Amplified ki-ras oncogene elimination as the basis for normal reversion in malignant adrenocortical cells (1985)
- Authors:
- USP affiliated authors: ARMELIN, HUGO AGUIRRE - IQ ; KIMURA, EICO - IQ
- Unidade: IQ
- Assunto: ONCOGENES
- Language: Português
- Imprenta:
- Source:
- Título: Arquivos de Biologia e Tecnologia
- Volume/Número/Paginação/Ano: v.28, n.1 , p.34, 1985
-
ABNT
KIMURA, Eico e ARMELIN, Hugo Aguirre. Amplified ki-ras oncogene elimination as the basis for normal reversion in malignant adrenocortical cells. Arquivos de Biologia e Tecnologia, v. 28, n. 1 , p. 34, 1985Tradução . . Acesso em: 27 jan. 2026. -
APA
Kimura, E., & Armelin, H. A. (1985). Amplified ki-ras oncogene elimination as the basis for normal reversion in malignant adrenocortical cells. Arquivos de Biologia e Tecnologia, 28( 1 ), 34. -
NLM
Kimura E, Armelin HA. Amplified ki-ras oncogene elimination as the basis for normal reversion in malignant adrenocortical cells. Arquivos de Biologia e Tecnologia. 1985 ;28( 1 ): 34.[citado 2026 jan. 27 ] -
Vancouver
Kimura E, Armelin HA. Amplified ki-ras oncogene elimination as the basis for normal reversion in malignant adrenocortical cells. Arquivos de Biologia e Tecnologia. 1985 ;28( 1 ): 34.[citado 2026 jan. 27 ] - Kinetcs of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth dibuthyl camp and phorbol ester in mouse adrenocortical cells
- Relevance of c-fos proto-oncogene induction for the steroidogenic response to acth, da amp and phorbol ester in adrenocortical cells
- Induction of fos and jun proto-ancogenes by the adrenocorticotropic hormone (ACTH) : possible biological implications: possible biological implications
- Growth control in mouse y-1 adrenocortical tumor cells: role of c-ki-ras amplification and effects of alth
- Phorbol ester mimics acth action in corticoadrenal cells stimulating steroidogenesis blocking cell cycle, changing cell shape, and inducing c-fos proto-oncogene expression
- Transcriptional pattern of c-fos oncogene in mouse malignant adrenocortical cell cultures
- Phorbol ester mimicks adrenocorticotropic hormone (acth) action in adrenocortical mouse cells
- Synthesis and secretion of pdgf-like factors by mouse corticoadrenal cells stimulated by acth and a protein kinase c-activating phorbol ester
- Protein kinase a and c in acth mechanisms of action
- Dna synthesis inhibition by adrenocorticotropic hormone (acth) and a phorbol ester in y-1 corticoadrenal cells
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