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Trabalho de evento-resumo periodico
Effect of long-term consumption of partially oxidized omega-6 fatty acids on oxidative stress and inflammation biomarkers associated with atherosclerosis in LDLr(-/-) mice (2020)
Nogueira, Marina; Scolaro, Bianca ; Vieira, Thiago; Teixeira, Pryscila Dryelle Sousa ; Donato Junior, José ; Cogliati, Bruno ; Milne, Ginger ; Castro, Inar Alves de
Source: Free Radical Biology and Medicine. Conference titles: SfRBM Annual Conference. Unidades: ICB, FMVZ
Subjects: ARTERIOSCLEROSE, ÁCIDOS GRAXOS OMEGA 6, BIOMARCADORES, MICROBIOLOGIA, FISIOLOGIA, ESTRESSE OXIDATIVO, CAMUNDONGOS, INFLAMAÇÃO
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ABNT
NOGUEIRA, Marina et al. Effect of long-term consumption of partially oxidized omega-6 fatty acids on oxidative stress and inflammation biomarkers associated with atherosclerosis in LDLr(-/-) mice. Free Radical Biology and Medicine. New York: Elsevier. Disponível em: https://doi.org/10.1016/j.freeradbiomed.2020.10.243. Acesso em: 12 ago. 2024. , 2020
APA
Nogueira, M., Scolaro, B., Vieira, T., Teixeira, P. D. S., Donato Junior, J., Cogliati, B., et al. (2020). Effect of long-term consumption of partially oxidized omega-6 fatty acids on oxidative stress and inflammation biomarkers associated with atherosclerosis in LDLr(-/-) mice. Free Radical Biology and Medicine. New York: Elsevier. doi:10.1016/j.freeradbiomed.2020.10.243
NLM
Nogueira M, Scolaro B, Vieira T, Teixeira PDS, Donato Junior J, Cogliati B, Milne G, Castro IA de. Effect of long-term consumption of partially oxidized omega-6 fatty acids on oxidative stress and inflammation biomarkers associated with atherosclerosis in LDLr(-/-) mice [Internet]. Free Radical Biology and Medicine. 2020 ; 159 S94-S95.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2020.10.243
Vancouver
Nogueira M, Scolaro B, Vieira T, Teixeira PDS, Donato Junior J, Cogliati B, Milne G, Castro IA de. Effect of long-term consumption of partially oxidized omega-6 fatty acids on oxidative stress and inflammation biomarkers associated with atherosclerosis in LDLr(-/-) mice [Internet]. Free Radical Biology and Medicine. 2020 ; 159 S94-S95.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2020.10.243
Artigo de periodico
Heat shock protein B1 is a key mediator of prolactin-induced beta-cell cytoprotection against oxidative stress (2019)
Terra, Letícia F.; Wailemann, Rosângela A. M; Santos, Ancély Ferreira dos ; Gomes, Vinícius M.; Silva, Railmara Pereira da; Laporte, Anna; Meotti, Flavia Carla ; Terra, Walter Ribeiro ; Palmisano, Giuseppe ; Lortz, Stephan; Labriola, Leticia
Source: Free Radical Biology and Medicine. Unidades: IQ, ICB
Subjects: ESTRESSE OXIDATIVO, DIABETES MELLITUS, PARASITOLOGIA, PROLACTINA, ESTRESSE OXIDATIVO, PROTEÍNAS, ILHOTAS DE LANGERHANS, APOPTOSE, PROTEÍNAS DO CHOQUE TÉRMICO, CITOCINAS, ESPECTROMETRIA DE MASSAS, ANTIOXIDANTES
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TERRA, Letícia F. et al. Heat shock protein B1 is a key mediator of prolactin-induced beta-cell cytoprotection against oxidative stress. Free Radical Biology and Medicine, v. 134, p. 394-405, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2019.01.023. Acesso em: 12 ago. 2024.
