Acth modulates gene transcription in adrenocortical cells via ap-1 complex (1992)
- Authors:
- USP affiliated authors: ARMELIN, HUGO AGUIRRE - IQ ; KIMURA, EICO - IQ ; SOGAYAR, MARI CLEIDE - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; CITOLOGIA; BIOLOGIA CELULAR; ONCOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Resumos
- Conference titles: Reuniao Anual da Sociedade Brasileira de Bioquimica e Biologia Molecular
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ABNT
KIMURA, E et al. Acth modulates gene transcription in adrenocortical cells via ap-1 complex. 1992, Anais.. São Paulo: Sbbq, 1992. . Acesso em: 02 nov. 2024. -
APA
Kimura, E., Sonobe, M. H., Armelin, M. C. S., & Armelin, H. A. (1992). Acth modulates gene transcription in adrenocortical cells via ap-1 complex. In Resumos. São Paulo: Sbbq. -
NLM
Kimura E, Sonobe MH, Armelin MCS, Armelin HA. Acth modulates gene transcription in adrenocortical cells via ap-1 complex. Resumos. 1992 ;[citado 2024 nov. 02 ] -
Vancouver
Kimura E, Sonobe MH, Armelin MCS, Armelin HA. Acth modulates gene transcription in adrenocortical cells via ap-1 complex. Resumos. 1992 ;[citado 2024 nov. 02 ] - Control of function and proliferation by acth in mouse adrenocortical cells: induction of fos and jun proto-oncogene families
- Induction of fos and jun proteins by adrenocorticoprotein and phorbol ester but not by 3',5'-ciclic adenosine monophosphate derivates
- Transcriptional pattern of c-fos oncogene in mouse malignant adrenocortical cell cultures
- Phorbol ester mimicks adrenocorticotropic hormone (acth) action in adrenocortical mouse cells
- Role of proto-oncogene c-ki-ras amplification and overexpression in the malignancy of y-1 adrenocortical tumor cells
- Kinetcs of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth dibuthyl camp and phorbol ester in mouse adrenocortical cells
- Relevance of c-fos proto-oncogene induction for the steroidogenic response to acth, da amp and phorbol ester in adrenocortical cells
- Kinetics of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth, dcamp and phorbol ester in mouse adrenocortical cells
- Amplified ki-ras oncogene elimination as the basis for normal reversion in malignant adrenocortical cells
- Phorbol ester mimics acth action in corticoadrenal cells stimulating steroidogenesis blocking cell cycle, changing cell shape, and inducing c-fos proto-oncogene expression
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