NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes (2017)
- Authors:
- USP affiliated authors: PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - FMRP ; CARNEIRO, FERNANDO SILVA - FMRP ; SILVA, JOÃO SANTANA DA - FMRP
- Unidade: FMRP
- DOI: 10.21767/2471-8513.100033
- Subjects: DIABETES MELLITUS; INSULINA; IMUNOLOGIA
- Keywords: Insulin; Diabetes mellitus; Immune response
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Autoimmune Disorders
- ISSN: 2471-8513
- Volume/Número/Paginação/Ano: v. 3, n. 2, art. 30, 2017
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
CARLOS, Daniela et al. NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes. Journal of Autoimmune Disorders, v. 3, n. 2, 2017Tradução . . Disponível em: https://doi.org/10.21767/2471-8513.100033. Acesso em: 17 out. 2024. -
APA
Carlos, D., Costa, F. R. C., Leite, J. A., André, C. A., Tostes, R. de C. A., Carneiro, F. S., et al. (2017). NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes. Journal of Autoimmune Disorders, 3( 2). doi:10.21767/2471-8513.100033 -
NLM
Carlos D, Costa FRC, Leite JA, André CA, Tostes R de CA, Carneiro FS, Câmara NOS, Silva JS da. NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes [Internet]. Journal of Autoimmune Disorders. 2017 ; 3( 2):[citado 2024 out. 17 ] Available from: https://doi.org/10.21767/2471-8513.100033 -
Vancouver
Carlos D, Costa FRC, Leite JA, André CA, Tostes R de CA, Carneiro FS, Câmara NOS, Silva JS da. NLRP3 inflammasome: From pathogenesis to therapeutic strategies in Type 1 Diabetes [Internet]. Journal of Autoimmune Disorders. 2017 ; 3( 2):[citado 2024 out. 17 ] Available from: https://doi.org/10.21767/2471-8513.100033 - NACHT, LRR and PYD domains-containing protein 3(NLRP3) crucial role of the cavernous tissue tone modulation in mice
- O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels
- Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms
- Angiotensin (1-7) inhibits Ang II-mediated ERK1/2 activation by stimulating MKP-1 activation in vascular smooth muscle cells
- O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway
- Mesenteric arteries from stroke-prone spontaneously hypertensive rats exhibit an increase in nitric-oxide-dependent vasorelaxation
- Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery
- Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats
- Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis
- G protein-coupled estrogen receptor contributes to the vascular effects of aldosterone and type two diabetes-associated vascular dysfunction
Informações sobre o DOI: 10.21767/2471-8513.100033 (Fonte: oaDOI API)
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