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Artigo de periodico
NLRP3 activation contributes to endothelin-1-induced erectile dysfunction (2023)
Fais, Rafael Sobrano; Costa, Rafael Menezes da ; Mendes, Allan Carvalho; Mestriner, Fabiola Leslie Antunes Cardoso; Comerma-Steffensen, Simón Gabriel; Tostes, Rita de Cássia Aleixo ; Simonsen, Ulf; Carneiro, Fernando Silva
Source: Journal of Cellular and Molecular Medicine. Unidade: FMRP
Subjects: ANTAGONISTAS, ENDOTELINAS, DISFUNÇÃO ERÉTIL, ESPÉCIES REATIVAS DE OXIGÊNIO, INFLAMAÇÃO
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ABNT
FAIS, Rafael Sobrano et al. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction. Journal of Cellular and Molecular Medicine, v. 27, n. 1, p. 1-14, 2023Tradução . . Disponível em: https://doi.org/10.1111/jcmm.17463. Acesso em: 13 nov. 2024.
APA
Fais, R. S., Costa, R. M. da, Mendes, A. C., Mestriner, F. L. A. C., Comerma-Steffensen, S. G., Tostes, R. de C. A., et al. (2023). NLRP3 activation contributes to endothelin-1-induced erectile dysfunction. Journal of Cellular and Molecular Medicine, 27( 1), 1-14. doi:10.1111/jcmm.17463
NLM
Fais RS, Costa RM da, Mendes AC, Mestriner FLAC, Comerma-Steffensen SG, Tostes R de CA, Simonsen U, Carneiro FS. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction [Internet]. Journal of Cellular and Molecular Medicine. 2023 ; 27( 1): 1-14.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1111/jcmm.17463
Vancouver
Fais RS, Costa RM da, Mendes AC, Mestriner FLAC, Comerma-Steffensen SG, Tostes R de CA, Simonsen U, Carneiro FS. NLRP3 activation contributes to endothelin-1-induced erectile dysfunction [Internet]. Journal of Cellular and Molecular Medicine. 2023 ; 27( 1): 1-14.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1111/jcmm.17463
Artigo de periodico
Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy (2022)
Santos, Jeimison D.; Oliveira Neto, José Teles de ; Barros, Paula Rodrigues de; Damasceno, Luis Eduardo Alves; Lautherbach, Natalia; Assis, Ana Paula de; Silva, Carlos Alberto Aguiar da; Sorgi, Carlos Arterio ; Faccioli, Lucia Helena ; Kettelhut, Isis do Carmo ; Salgado, Hélio César ; Carneiro, Fernando Silva ; Alves-Filho, José Carlos ; Tostes, Rita de Cássia Aleixo
Source: American Journal of Physiology-Heart and Circulatory Physiology. Unidades: FMRP, FFCLRP, FCFRP
Subjects: DOENÇAS CARDIOVASCULARES, FARMACOTERAPIA, HORMÔNIOS, ESTEROIDES, TESTOSTERONA
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ABNT
SANTOS, Jeimison D. et al. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, v. 323, p. H322-H335, 2022Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00182.2022. Acesso em: 13 nov. 2024.
APA
Santos, J. D., Oliveira Neto, J. T. de, Barros, P. R. de, Damasceno, L. E. A., Lautherbach, N., Assis, A. P. de, et al. (2022). Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy. American Journal of Physiology-Heart and Circulatory Physiology, 323, H322-H335. doi:10.1152/ajpheart.00182.2022
NLM
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Vancouver
Santos JD, Oliveira Neto JT de, Barros PR de, Damasceno LEA, Lautherbach N, Assis AP de, Silva CAA da, Sorgi CA, Faccioli LH, Kettelhut I do C, Salgado HC, Carneiro FS, Alves-Filho JC, Tostes R de CA. Th17 cell-linked mechanisms mediate vascular dysfunction induced by testosterone in a mouse model of gender-affirming hormone therapy [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2022 ; 323 H322-H335.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajpheart.00182.2022
Artigo de periodico
Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress (2022)
Costa, Rafael Menezes da ; Alves-Lopes, Rhéure ; Alves, Juliano Vilela ; Servian, Carolina P.; Mestriner, Fabiola Leslie Antunes Cardoso; Carneiro, Fernando Silva ; Lobato, Núbia de S.; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: OBESIDADE, ESTRESSE OXIDATIVO, TESTOSTERONA, DOENÇAS VASCULARES
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ABNT
COSTA, Rafael Menezes da et al. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, v. 13, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphys.2022.837603. Acesso em: 13 nov. 2024.
