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Artigo de periodico
Reversion of cardiovascular remodelling in renovascular hypertensive 2K‐1C rats by renin-angiotensin system inhibitors (2020)
Corrêa, José Wilson do Nascimento; Prado, Cibele Maria ; Riul, Maria Elena; Araújo, Alice Valença; Rossi, Marcos Antonio; Bendhack, Lusiane Maria
Source: Clinical and Experimental Pharmacology and Physiology. Unidades: FZEA, FCFRP
Subjects: HIPERTENSÃO, SISTEMA RENINA-ANGIOTENSINA, MODELOS ANIMAIS
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CORRÊA, José Wilson do Nascimento et al. Reversion of cardiovascular remodelling in renovascular hypertensive 2K‐1C rats by renin-angiotensin system inhibitors. Clinical and Experimental Pharmacology and Physiology, v. 47, n. 12, p. 1965-1977, 2020Tradução . . Disponível em: https://doi.org/10.1111/1440-1681.13384. Acesso em: 11 out. 2024.
APA
Corrêa, J. W. do N., Prado, C. M., Riul, M. E., Araújo, A. V., Rossi, M. A., & Bendhack, L. M. (2020). Reversion of cardiovascular remodelling in renovascular hypertensive 2K‐1C rats by renin-angiotensin system inhibitors. Clinical and Experimental Pharmacology and Physiology, 47( 12), 1965-1977. doi:10.1111/1440-1681.13384
NLM
Corrêa JW do N, Prado CM, Riul ME, Araújo AV, Rossi MA, Bendhack LM. Reversion of cardiovascular remodelling in renovascular hypertensive 2K‐1C rats by renin-angiotensin system inhibitors [Internet]. Clinical and Experimental Pharmacology and Physiology. 2020 ; 47( 12): 1965-1977.[citado 2024 out. 11 ] Available from: https://doi.org/10.1111/1440-1681.13384
Vancouver
Corrêa JW do N, Prado CM, Riul ME, Araújo AV, Rossi MA, Bendhack LM. Reversion of cardiovascular remodelling in renovascular hypertensive 2K‐1C rats by renin-angiotensin system inhibitors [Internet]. Clinical and Experimental Pharmacology and Physiology. 2020 ; 47( 12): 1965-1977.[citado 2024 out. 11 ] Available from: https://doi.org/10.1111/1440-1681.13384
Artigo de periodico
Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase (2017)
Dal Secco, Daniela; DalBó, Silvia; Lautherbach, Natalia Ennes da Silva; Gava, Fábio N.; Celes, Mara Rúbia Nunes; Benedet, Patricia de OLiveira; Souza, Adriana Helena de; Akinaga, Juliana; Lima, Vanessa; Silva, Katiussia P.; Kiguti, Luiz Ricardo A.; Rossi, Marcos Antonio; Kettelhut, Isis do Carmo; Pupo, André Sampaio; Cunha, Fernando de Queiroz; Assreuy, Jamil
Source: American Journal of Physiology-Heart and Circulatory Physiology. Unidade: FMRP
Subjects: ATIVAÇÃO ENZIMÁTICA, INSUFICIÊNCIA CARDÍACA, ÓXIDO NÍTRICO, SEPSE, TRANSDUÇÃO DE SINAL CELULAR
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DAL SECCO, Daniela et al. Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase. American Journal of Physiology-Heart and Circulatory Physiology, v. 313, n. 1, p. H149-H163, 2017Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00052.2016. Acesso em: 11 out. 2024.
APA
Dal Secco, D., DalBó, S., Lautherbach, N. E. da S., Gava, F. N., Celes, M. R. N., Benedet, P. de O. L., et al. (2017). Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase. American Journal of Physiology-Heart and Circulatory Physiology, 313( 1), H149-H163. doi:10.1152/ajpheart.00052.2016
NLM
Dal Secco D, DalBó S, Lautherbach NE da S, Gava FN, Celes MRN, Benedet P de OL, Souza AH de, Akinaga J, Lima V, Silva KP, Kiguti LRA, Rossi MA, Kettelhut I do C, Pupo AS, Cunha F de Q, Assreuy J. Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2017 ; 313( 1): H149-H163.[citado 2024 out. 11 ] Available from: https://doi.org/10.1152/ajpheart.00052.2016
Vancouver
Dal Secco D, DalBó S, Lautherbach NE da S, Gava FN, Celes MRN, Benedet P de OL, Souza AH de, Akinaga J, Lima V, Silva KP, Kiguti LRA, Rossi MA, Kettelhut I do C, Pupo AS, Cunha F de Q, Assreuy J. Cardiac hyporesponsiveness in severe sepsis is associated with nitric oxide-dependent activation of G protein receptor kinase [Internet]. American Journal of Physiology-Heart and Circulatory Physiology. 2017 ; 313( 1): H149-H163.[citado 2024 out. 11 ] Available from: https://doi.org/10.1152/ajpheart.00052.2016
Artigo de periodico
Short-term and long-term models of doxorubicin-induced cardiomyopathy in rats: a comparison of functional and histopathological changes (2017)
O’Connell, João Lucas; Romano, Minna Moreira Dias; Pulicic, Erica C. Campos; Carvalho, Eduardo E. V.; Souza, Fernanda R. de; Tanaka, Denise M.; Maciel, Benedito Carlos; Salgado, Hélio Cesar; Fazan-Júnior, Rubens; Rossi, Marcos Antonio; Simões, Marcus Vinícius
Source: Experimental and Toxicologic Pathology. Unidade: FMRP
Subjects: INSUFICIÊNCIA CARDÍACA, VENTRÍCULO CARDÍACO, ANTIBIÓTICOS, CORAÇÃO, MODELOS ANIMAIS DE DOENÇAS
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O’CONNELL, João Lucas et al. Short-term and long-term models of doxorubicin-induced cardiomyopathy in rats: a comparison of functional and histopathological changes. Experimental and Toxicologic Pathology, v. 69, n. 4, p. 213-219, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.etp.2017.01.004. Acesso em: 11 out. 2024.
