CD28 is required for T cell activation and IFN-gamma production by CD4+ and CD8+ T cells in response to Trypanosoma cruzi infection (2004)
- Authors:
- USP affiliated authors: RUSSO, MOMTCHILO - ICB ; CUNHA, FERNANDO DE QUEIROZ - FMRP ; RIZZO, LUIZ VICENTE - ICB ; SILVA, JOÃO SANTANA DA - FMRP
- Unidades: ICB; FMRP
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Microbes and Infection
- Volume/Número/Paginação/Ano: v. 6, p. 1133-1144, 2004
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ABNT
MARTINS, Gislâine Aparecida et al. CD28 is required for T cell activation and IFN-gamma production by CD4+ and CD8+ T cells in response to Trypanosoma cruzi infection. Microbes and Infection, v. 6, p. 1133-1144, 2004Tradução . . Acesso em: 13 fev. 2026. -
APA
Martins, G. A., Campanelli, A. P., Silva, R. B., Takodoro, C. E., Russo, M., Cunha, F. de Q., et al. (2004). CD28 is required for T cell activation and IFN-gamma production by CD4+ and CD8+ T cells in response to Trypanosoma cruzi infection. Microbes and Infection, 6, 1133-1144. -
NLM
Martins GA, Campanelli AP, Silva RB, Takodoro CE, Russo M, Cunha F de Q, Rizzo LV, Silva JS da. CD28 is required for T cell activation and IFN-gamma production by CD4+ and CD8+ T cells in response to Trypanosoma cruzi infection. Microbes and Infection. 2004 ; 6 1133-1144.[citado 2026 fev. 13 ] -
Vancouver
Martins GA, Campanelli AP, Silva RB, Takodoro CE, Russo M, Cunha F de Q, Rizzo LV, Silva JS da. CD28 is required for T cell activation and IFN-gamma production by CD4+ and CD8+ T cells in response to Trypanosoma cruzi infection. Microbes and Infection. 2004 ; 6 1133-1144.[citado 2026 fev. 13 ] - Mice deficient in (CD28-/-) are highly susceptible to Trypanosoma cruzi infection
- Absence of CD28 leads to increased susceptibility to experimental Trypanosoma cruzi infection
- CTLA-4 blockage increases reistance to infection with the intracellular protozaon Trypanossoma cruzi
- CD28 is requered for protective immunity against the intracellular protozoan Trypanosoma cruzi
- Absence of CD28 leads to increased susceptibility to experimental Trypanosoma cruzi infections
- Regulatory T cells migrate to airways via CCR4 and attenuate the severity of airway allergic inflammation
- Toll-like receptor 4 signaling leads to neutrophil migration impairment in polymicrobial sepsis
- Leukotriene'B IND.4'is essential for selective eosinophil recruitment following allergen challenge of 'CD4 POT.+' cells in a model of chronic eosinophilic inflammation
- Tnf-gamma and tnf-alpha mediated resistance to trypanosoma cruzi through no production
- Nitric oxide is involved in control of trypanosoma cruzi - induced parasitemia and directely kills the parasite in vitro
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