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  • Source: Scientific Reports. Unidade: ICB

    Subjects: IMUNOLOGIA, CAMUNDONGOS, RATOS, PARACOCCIDIOIDES, CITOCINAS, LIGANTES, PARACOCCIDIOIDOMICOSE, ANTIFÚNGICOS

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    • ABNT

      ARAÚJO, Eliseu Frank de et al. Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses. Scientific Reports, v. 10, p. 16 , 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-68322-6. Acesso em: 24 ago. 2024.
    • APA

      Araújo, E. F. de, Preite, N. W., Veldhoen, M., Loures, F. V., & Calich, V. L. G. (2020). Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses. Scientific Reports, 10, 16 . doi:10.1038/s41598-020-68322-6
    • NLM

      Araújo EF de, Preite NW, Veldhoen M, Loures FV, Calich VLG. Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses [Internet]. Scientific Reports. 2020 ; 10 16 .[citado 2024 ago. 24 ] Available from: https://doi.org/10.1038/s41598-020-68322-6
    • Vancouver

      Araújo EF de, Preite NW, Veldhoen M, Loures FV, Calich VLG. Pulmonary paracoccidioidomycosis in AhR deficient hosts is severe and associated with defective Treg and Th22 responses [Internet]. Scientific Reports. 2020 ; 10 16 .[citado 2024 ago. 24 ] Available from: https://doi.org/10.1038/s41598-020-68322-6
  • Source: Frontiers in Immunology. Unidade: ICB

    Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE, CÉLULAS DENDRÍTICAS, ATIVAÇÃO ENZIMÁTICA, ENZIMAS OXIRREDUTORAS

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      ARAÚJO, Eliseu Frank de et al. Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen. Frontiers in Immunology, v. 8, n. 1522, p. 1-14, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.01522. Acesso em: 24 ago. 2024.
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      Araújo, E. F. de, Loures, F. V., Feriotti, C., Costa, T., Vacca, C., Puccetti, P., et al. (2017). Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen. Frontiers in Immunology, 8( 1522), 1-14. doi:10.3389/fimmu.2017.01522
    • NLM

      Araújo EF de, Loures FV, Feriotti C, Costa T, Vacca C, Puccetti P, Romani L, Calich VLG. Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen [Internet]. Frontiers in Immunology. 2017 ; 8( 1522): 1-14.[citado 2024 ago. 24 ] Available from: https://doi.org/10.3389/fimmu.2017.01522
    • Vancouver

      Araújo EF de, Loures FV, Feriotti C, Costa T, Vacca C, Puccetti P, Romani L, Calich VLG. Disease tolerance mediated by phosphorylated indoleamine-2,3 dioxygenase confers resistance to a primary fungal pathogen [Internet]. Frontiers in Immunology. 2017 ; 8( 1522): 1-14.[citado 2024 ago. 24 ] Available from: https://doi.org/10.3389/fimmu.2017.01522
  • Source: PLoS Pathogens. Unidade: ICB

    Assunto: IMUNOLOGIA

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      ARAÚJO, Eliseu Frank de et al. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner. PLoS Pathogens, v. 12, n. 12, p. 1-29, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1006115. Acesso em: 24 ago. 2024.
    • APA

      Araújo, E. F. de, Medeiros, D. H., Galdino, N. A. de L., Condino-Neto, A., Calich, V. L. G., & Loures, F. V. (2016). Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner. PLoS Pathogens, 12( 12), 1-29. doi:10.1371/journal.ppat.1006115
    • NLM

      Araújo EF de, Medeiros DH, Galdino NA de L, Condino-Neto A, Calich VLG, Loures FV. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner [Internet]. PLoS Pathogens. 2016 ; 12( 12): 1-29.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1371/journal.ppat.1006115
    • Vancouver

      Araújo EF de, Medeiros DH, Galdino NA de L, Condino-Neto A, Calich VLG, Loures FV. Tolerogenic plasmacytoid dendritic cells control Paracoccidioides brasiliensis infection by inducting regulatory T cells in an IDO-dependent manner [Internet]. PLoS Pathogens. 2016 ; 12( 12): 1-29.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1371/journal.ppat.1006115
  • Source: Frontiers in Microbiology. Unidade: ICB

    Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE (RESISTÊNCIA), INFLAMAÇÃO

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      FERIOTTI, Claudia et al. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis. Frontiers in Microbiology, v. 6, n. 913, p. 1-13, 2015Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2015.00913. Acesso em: 24 ago. 2024.
    • APA

      Feriotti, C., Bazan, S. B., Loures, F. V., Araújo, E. F. de, Costa, T. A. da, & Calich, V. L. G. (2015). Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis. Frontiers in Microbiology, 6( 913), 1-13. doi:10.3389/fmicb.2015.00913
    • NLM

      Feriotti C, Bazan SB, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis [Internet]. Frontiers in Microbiology. 2015 ; 6( 913): 1-13.[citado 2024 ago. 24 ] Available from: https://doi.org/10.3389/fmicb.2015.00913
    • Vancouver

      Feriotti C, Bazan SB, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Expression of dectin-1 and enhanced activation of NALP3 inflammasome are associated with resistance to paracoccidioidomycosis [Internet]. Frontiers in Microbiology. 2015 ; 6( 913): 1-13.[citado 2024 ago. 24 ] Available from: https://doi.org/10.3389/fmicb.2015.00913
  • Source: Frontiers in Microbiology. Unidade: ICB

    Subjects: IMUNOLOGIA, PARACOCCIDIOIDES, PARACOCCIDIOIDES BRASILIENSIS, CÉLULAS DENDRITICAS

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      LOURES, Flávio Vieira et al. TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells. Frontiers in Microbiology, v. 6, n. 261, p. 1-11, 2015Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2015.00261. Acesso em: 24 ago. 2024.
    • APA

      Loures, F. V., Araújo, E. F. de, Feriotti, C., Bazan, S. B., & Calich, V. L. G. (2015). TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells. Frontiers in Microbiology, 6( 261), 1-11. doi:10.3389/fmicb.2015.00261
    • NLM

      Loures FV, Araújo EF de, Feriotti C, Bazan SB, Calich VLG. TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells [Internet]. Frontiers in Microbiology. 2015 ; 6( 261): 1-11.[citado 2024 ago. 24 ] Available from: https://doi.org/10.3389/fmicb.2015.00261
    • Vancouver

      Loures FV, Araújo EF de, Feriotti C, Bazan SB, Calich VLG. TLR-4 cooperates with Dectin-1 and mannose receptor to expand Th17 and Tc17 cells induced by Paracoccidioides brasiliensis stimulated dendritic cells [Internet]. Frontiers in Microbiology. 2015 ; 6( 261): 1-11.[citado 2024 ago. 24 ] Available from: https://doi.org/10.3389/fmicb.2015.00261
  • Source: PLoS Neglected Tropical Diseases. Unidade: ICB

    Subjects: IMUNOLOGIA, PARACOCCIDIOIDOMICOSE, IMUNIDADE CELULAR, CAMUNDONGOS

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      BAZAN, Silvia Boschi et al. Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis. PLoS Neglected Tropical Diseases, v. 9, n. 10, p. 1-24, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0004189. Acesso em: 24 ago. 2024.
    • APA

      Bazan, S. B., Costa, T. A. da, Araújo, E. F. de, Feriotti, C., Loures, F. V., Pretel, F. D., & Calich, V. L. G. (2015). Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis. PLoS Neglected Tropical Diseases, 9( 10), 1-24. doi:10.1371/journal.pntd.0004189
    • NLM

      Bazan SB, Costa TA da, Araújo EF de, Feriotti C, Loures FV, Pretel FD, Calich VLG. Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis [Internet]. PLoS Neglected Tropical Diseases. 2015 ; 9( 10): 1-24.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1371/journal.pntd.0004189
    • Vancouver

      Bazan SB, Costa TA da, Araújo EF de, Feriotti C, Loures FV, Pretel FD, Calich VLG. Loss- and gain-of-function approaches indicate a dual role exerted by regulatory T cells in pulmonary paracoccidioidomycosis [Internet]. PLoS Neglected Tropical Diseases. 2015 ; 9( 10): 1-24.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1371/journal.pntd.0004189
  • Source: Mediators of Inflammation. Unidade: ICB

