FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
- Authors:
- Borges, Vanessa de Fátima
- Galant, Leticia Selinger
- Kanashiro, Alexandre
- Castanheira, Fernanda Vargas e Silva
- Monteiro, Valter Vinícius Silva
- Duarte, Diego Ângelo
- Rodrigues, Filipe Camargo
- Silva, Camila Meirelles de Souza
- Schneider, Ayda Henriques
- Cebinelli, Guilherme Cesar Martelossi
- Lima, Mikhael Haruo Fernandes de
- Viola, João Paulo de Biaso
- Cunha, Thiago Mattar
- Costa Neto, Claudio Miguel da
- Alves Filho, José Carlos Farias
- Pupo, André Sampaio
- Cunha, Fernando de Queiroz
- USP affiliated authors: CUNHA, THIAGO MATTAR - FMRP ; COSTA NETO, CLAUDIO MIGUEL DA - FMRP ; ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP ; RODRIGUES, FILIPE CAMARGO - FMRP ; SCHNEIDER, AYDA HENRIQUES - FMRP ; CEBINELLI, GUILHERME CESAR MARTELOSSI - FMRP
- Unidade: FMRP
- DOI: 10.1007/s00011-022-01669-w
- Subjects: NEUTRÓFILOS; SEPSE; FÁRMACOS IMUNOSSUPRESSORES
- Keywords: CXCL2; CXCR2; FK506; Neutrophil migration; Sepsis; Tacrolimus
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher place: Heidelberg
- Date published: 2023
- Source:
- Título: Inflammation Research
- ISSN: 1023-3830
- Volume/Número/Paginação/Ano: v. 72, n. 2, p.203-215, 2023
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: green
- Licença: other-oa
-
ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 28 dez. 2025. -
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w -
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2025 dez. 28 ] Available from: https://doi.org/10.1007/s00011-022-01669-w -
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2025 dez. 28 ] Available from: https://doi.org/10.1007/s00011-022-01669-w - CCR2-deficient mice are protected to sepsis by the disruption of the inflammatory monocytes emigration from the bone marrow
- Toll-like receptor 9 activation in neutrophils impairs chemotaxis and reduces sepsis outcome
- Superoxide anion-induced pain and inflammation depends on TNFα/TNFR1 signaling in mice
- The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β
- Trans-chalcone attenuates pain and inflammation in experimental acute gout arthritis in mice
- Hesperidin methylchalcone suppresses experimental gout arthritis in mice by inhibiting NF-KB activation
- Toll like receptor 9 deficiency in neutrophil improves sepsis outcome preventing down-regulation of CXCR2
- Splenectomy increases neutrophil migration but does not affect mortality in lethal sepsis
- DF2755A, a novel non-competitive allosteric inhibitor of CXCR1/2, reduces inflammatory and post-operative pain
- Pyrrolidine dithiocarbamate inhibits superoxide anion-induced pain and inflammation in the paw skin and spinal cord by targeting NF-kB and oxidative stress
Informações sobre o DOI: 10.1007/s00011-022-01669-w (Fonte: oaDOI API)
Download do texto completo
| Tipo | Nome | Link | |
|---|---|---|---|
 |
003152951.pdf |
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