APA
Terra, L. F., Wailemann, R. A. M., Santos, A. F. dos, Gomes, V. M., Silva, R. P. da, Laporte, A., et al. (2019). Heat shock protein B1 is a key mediator of prolactin-induced beta-cell cytoprotection against oxidative stress. Free Radical Biology and Medicine, 134, 394-405. doi:10.1016/j.freeradbiomed.2019.01.023
NLM
Terra LF, Wailemann RAM, Santos AF dos, Gomes VM, Silva RP da, Laporte A, Meotti FC, Terra WR, Palmisano G, Lortz S, Labriola L. Heat shock protein B1 is a key mediator of prolactin-induced beta-cell cytoprotection against oxidative stress [Internet]. Free Radical Biology and Medicine. 2019 ; 134 394-405.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2019.01.023
Vancouver
Terra LF, Wailemann RAM, Santos AF dos, Gomes VM, Silva RP da, Laporte A, Meotti FC, Terra WR, Palmisano G, Lortz S, Labriola L. Heat shock protein B1 is a key mediator of prolactin-induced beta-cell cytoprotection against oxidative stress [Internet]. Free Radical Biology and Medicine. 2019 ; 134 394-405.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2019.01.023
Artigo de periodico
Nitrite treatment downregulates vascular MMP-2 activity and inhibits vascular remodeling in hypertension independently of its antihypertensive effects (2019)
Rizzi, Elen; Amaral, Jefferson Henrich; Guimarães, Danielle Aparecida; Conde-Tella, Sandra de Oliveira; Pinheiro, Lucas Cézar ; Gerlach, Raquel Fernanda; Castro, Michele Mazzaron de; Tanus-Santos, José Eduardo
Source: Free Radical Biology and Medicine. Unidades: FMRP, EERP, FORP
Subjects: NITRITOS, ESTRESSE OXIDATIVO, HIPERTENSÃO
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ABNT
RIZZI, Elen et al. Nitrite treatment downregulates vascular MMP-2 activity and inhibits vascular remodeling in hypertension independently of its antihypertensive effects. Free Radical Biology and Medicine, v. 130, p. 234-243, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2018.11.002. Acesso em: 12 ago. 2024.
APA
Rizzi, E., Amaral, J. H., Guimarães, D. A., Conde-Tella, S. de O., Pinheiro, L. C., Gerlach, R. F., et al. (2019). Nitrite treatment downregulates vascular MMP-2 activity and inhibits vascular remodeling in hypertension independently of its antihypertensive effects. Free Radical Biology and Medicine, 130, 234-243. doi:10.1016/j.freeradbiomed.2018.11.002
NLM
Rizzi E, Amaral JH, Guimarães DA, Conde-Tella S de O, Pinheiro LC, Gerlach RF, Castro MM de, Tanus-Santos JE. Nitrite treatment downregulates vascular MMP-2 activity and inhibits vascular remodeling in hypertension independently of its antihypertensive effects [Internet]. Free Radical Biology and Medicine. 2019 ; 130 234-243.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.11.002
Vancouver
Rizzi E, Amaral JH, Guimarães DA, Conde-Tella S de O, Pinheiro LC, Gerlach RF, Castro MM de, Tanus-Santos JE. Nitrite treatment downregulates vascular MMP-2 activity and inhibits vascular remodeling in hypertension independently of its antihypertensive effects [Internet]. Free Radical Biology and Medicine. 2019 ; 130 234-243.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.11.002
Artigo de periodico
The key role of UVA-light induced oxidative stress in human Xeroderma pigmentosum variant cells (2019)
Moreno, Natália Cestari ; Garcia, Camila Carrião Machado ; Munford, Veridiana ; Rocha, Clarissa Ribeiro Reily ; Pelegrini, Alessandra Luiza ; Corradi, Camila ; Sarasin, Alain R.; Menck, Carlos Frederico Martins
Source: Free Radical Biology and Medicine. Unidades: ICB, IQ, BIOINFORMÁTICA
Subjects: MICROBIOLOGIA, DANO AO DNA, DOENÇAS GENÉTICAS, PELE, ESTRESSE OXIDATIVO, DNA POLIMERASES, NEOPLASIAS CUTÂNEAS, ANTIOXIDANTES, CARCINOGÊNESE
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ABNT
MORENO, Natália Cestari et al. The key role of UVA-light induced oxidative stress in human Xeroderma pigmentosum variant cells. Free Radical Biology and Medicine, v. 131, p. 432-442, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2018.12.012. Acesso em: 12 ago. 2024.
