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Artigo de periodico
Global and local ancestry modulate APOE association with Alzheimer’s neuropathology and cognitive outcomes in an admixed sample (2022)
Naslavsky, Michel ; Suemoto, Claudia Kimie ; Brito, Luciano Abreu; Scliar, Marília Oliveira ; Ferretti-Rebustini, Renata Eloah de Lucena ; Rodriguez, Roberta Diehl; Leite, Renata Elaine Paraizo ; Araujo, Nathalia Matta; Borda, Victor; Tarazona-Santos, Eduardo; Jacob Filho, Wilson ; Pasqualucci, Carlos Augusto ; Nitrini, Ricardo ; Yaffe, Kristine; Zatz, Mayana ; Grinberg, Lea T.
Fonte: Molecular Psychiatry. Unidades: IB, FM, EE
Assuntos: ANCESTRAIS, DOENÇA DE ALZHEIMER, DEMÊNCIA
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ABNT
NASLAVSKY, Michel et al. Global and local ancestry modulate APOE association with Alzheimer’s neuropathology and cognitive outcomes in an admixed sample. Molecular Psychiatry, v. 27, p. 4800–4808, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41380-022-01729-x. Acesso em: 28 jun. 2025.
APA
Naslavsky, M., Suemoto, C. K., Brito, L. A., Scliar, M. O., Ferretti-Rebustini, R. E. de L., Rodriguez, R. D., et al. (2022). Global and local ancestry modulate APOE association with Alzheimer’s neuropathology and cognitive outcomes in an admixed sample. Molecular Psychiatry, 27, 4800–4808. doi:10.1038/s41380-022-01729-x
NLM
Naslavsky M, Suemoto CK, Brito LA, Scliar MO, Ferretti-Rebustini RE de L, Rodriguez RD, Leite REP, Araujo NM, Borda V, Tarazona-Santos E, Jacob Filho W, Pasqualucci CA, Nitrini R, Yaffe K, Zatz M, Grinberg LT. Global and local ancestry modulate APOE association with Alzheimer’s neuropathology and cognitive outcomes in an admixed sample [Internet]. Molecular Psychiatry. 2022 ; 27 4800–4808.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-022-01729-x
Vancouver
Naslavsky M, Suemoto CK, Brito LA, Scliar MO, Ferretti-Rebustini RE de L, Rodriguez RD, Leite REP, Araujo NM, Borda V, Tarazona-Santos E, Jacob Filho W, Pasqualucci CA, Nitrini R, Yaffe K, Zatz M, Grinberg LT. Global and local ancestry modulate APOE association with Alzheimer’s neuropathology and cognitive outcomes in an admixed sample [Internet]. Molecular Psychiatry. 2022 ; 27 4800–4808.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-022-01729-x
Artigo de periodico
Mutations in trpγ, the homologue of TRPC6 autism candidate gene, causes autism-like behavioral deficits in Drosophila (2022)
Palacios-Muñoz, Angelina ; Moreira, Danielle de Paula; Silva, Valeria ; García, Isaac E; Aboitiz, Francisco; Zarrei, Mehdi; Campos, Gabriele ; Rennie, Olivia ; Howe, Jennifer L; Anagnostou, Evdokia; Ambrozewic, Patricia; Scherer, Stephen W ; Passos-Bueno, Maria Rita ; Ewer, John
Fonte: Molecular Psychiatry. Unidade: IB
Assuntos: TRANSTORNO DO ESPECTRO AUTISTA, NEUROBIOLOGIA, MUTAÇÃO GENÉTICA
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PALACIOS-MUÑOZ, Angelina et al. Mutations in trpγ, the homologue of TRPC6 autism candidate gene, causes autism-like behavioral deficits in Drosophila. Molecular Psychiatry, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41380-022-01555-1. Acesso em: 28 jun. 2025.
