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Artigo de periodico
FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
Borges, Vanessa de Fátima; Galant, Leticia Selinger; Kanashiro, Alexandre ; Castanheira, Fernanda Vargas e Silva; Monteiro, Valter Vinícius Silva; Duarte, Diego Ângelo; Rodrigues, Filipe Camargo; Silva, Camila Meirelles de Souza; Schneider, Ayda Henriques ; Cebinelli, Guilherme Cesar Martelossi ; Lima, Mikhael Haruo Fernandes de; Viola, João Paulo de Biaso; Cunha, Thiago Mattar ; Costa Neto, Claudio Miguel da ; Alves Filho, José Carlos Farias ; Pupo, André Sampaio; Cunha, Fernando de Queiroz
Source: Inflammation Research. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 15 nov. 2024.
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Artigo de periodico
COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis (2023)
Barreto, Ester A.; Cruz, Amanda S.; Veras, Flávio Protásio ; Martins, Ronaldo Bragança ; Bernardelli, Rafaella S.; Paiva, Isadora Marques ; Lima, Thais Melquiades de; Singh, Youvika ; Guimaraes, Raphael C.; Damasceno, Samara; Pereira, Nayara; Alves, João Manoel; Gonçalves, Tiago T.; Forato, Julia; Muraro, Stéfanie P.; Souza, Gabriela F.; Batah, Sabrina Setembre ; Proença-Modena, José L.; Mori, Marcelo A.; Cunha, Fernando de Queiroz ; Louzada Júnior, Paulo ; Cunha, Thiago Mattar ; Nakaya, Helder Takashi Imoto ; Fabro, Alexandre Todorovic ; Oliveira, Renê Donizeti Ribeiro de; Arruda, Eurico ; Rea, Rosângela; Réa Neto, Álvaro; Silva, Miguel M Fernandes da; Leiria, Luiz Osório Silveira
Source: Proceedings of the National Academy of Sciences. Unidades: FMRP, FCF
Subjects: GLICOSE, COVID-19, GLICOGENOSE, HIPERGLICEMIA, ESTUDOS RETROSPECTIVOS
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BARRETO, Ester A. et al. COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis. Proceedings of the National Academy of Sciences, v. 120, n. 21, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1073/pnas.2217119120. Acesso em: 15 nov. 2024.
APA
Barreto, E. A., Cruz, A. S., Veras, F. P., Martins, R. B., Bernardelli, R. S., Paiva, I. M., et al. (2023). COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis. Proceedings of the National Academy of Sciences, 120( 21), 1-10. doi:10.1073/pnas.2217119120
NLM
Barreto EA, Cruz AS, Veras FP, Martins RB, Bernardelli RS, Paiva IM, Lima TM de, Singh Y, Guimaraes RC, Damasceno S, Pereira N, Alves JM, Gonçalves TT, Forato J, Muraro SP, Souza GF, Batah SS, Proença-Modena JL, Mori MA, Cunha F de Q, Louzada Júnior P, Cunha TM, Nakaya HTI, Fabro AT, Oliveira RDR de, Arruda E, Rea R, Réa Neto Á, Silva MMF da, Leiria LOS. COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis [Internet]. Proceedings of the National Academy of Sciences. 2023 ; 120( 21): 1-10.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1073/pnas.2217119120
Vancouver
Barreto EA, Cruz AS, Veras FP, Martins RB, Bernardelli RS, Paiva IM, Lima TM de, Singh Y, Guimaraes RC, Damasceno S, Pereira N, Alves JM, Gonçalves TT, Forato J, Muraro SP, Souza GF, Batah SS, Proença-Modena JL, Mori MA, Cunha F de Q, Louzada Júnior P, Cunha TM, Nakaya HTI, Fabro AT, Oliveira RDR de, Arruda E, Rea R, Réa Neto Á, Silva MMF da, Leiria LOS. COVID-19-related hyperglycemia is associated with infection of hepatocytes and stimulation of gluconeogenesis [Internet]. Proceedings of the National Academy of Sciences. 2023 ; 120( 21): 1-10.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1073/pnas.2217119120
Artigo de periodico
Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection (2023)
Moreira, Thaiane Pinto; Sousa, Carla Daiane Ferreira de; Costa, Victor Rodrigues de Melo; Queiroz Junior, Celso Martins; Santos, Franciele Martins; Bonilha, Caio Santos ; Ésper, Lísia Maria; Nogueira, Mauricio Lacerda ; Cunha, Thiago Mattar ; Teixeira, Mauro Martins ; Costa, Vivian Vasconcelos; Souza, Daniele da Glória de
Source: Immunology. Unidade: FMRP
Subjects: ARTRALGIA, VÍRUS CHIKUNGUNYA, NECROSE, REPLICAÇÃO VIRAL
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MOREIRA, Thaiane Pinto et al. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection. Immunology, v. 168, n. 3, p. 444-458, 2023Tradução . . Disponível em: https://doi.org/10.1111/imm.13583. Acesso em: 15 nov. 2024.