APA
Moreno, N. C., Garcia, C. C. M., Munford, V., Rocha, C. R. R., Pelegrini, A. L., Corradi, C., et al. (2019). The key role of UVA-light induced oxidative stress in human Xeroderma pigmentosum variant cells. Free Radical Biology and Medicine, 131, 432-442. doi:10.1016/j.freeradbiomed.2018.12.012
NLM
Moreno NC, Garcia CCM, Munford V, Rocha CRR, Pelegrini AL, Corradi C, Sarasin AR, Menck CFM. The key role of UVA-light induced oxidative stress in human Xeroderma pigmentosum variant cells [Internet]. Free Radical Biology and Medicine. 2019 ; 131 432-442.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.12.012
Vancouver
Moreno NC, Garcia CCM, Munford V, Rocha CRR, Pelegrini AL, Corradi C, Sarasin AR, Menck CFM. The key role of UVA-light induced oxidative stress in human Xeroderma pigmentosum variant cells [Internet]. Free Radical Biology and Medicine. 2019 ; 131 432-442.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.12.012
Artigo de periodico
Carbon Dioxide-catalyzed peroxynitrite reactivity – The Resilience of the radical mechanism after two decades of research (2019)
Augusto, Ohara ; Goldstein, Sara; Hurst, James K; Lind, Johan; Lymar, Sergei V; Merenyi, Gabor; Radi, Rafael
Source: Free Radical Biology and Medicine. Unidade: IQ
Subjects: DIÓXIDO DE CARBONO, ESTRESSE OXIDATIVO
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ABNT
AUGUSTO, Ohara et al. Carbon Dioxide-catalyzed peroxynitrite reactivity – The Resilience of the radical mechanism after two decades of research. Free Radical Biology and Medicine, v. 135, p. 210-215, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2019.02.026. Acesso em: 12 ago. 2024.
APA
Augusto, O., Goldstein, S., Hurst, J. K., Lind, J., Lymar, S. V., Merenyi, G., & Radi, R. (2019). Carbon Dioxide-catalyzed peroxynitrite reactivity – The Resilience of the radical mechanism after two decades of research. Free Radical Biology and Medicine, 135, 210-215. doi:10.1016/j.freeradbiomed.2019.02.026
NLM
Augusto O, Goldstein S, Hurst JK, Lind J, Lymar SV, Merenyi G, Radi R. Carbon Dioxide-catalyzed peroxynitrite reactivity – The Resilience of the radical mechanism after two decades of research [Internet]. Free Radical Biology and Medicine. 2019 ; 135 210-215.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2019.02.026
Vancouver
Augusto O, Goldstein S, Hurst JK, Lind J, Lymar SV, Merenyi G, Radi R. Carbon Dioxide-catalyzed peroxynitrite reactivity – The Resilience of the radical mechanism after two decades of research [Internet]. Free Radical Biology and Medicine. 2019 ; 135 210-215.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2019.02.026
Artigo de periodico
Targeting mitochondrial dysfunction and oxidative stress in heart failure: challenges and opportunities (2018)
Kiyuna, Ligia Akemi; Albuquerque, Rudá Prestes e; Chen, Che-Hong; Rosen, Daria Mochly; Ferreira, Julio Cesar Batista
Source: Free Radical Biology and Medicine. Unidade: ICB
Subjects: BIOENERGÉTICA, MITOCÔNDRIAS, ALDEÍDOS, INSUFICIÊNCIA CARDÍACA, TREINAMENTO FÍSICO, ESTRESSE OXIDATIVO, BIOENERGÉTICA
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ABNT
KIYUNA, Ligia Akemi et al. Targeting mitochondrial dysfunction and oxidative stress in heart failure: challenges and opportunities. Free Radical Biology and Medicine, v. 129, p. 155-168, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2018.09.019. Acesso em: 12 ago. 2024.
