WNT/beta-catenin pathway dysregulation in childhood adrenocortical tumors harboring the R337H p53 mutation (2010)
- Authors:
- USP affiliated authors: CASTRO, MARGARET DE - FMRP ; MARTINELLI JUNIOR, CARLOS EDUARDO - FMRP ; TONE, LUIZ GONZAGA - FMRP ; SCRIDELI, CARLOS ALBERTO - FMRP
- Unidade: FMRP
- Subjects: ENDOCRINOPATIAS; NEOPLASIAS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Advance Program
- Volume/Número/Paginação/Ano: 2010
- Conference titles: Annual Meeting and Expo of the Endocrine Society (ENDO)
-
ABNT
LEAL, L. F.; MERMEJO, L. M.; MARTINELLI JUNIOR, Carlos Eduardo; et al. WNT/beta-catenin pathway dysregulation in childhood adrenocortical tumors harboring the R337H p53 mutation. Anais.. San Diego: [s.n.], 2010. -
APA
Leal, L. F., Mermejo, L. M., Martinelli Junior, C. E., Moreira, A. C., Tone, L. G., Scrideli, C. A., et al. (2010). WNT/beta-catenin pathway dysregulation in childhood adrenocortical tumors harboring the R337H p53 mutation. In Advance Program. San Diego. -
NLM
Leal LF, Mermejo LM, Martinelli Junior CE, Moreira AC, Tone LG, Scrideli CA, Castro M de, Antonini SR. WNT/beta-catenin pathway dysregulation in childhood adrenocortical tumors harboring the R337H p53 mutation. Advance Program. 2010 ; -
Vancouver
Leal LF, Mermejo LM, Martinelli Junior CE, Moreira AC, Tone LG, Scrideli CA, Castro M de, Antonini SR. WNT/beta-catenin pathway dysregulation in childhood adrenocortical tumors harboring the R337H p53 mutation. Advance Program. 2010 ; - AMG 900, an aurora kinases inhibitor, enhances the chemosensitivity to topoisomerase II inhibitors and modulates gene expression in H295A adrenocortical tumor cell line
- R337H P53 mutation and the expression of microRNAs in adrenocortical tumor
- A novel nonsense mutation in the mineralocorticoid receptor gene in a family with a renal form od type 1 pseudohypoaldosteronism (PHA-1)
- Differential microRNAs expressions are associated with relapse and TP53 P.R377H mutation in pediatrics adrenocortical tumors
- High expression of miR-449 in childhood adrenocortical tumors seems to be due to a post-transcriptional event embedded in a tumor suppressive program governed by p53
- Molecular characterization of Wnt pathway and function of β-catenin overexpression in medulloblastoma cell lines
- Tetraploidization in Wilms tumor in an infant
- Alternative targets for medulloblastoma treatment: polo-like kinase 1
- Avaliação da expressão dos MIR-181B-5P, MIR-100-5P e MIR-125A-5P em amostras tumorais de osteossarcoma
- Effects of zebularine alone or in combination with the histone deacetylase inhibitor SAHA on medulloblastoma cells
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