APA
Costa, R. M. da, Alves-Lopes, R., Alves, J. V., Servian, C. P., Mestriner, F. L. A. C., Carneiro, F. S., et al. (2022). Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, 13, 1-14. doi:10.3389/fphys.2022.837603
NLM
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Vancouver
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Artigo de periodico
Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction (2022)
Silva, Josiane Fernandes da; Bolsoni, Juliana A.; Costa, Rafael Menezes da ; Alves, Juliano Vilela ; Bressan, Alecsander Fabrício Moreira ; Silva, Luiz Eduardo Virgilio da ; Costa, Tiago Januário da ; Oliveira, Antonio E. R.; Manzato, Carla Pavan; Silva, Carlos Alberto Aguiar da; Fazan Júnior, Rubens ; Cunha, Fernando de Queiroz ; Nakaya, Helder Takashi Imoto ; Carneiro, Fernando Silva ; Tostes, Rita de Cássia Aleixo
Source: British Journal of Pharmacology. Unidades: FMRP, FCF
Subjects: GORDURAS NA DIETA, CÉLULAS ENDOTELIAIS, ENDOTÉLIO VASCULAR, OBESIDADE, HIDROCARBONETOS, VASODILATAÇÃO
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ABNT
SILVA, Josiane Fernandes da et al. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction. British Journal of Pharmacology, v. 179, n. 12, p. 2938-2952, 2022Tradução . . Disponível em: https://doi.org/10.1111/bph.15789. Acesso em: 13 nov. 2024.
APA
Silva, J. F. da, Bolsoni, J. A., Costa, R. M. da, Alves, J. V., Bressan, A. F. M., Silva, L. E. V. da, et al. (2022). Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction. British Journal of Pharmacology, 179( 12), 2938-2952. doi:10.1111/bph.15789
NLM
Silva JF da, Bolsoni JA, Costa RM da, Alves JV, Bressan AFM, Silva LEV da, Costa TJ da, Oliveira AER, Manzato CP, Silva CAA da, Fazan Júnior R, Cunha F de Q, Nakaya HTI, Carneiro FS, Tostes R de CA. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction [Internet]. British Journal of Pharmacology. 2022 ; 179( 12): 2938-2952.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1111/bph.15789
Vancouver
Silva JF da, Bolsoni JA, Costa RM da, Alves JV, Bressan AFM, Silva LEV da, Costa TJ da, Oliveira AER, Manzato CP, Silva CAA da, Fazan Júnior R, Cunha F de Q, Nakaya HTI, Carneiro FS, Tostes R de CA. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction [Internet]. British Journal of Pharmacology. 2022 ; 179( 12): 2938-2952.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1111/bph.15789
Artigo de periodico
Vasoconstrictor and hemodynamic effects of a methanolic extract from Rhinella marina toad poison (2022)
Santos, Cintia Vieira dos; Kerkhoff, Jacqueline; Tomazelli, Caroline Aparecida ; Wenceslau, Camilla Ferreira; Sinhorin, Adilson Paulo; Rodrigues, Domingos de Jesus; Carneiro, Fernando Silva ; Bomfim, Gisele Facholi
Source: Toxicon. Unidades: FMRP, ICB
Subjects: ADENOSINA TRIFOSFATO, CANAIS DE CÁLCIO, ENDOTÉLIO, ÓXIDO NÍTRICO, PROSTAGLANDINAS, SEROTONINA
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ABNT
SANTOS, Cintia Vieira dos et al. Vasoconstrictor and hemodynamic effects of a methanolic extract from Rhinella marina toad poison. Toxicon, v. 218, p. 57-65, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.toxicon.2022.08.018. Acesso em: 13 nov. 2024.