APA
Palacios-Muñoz, A., Moreira, D. de P., Silva, V., García, I. E., Aboitiz, F., Zarrei, M., et al. (2022). Mutations in trpγ, the homologue of TRPC6 autism candidate gene, causes autism-like behavioral deficits in Drosophila. Molecular Psychiatry. doi:10.1038/s41380-022-01555-1
NLM
Palacios-Muñoz A, Moreira D de P, Silva V, García IE, Aboitiz F, Zarrei M, Campos G, Rennie O, Howe JL, Anagnostou E, Ambrozewic P, Scherer SW, Passos-Bueno MR, Ewer J. Mutations in trpγ, the homologue of TRPC6 autism candidate gene, causes autism-like behavioral deficits in Drosophila [Internet]. Molecular Psychiatry. 2022 ;[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-022-01555-1
Vancouver
Palacios-Muñoz A, Moreira D de P, Silva V, García IE, Aboitiz F, Zarrei M, Campos G, Rennie O, Howe JL, Anagnostou E, Ambrozewic P, Scherer SW, Passos-Bueno MR, Ewer J. Mutations in trpγ, the homologue of TRPC6 autism candidate gene, causes autism-like behavioral deficits in Drosophila [Internet]. Molecular Psychiatry. 2022 ;[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-022-01555-1
Artigo de periodico
Global DNA methylation changes in adults with attention deficit-hyperactivity disorder and its comorbidity with bipolar disorder: links with polygenic scores (2022)
Müller, Diana ; Grevet, Eugenio H. ; Silva, Natasha A. Figueira da; Bandeira, Cibele E. ; Barbosa, Eduardo; Vitola, Eduardo S. ; Charão, Mariele F. ; Linden, Rafael ; Rohde, Luis A. ; Ramos, João K. N.; Silva, Bruna S. da ; Bau, Claiton H. D. ; Rovaris, Diego Luiz
Fonte: Molecular Psychiatry. Unidade: ICB
Assuntos: FISIOLOGIA, TRANSTORNO DO DEFICIT DE ATENÇÃO COM HIPERATIVIDADE, TRANSTORNO BIPOLAR, DISTÚRBIOS PSICOLÓGICOS, HERANÇA GENÉTICA, DNA
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ABNT
MÜLLER, Diana et al. Global DNA methylation changes in adults with attention deficit-hyperactivity disorder and its comorbidity with bipolar disorder: links with polygenic scores. Molecular Psychiatry, v. 27, p. 2485–2491, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41380-022-01493-y. Acesso em: 28 jun. 2025.
APA
Müller, D., Grevet, E. H., Silva, N. A. F. da, Bandeira, C. E., Barbosa, E., Vitola, E. S., et al. (2022). Global DNA methylation changes in adults with attention deficit-hyperactivity disorder and its comorbidity with bipolar disorder: links with polygenic scores. Molecular Psychiatry, 27, 2485–2491. doi:10.1038/s41380-022-01493-y
NLM
Müller D, Grevet EH, Silva NAF da, Bandeira CE, Barbosa E, Vitola ES, Charão MF, Linden R, Rohde LA, Ramos JKN, Silva BS da, Bau CHD, Rovaris DL. Global DNA methylation changes in adults with attention deficit-hyperactivity disorder and its comorbidity with bipolar disorder: links with polygenic scores [Internet]. Molecular Psychiatry. 2022 ; 27 2485–2491.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-022-01493-y
Vancouver
Müller D, Grevet EH, Silva NAF da, Bandeira CE, Barbosa E, Vitola ES, Charão MF, Linden R, Rohde LA, Ramos JKN, Silva BS da, Bau CHD, Rovaris DL. Global DNA methylation changes in adults with attention deficit-hyperactivity disorder and its comorbidity with bipolar disorder: links with polygenic scores [Internet]. Molecular Psychiatry. 2022 ; 27 2485–2491.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-022-01493-y
Artigo de periodico
The 90 plus: longevity and COVID-19 survival (2022)
Zatz, Mayana ; Silva, Monize V. R ; Castro, Mateus V. de ; Naslavsky, Michel
Fonte: Molecular Psychiatry. Unidade: IB
Assuntos: LONGEVIDADE, ENVELHECIMENTO DA POPULAÇÃO, GENÉTICA MÉDICA, SAÚDE DO IDOSO, COVID-19
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ABNT
ZATZ, Mayana et al. The 90 plus: longevity and COVID-19 survival. Molecular Psychiatry, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41380-022-01461-6. Acesso em: 28 jun. 2025.
