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Filtros : "Martinelli Júnior, Carlos Eduardo" "Alemanha" Limpar

Filtros

USP affiliated authors
- antonini, sonir roberto rauber (2)
- castro, margaret de (2)
- martinelli junior, carlos eduardo (2)
- moreira, ayrton custodio (2)
- scrideli, carlos alberto (2)

Trabalho de evento-resumo
Abnormal activation of the WNT/B-catenin pathway in childhood adrenocortical tumors is independent of CTNNB1 mutations (2011)
Leal, L. F.; Mermejo, L. M.; Ramalho, Leandra Naíra Zambelli; Martinelli Júnior, Carlos Eduardo; Yunes, J. A.; Seidinger, A. L.; Mastellaro, M. J.; Brandalise, S. R.; Moreira, Ayrton Custódio; Tone, Luiz Gonzaga; Scrideli, Carlos Alberto; Castro, Margaret de; Antonini, Sonir Roberto Rauber
Source: Abstracts. Conference titles: International Adrenal Cancer Symposium. Unidade: FMRP
Subjects: CRIANÇAS, MUTAÇÃO GENÉTICA, NEOPLASIAS DO CÓRTEX SUPRARRENAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEAL, L. F. et al. Abnormal activation of the WNT/B-catenin pathway in childhood adrenocortical tumors is independent of CTNNB1 mutations. 2011, Anais.. Würzburg: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2011. . Acesso em: 18 nov. 2024.
APA
Leal, L. F., Mermejo, L. M., Ramalho, L. N. Z., Martinelli Júnior, C. E., Yunes, J. A., Seidinger, A. L., et al. (2011). Abnormal activation of the WNT/B-catenin pathway in childhood adrenocortical tumors is independent of CTNNB1 mutations. In Abstracts. Würzburg: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Leal LF, Mermejo LM, Ramalho LNZ, Martinelli Júnior CE, Yunes JA, Seidinger AL, Mastellaro MJ, Brandalise SR, Moreira AC, Tone LG, Scrideli CA, Castro M de, Antonini SRR. Abnormal activation of the WNT/B-catenin pathway in childhood adrenocortical tumors is independent of CTNNB1 mutations. Abstracts. 2011 ;[citado 2024 nov. 18 ]
Vancouver
Leal LF, Mermejo LM, Ramalho LNZ, Martinelli Júnior CE, Yunes JA, Seidinger AL, Mastellaro MJ, Brandalise SR, Moreira AC, Tone LG, Scrideli CA, Castro M de, Antonini SRR. Abnormal activation of the WNT/B-catenin pathway in childhood adrenocortical tumors is independent of CTNNB1 mutations. Abstracts. 2011 ;[citado 2024 nov. 18 ]
Trabalho de evento-resumo
High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53 (2011)
Colli, L. M.; Leal, L. F.; Mermejo, L. M.; Scrideli, Carlos Alberto; Tone, Luiz Gonzaga; Martinelli Júnior, Carlos Eduardo; Moreira, Ayrton Custódio; Antonini, Sonir Roberto Rauber; Castro, Margaret de
Source: Abstracts. Conference titles: International Adrenal Cancer Symposium. Unidade: FMRP
Subjects: CRIANÇAS, NEOPLASIAS DO CÓRTEX SUPRARRENAL, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COLLI, L. M. et al. High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53. 2011, Anais.. Würzburg: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2011. . Acesso em: 18 nov. 2024.
APA
Colli, L. M., Leal, L. F., Mermejo, L. M., Scrideli, C. A., Tone, L. G., Martinelli Júnior, C. E., et al. (2011). High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53. In Abstracts. Würzburg: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Colli LM, Leal LF, Mermejo LM, Scrideli CA, Tone LG, Martinelli Júnior CE, Moreira AC, Antonini SRR, Castro M de. High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53. Abstracts. 2011 ;[citado 2024 nov. 18 ]
Vancouver
Colli LM, Leal LF, Mermejo LM, Scrideli CA, Tone LG, Martinelli Júnior CE, Moreira AC, Antonini SRR, Castro M de. High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53. Abstracts. 2011 ;[citado 2024 nov. 18 ]

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