Cancer cell death induced by FGF2 and FGFR (2006)
- Authors:
- Autor USP: ARMELIN, HUGO AGUIRRE - IQ
- Unidade: IQ
- Assunto: BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Publisher: Cold Spring Harbor Laboratory
- Publisher place: New York
- Date published: 2006
- Source:
- Título: Abstracts
- Conference titles: Cold Spring Harbor Symposium on Quantitative Biology: Regulatory RNAs
-
ABNT
SALOTTI, Jacqueline et al. Cancer cell death induced by FGF2 and FGFR. 2006, Anais.. New York: Cold Spring Harbor Laboratory, 2006. . Acesso em: 06 fev. 2026. -
APA
Salotti, J., Costa, É. T., Koga, M. M., & Armelin, H. A. (2006). Cancer cell death induced by FGF2 and FGFR. In Abstracts. New York: Cold Spring Harbor Laboratory. -
NLM
Salotti J, Costa ÉT, Koga MM, Armelin HA. Cancer cell death induced by FGF2 and FGFR. Abstracts. 2006 ;[citado 2026 fev. 06 ] -
Vancouver
Salotti J, Costa ÉT, Koga MM, Armelin HA. Cancer cell death induced by FGF2 and FGFR. Abstracts. 2006 ;[citado 2026 fev. 06 ] - Selection of normal revertants from tumorigenic balb 3t3 mouse cell lines carrying the human ej-ras oncogene
- Selection of FGF2-resistant cell sublines led to separation between classical mitogenic signaling and novel cytostatic mechanisms of FGF2 in K-Ras-driven mouse Y1 adrenocortical tumor cells
- Regulation of 'G IND.0'-> 'G IND.1'-> s transition a genetic approach
- Antiproliferative effects of FGF2 on human embryonic kidney cell lines (HEK293) displaying a ras-dependent malignant phenotype
- Pkc role in acth mechanism of action
- Steroidogenesis regulation by phorbol esther in mouse y-1 adrenocortical cells
- Pattern of c-jun expression in malignant transformation by the human ej-ras oncogene
- Acth inhibits a ras-dependent anti-apoptotic and mitogenic pathway in mouse Y1 adrenocortical cells
- Analysis of FGF2-signaling by RNAi in the mouse Y adrenocortical cell line
- Role of growth factor inducible early response genes (c-fos, c-iin, c-myc, je, kc) in balb-3t3 cell's growth control and viral transformation
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