APA
Zatz, M., Silva, M. V. R., Castro, M. V. de, & Naslavsky, M. (2022). The 90 plus: longevity and COVID-19 survival. Molecular Psychiatry. doi:10.1038/s41380-022-01461-6
NLM
Zatz M, Silva MVR, Castro MV de, Naslavsky M. The 90 plus: longevity and COVID-19 survival [Internet]. Molecular Psychiatry. 2022 ;[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-022-01461-6
Vancouver
Zatz M, Silva MVR, Castro MV de, Naslavsky M. The 90 plus: longevity and COVID-19 survival [Internet]. Molecular Psychiatry. 2022 ;[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-022-01461-6
Artigo de periodico
ATP and spontaneous calcium oscillations control neural stem cell fate determination in Huntington’s disease: a novel approach for cell clock research (2021)
Glaser, Talita ; Shimojo, Hiromi; Ribeiro, Deidiane Elisa ; Martins, Patrícia Pereira Lopes; Beco, Renata Pereira; Kosinski, Michal; Sampaio, Vanessa Fernandes Arnaud ; Velloso, Juliana Correa ; Giacomelli, Ágatha Oliveira ; Lameu, Claudiana ; Santos, Ana Paula de Jesus; Souza, Hellio Danny Nobrega de ; Teng, Yang D; Kageyama, Ryoichiro; Ulrich, Henning
Fonte: Molecular Psychiatry. Unidade: IQ
Assuntos: EMBRIOGÊNESE, CÉLULAS-TRONCO, NEUROCIÊNCIAS
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ABNT
GLASER, Talita et al. ATP and spontaneous calcium oscillations control neural stem cell fate determination in Huntington’s disease: a novel approach for cell clock research. Molecular Psychiatry, v. 26, p. 2633–2650, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41380-020-0717-5. Acesso em: 28 jun. 2025.
APA
Glaser, T., Shimojo, H., Ribeiro, D. E., Martins, P. P. L., Beco, R. P., Kosinski, M., et al. (2021). ATP and spontaneous calcium oscillations control neural stem cell fate determination in Huntington’s disease: a novel approach for cell clock research. Molecular Psychiatry, 26, 2633–2650. doi:10.1038/s41380-020-0717-5
NLM
Glaser T, Shimojo H, Ribeiro DE, Martins PPL, Beco RP, Kosinski M, Sampaio VFA, Velloso JC, Giacomelli ÁO, Lameu C, Santos AP de J, Souza HDN de, Teng YD, Kageyama R, Ulrich H. ATP and spontaneous calcium oscillations control neural stem cell fate determination in Huntington’s disease: a novel approach for cell clock research [Internet]. Molecular Psychiatry. 2021 ; 26 2633–2650.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-020-0717-5
Vancouver
Glaser T, Shimojo H, Ribeiro DE, Martins PPL, Beco RP, Kosinski M, Sampaio VFA, Velloso JC, Giacomelli ÁO, Lameu C, Santos AP de J, Souza HDN de, Teng YD, Kageyama R, Ulrich H. ATP and spontaneous calcium oscillations control neural stem cell fate determination in Huntington’s disease: a novel approach for cell clock research [Internet]. Molecular Psychiatry. 2021 ; 26 2633–2650.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-020-0717-5
Artigo de periodico
The neuroendocrine modulation of global DNA methylation in neuropsychiatric disorders (2021)
Müller, Diana ; Grevet, Eugenio Horacio ; Silva, Bruna Santos da ; Charão, Mariele Feiffer ; Bau, Claiton H. D. ; Rovaris, Diego Luiz
Fonte: Molecular Psychiatry. Unidade: ICB
Assuntos: FISIOLOGIA, NEUROENDOCRINOLOGIA, HORMÔNIOS SEXUAIS, TRANSTORNOS MENTAIS, METILAÇÃO DE DNA, EPIGÊNESE GENÉTICA, CORTISOL
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MÜLLER, Diana et al. The neuroendocrine modulation of global DNA methylation in neuropsychiatric disorders. Molecular Psychiatry, v. 26, p. 66–69, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41380-020-00924-y. Acesso em: 28 jun. 2025.