APA
O’Connell, J. L., Romano, M. M. D., Pulicic, E. C. C., Carvalho, E. E. V., Souza, F. R. de, Tanaka, D. M., et al. (2017). Short-term and long-term models of doxorubicin-induced cardiomyopathy in rats: a comparison of functional and histopathological changes. Experimental and Toxicologic Pathology, 69( 4), 213-219. doi:10.1016/j.etp.2017.01.004
NLM
O’Connell JL, Romano MMD, Pulicic ECC, Carvalho EEV, Souza FR de, Tanaka DM, Maciel BC, Salgado HC, Fazan-Júnior R, Rossi MA, Simões MV. Short-term and long-term models of doxorubicin-induced cardiomyopathy in rats: a comparison of functional and histopathological changes [Internet]. Experimental and Toxicologic Pathology. 2017 ; 69( 4): 213-219.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.etp.2017.01.004
Vancouver
O’Connell JL, Romano MMD, Pulicic ECC, Carvalho EEV, Souza FR de, Tanaka DM, Maciel BC, Salgado HC, Fazan-Júnior R, Rossi MA, Simões MV. Short-term and long-term models of doxorubicin-induced cardiomyopathy in rats: a comparison of functional and histopathological changes [Internet]. Experimental and Toxicologic Pathology. 2017 ; 69( 4): 213-219.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.etp.2017.01.004
Trabalho de evento-resumo
A control pathway for the promiscuous gene expression in medullary thymic epithelial cells involving aire and microRNAs (2015)
Oliveira, Ernna H.; Macedo, Claudia; Collares, Cristhiana A.; Donate, Paula Barbim; Rossi, Marcos Antonio; Sakamoto-Hojo, Elza Tiemi; Donadi, Eduardo Antônio; Passos, Geraldo Aleixo Silva
Source: Abstracts. Conference titles: International Venice Thymus Meeting. Unidades: FMRP, FFCLRP, FORP
Subjects: CÉLULAS EPITELIAIS, EXPRESSÃO GÊNICA, TIMO (GLÂNDULA ENDÓCRINA)
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ABNT
OLIVEIRA, Ernna H. et al. A control pathway for the promiscuous gene expression in medullary thymic epithelial cells involving aire and microRNAs. 2015, Anais.. Venice: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2015. . Acesso em: 11 out. 2024.
APA
Oliveira, E. H., Macedo, C., Collares, C. A., Donate, P. B., Rossi, M. A., Sakamoto-Hojo, E. T., et al. (2015). A control pathway for the promiscuous gene expression in medullary thymic epithelial cells involving aire and microRNAs. In Abstracts. Venice: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Oliveira EH, Macedo C, Collares CA, Donate PB, Rossi MA, Sakamoto-Hojo ET, Donadi EA, Passos GAS. A control pathway for the promiscuous gene expression in medullary thymic epithelial cells involving aire and microRNAs. Abstracts. 2015 ;[citado 2024 out. 11 ]
Vancouver
Oliveira EH, Macedo C, Collares CA, Donate PB, Rossi MA, Sakamoto-Hojo ET, Donadi EA, Passos GAS. A control pathway for the promiscuous gene expression in medullary thymic epithelial cells involving aire and microRNAs. Abstracts. 2015 ;[citado 2024 out. 11 ]
Artigo de periodico
The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice (2014)
Canavaci, Adriana Monte Cassiano; Sorgi, Carlos Artério; Martins, Vicente de Paulo; Morais, Fabiana Rosseto; Sousa, Érika V. G. de; Trindade, Bruno Caetano; Cunha, Fernando de Queiróz; Rossi, Marcos Antonio; Aronoff, David M.; Faccioli, Lúcia Helena; Nomizo, Auro
Source: Mediators of Inflammation. Unidades: FCFRP, FMRP
Subjects: TRYPANOSOMA CRUZI, INTERLEUCINAS, ANTIPARASITÁRIOS (FARMACOLOGIA)
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CANAVACI, Adriana Monte Cassiano et al. The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice. Mediators of Inflammation, v. 2014, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/893634. Acesso em: 11 out. 2024.
