Effects of interactions between FGF2 and PMA in cell cycle arrested BALB 3T3 fibroblasts (1999)
- Authors:
- Autor USP: ARMELIN, HUGO AGUIRRE - IQ
- Unidade: IQ
- Assunto: BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Source:
- Título: Programa e Resumos
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular
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ABNT
COSTA, E. T. e LOTFI, Claudimara Ferini Pacicco e ARMELIN, Hugo Aguirre. Effects of interactions between FGF2 and PMA in cell cycle arrested BALB 3T3 fibroblasts. 1999, Anais.. São Paulo: SBBq, 1999. . Acesso em: 11 out. 2024. -
APA
Costa, E. T., Lotfi, C. F. P., & Armelin, H. A. (1999). Effects of interactions between FGF2 and PMA in cell cycle arrested BALB 3T3 fibroblasts. In Programa e Resumos. São Paulo: SBBq. -
NLM
Costa ET, Lotfi CFP, Armelin HA. Effects of interactions between FGF2 and PMA in cell cycle arrested BALB 3T3 fibroblasts. Programa e Resumos. 1999 ;[citado 2024 out. 11 ] -
Vancouver
Costa ET, Lotfi CFP, Armelin HA. Effects of interactions between FGF2 and PMA in cell cycle arrested BALB 3T3 fibroblasts. Programa e Resumos. 1999 ;[citado 2024 out. 11 ] - Selection of normal revertants from tumorigenic balb 3t3 mouse cell lines carrying the human ej-ras oncogene
- Regulation of cyclin D1 and CDKs in mouse adrenocortical cells by the adrenocorticotropic hormone (ACTH)
- Effects of ACTH balb 3T3 fibroblasts and AR-1 cells transfected with the ACTH receptor
- Synthesis of FGF and PDGF by the mouse Y-1 adrenocortical cell line
- Divergence between insulin and FGF signals in Ras-3T3 fibroblasts
- Revertants of tumorigenic mouse cell lines transformed by the human ej-ras oncogene
- Pma induction of tumorigenic potential in balb 3t3 mouse cells carrying the human ej-ras oncogene
- Regulation of expression of fos, jun an myc protooncogenes and activity of ap.1 transcription factor in y-1 adrenocortical cell effects of acth dcamp and phorbol ester
- Role of c-Ki-Ras protein overexpression in adrenocortical Y-1 cells: a study of signal transduction
- Understanding the mechanisms of cell cycle controle: links in the signaling network between mitogenic and cell death pathways
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