Analysis of DAX1 and SF1 genes and their interaction with genes involved in stem cell maintenance in adrenocortical tumors (2015)
- Authors:
- USP affiliated authors: ANTONINI, SONIR ROBERTO RAUBER - FMRP ; CASTRO, MARGARET DE - FMRP ; MOREIRA, AYRTON CUSTODIO - FMRP ; MARTINELLI JUNIOR, CARLOS EDUARDO - FMRP ; SCRIDELI, CARLOS ALBERTO - FMRP ; RAMALHO, LEANDRA NAIRA ZAMBELLI - FMRP
- Unidade: FMRP
- Subjects: CÉLULAS-TRONCO; EXPRESSÃO GÊNICA; ENDOCRINOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Hormone Research in Paediatrics
- ISSN: 1663-2818
- Volume/Número/Paginação/Ano: v. 84, suppl. 2, p. 15, res. 020, 2015
- Conference titles: Congress of the Latin American Society of Pediatric Endocrinology
-
ABNT
CAVALCANTI, M. et al. Analysis of DAX1 and SF1 genes and their interaction with genes involved in stem cell maintenance in adrenocortical tumors. Hormone Research in Paediatrics. Basel: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 27 fev. 2026. , 2015 -
APA
Cavalcanti, M., Leal, L., Lacchini, F., Martinelli Junior, C. E., Scrideli, C. A., Tucci Júnior, S., et al. (2015). Analysis of DAX1 and SF1 genes and their interaction with genes involved in stem cell maintenance in adrenocortical tumors. Hormone Research in Paediatrics. Basel: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Cavalcanti M, Leal L, Lacchini F, Martinelli Junior CE, Scrideli CA, Tucci Júnior S, Molina C, Yunes J, Mastellaro. M., Brandalise S, Cardinalli I, Moreira AC, Ramalho LNZ, Castro M de, Antonini SRR. Analysis of DAX1 and SF1 genes and their interaction with genes involved in stem cell maintenance in adrenocortical tumors. Hormone Research in Paediatrics. 2015 ; 84 15.[citado 2026 fev. 27 ] -
Vancouver
Cavalcanti M, Leal L, Lacchini F, Martinelli Junior CE, Scrideli CA, Tucci Júnior S, Molina C, Yunes J, Mastellaro. M., Brandalise S, Cardinalli I, Moreira AC, Ramalho LNZ, Castro M de, Antonini SRR. Analysis of DAX1 and SF1 genes and their interaction with genes involved in stem cell maintenance in adrenocortical tumors. Hormone Research in Paediatrics. 2015 ; 84 15.[citado 2026 fev. 27 ] - Abnormal activation of the WNT/B-catenin pathway in childhood adrenocortical tumors is independent of CTNNB1 mutations
- Wnt/β-catenin pathway deregulation in childhood adrenocortical tumors
- Wnt/β-catenin pathway deregulation in childhood adrenocortical tumors
- Abnormal activation of the Wint/β-catenin pathway in childhood adrenocortical tumors (ACTs)
- P53 and CTNNB1 mutations reveal different mechanisms/pattern between pediatric and adults adrenocortical tumors
- Salivary cortisol as a diagnostic tool of hypercortisolism in primary pigmented nodular adrenocortical disease (ppnad)
- Non-radioactive strategies on the diagnosis of congenital adrenal hyperplasia due to 21 hydroxylase deficiency (CAH-210HD)
- OTX2 gene mutations are rare in patients with combined pituitary hormone deficiency (CPHD) and /or midline cerebral defects (MCD)
- High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53
- Higher expression of the oncogene YAP1, a Wnt/β-catenin target, is associated with poor outcome in pediatric patients with adrenocortical tumors
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