APA
Canavaci, A. M. C., Sorgi, C. A., Martins, V. de P., Morais, F. R., Sousa, É. V. G. de, Trindade, B. C., et al. (2014). The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice. Mediators of Inflammation, 2014. doi:10.1155/2014/893634
NLM
Canavaci AMC, Sorgi CA, Martins V de P, Morais FR, Sousa ÉVG de, Trindade BC, Cunha F de Q, Rossi MA, Aronoff DM, Faccioli LH, Nomizo A. The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice [Internet]. Mediators of Inflammation. 2014 ; 2014[citado 2024 out. 11 ] Available from: https://doi.org/10.1155/2014/893634
Vancouver
Canavaci AMC, Sorgi CA, Martins V de P, Morais FR, Sousa ÉVG de, Trindade BC, Cunha F de Q, Rossi MA, Aronoff DM, Faccioli LH, Nomizo A. The acute phase of Trypanosoma cruzi infection is attenuated in 5-lipoxygenase-deficient mice [Internet]. Mediators of Inflammation. 2014 ; 2014[citado 2024 out. 11 ] Available from: https://doi.org/10.1155/2014/893634
Artigo de periodico
CCR5 controls immune and metabolic functions during Toxoplasma gondii infection (2014)
Bonfá, Giuliano; Souza, Maria do Carmo; Fonseca, Denise Morais da ; Mineo, Tiago Wilson Patriarca; Rossi, Marcos Antonio; Silva, Neide Maria; Silva, João Santana; Cardoso, Cristina Ribeiro de Barros
Source: PLOs One. Unidades: FMRP, FCFRP
Subjects: CITOCINAS (METABOLISMO), CITOMETRIA DE FLUXO, ÍLEO (IMUNOLOGIA), NEUTRÓFILOS, TOXOPLASMOSE
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BONFÁ, Giuliano et al. CCR5 controls immune and metabolic functions during Toxoplasma gondii infection. PLOs One, v. 9, n. 8, p. e104736-1 - e104736-13, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0104736. Acesso em: 11 out. 2024.
APA
Bonfá, G., Souza, M. do C., Fonseca, D. M. da, Mineo, T. W. P., Rossi, M. A., Silva, N. M., et al. (2014). CCR5 controls immune and metabolic functions during Toxoplasma gondii infection. PLOs One, 9( 8), e104736-1 - e104736-13. doi:10.1371/journal.pone.0104736
NLM
Bonfá G, Souza M do C, Fonseca DM da, Mineo TWP, Rossi MA, Silva NM, Silva JS, Cardoso CR de B. CCR5 controls immune and metabolic functions during Toxoplasma gondii infection [Internet]. PLOs One. 2014 ; 9( 8): e104736-1 - e104736-13.[citado 2024 out. 11 ] Available from: https://doi.org/10.1371/journal.pone.0104736
Vancouver
Bonfá G, Souza M do C, Fonseca DM da, Mineo TWP, Rossi MA, Silva NM, Silva JS, Cardoso CR de B. CCR5 controls immune and metabolic functions during Toxoplasma gondii infection [Internet]. PLOs One. 2014 ; 9( 8): e104736-1 - e104736-13.[citado 2024 out. 11 ] Available from: https://doi.org/10.1371/journal.pone.0104736
Artigo de periodico
Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection (2014)
Gutierrez, Fredy R. S; Pavanelli, Wander R; Medina, Tiago S; Silva, Grace K; Mariano, Flávia S; Guedes, Paulo M. M.; Mineo, Tiago W. P.; Rossi, Marcos Antonio; Cunha, Fernando de Queiroz; Silva, João Santana da
Source: Microbes and Infection. Unidade: FMRP
Subjects: TRYPANOSOMA CRUZI, OXIGENASE, LINFÓCITOS T
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GUTIERREZ, Fredy R. S et al. Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection. Microbes and Infection, v. 16, n. 1, p. 28-39, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2013.10.007. Acesso em: 11 out. 2024.
APA
Gutierrez, F. R. S., Pavanelli, W. R., Medina, T. S., Silva, G. K., Mariano, F. S., Guedes, P. M. M., et al. (2014). Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection. Microbes and Infection, 16( 1), 28-39. doi:10.1016/j.micinf.2013.10.007
NLM
Gutierrez FRS, Pavanelli WR, Medina TS, Silva GK, Mariano FS, Guedes PMM, Mineo TWP, Rossi MA, Cunha F de Q, Silva JS da. Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 1): 28-39.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.micinf.2013.10.007
Vancouver
Gutierrez FRS, Pavanelli WR, Medina TS, Silva GK, Mariano FS, Guedes PMM, Mineo TWP, Rossi MA, Cunha F de Q, Silva JS da. Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 1): 28-39.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.micinf.2013.10.007
Artigo de periodico
Role of dystrophin in acute Trypanosoma cruzi infection (2014)
Malvestio, Lygia M; Celes, Mara R. N; Milanezi, Cristiane Maria; Silva, João Santana da; Jelicks, Linda A; Tanowitz, Hebert B; Rossi, Marcos Antonio; Prado, Cibele Maria
Source: Microbes and Infection. Unidade: FMRP
Subjects: DOENÇA DE CHAGAS, INFLAMAÇÃO (METABOLISMO), TRYPANOSOMA CRUZI, MIÓCITOS DE MÚSCULO LISO
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ABNT
MALVESTIO, Lygia M et al. Role of dystrophin in acute Trypanosoma cruzi infection. Microbes and Infection, v. 16, n. 9, p. 768-777, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2014.07.010. Acesso em: 11 out. 2024.