APA
Santos, C. V. dos, Kerkhoff, J., Tomazelli, C. A., Wenceslau, C. F., Sinhorin, A. P., Rodrigues, D. de J., et al. (2022). Vasoconstrictor and hemodynamic effects of a methanolic extract from Rhinella marina toad poison. Toxicon, 218, 57-65. doi:10.1016/j.toxicon.2022.08.018
NLM
Santos CV dos, Kerkhoff J, Tomazelli CA, Wenceslau CF, Sinhorin AP, Rodrigues D de J, Carneiro FS, Bomfim GF. Vasoconstrictor and hemodynamic effects of a methanolic extract from Rhinella marina toad poison [Internet]. Toxicon. 2022 ; 218 57-65.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.toxicon.2022.08.018
Vancouver
Santos CV dos, Kerkhoff J, Tomazelli CA, Wenceslau CF, Sinhorin AP, Rodrigues D de J, Carneiro FS, Bomfim GF. Vasoconstrictor and hemodynamic effects of a methanolic extract from Rhinella marina toad poison [Internet]. Toxicon. 2022 ; 218 57-65.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.toxicon.2022.08.018
Artigo de periodico
Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage (2022)
Costa, Tiago Januário da ; Potje, Simone Regina ; Silva, Thais Fernanda de Campos Fraga da; Silva Neto, Júlio Alves da; Barros, Paula Rodrigues de; Rodrigues, Daniel; Machado, Mirele Resende; Martins Júnior, Ronaldo Bragança; Eichler, Rosangela Aparecida dos Santos; Benatti, Maira N.; Sá, Keyla Santos Guedes de; Almado, Carlos Eduardo Lopes; Castro, Italo A.; Pontelli, Marjorie C.; Serra, Leonardo La; Carneiro, Fernando Silva ; Becari, Christiane; Louzada Júnior, Paulo; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões; Arruda, Eurico; Martins, Maria Auxiliadora; Giachini, Fernanda R. C.; Bonato, Vânia Luiza Deperon; Zachara, Natasha E.; Bomfim, Gisele F.; Tostes, Rita de Cássia Aleixo
Source: Vascular Pharmacology. Unidades: FCFRP, ICB, FMRP
Subjects: COVID-19, MITOCÔNDRIAS, CÉLULAS ENDOTELIAIS, DNA MITOCONDRIAL
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ABNT
COSTA, Tiago Januário da et al. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, v. 142, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2021.106946. Acesso em: 13 nov. 2024.
APA
Costa, T. J. da, Potje, S. R., Silva, T. F. de C. F. da, Silva Neto, J. A. da, Barros, P. R. de, Rodrigues, D., et al. (2022). Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, 142. doi:10.1016/j.vph.2021.106946
NLM
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Vancouver
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Artigo de periodico
Treatment with nitrite prevents reactive oxygen species generation in the corpora cavernosa and restores intracavernosal pressure in hypertensive rats (2020)
Gonzaga, Natália de Almeida ; Vale, Gabriel Tavares do ; Silva, Carla Beatriz Pereira da ; Pinheiro, Lucas Cezar ; Leite, Letícia N.; Carneiro, Fernando Silva ; Tanus-Santos, José Eduardo ; Tirapelli, Carlos Renato
Source: Nitric Oxide. Unidades: EERP, FMRP
Subjects: DISFUNÇÃO ERÉTIL, HIPERTENSÃO, NITRITOS, ESPÉCIES REATIVAS DE OXIGÊNIO, ANTIOXIDANTES
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ABNT
GONZAGA, Natália de Almeida et al. Treatment with nitrite prevents reactive oxygen species generation in the corpora cavernosa and restores intracavernosal pressure in hypertensive rats. Nitric Oxide, v. 94, p. 19-26, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2019.10.006. Acesso em: 13 nov. 2024.
APA
Gonzaga, N. de A., Vale, G. T. do, Silva, C. B. P. da, Pinheiro, L. C., Leite, L. N., Carneiro, F. S., et al. (2020). Treatment with nitrite prevents reactive oxygen species generation in the corpora cavernosa and restores intracavernosal pressure in hypertensive rats. Nitric Oxide, 94, 19-26. doi:10.1016/j.niox.2019.10.006
NLM
Gonzaga N de A, Vale GT do, Silva CBP da, Pinheiro LC, Leite LN, Carneiro FS, Tanus-Santos JE, Tirapelli CR. Treatment with nitrite prevents reactive oxygen species generation in the corpora cavernosa and restores intracavernosal pressure in hypertensive rats [Internet]. Nitric Oxide. 2020 ; 94 19-26.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.niox.2019.10.006
Vancouver
Gonzaga N de A, Vale GT do, Silva CBP da, Pinheiro LC, Leite LN, Carneiro FS, Tanus-Santos JE, Tirapelli CR. Treatment with nitrite prevents reactive oxygen species generation in the corpora cavernosa and restores intracavernosal pressure in hypertensive rats [Internet]. Nitric Oxide. 2020 ; 94 19-26.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.niox.2019.10.006
Artigo de periodico
Angiotensin (1-7)-attenuated vasoconstriction is associated with the interleukin-10 signaling pathway (2020)
Freitas, Raiany Alves de; Júnior, Roberto R. P.; Justina, Vanessa Dela; Bressan, Alecsander Fabrício Moreira ; Bomfim, Gisele Facholi; Carneiro, Fernando Silva ; Giachini, Fernanda Regina; Lima, Victor Vitorino
Source: Life Sciences. Unidade: FMRP
Subjects: ANGIOTENSINAS, ANGIOTENSINA II, SISTEMA RENINA-ANGIOTENSINA, FOSFATASE ALCALINA
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ABNT
FREITAS, Raiany Alves de et al. Angiotensin (1-7)-attenuated vasoconstriction is associated with the interleukin-10 signaling pathway. Life Sciences, v. 262, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2020.118552. Acesso em: 13 nov. 2024.
