Abnormal activation of the WNT/B-catenin pathway in childhood adrenocortical tumors is independent of CTNNB1 mutations (2011)
- Authors:
- USP affiliated authors: ANTONINI, SONIR ROBERTO RAUBER - FMRP ; RAMALHO, LEANDRA NAIRA ZAMBELLI - FMRP ; MARTINELLI JUNIOR, CARLOS EDUARDO - FMRP ; MOREIRA, AYRTON CUSTODIO - FMRP ; TONE, LUIZ GONZAGA - FMRP ; CASTRO, MARGARET DE - FMRP ; SCRIDELI, CARLOS ALBERTO - FMRP
- Unidade: FMRP
- Subjects: CRIANÇAS; MUTAÇÃO GENÉTICA; NEOPLASIAS DO CÓRTEX SUPRARRENAL
- Language: Inglês
- Imprenta:
- Source:
- Título: Abstracts
- Conference titles: International Adrenal Cancer Symposium
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ABNT
LEAL, L. F. et al. Abnormal activation of the WNT/B-catenin pathway in childhood adrenocortical tumors is independent of CTNNB1 mutations. 2011, Anais.. Würzburg: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2011. . Acesso em: 03 jan. 2026. -
APA
Leal, L. F., Mermejo, L. M., Ramalho, L. N. Z., Martinelli Júnior, C. E., Yunes, J. A., Seidinger, A. L., et al. (2011). Abnormal activation of the WNT/B-catenin pathway in childhood adrenocortical tumors is independent of CTNNB1 mutations. In Abstracts. Würzburg: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Leal LF, Mermejo LM, Ramalho LNZ, Martinelli Júnior CE, Yunes JA, Seidinger AL, Mastellaro MJ, Brandalise SR, Moreira AC, Tone LG, Scrideli CA, Castro M de, Antonini SRR. Abnormal activation of the WNT/B-catenin pathway in childhood adrenocortical tumors is independent of CTNNB1 mutations. Abstracts. 2011 ;[citado 2026 jan. 03 ] -
Vancouver
Leal LF, Mermejo LM, Ramalho LNZ, Martinelli Júnior CE, Yunes JA, Seidinger AL, Mastellaro MJ, Brandalise SR, Moreira AC, Tone LG, Scrideli CA, Castro M de, Antonini SRR. Abnormal activation of the WNT/B-catenin pathway in childhood adrenocortical tumors is independent of CTNNB1 mutations. Abstracts. 2011 ;[citado 2026 jan. 03 ] - Wnt/β-catenin pathway deregulation in childhood adrenocortical tumors
- P53 and CTNNB1 mutations reveal different mechanisms/pattern between pediatric and adults adrenocortical tumors
- High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53
- Abnormal activation of the Wint/β-catenin pathway in childhood adrenocortical tumors (ACTs)
- Wnt/β-catenin pathway deregulation in childhood adrenocortical tumors
- Analysis of DAX1 and SF1 genes and their interaction with genes involved in stem cell maintenance in adrenocortical tumors
- AMG 900, an aurora kinases inhibitor, enhances the chemosensitivity to topoisomerase II inhibitors and modulates gene expression in H295A adrenocortical tumor cell line
- Noncanonical and canonical Wnt pathways abnormalities in pediatric and adult adrenocortical tumors
- Higher expression of the oncogene YAP1, a Wnt/β-catenin target, is associated with poor outcome in pediatric patients with adrenocortical tumors
- Anormalidades da via Sonic Hedgehog (SHH) em tumores adrenocorticais (TAC)
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