APA
Malvestio, L. M., Celes, M. R. N., Milanezi, C. M., Silva, J. S. da, Jelicks, L. A., Tanowitz, H. B., et al. (2014). Role of dystrophin in acute Trypanosoma cruzi infection. Microbes and Infection, 16( 9), 768-777. doi:10.1016/j.micinf.2014.07.010
NLM
Malvestio LM, Celes MRN, Milanezi CM, Silva JS da, Jelicks LA, Tanowitz HB, Rossi MA, Prado CM. Role of dystrophin in acute Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 9): 768-777.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.micinf.2014.07.010
Vancouver
Malvestio LM, Celes MRN, Milanezi CM, Silva JS da, Jelicks LA, Tanowitz HB, Rossi MA, Prado CM. Role of dystrophin in acute Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 9): 768-777.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.micinf.2014.07.010
Artigo de periodico
Involvement of the microvasculature in the pathogenesis of terlipressin-related myocardial infarction (2014)
Carmo, Lucas S.; Baima, Diego C.; Blefari, Valdecir; Zonta, Vaniela; Troncon, Luiz Ernesto de Almeida; Rossi, Marcos Antonio
Source: European Heart Journal: Acute Cardiovascular Care. Unidade: FMRP
Subjects: CIRROSE HEPÁTICA, MICROCIRCULAÇÃO, INFARTO DO MIOCÁRDIO, HEMORRAGIA, VASOPRESSINAS
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ABNT
CARMO, Lucas S. et al. Involvement of the microvasculature in the pathogenesis of terlipressin-related myocardial infarction. European Heart Journal: Acute Cardiovascular Care, 2014Tradução . . Disponível em: https://doi.org/10.1177/2048872614534921. Acesso em: 11 out. 2024.
APA
Carmo, L. S., Baima, D. C., Blefari, V., Zonta, V., Troncon, L. E. de A., & Rossi, M. A. (2014). Involvement of the microvasculature in the pathogenesis of terlipressin-related myocardial infarction. European Heart Journal: Acute Cardiovascular Care. doi:10.1177/2048872614534921
NLM
Carmo LS, Baima DC, Blefari V, Zonta V, Troncon LE de A, Rossi MA. Involvement of the microvasculature in the pathogenesis of terlipressin-related myocardial infarction [Internet]. European Heart Journal: Acute Cardiovascular Care. 2014 ;[citado 2024 out. 11 ] Available from: https://doi.org/10.1177/2048872614534921
Vancouver
Carmo LS, Baima DC, Blefari V, Zonta V, Troncon LE de A, Rossi MA. Involvement of the microvasculature in the pathogenesis of terlipressin-related myocardial infarction [Internet]. European Heart Journal: Acute Cardiovascular Care. 2014 ;[citado 2024 out. 11 ] Available from: https://doi.org/10.1177/2048872614534921
Artigo de periodico
Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats (2013)
Cáu, Stêfany Bruno de Assis; Barato, Renan C.; Celes, Mara Rúbia Nunes; Muniz, Jaqueline J.; Rossi, Marcos Antonio; Tanus-Santos, José Eduardo
Source: Cardiovascular Drugs and Therapy. Unidade: FMRP
Subjects: EMBOLIA PULMONAR, DOENÇAS CARDIOVASCULARES, METALOPROTEINASES, MODELOS ANIMAIS
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CÁU, Stêfany Bruno de Assis et al. Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats. Cardiovascular Drugs and Therapy, v. 27, n. 4, p. 259-267, 2013Tradução . . Disponível em: https://doi.org/10.1007/s10557-013-6458-9. Acesso em: 11 out. 2024.