    Subjects: IMUNOLOGIA, PARACOCCIDIOIDES BRASILIENSIS, MACRÓFAGOS, INFECÇÕES RESPIRATÓRIAS

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      RIBEIRO, Laura Rossetti Rios et al. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis. Mediators of Inflammation, v. 2015, p. 1-17, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/852574. Acesso em: 24 ago. 2024.
    • APA

      Ribeiro, L. R. R., Loures, F. V., Araújo, E. F. de, Feriotti, C., Costa, T. A. da, Serezani, C. H., et al. (2015). Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis. Mediators of Inflammation, 2015, 1-17. doi:10.1155/2015/852574
    • NLM

      Ribeiro LRR, Loures FV, Araújo EF de, Feriotti C, Costa TA da, Serezani CH, Jancar S, Calich VLG. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis [Internet]. Mediators of Inflammation. 2015 ; 2015 1-17.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1155/2015/852574
    • Vancouver

      Ribeiro LRR, Loures FV, Araújo EF de, Feriotti C, Costa TA da, Serezani CH, Jancar S, Calich VLG. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis [Internet]. Mediators of Inflammation. 2015 ; 2015 1-17.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1155/2015/852574
  • Source: Journal of Infectious Diseases. Unidade: ICB

    Subjects: IMUNOLOGIA, MACRÓFAGOS, PARACOCCIDIOIDOMICOSE

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      LOURES, Flávio V. et al. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis. Journal of Infectious Diseases, v. 210, n. 5, p. 762-773, 2014Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiu136. Acesso em: 24 ago. 2024.
    • APA

      Loures, F. V., Araújo, E. F. de, Feriotti, C., Bazan, S. B., Costa, T. A. da, Brown, G. D., & Calich, V. L. G. (2014). Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis. Journal of Infectious Diseases, 210( 5), 762-773. doi:10.1093/infdis/jiu136
    • NLM

      Loures FV, Araújo EF de, Feriotti C, Bazan SB, Costa TA da, Brown GD, Calich VLG. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis [Internet]. Journal of Infectious Diseases. 2014 ; 210( 5): 762-773.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1093/infdis/jiu136
    • Vancouver

      Loures FV, Araújo EF de, Feriotti C, Bazan SB, Costa TA da, Brown GD, Calich VLG. Dectin-1 induces M1 macrophages and prominent expansion of CD8+IL-17+ cells in pulmonary Paracoccidioidomycosis [Internet]. Journal of Infectious Diseases. 2014 ; 210( 5): 762-773.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1093/infdis/jiu136
  • Source: PLOS ONE. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FERIOTTI, Claudia et al. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection. PLOS ONE, v. 8, n. 1, p. 1-17, 2013Tradução . . Disponível em: https://doi.org/10.1590/S0004-27302013000100003. Acesso em: 24 ago. 2024.
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      Feriotti, C., Loures, F. V., Araújo, E. F. de, Costa, T. A. da, & Calich, V. L. G. (2013). Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection. PLOS ONE, 8( 1), 1-17. doi:10.1590/S0004-27302013000100003
    • NLM

      Feriotti C, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection [Internet]. PLOS ONE. 2013 ; 8( 1): 1-17.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1590/S0004-27302013000100003
    • Vancouver

      Feriotti C, Loures FV, Araújo EF de, Costa TA da, Calich VLG. Mannosyl-recognizing receptors induce an M1-like phenotype in macrophages of susceptible mice but an M2-like phenotype in mice resistant to a fungal infection [Internet]. PLOS ONE. 2013 ; 8( 1): 1-17.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1590/S0004-27302013000100003
  • Source: Infection and Immunity. Unidade: ICB

    Assunto: IMUNOLOGIA

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      LOURES, Flávio Vieira et al. MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection. Infection and Immunity, v. 79, n. 6, p. 2470-2480, 2011Tradução . . Disponível em: https://doi.org/10.1128/IAI.00375-10. Acesso em: 24 ago. 2024.
    • APA

      Loures, F. V., Pina, A., Felonato, M., Feriotti, C., Araújo, E. F. de, & Calich, V. L. G. (2011). MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection. Infection and Immunity, 79( 6), 2470-2480. doi:10.1128/IAI.00375-10
    • NLM