APA
Kiyuna, L. A., Albuquerque, R. P. e, Chen, C. -H., Rosen, D. M., & Ferreira, J. C. B. (2018). Targeting mitochondrial dysfunction and oxidative stress in heart failure: challenges and opportunities. Free Radical Biology and Medicine, 129, 155-168. doi:10.1016/j.freeradbiomed.2018.09.019
NLM
Kiyuna LA, Albuquerque RP e, Chen C-H, Rosen DM, Ferreira JCB. Targeting mitochondrial dysfunction and oxidative stress in heart failure: challenges and opportunities [Internet]. Free Radical Biology and Medicine. 2018 ; 129 155-168.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.09.019
Vancouver
Kiyuna LA, Albuquerque RP e, Chen C-H, Rosen DM, Ferreira JCB. Targeting mitochondrial dysfunction and oxidative stress in heart failure: challenges and opportunities [Internet]. Free Radical Biology and Medicine. 2018 ; 129 155-168.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.09.019
Artigo de periodico
Erythrocyte SOD1 activity, but not SOD1 polymorphisms, is associated with ICU mortality in patients with septic shock (2018)
Costa, Nara Aline; Rogero, Marcelo Macedo ; Norde, Marina Maintinguer; Minicucci, Marcos Ferreira; Cunha, Natália Baraldi; Gut, Ana Lúcia; Azevedo, Paula Schmidt; Polegato, Bertha Furlan; Zornoff, Leonardo Antonio Mamede; Paiva, Sérgio Alberto Rupp de; Reis, Bruna Zavarize ; Fernandes, Ana Angelica Henrique
Source: Free Radical Biology and Medicine. Unidades: FSP, FCF
Subjects: CHOQUE SÉPTICO, MORTALIDADE, UNIDADES DE TERAPIA INTENSIVA, ESTRESSE OXIDATIVO
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ABNT
COSTA, Nara Aline et al. Erythrocyte SOD1 activity, but not SOD1 polymorphisms, is associated with ICU mortality in patients with septic shock. Free Radical Biology and Medicine, v. 124, p. 199-204, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2018.06.013. Acesso em: 12 ago. 2024.
APA
Costa, N. A., Rogero, M. M., Norde, M. M., Minicucci, M. F., Cunha, N. B., Gut, A. L., et al. (2018). Erythrocyte SOD1 activity, but not SOD1 polymorphisms, is associated with ICU mortality in patients with septic shock. Free Radical Biology and Medicine, 124, 199-204. doi:10.1016/j.freeradbiomed.2018.06.013
NLM
Costa NA, Rogero MM, Norde MM, Minicucci MF, Cunha NB, Gut AL, Azevedo PS, Polegato BF, Zornoff LAM, Paiva SAR de, Reis BZ, Fernandes AAH. Erythrocyte SOD1 activity, but not SOD1 polymorphisms, is associated with ICU mortality in patients with septic shock [Internet]. Free Radical Biology and Medicine. 2018 ;124 199-204.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.06.013
Vancouver
Costa NA, Rogero MM, Norde MM, Minicucci MF, Cunha NB, Gut AL, Azevedo PS, Polegato BF, Zornoff LAM, Paiva SAR de, Reis BZ, Fernandes AAH. Erythrocyte SOD1 activity, but not SOD1 polymorphisms, is associated with ICU mortality in patients with septic shock [Internet]. Free Radical Biology and Medicine. 2018 ;124 199-204.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2018.06.013
Artigo de periodico
Allelic variations in the CYBA gene of NADPH oxidase and risk of kidney complications in patients with type 1 diabetes (2015)
Patente, Thiago A.; Mohammedi, Kamel; Bellili-Muñoz, Naïma; Driss, Fathi; Sanchez, Manuel; Fumeron, Frédéric; Roussel, Ronan; Hadjadj, Samy; Corrêa-Giannella, Maria Lúcia; Marre, Michel; Velho, Gilberto
Source: Free Radical Biology and Medicine. Unidade: FM
Subjects: ESTUDOS PROSPECTIVOS, ESTUDOS DE COORTES, NEFROPATIAS (ESTÁGIO), ESTRESSE OXIDATIVO, DIABETES MELLITUS
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ABNT
PATENTE, Thiago A. et al. Allelic variations in the CYBA gene of NADPH oxidase and risk of kidney complications in patients with type 1 diabetes. Free Radical Biology and Medicine, v. 86, p. 16-24, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2015.04.002. Acesso em: 12 ago. 2024.