APA
Cáu, S. B. de A., Barato, R. C., Celes, M. R. N., Muniz, J. J., Rossi, M. A., & Tanus-Santos, J. E. (2013). Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats. Cardiovascular Drugs and Therapy, 27( 4), 259-267. doi:10.1007/s10557-013-6458-9
NLM
Cáu SB de A, Barato RC, Celes MRN, Muniz JJ, Rossi MA, Tanus-Santos JE. Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats [Internet]. Cardiovascular Drugs and Therapy. 2013 ; 27( 4): 259-267.[citado 2024 out. 11 ] Available from: https://doi.org/10.1007/s10557-013-6458-9
Vancouver
Cáu SB de A, Barato RC, Celes MRN, Muniz JJ, Rossi MA, Tanus-Santos JE. Doxycycline prevents acute pulmonary embolism-induced mortality and right ventricular deformation in rats [Internet]. Cardiovascular Drugs and Therapy. 2013 ; 27( 4): 259-267.[citado 2024 out. 11 ] Available from: https://doi.org/10.1007/s10557-013-6458-9
Artigo de periodico
Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis (2013)
Czaikoski, Paula Giselle; Nascimento, Daniele Carvalho; Sônego, Fabiane; Freitas, Andressa de; Turato, Walter Miguel; Carvalho, Michel A. de; Santos, Raquel Souza; Oliveira, Gisele Pena de; Samary, Cynthia dos Santos; Tefe-Silva, Cristiane; Alves Filho, José Carlos Farias; Ferreira, Sérgio Henrique; Rossi, Marcos Antonio; Rocco, Patricia Rieken Macedo; Spiller, Fernando; Cunha, Fernando de Queiroz
Source: Shock. Unidades: FCF, FMRP
Subjects: OXIGENASE, INIBIDORES DE ENZIMAS, NEUTRÓFILOS, SEPSE, PNEUMONIA
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ABNT
CZAIKOSKI, Paula Giselle et al. Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis. Shock, v. 39, n. 4, p. 389-396, 2013Tradução . . Disponível em: https://doi.org/10.1097/SHK.0b013e31828bbcf9. Acesso em: 11 out. 2024.
APA
Czaikoski, P. G., Nascimento, D. C., Sônego, F., Freitas, A. de, Turato, W. M., Carvalho, M. A. de, et al. (2013). Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis. Shock, 39( 4), 389-396. doi:10.1097/SHK.0b013e31828bbcf9
NLM
Czaikoski PG, Nascimento DC, Sônego F, Freitas A de, Turato WM, Carvalho MA de, Santos RS, Oliveira GP de, Samary C dos S, Tefe-Silva C, Alves Filho JCF, Ferreira SH, Rossi MA, Rocco PRM, Spiller F, Cunha F de Q. Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis [Internet]. Shock. 2013 ; 39( 4): 389-396.[citado 2024 out. 11 ] Available from: https://doi.org/10.1097/SHK.0b013e31828bbcf9
Vancouver
Czaikoski PG, Nascimento DC, Sônego F, Freitas A de, Turato WM, Carvalho MA de, Santos RS, Oliveira GP de, Samary C dos S, Tefe-Silva C, Alves Filho JCF, Ferreira SH, Rossi MA, Rocco PRM, Spiller F, Cunha F de Q. Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis [Internet]. Shock. 2013 ; 39( 4): 389-396.[citado 2024 out. 11 ] Available from: https://doi.org/10.1097/SHK.0b013e31828bbcf9
Artigo de periodico
Persistence of epoxy-based sealer residues in dentin treated with different chemical removal protocols (2013)
Kuga, Milton Carlos ; Faria, Gisele ; Rossi, Marcos Antonio; Monteiro, Jardel Camilo do Carmo ; Bonetti-Filho, Idomeo; Berbert, Fábio Luiz Camargo Vilella; Keine, Kátia Cristina ; Só, Marcus Vinicius Reis
Source: Scanning. Unidade: FMRP
Subjects: MATERIAIS DENTÁRIOS (ANÁLISE), CIMENTOS DENTÁRIOS (REMOÇÃO), OBTURAÇÃO DO CANAL RADICULAR
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ABNT
KUGA, Milton Carlos et al. Persistence of epoxy-based sealer residues in dentin treated with different chemical removal protocols. Scanning, v. 35, n. 1, p. 17-21, 2013Tradução . . Disponível em: https://doi.org/10.1002/sca.21030. Acesso em: 11 out. 2024.
APA
Kuga, M. C., Faria, G., Rossi, M. A., Monteiro, J. C. do C., Bonetti-Filho, I., Berbert, F. L. C. V., et al. (2013). Persistence of epoxy-based sealer residues in dentin treated with different chemical removal protocols. Scanning, 35( 1), 17-21. doi:10.1002/sca.21030
NLM
Kuga MC, Faria G, Rossi MA, Monteiro JC do C, Bonetti-Filho I, Berbert FLCV, Keine KC, Só MVR. Persistence of epoxy-based sealer residues in dentin treated with different chemical removal protocols [Internet]. Scanning. 2013 ; 35( 1): 17-21.[citado 2024 out. 11 ] Available from: https://doi.org/10.1002/sca.21030
Vancouver
Kuga MC, Faria G, Rossi MA, Monteiro JC do C, Bonetti-Filho I, Berbert FLCV, Keine KC, Só MVR. Persistence of epoxy-based sealer residues in dentin treated with different chemical removal protocols [Internet]. Scanning. 2013 ; 35( 1): 17-21.[citado 2024 out. 11 ] Available from: https://doi.org/10.1002/sca.21030
Artigo de periodico
Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy (2013)
Rizzi, Elen; Ceron, Carla S.; Guimarães, Danielle A.; Prado, Cibele Maria; Rossi, Marcos Antonio; Gerlach, Raquel Fernanda; Santos, José Eduardo Tanus dos
Source: Experimental and Molecular Pathology. Unidades: FMRP, FORP
Subjects: METALOPROTEINASES, HIPERTENSÃO, DOENÇAS CARDIOVASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RIZZI, Elen et al. Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy. Experimental and Molecular Pathology, v. 94, n. 1, p. 1-9, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.yexmp.2012.10.010. Acesso em: 11 out. 2024.