      Loures FV, Pina A, Felonato M, Feriotti C, Araújo EF de, Calich VLG. MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection [Internet]. Infection and Immunity. 2011 ; 79( 6): 2470-2480.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1128/IAI.00375-10
    • Vancouver

      Loures FV, Pina A, Felonato M, Feriotti C, Araújo EF de, Calich VLG. MyD88 signaling is required for efficient innate and adaptive immune responses to Paracoccidioides brasiliensis infection [Internet]. Infection and Immunity. 2011 ; 79( 6): 2470-2480.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1128/IAI.00375-10
  • Source: Infection and Immunity. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FELONATO, Maíra et al. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis. Infection and Immunity, v. 78, n. 11, p. 4922-4935, 2010Tradução . . Disponível em: https://doi.org/10.1128/IAI.00297-10. Acesso em: 24 ago. 2024.
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      Felonato, M., Pina, A., Bernardino, S., Loures, F. V., Araújo, E. F. de, & Calich, V. L. G. (2010). CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis. Infection and Immunity, 78( 11), 4922-4935. doi:10.1128/IAI.00297-10
    • NLM

      Felonato M, Pina A, Bernardino S, Loures FV, Araújo EF de, Calich VLG. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis [Internet]. Infection and Immunity. 2010 ; 78( 11): 4922-4935.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1128/IAI.00297-10
    • Vancouver

      Felonato M, Pina A, Bernardino S, Loures FV, Araújo EF de, Calich VLG. CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis [Internet]. Infection and Immunity. 2010 ; 78( 11): 4922-4935.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1128/IAI.00297-10
  • Source: Infection and Immunity. Unidade: ICB

    Assunto: IMUNOLOGIA

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      LOURES, Flávio Vieira et al. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity, v. 78, n. 3, p. 1078-1088, 2010Tradução . . Acesso em: 24 ago. 2024.
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      Loures, F. V., Pina, A., Felonato, M., Araújo, E. F. de, Leite, K. R. M., & Calich, V. L. G. (2010). Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity, 78( 3), 1078-1088.
    • NLM

      Loures FV, Pina A, Felonato M, Araújo EF de, Leite KRM, Calich VLG. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity. 2010 ; 78( 3): 1078-1088.[citado 2024 ago. 24 ]
    • Vancouver

      Loures FV, Pina A, Felonato M, Araújo EF de, Leite KRM, Calich VLG. Toll-Like receptor 4 signaling leads to severe fungal infection associated with enhanced proinflammatory immunity and impaired expansion of regulatory T cells. Infection and Immunity. 2010 ; 78( 3): 1078-1088.[citado 2024 ago. 24 ]
  • Unidade: ICB

    Subjects: PARACOCCIDIOIDOMICOSE, LINFÓCITOS, MACRÓFAGOS, CITOCINAS

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      ARAÚJO, Eliseu Frank de. A IDO controla a carga fúngica e a imunidade celular de camundongos suscetíveis e resistentes à infecção pelo Paracoccidioides brasiliensis. 2009. Dissertação (Mestrado) – Universidade de São Paulo, São Paulo, 2009. Disponível em: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-02022010-124420/. Acesso em: 24 ago. 2024.
    • APA

      Araújo, E. F. de. (2009). A IDO controla a carga fúngica e a imunidade celular de camundongos suscetíveis e resistentes à infecção pelo Paracoccidioides brasiliensis (Dissertação (Mestrado). Universidade de São Paulo, São Paulo. Recuperado de http://www.teses.usp.br/teses/disponiveis/42/42133/tde-02022010-124420/
    • NLM

      Araújo EF de. A IDO controla a carga fúngica e a imunidade celular de camundongos suscetíveis e resistentes à infecção pelo Paracoccidioides brasiliensis [Internet]. 2009 ;[citado 2024 ago. 24 ] Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-02022010-124420/
    • Vancouver

      Araújo EF de. A IDO controla a carga fúngica e a imunidade celular de camundongos suscetíveis e resistentes à infecção pelo Paracoccidioides brasiliensis [Internet]. 2009 ;[citado 2024 ago. 24 ] Available from: http://www.teses.usp.br/teses/disponiveis/42/42133/tde-02022010-124420/

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