APA
Moreira, T. P., Sousa, C. D. F. de, Costa, V. R. de M., Queiroz Junior, C. M., Santos, F. M., Bonilha, C. S., et al. (2023). Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection. Immunology, 168( 3), 444-458. doi:10.1111/imm.13583
NLM
Moreira TP, Sousa CDF de, Costa VR de M, Queiroz Junior CM, Santos FM, Bonilha CS, Ésper LM, Nogueira ML, Cunha TM, Teixeira MM, Costa VV, Souza D da G de. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection [Internet]. Immunology. 2023 ; 168( 3): 444-458.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1111/imm.13583
Vancouver
Moreira TP, Sousa CDF de, Costa VR de M, Queiroz Junior CM, Santos FM, Bonilha CS, Ésper LM, Nogueira ML, Cunha TM, Teixeira MM, Costa VV, Souza D da G de. Tumour necrosis factor plays a deleterious role in the pathogenesis of chikungunya virus infection [Internet]. Immunology. 2023 ; 168( 3): 444-458.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1111/imm.13583
Artigo de periodico
SARS-CoV-2 uses CD4 to infect T helper lymphocytes (2023)
Brunetti, Natalia S.; Silva, João Victor Virgilio da ; Veras, Flávio Protásio ; Tavares, Lucas Alves ; Castro, Ícaro Maia Santos de ; Silvestrini, Virgínia Campos; Oliveira, Eduardo Brandt de; Faça, Vitor Marcel ; Sposito, Andrei Carvalho ; Velloso, Licio Augusto; Silva, Luis Lamberti Pinto da ; Cunha, Thiago Mattar ; Nakaya, Helder Takashi Imoto ; Skaf, Munir Salomão ; Vieira, Pedro Manoel Mendes de Moraes ; Farias, Alessandro dos Santos
Source: eLife. Unidades: FMRP, FM, IQ, FCF, FFCLRP, ICB, ESALQ, BIOINFORMÁTICA
Subjects: LINFÓCITOS T, COVID-19, PULMÃO, CORONAVIRUS
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BRUNETTI, Natalia S. et al. SARS-CoV-2 uses CD4 to infect T helper lymphocytes. eLife, v. 12, p. 1-26, 2023Tradução . . Disponível em: https://doi.org/10.7554/eLife.84790. Acesso em: 15 nov. 2024.
APA
Brunetti, N. S., Silva, J. V. V. da, Veras, F. P., Tavares, L. A., Castro, Í. M. S. de, Silvestrini, V. C., et al. (2023). SARS-CoV-2 uses CD4 to infect T helper lymphocytes. eLife, 12, 1-26. doi:10.7554/eLife.84790
NLM
Brunetti NS, Silva JVV da, Veras FP, Tavares LA, Castro ÍMS de, Silvestrini VC, Oliveira EB de, Faça VM, Sposito AC, Velloso LA, Silva LLP da, Cunha TM, Nakaya HTI, Skaf MS, Vieira PMM de M, Farias A dos S. SARS-CoV-2 uses CD4 to infect T helper lymphocytes [Internet]. eLife. 2023 ; 12 1-26.[citado 2024 nov. 15 ] Available from: https://doi.org/10.7554/eLife.84790
Vancouver
Brunetti NS, Silva JVV da, Veras FP, Tavares LA, Castro ÍMS de, Silvestrini VC, Oliveira EB de, Faça VM, Sposito AC, Velloso LA, Silva LLP da, Cunha TM, Nakaya HTI, Skaf MS, Vieira PMM de M, Farias A dos S. SARS-CoV-2 uses CD4 to infect T helper lymphocytes [Internet]. eLife. 2023 ; 12 1-26.[citado 2024 nov. 15 ] Available from: https://doi.org/10.7554/eLife.84790
Artigo de periodico
Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production (2023)
Córneo, Emily S.; Veras, Flávio Protásio ; Gomes, Giovanni F.; Schneider, Ayda Henriques ; Silva, Bruna Manuella Souza; Almeida, Cícero José Luíz dos Ramos; Silva, Camila M.; Martins, Ronaldo B.; Batah, Sabrina Setembre ; Simon, Carla S.; Prestes, Gabriele da S.; Alves Filho, José Carlos Farias ; Arruda, Eurico ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de; Fabro, Alexandre Todorovic ; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Dal-Pizzol, Felipe
Source: Clinical Immunology. Unidades: FMRP, ESALQ
Subjects: COVID-19, NEUTRÓFILOS, CORONAVIRUS
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ABNT
CÓRNEO, Emily S. et al. Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production. Clinical Immunology, v. 257, p. 1-12, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.clim.2023.109836. Acesso em: 15 nov. 2024.
APA
Córneo, E. S., Veras, F. P., Gomes, G. F., Schneider, A. H., Silva, B. M. S., Almeida, C. J. L. dos R., et al. (2023). Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production. Clinical Immunology, 257, 1-12. doi:10.1016/j.clim.2023.109836
NLM
Córneo ES, Veras FP, Gomes GF, Schneider AH, Silva BMS, Almeida CJL dos R, Silva CM, Martins RB, Batah SS, Simon CS, Prestes G da S, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RDR de, Fabro AT, Cunha TM, Cunha F de Q, Dal-Pizzol F. Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production [Internet]. Clinical Immunology. 2023 ; 257 1-12.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.clim.2023.109836
Vancouver
Córneo ES, Veras FP, Gomes GF, Schneider AH, Silva BMS, Almeida CJL dos R, Silva CM, Martins RB, Batah SS, Simon CS, Prestes G da S, Alves Filho JCF, Arruda E, Louzada Júnior P, Oliveira RDR de, Fabro AT, Cunha TM, Cunha F de Q, Dal-Pizzol F. Enoxaparin improves COVID-19 by reducing Neutrophils Extracellular Traps (NETs) production [Internet]. Clinical Immunology. 2023 ; 257 1-12.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.clim.2023.109836
Artigo de periodico
Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT (2022)
Lima, Mikhael Haruo Fernandes de; Hiroki, Carlos Hiroji; Borges, Vanessa de Fátima; Cebinelli, Guilherme Cesar Martelossi ; Santos, Jessica; Rosa, Marcos Henrique; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner ; Gonçalves, Augusto Velozo; Quirino, Gustavo Fernando da Silva; Zamboni, Dario Simões ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Source: The Journal of Infectious Diseases. Unidade: FMRP
Subjects: IMUNOSSUPRESSÃO, SEPSE, MACRÓFAGOS, CITOMETRIA DE FLUXO
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LIMA, Mikhael Haruo Fernandes de et al. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, v. 225, n. 3, p. 531-541, 2022Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiab405. Acesso em: 15 nov. 2024.
