Synthesis of FGF and PDGF by the mouse Y-1 adrenocortical cell line (1999)
- Authors:
- Autor USP: ARMELIN, HUGO AGUIRRE - IQ
- Unidade: IQ
- Assunto: BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Source:
- Título: Programa e Resumos
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular
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ABNT
REBUSTINI, I. T. e FORTI, Fabio Luis e ARMELIN, Hugo Aguirre. Synthesis of FGF and PDGF by the mouse Y-1 adrenocortical cell line. 1999, Anais.. São Paulo: SBBq, 1999. . Acesso em: 04 mar. 2026. -
APA
Rebustini, I. T., Forti, F. L., & Armelin, H. A. (1999). Synthesis of FGF and PDGF by the mouse Y-1 adrenocortical cell line. In Programa e Resumos. São Paulo: SBBq. -
NLM
Rebustini IT, Forti FL, Armelin HA. Synthesis of FGF and PDGF by the mouse Y-1 adrenocortical cell line. Programa e Resumos. 1999 ;[citado 2026 mar. 04 ] -
Vancouver
Rebustini IT, Forti FL, Armelin HA. Synthesis of FGF and PDGF by the mouse Y-1 adrenocortical cell line. Programa e Resumos. 1999 ;[citado 2026 mar. 04 ] - Selection of normal revertants from tumorigenic balb 3t3 mouse cell lines carrying the human ej-ras oncogene
- Selection of FGF2-resistant cell sublines led to separation between classical mitogenic signaling and novel cytostatic mechanisms of FGF2 in K-Ras-driven mouse Y1 adrenocortical tumor cells
- Regulation of 'G IND.0'-> 'G IND.1'-> s transition a genetic approach
- Antiproliferative effects of FGF2 on human embryonic kidney cell lines (HEK293) displaying a ras-dependent malignant phenotype
- Pkc role in acth mechanism of action
- Steroidogenesis regulation by phorbol esther in mouse y-1 adrenocortical cells
- Pattern of c-jun expression in malignant transformation by the human ej-ras oncogene
- Acth inhibits a ras-dependent anti-apoptotic and mitogenic pathway in mouse Y1 adrenocortical cells
- Analysis of FGF2-signaling by RNAi in the mouse Y adrenocortical cell line
- Role of growth factor inducible early response genes (c-fos, c-iin, c-myc, je, kc) in balb-3t3 cell's growth control and viral transformation
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