APA
Freitas, R. A. de, Júnior, R. R. P., Justina, V. D., Bressan, A. F. M., Bomfim, G. F., Carneiro, F. S., et al. (2020). Angiotensin (1-7)-attenuated vasoconstriction is associated with the interleukin-10 signaling pathway. Life Sciences, 262. doi:10.1016/j.lfs.2020.118552
NLM
Freitas RA de, Júnior RRP, Justina VD, Bressan AFM, Bomfim GF, Carneiro FS, Giachini FR, Lima VV. Angiotensin (1-7)-attenuated vasoconstriction is associated with the interleukin-10 signaling pathway [Internet]. Life Sciences. 2020 ; 262[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2020.118552
Vancouver
Freitas RA de, Júnior RRP, Justina VD, Bressan AFM, Bomfim GF, Carneiro FS, Giachini FR, Lima VV. Angiotensin (1-7)-attenuated vasoconstriction is associated with the interleukin-10 signaling pathway [Internet]. Life Sciences. 2020 ; 262[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2020.118552
Artigo de periodico
Supraphysiological levels of testosterone induce vascular dysfunction via activation of the NLRP3 inflammasome (2020)
Alves, Juliano Vilela; Costa, Rafael Menezes da; Pereira, Camila André; Fedoce, Aline Garcia; Silva, Carlos Alberto Aguiar da; Carneiro, Fernando Silva ; Lobato, Núbia de Souza; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Immunology. Unidade: FMRP
Subjects: ANDRÓGENOS, AORTA TORÁCICA, INTERLEUCINA 1, MITOCÔNDRIAS, ESPÉCIES REATIVAS DE OXIGÊNIO, RECEPTORES ANDROGÊNICOS
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ABNT
ALVES, Juliano Vilela et al. Supraphysiological levels of testosterone induce vascular dysfunction via activation of the NLRP3 inflammasome. Frontiers in Immunology, v. 11, 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.01647. Acesso em: 13 nov. 2024.
APA
Alves, J. V., Costa, R. M. da, Pereira, C. A., Fedoce, A. G., Silva, C. A. A. da, Carneiro, F. S., et al. (2020). Supraphysiological levels of testosterone induce vascular dysfunction via activation of the NLRP3 inflammasome. Frontiers in Immunology, 11. doi:10.3389/fimmu.2020.01647
NLM
Alves JV, Costa RM da, Pereira CA, Fedoce AG, Silva CAA da, Carneiro FS, Lobato N de S, Tostes R de CA. Supraphysiological levels of testosterone induce vascular dysfunction via activation of the NLRP3 inflammasome [Internet]. Frontiers in Immunology. 2020 ; 11[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fimmu.2020.01647
Vancouver
Alves JV, Costa RM da, Pereira CA, Fedoce AG, Silva CAA da, Carneiro FS, Lobato N de S, Tostes R de CA. Supraphysiological levels of testosterone induce vascular dysfunction via activation of the NLRP3 inflammasome [Internet]. Frontiers in Immunology. 2020 ; 11[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fimmu.2020.01647
Artigo de periodico
Angiotensin (1-7) inhibits Ang II-mediated ERK1/2 activation by stimulating MKP-1 activation in vascular smooth muscle cells (2020)
Sousa-Lopes, Alejandra; Freitas, Raiany Alves de; Carneiro, Fernando Silva ; Nunes, Kenia Pedrosa; Allahdadi, Kyan James; Webb, Robert Clinton; Tostes, Rita de Cássia Aleixo ; Giachini, Fernanda Regina Casagrande; Lima, Victor Vitorino
Source: International Journal of Cellular and Molecular Medicine. Unidade: FMRP
Subjects: ANGIOTENSINAS, HIPERTENSÃO, HOMEOSTASE, SISTEMA NERVOSO CENTRAL
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ABNT
SOUSA-LOPES, Alejandra et al. Angiotensin (1-7) inhibits Ang II-mediated ERK1/2 activation by stimulating MKP-1 activation in vascular smooth muscle cells. International Journal of Cellular and Molecular Medicine, v. 9, n. 1, p. 50-61, 2020Tradução . . Disponível em: https://doi.org/10.22088/IJMCM.BUMS.9.1.50. Acesso em: 13 nov. 2024.
