Terpy) and sodium nitroprusside (SNP) in rat mesenteric resistance arteries. The Faseb Journal. 2010 ; 24[citado 2026 fev. 10 ] -
Vancouver
Araújo AV, Vercesi JA, Biazzotto JC, Silva RS da, Bendhack LM. The contribution of guanylyl cyclase and potassium channels to the relaxation induced by the NO donors [Ru(terpy)(bdq)NO+]3+ (Terpy) and sodium nitroprusside (SNP) in rat mesenteric resistance arteries. The Faseb Journal. 2010 ; 24[citado 2026 fev. 10 ] - Cellular mechanisms involved in the venodilation induced by nitric oxide donors
- Nitric oxide (NO) released form NO donors has hypotensive effect and induces rat aorta relaxation due to the activation of K+ channels, guanylyl-cyclase and SERCA
- The nitric oxide (NO) donors [Ru(terpy)(bdq)NO+]3+(TERPY) and sodium nitroprusside (SNP) have different cellular effects in rat basilar arteries relaxation
- Vascular smooth muscle relaxation induced by nitric oxide(NO) donor
- Endothelium negatively modulates the vascular relaxation induced by nitric oxide donor, due to uncoupling NO synthase
- Acetylcholine induces relaxation dependent on nitric oxide synthase and cyclooxigenase during sepsis
- Acetylcholine induces relaxation dependent on nitric oxide synthase (NOS) and an alternative pathway during sepsis
- New NO donor induces relaxation of mesenteric resistance arteries and reduces resistance of mesenteric bed of normotensive and 2K-1C hypertensive rats
- Vascular relaxation induced by nitric oxide donor in mesenteric artery from hypertensive (2K-1C) rats
- Reactive oxygen species are involved in the vascular relaxation induced by nitric oxide donor in mesenteric resistance artery from 2K-1C rats
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