Supraphysiological levels of testosterone induce vascular dysfunction via activation of the NLRP3 inflammasome (2020)
- Authors:
- USP affiliated authors: SILVA, CARLOS ALBERTO AGUIAR DA - FMRP ; CARNEIRO, FERNANDO SILVA - FMRP ; PASSAGLIA, RITA DE CASSIA ALEIXO TOSTES - FMRP ; ALVES, JULIANO VILELA - FMRP ; FEDOCE, ALINE GARCIA - FMRP
- Unidade: FMRP
- DOI: 10.3389/fimmu.2020.01647
- Subjects: ANDRÓGENOS; AORTA TORÁCICA; INTERLEUCINA 1; MITOCÔNDRIAS; ESPÉCIES REATIVAS DE OXIGÊNIO; RECEPTORES ANDROGÊNICOS
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Frontiers in Immunology
- ISSN: 1664-3224
- Volume/Número/Paginação/Ano: v. 11, art. 1647, 2020
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
ALVES, Juliano Vilela et al. Supraphysiological levels of testosterone induce vascular dysfunction via activation of the NLRP3 inflammasome. Frontiers in Immunology, v. 11, 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.01647. Acesso em: 21 fev. 2026. -
APA
Alves, J. V., Costa, R. M. da, Pereira, C. A., Fedoce, A. G., Silva, C. A. A. da, Carneiro, F. S., et al. (2020). Supraphysiological levels of testosterone induce vascular dysfunction via activation of the NLRP3 inflammasome. Frontiers in Immunology, 11. doi:10.3389/fimmu.2020.01647 -
NLM
Alves JV, Costa RM da, Pereira CA, Fedoce AG, Silva CAA da, Carneiro FS, Lobato N de S, Tostes R de CA. Supraphysiological levels of testosterone induce vascular dysfunction via activation of the NLRP3 inflammasome [Internet]. Frontiers in Immunology. 2020 ; 11[citado 2026 fev. 21 ] Available from: https://doi.org/10.3389/fimmu.2020.01647 -
Vancouver
Alves JV, Costa RM da, Pereira CA, Fedoce AG, Silva CAA da, Carneiro FS, Lobato N de S, Tostes R de CA. Supraphysiological levels of testosterone induce vascular dysfunction via activation of the NLRP3 inflammasome [Internet]. Frontiers in Immunology. 2020 ; 11[citado 2026 fev. 21 ] Available from: https://doi.org/10.3389/fimmu.2020.01647 - Toll-like receptor 9 (TLR9) plays a key role in the autonomic cardiac and baroreflex control of arterial pressure
- Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats
- O-Glycosylation with O-linked β-N-acetylglucosamine increases vascular contraction: possible modulatory role on Interleukin-10 signaling pathway
- Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms
- O -linked N -acetyl-glucosamine deposition in placental proteins varies according to maternal glycemic levels
- Erectile dysfunction in heart failure rats is associated with increased neurogenic contractions in cavernous tissue and internal pudendal artery
- NACHT, LRR and PYD domains-containing protein 3(NLRP3) crucial role of the cavernous tissue tone modulation in mice
- Mesenteric arteries from stroke-prone spontaneously hypertensive rats exhibit an increase in nitric-oxide-dependent vasorelaxation
- Angiotensin (1-7) inhibits Ang II-mediated ERK1/2 activation by stimulating MKP-1 activation in vascular smooth muscle cells
- Bonus effects of antidiabetic drugs: possible beneficial effects on endothelial dysfunction, vascular inflammation and atherosclerosis
Informações sobre o DOI: 10.3389/fimmu.2020.01647 (Fonte: oaDOI API)
Download do texto completo
| Tipo | Nome | Link | |
|---|---|---|---|
| 003036751.pdf | Direct link |
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
