The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration (2004)
- Authors:
- USP affiliated authors: ARNHOLD, IVO JORGE PRADO - FM ; MENDONCA, BERENICE BILHARINHO DE - FM
- Unidade: FM
- DOI: 10.1111/j.1365-2265.2004.01930.x
- Assunto: ENDOCRINOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Clinical Endocrinology
- Volume/Número/Paginação/Ano: v. 60, p. 36-40, 2004
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
JORGE, Alexander A. L. et al. The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration. Clinical Endocrinology, v. 60, p. 36-40, 2004Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2265.2004.01930.x. Acesso em: 20 set. 2024. -
APA
Jorge, A. A. L., Souza, S. C. A. L., Arnhold, I. J. P., & Mendonça, B. B. (2004). The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration. Clinical Endocrinology, 60, 36-40. doi:10.1111/j.1365-2265.2004.01930.x -
NLM
Jorge AAL, Souza SCAL, Arnhold IJP, Mendonça BB. The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration [Internet]. Clinical Endocrinology. 2004 ; 60 36-40.[citado 2024 set. 20 ] Available from: https://doi.org/10.1111/j.1365-2265.2004.01930.x -
Vancouver
Jorge AAL, Souza SCAL, Arnhold IJP, Mendonça BB. The first homozygous mutation (S226I) in the highly-conserved WSXWS-like motif of the GH receptor causing Laron syndrome: supression of GH secretion by GnRH anlogue therapy not restored by dihydrotestosterone administration [Internet]. Clinical Endocrinology. 2004 ; 60 36-40.[citado 2024 set. 20 ] Available from: https://doi.org/10.1111/j.1365-2265.2004.01930.x - Molecular analysis of SRY gene in Brazilian 46,XX sex reversed patients: absense of SRY sequence in gonadal tissue
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Informações sobre o DOI: 10.1111/j.1365-2265.2004.01930.x (Fonte: oaDOI API)
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