APA
Lima, M. H. F. de, Hiroki, C. H., Borges, V. de F., Cebinelli, G. C. M., Santos, J., Rosa, M. H., et al. (2022). Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, 225( 3), 531-541. doi:10.1093/infdis/jiab405
NLM
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1093/infdis/jiab405
Vancouver
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1093/infdis/jiab405
Artigo de periodico
Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons (2022)
Yang, Nicole J.; Isensee, Jörg; Neel, Dylan V; Quadros, Andreza Urba de; Zhang, Han-Xiong Bear; Lauzadis, Justas; Liu, Sai Man; Shiers, Stephanie; Belu, Andreea; Cunha, Thiago Mattar
Source: Nature Neuroscience. Unidade: FMRP
Subjects: TOXINAS, BACTÉRIAS, GÂNGLIOS ESPINHAIS, CÉLULAS-TRONCO, NOCICEPTORES, DOR, PEPTÍDEOS
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YANG, Nicole J. et al. Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons. Nature Neuroscience, v. 25, n. 2, p. 168-179, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41593-021-00973-8. Acesso em: 15 nov. 2024.
APA
Yang, N. J., Isensee, J., Neel, D. V., Quadros, A. U. de, Zhang, H. -X. B., Lauzadis, J., et al. (2022). Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons. Nature Neuroscience, 25( 2), 168-179. doi:10.1038/s41593-021-00973-8
NLM
Yang NJ, Isensee J, Neel DV, Quadros AU de, Zhang H-XB, Lauzadis J, Liu SM, Shiers S, Belu A, Cunha TM. Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons [Internet]. Nature Neuroscience. 2022 ; 25( 2): 168-179.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1038/s41593-021-00973-8
Vancouver
Yang NJ, Isensee J, Neel DV, Quadros AU de, Zhang H-XB, Lauzadis J, Liu SM, Shiers S, Belu A, Cunha TM. Anthrax toxins regulate pain signaling and can deliver molecular cargoes into ANTXR2+ DRG sensory neurons [Internet]. Nature Neuroscience. 2022 ; 25( 2): 168-179.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1038/s41593-021-00973-8
Artigo de periodico
Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice (2022)
Castro, Paula A. T. S.; Barbosa, Germanna M.; Machanocker, Dafiner H.; Peres, Raphael S.; Cunha, Thiago Mattar ; Cunha, Jonathan E.; Oliveira, Francisco F. B.; Ramalho, Fernando Silva ; Russo, Thiago L.; Cunha, Fernando de Queiroz ; Salvini, Tania F.
Source: Plos one. Unidade: FMRP
Subjects: CRIOTERAPIA, INFLAMAÇÃO, ARTRITE, JOELHO
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ABNT
CASTRO, Paula A. T. S. et al. Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice. Plos one, v. 17, n. 1, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0261667. Acesso em: 15 nov. 2024.
APA
Castro, P. A. T. S., Barbosa, G. M., Machanocker, D. H., Peres, R. S., Cunha, T. M., Cunha, J. E., et al. (2022). Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice. Plos one, 17( 1), 1-14. doi:10.1371/journal.pone.0261667
NLM
Castro PATS, Barbosa GM, Machanocker DH, Peres RS, Cunha TM, Cunha JE, Oliveira FFB, Ramalho FS, Russo TL, Cunha F de Q, Salvini TF. Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice [Internet]. Plos one. 2022 ; 17( 1): 1-14.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1371/journal.pone.0261667
Vancouver
Castro PATS, Barbosa GM, Machanocker DH, Peres RS, Cunha TM, Cunha JE, Oliveira FFB, Ramalho FS, Russo TL, Cunha F de Q, Salvini TF. Clinical-like cryotherapy in acute knee arthritis of the knee improves inflammation signs, pain, joint swelling, and motor performance in mice [Internet]. Plos one. 2022 ; 17( 1): 1-14.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1371/journal.pone.0261667
Artigo de periodico
Angiotensin type 2 receptor antagonism as a new target to manage gout (2022)
Vieira, Thiago Neves; Saraiva, André L. Lopes; Guimarães, Rafaela Mano ; Luiz, João Paulo Mesquita ; Pinto, Larissa Garcia; Ávila, Veridiana de Melo Rodrigues; Goulart, Luiz Ricardo ; Cunha Júnior, Jair Pereira; McNaughton, Peter Anthony; Cunha, Thiago Mattar ; Ferreira, Juliano; Silva, Cássia Regina
Source: Inflammopharmacology. Unidade: FMRP
Subjects: DOR, FARMACOTERAPIA, ANGIOTENSINA II, ANTI-INFLAMATÓRIOS, ANTIOXIDANTES, ARTRITE, ÁCIDO ÚRICO
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ABNT
VIEIRA, Thiago Neves et al. Angiotensin type 2 receptor antagonism as a new target to manage gout. Inflammopharmacology, v. 30, n. 6, p. 2399-2410, 2022Tradução . . Disponível em: https://doi.org/10.1007/s10787-022-01076-x. Acesso em: 15 nov. 2024.
APA
Vieira, T. N., Saraiva, A. L. L., Guimarães, R. M., Luiz, J. P. M., Pinto, L. G., Ávila, V. de M. R., et al. (2022). Angiotensin type 2 receptor antagonism as a new target to manage gout. Inflammopharmacology, 30( 6), 2399-2410. doi:10.1007/s10787-022-01076-x
NLM
Vieira TN, Saraiva ALL, Guimarães RM, Luiz JPM, Pinto LG, Ávila V de MR, Goulart LR, Cunha Júnior JP, McNaughton PA, Cunha TM, Ferreira J, Silva CR. Angiotensin type 2 receptor antagonism as a new target to manage gout [Internet]. Inflammopharmacology. 2022 ; 30( 6): 2399-2410.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s10787-022-01076-x
Vancouver
Vieira TN, Saraiva ALL, Guimarães RM, Luiz JPM, Pinto LG, Ávila V de MR, Goulart LR, Cunha Júnior JP, McNaughton PA, Cunha TM, Ferreira J, Silva CR. Angiotensin type 2 receptor antagonism as a new target to manage gout [Internet]. Inflammopharmacology. 2022 ; 30( 6): 2399-2410.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1007/s10787-022-01076-x
Artigo de periodico
Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts (2022)
Charlie-Silva, Ives ; Feitosa, Natália M.; Pontes, Leticia Gomes de ; Fernandes, Bianca Helena Ventura ; Nóbrega, Rafael Henrique ; Gomes, Juliana Moreira Mendonça; Prata, Mariana N. L.; Ferraris, Fausto k; Freitas, Anderson Zanardi de ; Ribeiro, Martha Simões ; Cunha, Thiago Mattar
Source: Frontiers in Immunology. Unidades: FM, ICB, IPEN, FMRP
Subjects: PROTEÍNAS, CARRAGENINA, GLICOSAMINOGLICANAS, INFLAMAÇÃO, LEUCÓCITOS, NEUTRÓFILOS, PROTEÔMICA
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CHARLIE-SILVA, Ives et al. Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts. Frontiers in Immunology, v. 13, p. 1-21, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.1019201. Acesso em: 15 nov. 2024.