APA
Sousa-Lopes, A., Freitas, R. A. de, Carneiro, F. S., Nunes, K. P., Allahdadi, K. J., Webb, R. C., et al. (2020). Angiotensin (1-7) inhibits Ang II-mediated ERK1/2 activation by stimulating MKP-1 activation in vascular smooth muscle cells. International Journal of Cellular and Molecular Medicine, 9( 1), 50-61. doi:10.22088/IJMCM.BUMS.9.1.50
NLM
Sousa-Lopes A, Freitas RA de, Carneiro FS, Nunes KP, Allahdadi KJ, Webb RC, Tostes R de CA, Giachini FRC, Lima VV. Angiotensin (1-7) inhibits Ang II-mediated ERK1/2 activation by stimulating MKP-1 activation in vascular smooth muscle cells [Internet]. International Journal of Cellular and Molecular Medicine. 2020 ; 9( 1): 50-61.[citado 2024 nov. 13 ] Available from: https://doi.org/10.22088/IJMCM.BUMS.9.1.50
Vancouver
Sousa-Lopes A, Freitas RA de, Carneiro FS, Nunes KP, Allahdadi KJ, Webb RC, Tostes R de CA, Giachini FRC, Lima VV. Angiotensin (1-7) inhibits Ang II-mediated ERK1/2 activation by stimulating MKP-1 activation in vascular smooth muscle cells [Internet]. International Journal of Cellular and Molecular Medicine. 2020 ; 9( 1): 50-61.[citado 2024 nov. 13 ] Available from: https://doi.org/10.22088/IJMCM.BUMS.9.1.50
Artigo de periodico
The inflammasome NLRP3 plays a dual role on mouse corpora cavernosa relaxation (2019)
Fais, Rafael Sobrano; Rodrigues, Fernanda Luciano; Pereira, Camila André; Mendes, Allan Carvalho; Mestriner, Fabiola Leslie Antunes Cardoso; Tostes, Rita de Cássia Aleixo ; Carneiro, Fernando Silva
Source: Scientific Reports. Unidade: FMRP
Subjects: DISFUNÇÃO ERÉTIL, INFLAMAÇÃO, PROTEÍNAS
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ABNT
FAIS, Rafael Sobrano et al. The inflammasome NLRP3 plays a dual role on mouse corpora cavernosa relaxation. Scientific Reports, v. 9, n. 1, 2019Tradução . . Disponível em: https://doi.org/10.1038/s41598-019-52831-0. Acesso em: 13 nov. 2024.
APA
Fais, R. S., Rodrigues, F. L., Pereira, C. A., Mendes, A. C., Mestriner, F. L. A. C., Tostes, R. de C. A., & Carneiro, F. S. (2019). The inflammasome NLRP3 plays a dual role on mouse corpora cavernosa relaxation. Scientific Reports, 9( 1). doi:10.1038/s41598-019-52831-0
NLM
Fais RS, Rodrigues FL, Pereira CA, Mendes AC, Mestriner FLAC, Tostes R de CA, Carneiro FS. The inflammasome NLRP3 plays a dual role on mouse corpora cavernosa relaxation [Internet]. Scientific Reports. 2019 ; 9( 1):[citado 2024 nov. 13 ] Available from: https://doi.org/10.1038/s41598-019-52831-0
Vancouver
Fais RS, Rodrigues FL, Pereira CA, Mendes AC, Mestriner FLAC, Tostes R de CA, Carneiro FS. The inflammasome NLRP3 plays a dual role on mouse corpora cavernosa relaxation [Internet]. Scientific Reports. 2019 ; 9( 1):[citado 2024 nov. 13 ] Available from: https://doi.org/10.1038/s41598-019-52831-0
Artigo de periodico
Hypertension: a new treatment for an old disease? Targeting the immune system (2019)
Bomfim, Gisele Facholi; Cau, Stefany Bruno Assis; Bruno, Alexandre Santos; Fedoce, Aline Garcia; Carneiro, Fernando Silva
Source: British Journal of Pharmacology. Unidade: FMRP
Subjects: HIPERTENSÃO, FARMACOLOGIA, SISTEMA IMUNE
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ABNT
BOMFIM, Gisele Facholi et al. Hypertension: a new treatment for an old disease? Targeting the immune system. British Journal of Pharmacology, v. 176, n. 12, p. 2028-2048, 2019Tradução . . Disponível em: https://doi.org/10.1111/bph.14436. Acesso em: 13 nov. 2024.