APA
Rizzi, E., Ceron, C. S., Guimarães, D. A., Prado, C. M., Rossi, M. A., Gerlach, R. F., & Santos, J. E. T. dos. (2013). Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy. Experimental and Molecular Pathology, 94( 1), 1-9. doi:10.1016/j.yexmp.2012.10.010
NLM
Rizzi E, Ceron CS, Guimarães DA, Prado CM, Rossi MA, Gerlach RF, Santos JET dos. Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy [Internet]. Experimental and Molecular Pathology. 2013 ; 94( 1): 1-9.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.yexmp.2012.10.010
Vancouver
Rizzi E, Ceron CS, Guimarães DA, Prado CM, Rossi MA, Gerlach RF, Santos JET dos. Temporal changes in cardiac matrix metalloproteinase activity, oxidative stress, and TGF-'beta' in renovascular hypertension-induced cardiac hypertrophy [Internet]. Experimental and Molecular Pathology. 2013 ; 94( 1): 1-9.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.yexmp.2012.10.010
Artigo de periodico
B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection (2013)
Tristão, Fabrine Sales Massafera; Panagio, Luciano Aparecido; Rocha, F. A.; Cavassani, K. A.; Moreira, A. P.; Rossi, Marcos Antonio; Silva, João Santana da
Source: Mycopathologia. Unidade: FMRP
Subjects: PARACOCCIDIOIDES BRASILIENSIS (IMUNOLOGIA), LINFÓCITOS B (DEFICIÊNCIA), CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TRISTÃO, Fabrine Sales Massafera et al. B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection. Mycopathologia, v. 176, n. 1-2, p. 1-10, 2013Tradução . . Disponível em: https://doi.org/10.1007/s11046-013-9671-y. Acesso em: 11 out. 2024.
APA
Tristão, F. S. M., Panagio, L. A., Rocha, F. A., Cavassani, K. A., Moreira, A. P., Rossi, M. A., & Silva, J. S. da. (2013). B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection. Mycopathologia, 176( 1-2), 1-10. doi:10.1007/s11046-013-9671-y
NLM
Tristão FSM, Panagio LA, Rocha FA, Cavassani KA, Moreira AP, Rossi MA, Silva JS da. B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection [Internet]. Mycopathologia. 2013 ; 176( 1-2): 1-10.[citado 2024 out. 11 ] Available from: https://doi.org/10.1007/s11046-013-9671-y
Vancouver
Tristão FSM, Panagio LA, Rocha FA, Cavassani KA, Moreira AP, Rossi MA, Silva JS da. B cell-deficient mice display enhanced susceptibility to Paracoccidioides brasiliensis infection [Internet]. Mycopathologia. 2013 ; 176( 1-2): 1-10.[citado 2024 out. 11 ] Available from: https://doi.org/10.1007/s11046-013-9671-y
Artigo de periodico
Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure (2013)
Lataro, Renata Maria; Silva, Carlos Alberto Aguiar da; Fazan Júnior, Rubens; Rossi, Marcos Antonio; Prado, Cibele Maria; Godinho, Rosely Oliveira; Salgado, Helio Cesar
Source: American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. Unidade: FMRP
Subjects: CORAÇÃO (FISIOPATOLOGIA;EFEITOS DE DROGAS), INIBIDORES DE ENZIMAS (FARMACOLOGIA), SISTEMA NERVOSO PARASSIMPÁTICO, VENTRÍCULO CARDÍACO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LATARO, Renata Maria et al. Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, v. 305, n. 8, p. R908-R916, 2013Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00102.2013. Acesso em: 11 out. 2024.