APA
Charlie-Silva, I., Feitosa, N. M., Pontes, L. G. de, Fernandes, B. H. V., Nóbrega, R. H., Gomes, J. M. M., et al. (2022). Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts. Frontiers in Immunology, 13, 1-21. doi:10.3389/fimmu.2022.1019201
NLM
Charlie-Silva I, Feitosa NM, Pontes LG de, Fernandes BHV, Nóbrega RH, Gomes JMM, Prata MNL, Ferraris F k, Freitas AZ de, Ribeiro MS, Cunha TM. Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts [Internet]. Frontiers in Immunology. 2022 ; 13 1-21.[citado 2024 nov. 15 ] Available from: https://doi.org/10.3389/fimmu.2022.1019201
Vancouver
Charlie-Silva I, Feitosa NM, Pontes LG de, Fernandes BHV, Nóbrega RH, Gomes JMM, Prata MNL, Ferraris F k, Freitas AZ de, Ribeiro MS, Cunha TM. Plasma proteome responses in zebrafish following λ-carrageenan-induced inflammation are mediated by PMN leukocytes and correlate highly with their human counterparts [Internet]. Frontiers in Immunology. 2022 ; 13 1-21.[citado 2024 nov. 15 ] Available from: https://doi.org/10.3389/fimmu.2022.1019201
Artigo de periodico
Impact of microbiota depletion by antibiotics on SARS-CoV-2 infection of K18-hACE2 mice (2022)
Rodrigues, Patrícia Brito; Gomes, Giovanni Freitas; Angelim, Monara K. S. C.; Souza, Gabriela F.; Muraro, Stéfanie Primon ; Toledo-Teixeira, Daniel A.; Rattis, Bruna Amanda da Cruz; Passos, Amanda Stephane Cruz dos; Pral, Laís Passariello ; Rodovalho, Vinícius de Rezende ; Gomes, Arilson Bernardo dos Santos P.; Matheus, Valquíria Aparecida ; Antunes, André Saraiva Leão Marcelo ; Crunfli, Fernanda ; Antunes, Krist Helen; Souza, Ana Paula Duarte de; Consonni, Sílvio Roberto ; Leiria, Luiz Osório Silveira ; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Moraes-Vieira, Pedro Manoel Mendes de ; Modena, José Luiz Proenca ; Vinolo, Marco Aurelio Ramirez
Source: Cells. Unidade: FMRP
Subjects: COVID-19, MODELOS ANIMAIS DE DOENÇAS, QUIMIOTERÁPICOS, INFECÇÕES POR CORONAVIRUS, INFECÇÕES RESPIRATÓRIAS
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ABNT
RODRIGUES, Patrícia Brito et al. Impact of microbiota depletion by antibiotics on SARS-CoV-2 infection of K18-hACE2 mice. Cells, v. 11, n. 16, p. 1-16, 2022Tradução . . Disponível em: https://doi.org/10.3390/cells11162572. Acesso em: 15 nov. 2024.
APA
Rodrigues, P. B., Gomes, G. F., Angelim, M. K. S. C., Souza, G. F., Muraro, S. P., Toledo-Teixeira, D. A., et al. (2022). Impact of microbiota depletion by antibiotics on SARS-CoV-2 infection of K18-hACE2 mice. Cells, 11( 16), 1-16. doi:10.3390/cells11162572
NLM
Rodrigues PB, Gomes GF, Angelim MKSC, Souza GF, Muraro SP, Toledo-Teixeira DA, Rattis BA da C, Passos ASC dos, Pral LP, Rodovalho V de R, Gomes AB dos SP, Matheus VA, Antunes ASLM, Crunfli F, Antunes KH, Souza APD de, Consonni SR, Leiria LOS, Alves Filho JCF, Cunha TM, Moraes-Vieira PMM de, Modena JLP, Vinolo MAR. Impact of microbiota depletion by antibiotics on SARS-CoV-2 infection of K18-hACE2 mice [Internet]. Cells. 2022 ; 11( 16): 1-16.[citado 2024 nov. 15 ] Available from: https://doi.org/10.3390/cells11162572
Vancouver
Rodrigues PB, Gomes GF, Angelim MKSC, Souza GF, Muraro SP, Toledo-Teixeira DA, Rattis BA da C, Passos ASC dos, Pral LP, Rodovalho V de R, Gomes AB dos SP, Matheus VA, Antunes ASLM, Crunfli F, Antunes KH, Souza APD de, Consonni SR, Leiria LOS, Alves Filho JCF, Cunha TM, Moraes-Vieira PMM de, Modena JLP, Vinolo MAR. Impact of microbiota depletion by antibiotics on SARS-CoV-2 infection of K18-hACE2 mice [Internet]. Cells. 2022 ; 11( 16): 1-16.[citado 2024 nov. 15 ] Available from: https://doi.org/10.3390/cells11162572
Artigo de periodico
Skin-resident dendritic cells mediate postoperative pain via CCR4 on sensory neurons (2022)
Silva, Jaqueline Raymondi; Iftinca, Mircea; Gomes, Francisco Isaac Fernandes ; Segal, Julia P.; Smith, Olivia M. A.; Bannerman, Courtney A.; Mendes, Atlante Silva ; Defaye, Manon; Robinson, Madeline E. C.; Gilron, Ian; Cunha, Thiago Mattar ; Altier, Christophe; Ghasemlou, Nader
Source: Proceedings of the National Academy of Sciences. Unidade: FMRP
Subjects: POTENCIAIS DE AÇÃO, BIOMARCADORES, QUIMIOCINAS, MODELOS ANIMAIS DE DOENÇAS, PREDISPOSIÇÃO GENÉTICA PARA DOENÇA, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
SILVA, Jaqueline Raymondi et al. Skin-resident dendritic cells mediate postoperative pain via CCR4 on sensory neurons. Proceedings of the National Academy of Sciences, v. 119, n. 4, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.1073/pnas.2118238119. Acesso em: 15 nov. 2024.