APA
Bomfim, G. F., Cau, S. B. A., Bruno, A. S., Fedoce, A. G., & Carneiro, F. S. (2019). Hypertension: a new treatment for an old disease? Targeting the immune system. British Journal of Pharmacology, 176( 12), 2028-2048. doi:10.1111/bph.14436
NLM
Bomfim GF, Cau SBA, Bruno AS, Fedoce AG, Carneiro FS. Hypertension: a new treatment for an old disease? Targeting the immune system [Internet]. British Journal of Pharmacology. 2019 ; 176( 12): 2028-2048.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1111/bph.14436
Vancouver
Bomfim GF, Cau SBA, Bruno AS, Fedoce AG, Carneiro FS. Hypertension: a new treatment for an old disease? Targeting the immune system [Internet]. British Journal of Pharmacology. 2019 ; 176( 12): 2028-2048.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1111/bph.14436
Artigo de periodico
Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis (2018)
Pereira, Camila A.; Carneiro, Fernando Silva; Matsumoto, Takayuki; Passaglia, Rita de Cássia Aleixo Tostes
Source: Basic and Clinical Pharmacology and Toxicology. Unidade: FMRP
Subjects: DIABETES MELLITUS, FÁRMACOS, ENDOTÉLIO, ARTERIOSCLEROSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Camila A. et al. Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis. Basic and Clinical Pharmacology and Toxicology, v. 123, n. 5, p. 523-538, 2018Tradução . . Disponível em: https://doi.org/10.1111/bcpt.13054. Acesso em: 13 nov. 2024.
APA
Pereira, C. A., Carneiro, F. S., Matsumoto, T., & Passaglia, R. de C. A. T. (2018). Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis. Basic and Clinical Pharmacology and Toxicology, 123( 5), 523-538. doi:10.1111/bcpt.13054
NLM
Pereira CA, Carneiro FS, Matsumoto T, Passaglia R de CAT. Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis [Internet]. Basic and Clinical Pharmacology and Toxicology. 2018 ; 123( 5): 523-538.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1111/bcpt.13054
Vancouver
Pereira CA, Carneiro FS, Matsumoto T, Passaglia R de CAT. Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis [Internet]. Basic and Clinical Pharmacology and Toxicology. 2018 ; 123( 5): 523-538.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1111/bcpt.13054
Artigo de periodico
Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism (2018)
Nascimento, M.; Piran, R.; Da Costa, R. M.; Giordani, M. A.; Carneiro, Fernando Silva; Aguiar, D. H.; Dias, M. C.; Sugizaki, M. M.; Luvizotto, R. A.; Nascimento, A. F.; Bomfim, G. F.
Source: Life Sciences. Unidade: FMRP
Subjects: AORTA, FÍGADO, PRESSÃO SANGUÍNEA, ENDOTÉLIO VASCULAR, DOENÇAS VASCULARES, RATOS WISTAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NASCIMENTO, M. et al. Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism. Life Sciences, v. 212, p. 168-175, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.09.051. Acesso em: 13 nov. 2024.
APA
Nascimento, M., Piran, R., Da Costa, R. M., Giordani, M. A., Carneiro, F. S., Aguiar, D. H., et al. (2018). Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism. Life Sciences, 212, 168-175. doi:10.1016/j.lfs.2018.09.051
NLM
Nascimento M, Piran R, Da Costa RM, Giordani MA, Carneiro FS, Aguiar DH, Dias MC, Sugizaki MM, Luvizotto RA, Nascimento AF, Bomfim GF. Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism [Internet]. Life Sciences. 2018 ; 212 168-175.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2018.09.051
Vancouver
Nascimento M, Piran R, Da Costa RM, Giordani MA, Carneiro FS, Aguiar DH, Dias MC, Sugizaki MM, Luvizotto RA, Nascimento AF, Bomfim GF. Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism [Internet]. Life Sciences. 2018 ; 212 168-175.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2018.09.051
Artigo de periodico
O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway (2018)
Miguez, Jéssica S. G.; Dela Justina, Vanessa; Bressan, Alecsander F. M.; Marchi, Patrícia G. F.; França, Adenilda C. Honorio; Carneiro, Fernando Silva; Webb, R. Clinton; Passaglia, Rita de Cássia Aleixo Tostes; Giachini, Fernanda R.; Lima, Victor V.
Source: Life Sciences. Unidade: FMRP
Subjects: CITOCINAS, INTERLEUCINA 10, TRANSDUÇÃO DE SINAL CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MIGUEZ, Jéssica S. G. et al. O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway. Life Sciences, v. 209, p. 78-84, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.07.058. Acesso em: 13 nov. 2024.