APA
Lataro, R. M., Silva, C. A. A. da, Fazan Júnior, R., Rossi, M. A., Prado, C. M., Godinho, R. O., & Salgado, H. C. (2013). Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, 305( 8), R908-R916. doi:10.1152/ajpregu.00102.2013
NLM
Lataro RM, Silva CAA da, Fazan Júnior R, Rossi MA, Prado CM, Godinho RO, Salgado HC. Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2013 ; 305( 8): R908-R916.[citado 2024 out. 11 ] Available from: https://doi.org/10.1152/ajpregu.00102.2013
Vancouver
Lataro RM, Silva CAA da, Fazan Júnior R, Rossi MA, Prado CM, Godinho RO, Salgado HC. Increase in parasympathetic tone by pyridostigmine prevents ventricular dysfunction during the onset of heart failure [Internet]. American Journal of Physiology: Regulatory, Integrative and Comparative Physiology. 2013 ; 305( 8): R908-R916.[citado 2024 out. 11 ] Available from: https://doi.org/10.1152/ajpregu.00102.2013
Artigo de periodico
Fulminant gastrointestinal hemorrhage due to Strongyloides stercoralis hyperinfection in an AIDS patient (2013)
Bollela, Valdes Roberto; Feliciano, Cinara Silva; Teixeira, André Costa; Junqueira, Ana Carolina Ribeiro; Rossi, Marcos Antonio
Source: Revista da Sociedade Brasileira de Medicina Tropical. Unidade: FMRP
Subjects: STRONGYLIDA, HEMORRAGIA, GASTROENTEROLOGIA (COMPLICAÇÕES), SÍNDROME DE IMUNODEFICIÊNCIA ADQUIRIDA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOLLELA, Valdes Roberto et al. Fulminant gastrointestinal hemorrhage due to Strongyloides stercoralis hyperinfection in an AIDS patient. Revista da Sociedade Brasileira de Medicina Tropical, v. 46, n. 1, p. 111-113, 2013Tradução . . Disponível em: https://doi.org/10.1590/0037-868215522013. Acesso em: 11 out. 2024.
APA
Bollela, V. R., Feliciano, C. S., Teixeira, A. C., Junqueira, A. C. R., & Rossi, M. A. (2013). Fulminant gastrointestinal hemorrhage due to Strongyloides stercoralis hyperinfection in an AIDS patient. Revista da Sociedade Brasileira de Medicina Tropical, 46( 1), 111-113. doi:10.1590/0037-868215522013
NLM
Bollela VR, Feliciano CS, Teixeira AC, Junqueira ACR, Rossi MA. Fulminant gastrointestinal hemorrhage due to Strongyloides stercoralis hyperinfection in an AIDS patient [Internet]. Revista da Sociedade Brasileira de Medicina Tropical. 2013 ; 46( 1): 111-113.[citado 2024 out. 11 ] Available from: https://doi.org/10.1590/0037-868215522013
Vancouver
Bollela VR, Feliciano CS, Teixeira AC, Junqueira ACR, Rossi MA. Fulminant gastrointestinal hemorrhage due to Strongyloides stercoralis hyperinfection in an AIDS patient [Internet]. Revista da Sociedade Brasileira de Medicina Tropical. 2013 ; 46( 1): 111-113.[citado 2024 out. 11 ] Available from: https://doi.org/10.1590/0037-868215522013
Artigo de periodico
Sepsis: going to the heart of the matter (2013)
Celes, Mara Rúbia Nunes; Prado, Cibele Maria; Rossi, Marcos Antonio
Source: Pathobiology. Unidade: FMRP
Subjects: SEPSE, MIOCARDIOPATIAS, CORAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CELES, Mara Rúbia Nunes e PRADO, Cibele Maria e ROSSI, Marcos Antonio. Sepsis: going to the heart of the matter. Pathobiology, v. 80, n. 2, p. 70-86, 2013Tradução . . Disponível em: https://doi.org/10.1159/000341640. Acesso em: 11 out. 2024.
APA
Celes, M. R. N., Prado, C. M., & Rossi, M. A. (2013). Sepsis: going to the heart of the matter. Pathobiology, 80( 2), 70-86. doi:10.1159/000341640
NLM
Celes MRN, Prado CM, Rossi MA. Sepsis: going to the heart of the matter [Internet]. Pathobiology. 2013 ; 80( 2): 70-86.[citado 2024 out. 11 ] Available from: https://doi.org/10.1159/000341640
Vancouver
Celes MRN, Prado CM, Rossi MA. Sepsis: going to the heart of the matter [Internet]. Pathobiology. 2013 ; 80( 2): 70-86.[citado 2024 out. 11 ] Available from: https://doi.org/10.1159/000341640
Artigo de periodico
The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice (2013)
Tristão, Fabrine Sales Massafera; Rocha, F. A.; Dias, F. C.; Rossi, Marcos Antonio; Silva, João Santana da
Source: Brazilian Journal of Medical and Biological Research. Unidade: FMRP
Subjects: PULMÃO, PARACOCCIDIOIDES BRASILIENSIS, MODELOS ANIMAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TRISTÃO, Fabrine Sales Massafera et al. The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice. Brazilian Journal of Medical and Biological Research, v. 46, n. 10, p. 839-843, 2013Tradução . . Disponível em: https://doi.org/10.1590/1414-431X20133140. Acesso em: 11 out. 2024.