APA
Silva, J. R., Iftinca, M., Gomes, F. I. F., Segal, J. P., Smith, O. M. A., Bannerman, C. A., et al. (2022). Skin-resident dendritic cells mediate postoperative pain via CCR4 on sensory neurons. Proceedings of the National Academy of Sciences, 119( 4), 1-10. doi:10.1073/pnas.2118238119
NLM
Silva JR, Iftinca M, Gomes FIF, Segal JP, Smith OMA, Bannerman CA, Mendes AS, Defaye M, Robinson MEC, Gilron I, Cunha TM, Altier C, Ghasemlou N. Skin-resident dendritic cells mediate postoperative pain via CCR4 on sensory neurons [Internet]. Proceedings of the National Academy of Sciences. 2022 ; 119( 4): 1-10.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1073/pnas.2118238119
Vancouver
Silva JR, Iftinca M, Gomes FIF, Segal JP, Smith OMA, Bannerman CA, Mendes AS, Defaye M, Robinson MEC, Gilron I, Cunha TM, Altier C, Ghasemlou N. Skin-resident dendritic cells mediate postoperative pain via CCR4 on sensory neurons [Internet]. Proceedings of the National Academy of Sciences. 2022 ; 119( 4): 1-10.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1073/pnas.2118238119
Artigo de periodico
Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice (2022)
Maganin, Alexandre Gomes de Macedo ; Souza, Guilherme Rabelo de; Fonseca, Miriam D.; Lopes, Alexandre H.; Guimarães, Rafaela Mano ; Dagostin, André; Cecílio, Nerry T.; Mendes, Atlante Silva ; Gonçalves, William Antônio; Silva, Conceição Elidianne Aníbal; Gomes, Francisco Isaac Fernandes ; Marques, Lucas Maciel Mauriz ; Silva, Rangel L.; Arruda, Leticia M.; Santana, Denis A.; Lemos, Henrique; Huang, Lei; Ferreira, Marcela Davoli; Santana-Coelho, Danielle; Sant’Anna, Morena Brasil Martins; Kusuda, Ricardo; Talbot, Jhimmy; Pacholczyk, Gabriela; Buqui, Gabriela A.; Lopes, Norberto Peporine ; Alves Filho, José Carlos Farias ; Leão, Ricardo Maurício Xavier; O’Connor, Jason C.; Cunha, Fernando de Queiroz ; Mellor, Andrew; Cunha, Thiago Mattar
Source: Journal of Clinical Investigation. Unidades: FCFRP, FMRP, FCF
Subjects: CÉLULAS DENDRÍTICAS, METABÓLITOS, DOENÇAS DO SISTEMA NERVOSO PERIFÉRICO, NERVOS PERIFÉRICOS, DOR CRÔNICA
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ABNT
MAGANIN, Alexandre Gomes de Macedo et al. Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice. Journal of Clinical Investigation, v. 132, n. 23, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.1172/JCI153805. Acesso em: 15 nov. 2024.
APA
Maganin, A. G. de M., Souza, G. R. de, Fonseca, M. D., Lopes, A. H., Guimarães, R. M., Dagostin, A., et al. (2022). Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice. Journal of Clinical Investigation, 132( 23), 1-17. doi:10.1172/JCI153805
NLM
Maganin AG de M, Souza GR de, Fonseca MD, Lopes AH, Guimarães RM, Dagostin A, Cecílio NT, Mendes AS, Gonçalves WA, Silva CEA, Gomes FIF, Marques LMM, Silva RL, Arruda LM, Santana DA, Lemos H, Huang L, Ferreira MD, Santana-Coelho D, Sant’Anna MBM, Kusuda R, Talbot J, Pacholczyk G, Buqui GA, Lopes NP, Alves Filho JCF, Leão RMX, O’Connor JC, Cunha F de Q, Mellor A, Cunha TM. Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice [Internet]. Journal of Clinical Investigation. 2022 ; 132( 23): 1-17.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1172/JCI153805
Vancouver
Maganin AG de M, Souza GR de, Fonseca MD, Lopes AH, Guimarães RM, Dagostin A, Cecílio NT, Mendes AS, Gonçalves WA, Silva CEA, Gomes FIF, Marques LMM, Silva RL, Arruda LM, Santana DA, Lemos H, Huang L, Ferreira MD, Santana-Coelho D, Sant’Anna MBM, Kusuda R, Talbot J, Pacholczyk G, Buqui GA, Lopes NP, Alves Filho JCF, Leão RMX, O’Connor JC, Cunha F de Q, Mellor A, Cunha TM. Meningeal dendritic cells drive neuropathic pain through elevation of the kynurenine metabolic pathway in mice [Internet]. Journal of Clinical Investigation. 2022 ; 132( 23): 1-17.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1172/JCI153805
Artigo de periodico
Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition (2022)
Franchin, Marcelo ; Rosalen, Pedro Luiz ; Prado, Douglas da Silva ; Paraluppi, Murilo César; Silva, Rangel Leal ; Damasceno, Luis Eduardo Alves ; morelo, David Fernando Colon ; Napimoga, Marcelo Henrique; Cunha, Fernando de Queiroz ; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar
Source: European Journal of Pharmacology. Unidades: CENA, FMRP
Subjects: DIFERENCIAÇÃO CELULAR, ENCEFALOMIELITE AGUDA DISSEMINADA, DOENÇAS AUTOIMUNES, INFLAMAÇÃO, FARMACOTERAPIA, INTERLEUCINAS, PRÓPOLIS
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ABNT
FRANCHIN, Marcelo et al. Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition. European Journal of Pharmacology, v. 929, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2022.175127. Acesso em: 15 nov. 2024.