APA
Patente, T. A., Mohammedi, K., Bellili-Muñoz, N., Driss, F., Sanchez, M., Fumeron, F., et al. (2015). Allelic variations in the CYBA gene of NADPH oxidase and risk of kidney complications in patients with type 1 diabetes. Free Radical Biology and Medicine, 86, 16-24. doi:10.1016/j.freeradbiomed.2015.04.002
NLM
Patente TA, Mohammedi K, Bellili-Muñoz N, Driss F, Sanchez M, Fumeron F, Roussel R, Hadjadj S, Corrêa-Giannella ML, Marre M, Velho G. Allelic variations in the CYBA gene of NADPH oxidase and risk of kidney complications in patients with type 1 diabetes [Internet]. Free Radical Biology and Medicine. 2015 ; 86 16-24.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2015.04.002
Vancouver
Patente TA, Mohammedi K, Bellili-Muñoz N, Driss F, Sanchez M, Fumeron F, Roussel R, Hadjadj S, Corrêa-Giannella ML, Marre M, Velho G. Allelic variations in the CYBA gene of NADPH oxidase and risk of kidney complications in patients with type 1 diabetes [Internet]. Free Radical Biology and Medicine. 2015 ; 86 16-24.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2015.04.002
Artigo de periodico
'beta IND.1'-Adrenergic blockers exert antioxidant effects, reduce matrix metalloproteinase activity, and improve renovascular hypertension-induced cardiac hypertrophy (2014)
Rizzi, Elen; Guimarães, Danielle A.; Ceron, Carla S.; Prado, Cibele M.; Pinheiro, Lucas C.; Martins-Oliveira, Alisson; Gerlach, Raquel Fernanda; Tanus-Santos, José Eduardo
Source: Free Radical Biology and Medicine. Unidades: FORP, FMRP
Subjects: HIPERTROFIA VENTRICULAR ESQUERDA, ANTIOXIDANTES, RADICAIS LIVRES, ESTRESSE OXIDATIVO
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ABNT
RIZZI, Elen et al. 'beta IND.1'-Adrenergic blockers exert antioxidant effects, reduce matrix metalloproteinase activity, and improve renovascular hypertension-induced cardiac hypertrophy. Free Radical Biology and Medicine, v. 73, p. 308-317, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2014.05.024. Acesso em: 12 ago. 2024.
