Absence of CD28 leads to increased susceptibility to experimental Trypanosoma cruzi infections (2001)
- Authors:
- USP affiliated authors: RIZZO, LUIZ VICENTE - ICB ; SILVA, JOÃO SANTANA DA - FMRP
- Unidades: ICB; FMRP
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Scandinavian Journal of Immunology
- Volume/Número/Paginação/Ano: v. 54, n, supl. 1 p
- Conference titles: International Congress of Immunology
-
ABNT
MARTINS, G. A. et al. Absence of CD28 leads to increased susceptibility to experimental Trypanosoma cruzi infections. Scandinavian Journal of Immunology. Oxford: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 13 fev. 2026. , 2001 -
APA
Martins, G. A., Campanelli, A. P., Tadokoro, C. E., Vallochi, A. L., Rizzo, L. V., & Silva, J. S. da. (2001). Absence of CD28 leads to increased susceptibility to experimental Trypanosoma cruzi infections. Scandinavian Journal of Immunology. Oxford: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Martins GA, Campanelli AP, Tadokoro CE, Vallochi AL, Rizzo LV, Silva JS da. Absence of CD28 leads to increased susceptibility to experimental Trypanosoma cruzi infections. Scandinavian Journal of Immunology. 2001 ; 54[citado 2026 fev. 13 ] -
Vancouver
Martins GA, Campanelli AP, Tadokoro CE, Vallochi AL, Rizzo LV, Silva JS da. Absence of CD28 leads to increased susceptibility to experimental Trypanosoma cruzi infections. Scandinavian Journal of Immunology. 2001 ; 54[citado 2026 fev. 13 ] - Absence of CD28 leads to increased susceptibility to experimental Trypanosoma cruzi infection
- CTLA-4 blockage increases reistance to infection with the intracellular protozaon Trypanossoma cruzi
- CD28 is requered for protective immunity against the intracellular protozoan Trypanosoma cruzi
- Mice deficient in (CD28-/-) are highly susceptible to Trypanosoma cruzi infection
- Combined immunodeficiency in a chilhood with Cd3 deficiency: case report
- Development of CD4+T cell lines that suppress an antigen-specific immune response in vivo
- Oral tolerance mechanisms reside on reduction of pathogenic cytokine secretion rather than on down-regulation of costimulatory molecules
- Influência de antígenos alogeneicos e singeneicos na geração de células T reguladoras
- Effects of cyclooxygenase inhibitors treatment in murine model of trypanosoma cruzi infection
- Administration of m. Leprae hsp65 aggravates experimental autoimmune uveitis [eau] in mice
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