High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53 (2011)
- Authors:
- USP affiliated authors: ANTONINI, SONIR ROBERTO RAUBER - FMRP ; MARTINELLI JUNIOR, CARLOS EDUARDO - FMRP ; MOREIRA, AYRTON CUSTODIO - FMRP ; TONE, LUIZ GONZAGA - FMRP ; CASTRO, MARGARET DE - FMRP ; SCRIDELI, CARLOS ALBERTO - FMRP
- Unidade: FMRP
- Subjects: CRIANÇAS; NEOPLASIAS DO CÓRTEX SUPRARRENAL; EXPRESSÃO GÊNICA
- Language: Inglês
- Imprenta:
- Source:
- Título: Abstracts
- Conference titles: International Adrenal Cancer Symposium
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ABNT
COLLI, L. M. et al. High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53. 2011, Anais.. Würzburg: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2011. . Acesso em: 03 jan. 2026. -
APA
Colli, L. M., Leal, L. F., Mermejo, L. M., Scrideli, C. A., Tone, L. G., Martinelli Júnior, C. E., et al. (2011). High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53. In Abstracts. Würzburg: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Colli LM, Leal LF, Mermejo LM, Scrideli CA, Tone LG, Martinelli Júnior CE, Moreira AC, Antonini SRR, Castro M de. High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53. Abstracts. 2011 ;[citado 2026 jan. 03 ] -
Vancouver
Colli LM, Leal LF, Mermejo LM, Scrideli CA, Tone LG, Martinelli Júnior CE, Moreira AC, Antonini SRR, Castro M de. High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53. Abstracts. 2011 ;[citado 2026 jan. 03 ] - AMG 900, an aurora kinases inhibitor, enhances the chemosensitivity to topoisomerase II inhibitors and modulates gene expression in H295A adrenocortical tumor cell line
- Abnormal activation of the WNT/B-catenin pathway in childhood adrenocortical tumors is independent of CTNNB1 mutations
- Wnt/β-catenin pathway deregulation in childhood adrenocortical tumors
- OTX2 gene mutations are rare in patients with combined pituitary hormone deficiency (CPHD) and /or midline cerebral defects (MCD)
- P53 and CTNNB1 mutations reveal different mechanisms/pattern between pediatric and adults adrenocortical tumors
- Non-radioactive strategies on the diagnosis of congenital adrenal hyperplasia due to 21 hydroxylase deficiency (CAH-210HD)
- Salivary cortisol as a diagnostic tool of hypercortisolism in primary pigmented nodular adrenocortical disease (ppnad)
- Abnormal activation of the Wint/β-catenin pathway in childhood adrenocortical tumors (ACTs)
- Wnt/β-catenin pathway deregulation in childhood adrenocortical tumors
- Differential microRNAs expressions are associated with relapse and TP53 P.R377H mutation in pediatrics adrenocortical tumors
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