APA
Rizzi, E., Guimarães, D. A., Ceron, C. S., Prado, C. M., Pinheiro, L. C., Martins-Oliveira, A., et al. (2014). 'beta IND.1'-Adrenergic blockers exert antioxidant effects, reduce matrix metalloproteinase activity, and improve renovascular hypertension-induced cardiac hypertrophy. Free Radical Biology and Medicine, 73, 308-317. doi:10.1016/j.freeradbiomed.2014.05.024
NLM
Rizzi E, Guimarães DA, Ceron CS, Prado CM, Pinheiro LC, Martins-Oliveira A, Gerlach RF, Tanus-Santos JE. 'beta IND.1'-Adrenergic blockers exert antioxidant effects, reduce matrix metalloproteinase activity, and improve renovascular hypertension-induced cardiac hypertrophy [Internet]. Free Radical Biology and Medicine. 2014 ; 73 308-317.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2014.05.024
Vancouver
Rizzi E, Guimarães DA, Ceron CS, Prado CM, Pinheiro LC, Martins-Oliveira A, Gerlach RF, Tanus-Santos JE. 'beta IND.1'-Adrenergic blockers exert antioxidant effects, reduce matrix metalloproteinase activity, and improve renovascular hypertension-induced cardiac hypertrophy [Internet]. Free Radical Biology and Medicine. 2014 ; 73 308-317.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2014.05.024
Artigo de periodico
Caffeic acid phenethyl ester (CAPE) protects against oxidative stress induced by obesity in mice (2012)
Bezerra, Rosângela Maria Neves; Caetano, Aline Camila ; Veiga, L. F; Capaldi, F. R; Alencar, Severino Matias ; Azevedo , Ricardo Antunes de
Source: Free Radical Biology and Medicine. Unidade: ESALQ
Subjects: ANTIOXIDANTES, CAMUNDONGOS, ESTRESSE OXIDATIVO, OBESIDADE
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ABNT
BEZERRA, Rosângela Maria Neves et al. Caffeic acid phenethyl ester (CAPE) protects against oxidative stress induced by obesity in mice. Free Radical Biology and Medicine, v. 53 Supl. 1, p. S222, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2012.08.466. Acesso em: 12 ago. 2024.
APA
Bezerra, R. M. N., Caetano, A. C., Veiga, L. F., Capaldi, F. R., Alencar, S. M., & Azevedo , R. A. de. (2012). Caffeic acid phenethyl ester (CAPE) protects against oxidative stress induced by obesity in mice. Free Radical Biology and Medicine, 53 Supl. 1, S222. doi:10.1016/j.freeradbiomed.2012.08.466
NLM
Bezerra RMN, Caetano AC, Veiga LF, Capaldi FR, Alencar SM, Azevedo RA de. Caffeic acid phenethyl ester (CAPE) protects against oxidative stress induced by obesity in mice [Internet]. Free Radical Biology and Medicine. 2012 ; 53 Supl. 1 S222.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2012.08.466
Vancouver
Bezerra RMN, Caetano AC, Veiga LF, Capaldi FR, Alencar SM, Azevedo RA de. Caffeic acid phenethyl ester (CAPE) protects against oxidative stress induced by obesity in mice [Internet]. Free Radical Biology and Medicine. 2012 ; 53 Supl. 1 S222.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2012.08.466
Artigo de periodico
Tissue-, substrate-, and site-specific characteristics of mitochondrial reactive oxygen species generation (2009)
Tahara, Erich Birelli; Navarete, Felipe Donizeti Teston; Kowaltowski, Alicia Juliana
Source: Free Radical Biology and Medicine. Unidade: IQ
Subjects: RADICAIS LIVRES, ESTRESSE OXIDATIVO, MITOCÔNDRIAS
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ABNT
TAHARA, Erich Birelli e NAVARETE, Felipe Donizeti Teston e KOWALTOWSKI, Alicia Juliana. Tissue-, substrate-, and site-specific characteristics of mitochondrial reactive oxygen species generation. Free Radical Biology and Medicine, v. 46, n. 9, p. 1283-1297, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2009.02.008. Acesso em: 12 ago. 2024.
