Control of function and proliferation by acth in mouse adrenocortical cells: induction of fos and jun proto-oncogene families (1993)
- Authors:
- USP affiliated authors: ARMELIN, HUGO AGUIRRE - IQ ; KIMURA, EICO - IQ ; SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Subjects: CITOLOGIA; BIOLOGIA CELULAR; ONCOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Universidad de Chile
- Publisher place: Santiago
- Date published: 1993
- Source:
- Título do periódico: Abstracts
- Conference titles: Santiago Southern Summer Symposium
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ABNT
KIMURA, E; SONOBE, M S; ARMELIN, M C S; ARMELIN, Hugo Aguirre. Control of function and proliferation by acth in mouse adrenocortical cells: induction of fos and jun proto-oncogene families. Anais.. Santiago: Universidad de Chile, 1993. -
APA
Kimura, E., Sonobe, M. S., Armelin, M. C. S., & Armelin, H. A. (1993). Control of function and proliferation by acth in mouse adrenocortical cells: induction of fos and jun proto-oncogene families. In Abstracts. Santiago: Universidad de Chile. -
NLM
Kimura E, Sonobe MS, Armelin MCS, Armelin HA. Control of function and proliferation by acth in mouse adrenocortical cells: induction of fos and jun proto-oncogene families. Abstracts. 1993 ; -
Vancouver
Kimura E, Sonobe MS, Armelin MCS, Armelin HA. Control of function and proliferation by acth in mouse adrenocortical cells: induction of fos and jun proto-oncogene families. Abstracts. 1993 ; - Acth modulates gene transcription in adrenocortical cells via ap-1 complex
- Induction of fos and jun proteins by adrenocorticoprotein and phorbol ester but not by 3',5'-ciclic adenosine monophosphate derivates
- Kinetics of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth, dcamp and phorbol ester in mouse adrenocortical cells
- Relevance of c-fos proto-oncogene induction for the steroidogenic response to acth, da amp and phorbol ester in adrenocortical cells
- Kinetcs of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth dibuthyl camp and phorbol ester in mouse adrenocortical cells
- Transcriptional pattern of c-fos oncogene in mouse malignant adrenocortical cell cultures
- Role of proto-oncogene c-ki-ras amplification and overexpression in the malignancy of y-1 adrenocortical tumor cells
- Phorbol ester mimicks adrenocorticotropic hormone (acth) action in adrenocortical mouse cells
- Amplified ki-ras oncogene elimination as the basis for normal reversion in malignant adrenocortical cells
- Phorbol ester mimics acth action in corticoadrenal cells stimulating steroidogenesis blocking cell cycle, changing cell shape, and inducing c-fos proto-oncogene expression
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