APA
Miguez, J. S. G., Dela Justina, V., Bressan, A. F. M., Marchi, P. G. F., França, A. C. H., Carneiro, F. S., et al. (2018). O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway. Life Sciences, 209, 78-84. doi:10.1016/j.lfs.2018.07.058
NLM
Miguez JSG, Dela Justina V, Bressan AFM, Marchi PGF, França ACH, Carneiro FS, Webb RC, Passaglia R de CAT, Giachini FR, Lima VV. O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway [Internet]. Life Sciences. 2018 ; 209 78-84.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2018.07.058
Vancouver
Miguez JSG, Dela Justina V, Bressan AFM, Marchi PGF, França ACH, Carneiro FS, Webb RC, Passaglia R de CAT, Giachini FR, Lima VV. O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway [Internet]. Life Sciences. 2018 ; 209 78-84.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2018.07.058
Artigo de periodico
O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels (2018)
Dela Justina, Vanessa; Passos Junior, Rinaldo R. dos; Bressan, Alecsander F.; Passaglia, Rita de Cássia Aleixo Tostes; Carneiro, Fernando Silva; Soares, Thaigra S.; Volpato, Gustavo T.; Lima, Victor Vitorino; Martin, Sebastian San; Giachini, Fernanda R.
Source: Life Sciences. Unidade: FMRP
Subjects: GRAVIDEZ, HIPERGLICEMIA, TROFOBLASTO, CÉLULAS ENDOTELIAIS, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DELA JUSTINA, Vanessa et al. O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels. Life Sciences, v. 205, p. 18-25, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.05.013. Acesso em: 13 nov. 2024.
APA
Dela Justina, V., Passos Junior, R. R. dos, Bressan, A. F., Passaglia, R. de C. A. T., Carneiro, F. S., Soares, T. S., et al. (2018). O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels. Life Sciences, 205, 18-25. doi:10.1016/j.lfs.2018.05.013
NLM
Dela Justina V, Passos Junior RR dos, Bressan AF, Passaglia R de CAT, Carneiro FS, Soares TS, Volpato GT, Lima VV, Martin SS, Giachini FR. O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels [Internet]. Life Sciences. 2018 ; 205 18-25.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2018.05.013
Vancouver
Dela Justina V, Passos Junior RR dos, Bressan AF, Passaglia R de CAT, Carneiro FS, Soares TS, Volpato GT, Lima VV, Martin SS, Giachini FR. O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels [Internet]. Life Sciences. 2018 ; 205 18-25.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2018.05.013
Artigo de periodico
Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats (2017)
Justina, Vanessa Dela; Gonçalves, Jéssica S.; Freitas, Raiany Alves de; Fonseca, Adriany Dias; Volpato, Gustavo T.; Tostes, Rita de Cássia Aleixo; Carneiro, Fernando Silva; Giachini, Fernanda R.
Source: Inflammation. Unidade: FMRP
Subjects: CITOCINAS, HIPERGLICEMIA, PLACENTA, COMPLICAÇÕES NA GRAVIDEZ
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
JUSTINA, Vanessa Dela et al. Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats. Inflammation, v. 40, n. 5, p. 1773-1781, 2017Tradução . . Disponível em: https://doi.org/10.1007/s10753-017-0620-7. Acesso em: 13 nov. 2024.
APA
Justina, V. D., Gonçalves, J. S., Freitas, R. A. de, Fonseca, A. D., Volpato, G. T., Tostes, R. de C. A., et al. (2017). Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats. Inflammation, 40( 5), 1773-1781. doi:10.1007/s10753-017-0620-7
NLM
Justina VD, Gonçalves JS, Freitas RA de, Fonseca AD, Volpato GT, Tostes R de CA, Carneiro FS, Giachini FR. Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats [Internet]. Inflammation. 2017 ; 40( 5): 1773-1781.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1007/s10753-017-0620-7
Vancouver
Justina VD, Gonçalves JS, Freitas RA de, Fonseca AD, Volpato GT, Tostes R de CA, Carneiro FS, Giachini FR. Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats [Internet]. Inflammation. 2017 ; 40( 5): 1773-1781.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1007/s10753-017-0620-7
Artigo de periodico
Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species (2017)
Leite, Letícia N.; Vale, Gabriel T. do; Simplicio, Janaína A.; De Martinis, Bruno Spinosa; Carneiro, Fernando Silva; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: EERP, FFCLRP, FMRP
Subjects: ETANOL, ESTRESSE OXIDATIVO, DISFUNÇÃO ERÉTIL, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEITE, Letícia N. et al. Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species. European Journal of Pharmacology, v. 804, p. 82-93, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2017.03.024. Acesso em: 13 nov. 2024.
