CTLA-4 blockage increases reistance to infection with the intracellular protozaon Trypanossoma cruzi (2004)
- Authors:
- USP affiliated authors: SILVA, JOAO SANTANA DA - FMRP ; RIZZO, LUIZ VICENTE - ICB
- Unidades: FMRP; ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Immunology
- ISSN: 0022-1767
- Volume/Número/Paginação/Ano: v. 172, p. 4893-4901, 2004
-
ABNT
MARTINS, Gislâine Aparecida et al. CTLA-4 blockage increases reistance to infection with the intracellular protozaon Trypanossoma cruzi. Journal of Immunology, v. 172, p. 4893-4901, 2004Tradução . . Acesso em: 29 dez. 2025. -
APA
Martins, G. A., Tadoroko, C. E., Silva, R. B., Silva, J. S. da, & Rizzo, L. V. (2004). CTLA-4 blockage increases reistance to infection with the intracellular protozaon Trypanossoma cruzi. Journal of Immunology, 172, 4893-4901. -
NLM
Martins GA, Tadoroko CE, Silva RB, Silva JS da, Rizzo LV. CTLA-4 blockage increases reistance to infection with the intracellular protozaon Trypanossoma cruzi. Journal of Immunology. 2004 ; 172 4893-4901.[citado 2025 dez. 29 ] -
Vancouver
Martins GA, Tadoroko CE, Silva RB, Silva JS da, Rizzo LV. CTLA-4 blockage increases reistance to infection with the intracellular protozaon Trypanossoma cruzi. Journal of Immunology. 2004 ; 172 4893-4901.[citado 2025 dez. 29 ] - Absence of CD28 leads to increased susceptibility to experimental Trypanosoma cruzi infections
- CD28 is requered for protective immunity against the intracellular protozoan Trypanosoma cruzi
- Absence of CD28 leads to increased susceptibility to experimental Trypanosoma cruzi infection
- Mice deficient in (CD28-/-) are highly susceptible to Trypanosoma cruzi infection
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- Involvement of nitric oxide (NO) and TNF-A in the anemia and in the oxidative stress in erythrocytes from mice susceptible and resistant to Tripanossoma Cruzi infection
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