APA
Franchin, M., Rosalen, P. L., Prado, D. da S., Paraluppi, M. C., Silva, R. L., Damasceno, L. E. A., et al. (2022). Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition. European Journal of Pharmacology, 929, 1-8. doi:10.1016/j.ejphar.2022.175127
NLM
Franchin M, Rosalen PL, Prado D da S, Paraluppi MC, Silva RL, Damasceno LEA, morelo DFC, Napimoga MH, Cunha F de Q, Alves Filho JCF, Cunha TM. Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition [Internet]. European Journal of Pharmacology. 2022 ; 929 1-8.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.ejphar.2022.175127
Vancouver
Franchin M, Rosalen PL, Prado D da S, Paraluppi MC, Silva RL, Damasceno LEA, morelo DFC, Napimoga MH, Cunha F de Q, Alves Filho JCF, Cunha TM. Cinnamoyloxy-mammeisin, a coumarin from propolis of stingless bees, attenuates Th17 cell differentiation and autoimmune inflammation via STAT3 inhibition [Internet]. European Journal of Pharmacology. 2022 ; 929 1-8.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.ejphar.2022.175127
Artigo de periodico
Enhancing untargeted metabolomics using metadata-based source annotation (2022)
Gauglitz, Julia M.; West, Kiana A.; Bittremieux, Wout; Williams, Candace L.; Weldon, Kelly C.; Panitchpakdi, Morgan; Marques, Lucas Maciel Mauriz ; Silva, Rodrigo Moreira da ; Veras, Flávio Protásio ; Cunha, Thiago Mattar ; Oliveira, Renê Donizeti Ribeiro de; Louzada Júnior, Paulo ; Lopes, Norberto Peporine
Source: Nature Biotechnology. Unidades: FCFRP, FMRP
Subjects: METABOLÔMICA, METADADOS, ESPECTROMETRIA DE MASSAS, GENÉTICA
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ABNT
GAUGLITZ, Julia M. et al. Enhancing untargeted metabolomics using metadata-based source annotation. Nature Biotechnology, v. 40, n. 12, p. 1774-1779, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41587-022-01368-1. Acesso em: 15 nov. 2024.
APA
Gauglitz, J. M., West, K. A., Bittremieux, W., Williams, C. L., Weldon, K. C., Panitchpakdi, M., et al. (2022). Enhancing untargeted metabolomics using metadata-based source annotation. Nature Biotechnology, 40( 12), 1774-1779. doi:10.1038/s41587-022-01368-1
NLM
Gauglitz JM, West KA, Bittremieux W, Williams CL, Weldon KC, Panitchpakdi M, Marques LMM, Silva RM da, Veras FP, Cunha TM, Oliveira RDR de, Louzada Júnior P, Lopes NP. Enhancing untargeted metabolomics using metadata-based source annotation [Internet]. Nature Biotechnology. 2022 ; 40( 12): 1774-1779.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1038/s41587-022-01368-1
Vancouver
Gauglitz JM, West KA, Bittremieux W, Williams CL, Weldon KC, Panitchpakdi M, Marques LMM, Silva RM da, Veras FP, Cunha TM, Oliveira RDR de, Louzada Júnior P, Lopes NP. Enhancing untargeted metabolomics using metadata-based source annotation [Internet]. Nature Biotechnology. 2022 ; 40( 12): 1774-1779.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1038/s41587-022-01368-1
Artigo de periodico
The role of adenosine A1 receptor in the peripheral tramadol’s effect in the temporomandibular joint of rats (2021)
Abdalla, Henrique Ballassini; Napimoga, Marcelo Henrique; Maganin, Alexandre Gomes de Macedo; Lopes, Alexandre Hashimoto Pereira; Cunha, Thiago Mattar; Gill, Harvinder Singh; Napimoga, Juliana Trindade Clemente
Source: International Immunopharmacology. Unidade: FMRP
Subjects: ADENOSINA, ANALGÉSICOS, ANTI-INFLAMATÓRIOS, ARTRALGIA, MACRÓFAGOS, NOCICEPTIVIDADE
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ABNT
ABDALLA, Henrique Ballassini et al. The role of adenosine A1 receptor in the peripheral tramadol’s effect in the temporomandibular joint of rats. International Immunopharmacology, v. 97, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2021.107680. Acesso em: 15 nov. 2024.