APA
Leite, L. N., Vale, G. T. do, Simplicio, J. A., De Martinis, B. S., Carneiro, F. S., & Tirapelli, C. R. (2017). Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species. European Journal of Pharmacology, 804, 82-93. doi:10.1016/j.ejphar.2017.03.024
NLM
Leite LN, Vale GT do, Simplicio JA, De Martinis BS, Carneiro FS, Tirapelli CR. Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species [Internet]. European Journal of Pharmacology. 2017 ; 804 82-93.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.ejphar.2017.03.024
Vancouver
Leite LN, Vale GT do, Simplicio JA, De Martinis BS, Carneiro FS, Tirapelli CR. Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species [Internet]. European Journal of Pharmacology. 2017 ; 804 82-93.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.ejphar.2017.03.024
Artigo de periodico
Chronic treatment with fluoxetine modulates vascular adrenergic responses by inhibition of pre- and post synaptic mechanisms (2017)
Pereira, Camila A.; Rodrigues, Fernanda L.; Ruginsk, Silvia G.; Zanotto, Camila Z.; Rodrigues, José Antunes; Duarte, Diego A.; Costa Neto, Cláudio Miguel; Resstel, Leonardo Barbosa Moraes; Carneiro, Fernando Silva; Passaglia, Rita de Cássia Aleixo Tostes
Source: European Journal of Pharmacology. Unidade: FMRP
Subjects: CÁLCIO, NOREPINEFRINA, RECEPTORES ADRENÉRGICOS, SISTEMA NERVOSO SIMPÁTICO, ESTIMULAÇÃO ELÉTRICA, FREQUÊNCIA CARDÍACA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PEREIRA, Camila A. et al. Chronic treatment with fluoxetine modulates vascular adrenergic responses by inhibition of pre- and post synaptic mechanisms. European Journal of Pharmacology, v. 800, p. 70-80, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2017.02.029. Acesso em: 13 nov. 2024.
APA
Pereira, C. A., Rodrigues, F. L., Ruginsk, S. G., Zanotto, C. Z., Rodrigues, J. A., Duarte, D. A., et al. (2017). Chronic treatment with fluoxetine modulates vascular adrenergic responses by inhibition of pre- and post synaptic mechanisms. European Journal of Pharmacology, 800, 70-80. doi:10.1016/j.ejphar.2017.02.029
NLM
Pereira CA, Rodrigues FL, Ruginsk SG, Zanotto CZ, Rodrigues JA, Duarte DA, Costa Neto CM, Resstel LBM, Carneiro FS, Passaglia R de CAT. Chronic treatment with fluoxetine modulates vascular adrenergic responses by inhibition of pre- and post synaptic mechanisms [Internet]. European Journal of Pharmacology. 2017 ; 800 70-80.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.ejphar.2017.02.029
Vancouver
Pereira CA, Rodrigues FL, Ruginsk SG, Zanotto CZ, Rodrigues JA, Duarte DA, Costa Neto CM, Resstel LBM, Carneiro FS, Passaglia R de CAT. Chronic treatment with fluoxetine modulates vascular adrenergic responses by inhibition of pre- and post synaptic mechanisms [Internet]. European Journal of Pharmacology. 2017 ; 800 70-80.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.ejphar.2017.02.029
Artigo de periodico
NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes (2017)
Carlos, Daniela; Costa, Frederico R. C.; Leite, Jefferson A.; André, Camila A.; Tostes, Rita de Cássia Aleixo; Carneiro, Fernando Silva; Câmara, Niels Olsen Saraiva ; Silva, João Santana da
Source: Journal of Autoimmune Disorders. Unidade: FMRP
Subjects: DIABETES MELLITUS, INSULINA, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARLOS, Daniela et al. NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes. Journal of Autoimmune Disorders, v. 3, n. 2, 2017Tradução . . Disponível em: https://doi.org/10.21767/2471-8513.100033. Acesso em: 13 nov. 2024.
APA
Carlos, D., Costa, F. R. C., Leite, J. A., André, C. A., Tostes, R. de C. A., Carneiro, F. S., et al. (2017). NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes. Journal of Autoimmune Disorders, 3( 2). doi:10.21767/2471-8513.100033
NLM
Carlos D, Costa FRC, Leite JA, André CA, Tostes R de CA, Carneiro FS, Câmara NOS, Silva JS da. NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes [Internet]. Journal of Autoimmune Disorders. 2017 ; 3( 2):[citado 2024 nov. 13 ] Available from: https://doi.org/10.21767/2471-8513.100033
Vancouver
Carlos D, Costa FRC, Leite JA, André CA, Tostes R de CA, Carneiro FS, Câmara NOS, Silva JS da. NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes [Internet]. Journal of Autoimmune Disorders. 2017 ; 3( 2):[citado 2024 nov. 13 ] Available from: https://doi.org/10.21767/2471-8513.100033

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