APA
Tahara, E. B., Navarete, F. D. T., & Kowaltowski, A. J. (2009). Tissue-, substrate-, and site-specific characteristics of mitochondrial reactive oxygen species generation. Free Radical Biology and Medicine, 46( 9), 1283-1297. doi:10.1016/j.freeradbiomed.2009.02.008
NLM
Tahara EB, Navarete FDT, Kowaltowski AJ. Tissue-, substrate-, and site-specific characteristics of mitochondrial reactive oxygen species generation [Internet]. Free Radical Biology and Medicine. 2009 ; 46( 9): 1283-1297.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2009.02.008
Vancouver
Tahara EB, Navarete FDT, Kowaltowski AJ. Tissue-, substrate-, and site-specific characteristics of mitochondrial reactive oxygen species generation [Internet]. Free Radical Biology and Medicine. 2009 ; 46( 9): 1283-1297.[citado 2024 ago. 12 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2009.02.008
Trabalho de evento-resumo periodico
Classification of individuals with dyslipidemia controlled by statins according to plasma biomarkers of oxidative stress using cluster analysis (2009)
Botelho, Patrícia Borges; Fioratti, Cyntia Okoshi; Abdalla, Dulcinéia Saes Parra ; Bertolami, Marcelo Chiara; Castro, Inar Alves de
Source: Free Radical Biology and Medicine. Conference titles: Annual Meeting of the Society for Free Radical Biology and Medicine. Unidade: FCF
Subjects: DISLIPIDEMIAS, ESTRESSE OXIDATIVO, BIOMARCADORES
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ABNT
BOTELHO, Patrícia Borges et al. Classification of individuals with dyslipidemia controlled by statins according to plasma biomarkers of oxidative stress using cluster analysis. Free Radical Biology and Medicine. New York: Faculdade de Ciências Farmacêuticas, Universidade de São Paulo. . Acesso em: 12 ago. 2024. , 2009
APA
Botelho, P. B., Fioratti, C. O., Abdalla, D. S. P., Bertolami, M. C., & Castro, I. A. de. (2009). Classification of individuals with dyslipidemia controlled by statins according to plasma biomarkers of oxidative stress using cluster analysis. Free Radical Biology and Medicine. New York: Faculdade de Ciências Farmacêuticas, Universidade de São Paulo.
NLM
Botelho PB, Fioratti CO, Abdalla DSP, Bertolami MC, Castro IA de. Classification of individuals with dyslipidemia controlled by statins according to plasma biomarkers of oxidative stress using cluster analysis. Free Radical Biology and Medicine. 2009 ; 47 S55 res. 122.[citado 2024 ago. 12 ]
Vancouver
Botelho PB, Fioratti CO, Abdalla DSP, Bertolami MC, Castro IA de. Classification of individuals with dyslipidemia controlled by statins according to plasma biomarkers of oxidative stress using cluster analysis. Free Radical Biology and Medicine. 2009 ; 47 S55 res. 122.[citado 2024 ago. 12 ]
Trabalho de evento-resumo periodico
Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells (2008)
Sartori, Adriano; Garay-Malpartida, Humberto Miguel; Forni, Maria Fernanda Pereira de Araújo Demonte; Schumacher, Robert Ivan; Dutra, Fernando; Sogayar, Mari Cleide ; Bechara, Etelvino José Henriques
Source: Free Radical Biology and Medicine. Conference titles: Annual Meeting of the Society for Free Radical Biology and Medicine. Unidades: EACH, IQ
Subjects: ESTRESSE OXIDATIVO, BIOQUÍMICA, DIABETES MELLITUS
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ABNT
SARTORI, Adriano et al. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Free Radical Biology and Medicine. New York: Escola de Artes, Ciências e Humanidades, Universidade de São Paulo. . Acesso em: 12 ago. 2024. , 2008
APA
Sartori, A., Garay-Malpartida, H. M., Forni, M. F. P. de A. D., Schumacher, R. I., Dutra, F., Sogayar, M. C., & Bechara, E. J. H. (2008). Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Free Radical Biology and Medicine. New York: Escola de Artes, Ciências e Humanidades, Universidade de São Paulo.
NLM
Sartori A, Garay-Malpartida HM, Forni MFP de AD, Schumacher RI, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Free Radical Biology and Medicine. 2008 ; 45 S157 res. 426.[citado 2024 ago. 12 ]
Vancouver
Sartori A, Garay-Malpartida HM, Forni MFP de AD, Schumacher RI, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Free Radical Biology and Medicine. 2008 ; 45 S157 res. 426.[citado 2024 ago. 12 ]

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