APA
Abdalla, H. B., Napimoga, M. H., Maganin, A. G. de M., Lopes, A. H. P., Cunha, T. M., Gill, H. S., & Napimoga, J. T. C. (2021). The role of adenosine A1 receptor in the peripheral tramadol’s effect in the temporomandibular joint of rats. International Immunopharmacology, 97. doi:10.1016/j.intimp.2021.107680
NLM
Abdalla HB, Napimoga MH, Maganin AG de M, Lopes AHP, Cunha TM, Gill HS, Napimoga JTC. The role of adenosine A1 receptor in the peripheral tramadol’s effect in the temporomandibular joint of rats [Internet]. International Immunopharmacology. 2021 ; 97[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.intimp.2021.107680
Vancouver
Abdalla HB, Napimoga MH, Maganin AG de M, Lopes AHP, Cunha TM, Gill HS, Napimoga JTC. The role of adenosine A1 receptor in the peripheral tramadol’s effect in the temporomandibular joint of rats [Internet]. International Immunopharmacology. 2021 ; 97[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.intimp.2021.107680
Trabalho de evento-resumo periodico
Adult human brain slice cultures in the investigation of SARS-CoV-2 CNS infection (2021)
Almeida, Glaucia Maria ; Mendes, Niele Dias ; Santos, Marcelo Volpon ; Gondim, Guilherme Gozzoli Podolsky ; Oliveira, Ricardo Santos de ; Cunha, Thiago Mattar ; Arruda, Eurico ; Sebollela, Adriano Silva ; Neder, Luciano
Source: Journal of Neuropathology and Experimental Neurology. Conference titles: Annual Meeting of the American Association of Neuropathologists. Unidade: FMRP
Subjects: COVID-19, RECEPTORES CELULARES, DOENÇAS DO SISTEMA NERVOSO CENTRAL, BIOMARCADORES, EXPRESSÃO GÊNICA, CITOCININAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Glaucia Maria et al. Adult human brain slice cultures in the investigation of SARS-CoV-2 CNS infection. Journal of Neuropathology and Experimental Neurology. Cary: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1093/jnen/nlab042. Acesso em: 15 nov. 2024. , 2021
APA
Almeida, G. M., Mendes, N. D., Santos, M. V., Gondim, G. G. P., Oliveira, R. S. de, Cunha, T. M., et al. (2021). Adult human brain slice cultures in the investigation of SARS-CoV-2 CNS infection. Journal of Neuropathology and Experimental Neurology. Cary: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.1093/jnen/nlab042
NLM
Almeida GM, Mendes ND, Santos MV, Gondim GGP, Oliveira RS de, Cunha TM, Arruda E, Sebollela AS, Neder L. Adult human brain slice cultures in the investigation of SARS-CoV-2 CNS infection [Internet]. Journal of Neuropathology and Experimental Neurology. 2021 ; 80( 6): 586-587.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1093/jnen/nlab042
Vancouver
Almeida GM, Mendes ND, Santos MV, Gondim GGP, Oliveira RS de, Cunha TM, Arruda E, Sebollela AS, Neder L. Adult human brain slice cultures in the investigation of SARS-CoV-2 CNS infection [Internet]. Journal of Neuropathology and Experimental Neurology. 2021 ; 80( 6): 586-587.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1093/jnen/nlab042
Artigo de periodico
Chemogenetic modulation of sensory neurons reveals their regulating role in melanoma progression (2021)
Costa, Pedro A. C.; Silva, Walison N.; Prazeres, Pedro H. D. M.; Picoli, Caroline C.; Guardia, Gabriela D. A.; Costa, Alinne C.; Oliveira, Mariana A.; Guimarães, Pedro P. G.; Goncalves, Ricardo; Pinto, Mauro C. X.; Amorim, Jaime H.; Azevedo, Vasco A. C.; Resende, Rodrigo R.; Russo, Remo C.; Cunha, Thiago Mattar; Galante, Pedro Alexandre Favoretto; Mintz, Akiva; Birbrair, Alexander
Source: Acta Neuropathologica Communications. Unidades: FMRP, BIOINFORMÁTICA, FMVZ
Subjects: COMPORTAMENTO ANIMAL, BIÓPSIA, LINHAGEM CELULAR, LINFÓCITOS, MELANOMA, NEOVASCULARIZAÇÃO PATOLÓGICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Pedro A. C. et al. Chemogenetic modulation of sensory neurons reveals their regulating role in melanoma progression. Acta Neuropathologica Communications, v. 9, 2021Tradução . . Disponível em: https://doi.org/10.1186/s40478-021-01273-9. Acesso em: 15 nov. 2024.
APA
Costa, P. A. C., Silva, W. N., Prazeres, P. H. D. M., Picoli, C. C., Guardia, G. D. A., Costa, A. C., et al. (2021). Chemogenetic modulation of sensory neurons reveals their regulating role in melanoma progression. Acta Neuropathologica Communications, 9. doi:10.1186/s40478-021-01273-9
NLM
Costa PAC, Silva WN, Prazeres PHDM, Picoli CC, Guardia GDA, Costa AC, Oliveira MA, Guimarães PPG, Goncalves R, Pinto MCX, Amorim JH, Azevedo VAC, Resende RR, Russo RC, Cunha TM, Galante PAF, Mintz A, Birbrair A. Chemogenetic modulation of sensory neurons reveals their regulating role in melanoma progression [Internet]. Acta Neuropathologica Communications. 2021 ; 9[citado 2024 nov. 15 ] Available from: https://doi.org/10.1186/s40478-021-01273-9
Vancouver
Costa PAC, Silva WN, Prazeres PHDM, Picoli CC, Guardia GDA, Costa AC, Oliveira MA, Guimarães PPG, Goncalves R, Pinto MCX, Amorim JH, Azevedo VAC, Resende RR, Russo RC, Cunha TM, Galante PAF, Mintz A, Birbrair A. Chemogenetic modulation of sensory neurons reveals their regulating role in melanoma progression [Internet]. Acta Neuropathologica Communications. 2021 ; 9[citado 2024 nov. 15 ] Available from: https://doi.org/10.1186/s40478-021-01273-9
Artigo de periodico
Jararhagin, a snake venom metalloproteinase, induces mechanical hyperalgesia in mice with the neuroinflammatory contribution of spinal cord microglia and astrocytes (2021)
Ferraz, Camila R; Carvalho, Thacyana T.; Fattori, Victor; Santos, Telma Saraiva; Ribeiro, Felipe A. Pinho; Borghi, Sergio M.; Manchope, Marilia F.; Zaninelli, Tiago H.; Cunha, Thiago Mattar; Casagrande, Rúbia; Clissa, Patricia B.; Verri Júnior, Waldiceu A
Source: International Journal of Biological Macromolecules. Unidades: FMRP, BIOTECNOLOGIA
Subjects: ASTRÓCITOS, VENENOS DE ORIGEM ANIMAL, COBRAS, CITOCINAS, DOR, MEDULA ESPINHAL
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ABNT
FERRAZ, Camila R et al. Jararhagin, a snake venom metalloproteinase, induces mechanical hyperalgesia in mice with the neuroinflammatory contribution of spinal cord microglia and astrocytes. International Journal of Biological Macromolecules, v. 179, p. 610-619, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.ijbiomac.2021.02.178. Acesso em: 15 nov. 2024.
