Alvizi, Lucas 
; Brito, Luciano Abreu; Bischain, Bárbara; Silva, Camila Bassi Fernandes da; Ramos, Sofia Ligia Guimaraes; Kobayashi, Gerson Shigeru; Wang, Jaqueline; Passos-Bueno, Maria Rita
Fonte: BioRxiv. Unidades: IB, BIOINFORMÁTICA
Assuntos: MALFORMAÇÕES, FISSURA LÁBIOPALATINA, EXPRESSÃO GÊNICA, METILAÇÃO DE DNA
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ALVIZI, Lucas et al. Mir152 hypomethylation, potentially triggered by embryonic hypoxia, as a common mechanism for non-syndromic cleft lip/palate. BioRxiv, 2019Tradução . . Disponível em: https://doi.org/10.1101/850016. Acesso em: 18 nov. 2024.APA
Alvizi, L., Brito, L. A., Bischain, B., Silva, C. B. F. da, Ramos, S. L. G., Kobayashi, G. S., et al. (2019). Mir152 hypomethylation, potentially triggered by embryonic hypoxia, as a common mechanism for non-syndromic cleft lip/palate. BioRxiv. doi:10.1101/850016NLM
Alvizi L, Brito LA, Bischain B, Silva CBF da, Ramos SLG, Kobayashi GS, Wang J, Passos-Bueno MR. Mir152 hypomethylation, potentially triggered by embryonic hypoxia, as a common mechanism for non-syndromic cleft lip/palate [Internet]. BioRxiv. 2019 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.1101/850016Vancouver
Alvizi L, Brito LA, Bischain B, Silva CBF da, Ramos SLG, Kobayashi GS, Wang J, Passos-Bueno MR. Mir152 hypomethylation, potentially triggered by embryonic hypoxia, as a common mechanism for non-syndromic cleft lip/palate [Internet]. BioRxiv. 2019 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.1101/850016