APA
Tristão, F. S. M., Rocha, F. A., Dias, F. C., Rossi, M. A., & Silva, J. S. da. (2013). The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice. Brazilian Journal of Medical and Biological Research, 46( 10), 839-843. doi:10.1590/1414-431X20133140
NLM
Tristão FSM, Rocha FA, Dias FC, Rossi MA, Silva JS da. The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice [Internet]. Brazilian Journal of Medical and Biological Research. 2013 ; 46( 10): 839-843.[citado 2024 out. 11 ] Available from: https://doi.org/10.1590/1414-431X20133140
Vancouver
Tristão FSM, Rocha FA, Dias FC, Rossi MA, Silva JS da. The left lung is preferentially targeted during experimental paracoccidioidomycosis in C57BL/6 mice [Internet]. Brazilian Journal of Medical and Biological Research. 2013 ; 46( 10): 839-843.[citado 2024 out. 11 ] Available from: https://doi.org/10.1590/1414-431X20133140
Artigo de periodico
Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis (2013)
Celes, Mara R. N.; Malvestio, Lygia M.; Suadicani, Sylvia O.; Prado, Cibele Maria; Figueiredo, Maria J.; Campos, Erica C.; Freitas, Ana C. S.; Spray, David C.; Tanowitz, Herbert B.; Silva, João Santana da; Rossi, Marcos Antonio
Source: PLOS ONE. Unidade: FMRP
Subjects: CARDIOPATIAS, SEPSE, EXPERIMENTOS ANIMAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CELES, Mara R. N. et al. Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis. PLOS ONE, v. 8, n. 7, p. e68809-1-e68809-13, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0068809. Acesso em: 11 out. 2024.
APA
Celes, M. R. N., Malvestio, L. M., Suadicani, S. O., Prado, C. M., Figueiredo, M. J., Campos, E. C., et al. (2013). Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis. PLOS ONE, 8( 7), e68809-1-e68809-13. doi:10.1371/journal.pone.0068809
NLM
Celes MRN, Malvestio LM, Suadicani SO, Prado CM, Figueiredo MJ, Campos EC, Freitas ACS, Spray DC, Tanowitz HB, Silva JS da, Rossi MA. Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis [Internet]. PLOS ONE. 2013 ; 8( 7): e68809-1-e68809-13.[citado 2024 out. 11 ] Available from: https://doi.org/10.1371/journal.pone.0068809
Vancouver
Celes MRN, Malvestio LM, Suadicani SO, Prado CM, Figueiredo MJ, Campos EC, Freitas ACS, Spray DC, Tanowitz HB, Silva JS da, Rossi MA. Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis [Internet]. PLOS ONE. 2013 ; 8( 7): e68809-1-e68809-13.[citado 2024 out. 11 ] Available from: https://doi.org/10.1371/journal.pone.0068809
Artigo de periodico
Expression profile of peripheral tissue antigen genes in medullary thymic epithelial cells (mTECs) is dependent on mRNA levels of autoimmune regulator (Aire) (2013)
Oliveira, Ernna H.; Macedo, Claudia; Donate, Paula B.; Almeida, Renata S.; Pezzi, Nicole; Nguyen, Catherine; Rossi, Marcos Antonio; Sakamoto-Hojo, Elza Tiemi; Donadi, Eduardo Antônio; Passos, Geraldo Aleixo Silva
Source: Immunobiology. Unidades: FMRP, FFCLRP, FORP
Subjects: IMUNOLOGIA CELULAR, DIABETES MELLITUS INSULINO-DEPENDENTE, AUTOIMUNIDADE (REGULAÇÃO), EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Ernna H. et al. Expression profile of peripheral tissue antigen genes in medullary thymic epithelial cells (mTECs) is dependent on mRNA levels of autoimmune regulator (Aire). Immunobiology, v. 218, n. 1, p. 96-104, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2012.02.005. Acesso em: 11 out. 2024.
APA
Oliveira, E. H., Macedo, C., Donate, P. B., Almeida, R. S., Pezzi, N., Nguyen, C., et al. (2013). Expression profile of peripheral tissue antigen genes in medullary thymic epithelial cells (mTECs) is dependent on mRNA levels of autoimmune regulator (Aire). Immunobiology, 218( 1), 96-104. doi:10.1016/j.imbio.2012.02.005
NLM
Oliveira EH, Macedo C, Donate PB, Almeida RS, Pezzi N, Nguyen C, Rossi MA, Sakamoto-Hojo ET, Donadi EA, Passos GAS. Expression profile of peripheral tissue antigen genes in medullary thymic epithelial cells (mTECs) is dependent on mRNA levels of autoimmune regulator (Aire) [Internet]. Immunobiology. 2013 ; 218( 1): 96-104.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.imbio.2012.02.005
Vancouver
Oliveira EH, Macedo C, Donate PB, Almeida RS, Pezzi N, Nguyen C, Rossi MA, Sakamoto-Hojo ET, Donadi EA, Passos GAS. Expression profile of peripheral tissue antigen genes in medullary thymic epithelial cells (mTECs) is dependent on mRNA levels of autoimmune regulator (Aire) [Internet]. Immunobiology. 2013 ; 218( 1): 96-104.[citado 2024 out. 11 ] Available from: https://doi.org/10.1016/j.imbio.2012.02.005

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