Relevance of c-fos proto-oncogene induction for the steroidogenic response to acth, da amp and phorbol ester in adrenocortical cells (1993)
- Authors:
- USP affiliated authors: ARMELIN, HUGO AGUIRRE - IQ ; KIMURA, EICO - IQ
- Unidades: IQ; IQ
- Subjects: BIOQUÍMICA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Molecular and Cellular Biochemistry
- Volume/Número/Paginação/Ano: v.124, n.1 , p.23-32, 1993
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ABNT
KIMURA, E; FRIGERI, C K; ARMELIN, Hugo Aguirre. Relevance of c-fos proto-oncogene induction for the steroidogenic response to acth, da amp and phorbol ester in adrenocortical cells. Molecular and Cellular Biochemistry, Hague, v. 124, n. 1 , p. 23-32, 1993. -
APA
Kimura, E., Frigeri, C. K., & Armelin, H. A. (1993). Relevance of c-fos proto-oncogene induction for the steroidogenic response to acth, da amp and phorbol ester in adrenocortical cells. Molecular and Cellular Biochemistry, 124( 1 ), 23-32. -
NLM
Kimura E, Frigeri CK, Armelin HA. Relevance of c-fos proto-oncogene induction for the steroidogenic response to acth, da amp and phorbol ester in adrenocortical cells. Molecular and Cellular Biochemistry. 1993 ;124( 1 ): 23-32. -
Vancouver
Kimura E, Frigeri CK, Armelin HA. Relevance of c-fos proto-oncogene induction for the steroidogenic response to acth, da amp and phorbol ester in adrenocortical cells. Molecular and Cellular Biochemistry. 1993 ;124( 1 ): 23-32. - Transcriptional pattern of c-fos oncogene in mouse malignant adrenocortical cell cultures
- Role of proto-oncogene c-ki-ras amplification and overexpression in the malignancy of y-1 adrenocortical tumor cells
- Phorbol ester mimicks adrenocorticotropic hormone (acth) action in adrenocortical mouse cells
- Kinetics of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth, dcamp and phorbol ester in mouse adrenocortical cells
- Kinetcs of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth dibuthyl camp and phorbol ester in mouse adrenocortical cells
- Growth control in mouse y-1 adrenocortical tumor cells: role of c-ki-ras amplification and effects of alth
- Phorbol ester mimics acth action in corticoadrenal cells stimulating steroidogenesis blocking cell cycle, changing cell shape, and inducing c-fos proto-oncogene expression
- Amplified ki-ras oncogene elimination as the basis for normal reversion in malignant adrenocortical cells
- C-fos oncogene transcription pattern in mouse malignant adrenocortical cell cultures
- Dna synthesis inhibition by adrenocorticotropic hormone (acth) and a phorbol ester in y-1 corticoadrenal cells
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