APA
Ferraz, C. R., Carvalho, T. T., Fattori, V., Santos, T. S., Ribeiro, F. A. P., Borghi, S. M., et al. (2021). Jararhagin, a snake venom metalloproteinase, induces mechanical hyperalgesia in mice with the neuroinflammatory contribution of spinal cord microglia and astrocytes. International Journal of Biological Macromolecules, 179, 610-619. doi:10.1016/j.ijbiomac.2021.02.178
NLM
Ferraz CR, Carvalho TT, Fattori V, Santos TS, Ribeiro FAP, Borghi SM, Manchope MF, Zaninelli TH, Cunha TM, Casagrande R, Clissa PB, Verri Júnior WA. Jararhagin, a snake venom metalloproteinase, induces mechanical hyperalgesia in mice with the neuroinflammatory contribution of spinal cord microglia and astrocytes [Internet]. International Journal of Biological Macromolecules. 2021 ; 179 610-619.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.ijbiomac.2021.02.178
Vancouver
Ferraz CR, Carvalho TT, Fattori V, Santos TS, Ribeiro FAP, Borghi SM, Manchope MF, Zaninelli TH, Cunha TM, Casagrande R, Clissa PB, Verri Júnior WA. Jararhagin, a snake venom metalloproteinase, induces mechanical hyperalgesia in mice with the neuroinflammatory contribution of spinal cord microglia and astrocytes [Internet]. International Journal of Biological Macromolecules. 2021 ; 179 610-619.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1016/j.ijbiomac.2021.02.178
Artigo de periodico
Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients (2021)
Rodrigues, Tamara Silva ; Sá, Keyla Santos Guedes de ; Ishimoto, Adriene Yumi; Becerra, Amanda; Oliveira, Samuel; Almeida, Leticia; Gonçalves, Augusto V.; Perucello, Debora B.; Andrade, Warrison A. ; Castro, Ricardo; Veras, Flavio P.; Toller-Kawahisa, Juliana E.; Nascimento, Daniele C.; Lima, Mikhael H. F. de; Silva, Camila M. S.; Caetite, Diego B.; Martins, Ronaldo Bragança ; Castro, Italo A.; Pontelli, Marjorie C.; Barros, Fabio C. de; Amaral, Natália B. do; Giannini, Marcela C.; Bonjorno, Letícia P.; Lopes, Maria Isabel F.; Santana, Rodrigo de Carvalho ; Vilar, Fernando C.; Auxiliadora-Martins, Maria ; Luppino-Assad, Rodrigo; Almeida, Sergio C. L. de; Oliveira, Fabiola R. de; Batah, Sabrina Setembre ; Siyuan, Li; Benatti, Maira N.; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz ; Cunha, Larissa D.; Frantz, Fabiani Gai ; Kohlsdorf, Tiana ; Fabro, Alexandre Todorovic ; Arruda, Eurico ; Oliveira, Renê D. R. de; Louzada Júnior, Paulo ; Zamboni, Dario Simões
Source: Journal of Experimental Medicine. Unidades: FMRP, FCFRP, FFCLRP
Subjects: COVID-19, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRIGUES, Tamara Silva et al. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients. Journal of Experimental Medicine, v. 218, n. 3, 2021Tradução . . Disponível em: https://doi.org/10.1084/jem.20201707. Acesso em: 15 nov. 2024.
APA
Rodrigues, T. S., Sá, K. S. G. de, Ishimoto, A. Y., Becerra, A., Oliveira, S., Almeida, L., et al. (2021). Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients. Journal of Experimental Medicine, 218( 3). doi:10.1084/jem.20201707
NLM
Rodrigues TS, Sá KSG de, Ishimoto AY, Becerra A, Oliveira S, Almeida L, Gonçalves AV, Perucello DB, Andrade WA, Castro R, Veras FP, Toller-Kawahisa JE, Nascimento DC, Lima MHF de, Silva CMS, Caetite DB, Martins RB, Castro IA, Pontelli MC, Barros FC de, Amaral NB do, Giannini MC, Bonjorno LP, Lopes MIF, Santana R de C, Vilar FC, Auxiliadora-Martins M, Luppino-Assad R, Almeida SCL de, Oliveira FR de, Batah SS, Siyuan L, Benatti MN, Cunha TM, Alves Filho JCF, Cunha F de Q, Cunha LD, Frantz FG, Kohlsdorf T, Fabro AT, Arruda E, Oliveira RDR de, Louzada Júnior P, Zamboni DS. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients [Internet]. Journal of Experimental Medicine. 2021 ; 218( 3):[citado 2024 nov. 15 ] Available from: https://doi.org/10.1084/jem.20201707
Vancouver
Rodrigues TS, Sá KSG de, Ishimoto AY, Becerra A, Oliveira S, Almeida L, Gonçalves AV, Perucello DB, Andrade WA, Castro R, Veras FP, Toller-Kawahisa JE, Nascimento DC, Lima MHF de, Silva CMS, Caetite DB, Martins RB, Castro IA, Pontelli MC, Barros FC de, Amaral NB do, Giannini MC, Bonjorno LP, Lopes MIF, Santana R de C, Vilar FC, Auxiliadora-Martins M, Luppino-Assad R, Almeida SCL de, Oliveira FR de, Batah SS, Siyuan L, Benatti MN, Cunha TM, Alves Filho JCF, Cunha F de Q, Cunha LD, Frantz FG, Kohlsdorf T, Fabro AT, Arruda E, Oliveira RDR de, Louzada Júnior P, Zamboni DS. Inflammasomes are activated in response to SARS-CoV-2 infection and are associated with COVID-19 severity in patients [Internet]. Journal of Experimental Medicine. 2021 ; 218( 3):[citado 2024 nov. 15 ] Available from: https://doi.org/10.1084/jem.20201707

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