APA
Müller, D., Grevet, E. H., Silva, B. S. da, Charão, M. F., Bau, C. H. D., & Rovaris, D. L. (2021). The neuroendocrine modulation of global DNA methylation in neuropsychiatric disorders. Molecular Psychiatry, 26, 66–69. doi:10.1038/s41380-020-00924-y
NLM
Müller D, Grevet EH, Silva BS da, Charão MF, Bau CHD, Rovaris DL. The neuroendocrine modulation of global DNA methylation in neuropsychiatric disorders [Internet]. Molecular Psychiatry. 2021 ; 26 66–69.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-020-00924-y
Vancouver
Müller D, Grevet EH, Silva BS da, Charão MF, Bau CHD, Rovaris DL. The neuroendocrine modulation of global DNA methylation in neuropsychiatric disorders [Internet]. Molecular Psychiatry. 2021 ; 26 66–69.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-020-00924-y
Artigo de periodico
Transcriptome of iPSC-derived neuronal cells reveals a module of co-expressed genes consistently associated with autism spectrum disorder (2021)
Oliveira, Karina Griesi ; Fogo, Mariana Soares; Pinto, B. G. G; Alves, A. Y; Suzuki, A. M; Morales, A. G; Ezquina, Suzana ; Sosa, Omar Júlio ; Sutton, G. J; Franze, D. Y. Sunaga; Bueno, A. P; Seabra, G; Sardinha, L; Costa, S. S; Rosenberg, Carla ; Zachi, Elaine Cristina; Sertie, A. L; Souza, Daniel Martins de ; Reis, Eduardo Moraes ; Voineagu, I; Passos-Bueno, Maria Rita
Fonte: Molecular Psychiatry. Unidades: IB, IP, IQ
Assuntos: TRANSTORNO DO ESPECTRO AUTISTA, GENES
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ABNT
OLIVEIRA, Karina Griesi et al. Transcriptome of iPSC-derived neuronal cells reveals a module of co-expressed genes consistently associated with autism spectrum disorder. Molecular Psychiatry, v. 26, p. 1589–1605, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41380-020-0669-9. Acesso em: 28 jun. 2025.
APA
Oliveira, K. G., Fogo, M. S., Pinto, B. G. G., Alves, A. Y., Suzuki, A. M., Morales, A. G., et al. (2021). Transcriptome of iPSC-derived neuronal cells reveals a module of co-expressed genes consistently associated with autism spectrum disorder. Molecular Psychiatry, 26, 1589–1605. doi:10.1038/s41380-020-0669-9
NLM
Oliveira KG, Fogo MS, Pinto BGG, Alves AY, Suzuki AM, Morales AG, Ezquina S, Sosa OJ, Sutton GJ, Franze DYS, Bueno AP, Seabra G, Sardinha L, Costa SS, Rosenberg C, Zachi EC, Sertie AL, Souza DM de, Reis EM, Voineagu I, Passos-Bueno MR. Transcriptome of iPSC-derived neuronal cells reveals a module of co-expressed genes consistently associated with autism spectrum disorder [Internet]. Molecular Psychiatry. 2021 ; 26 1589–1605.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-020-0669-9
Vancouver
Oliveira KG, Fogo MS, Pinto BGG, Alves AY, Suzuki AM, Morales AG, Ezquina S, Sosa OJ, Sutton GJ, Franze DYS, Bueno AP, Seabra G, Sardinha L, Costa SS, Rosenberg C, Zachi EC, Sertie AL, Souza DM de, Reis EM, Voineagu I, Passos-Bueno MR. Transcriptome of iPSC-derived neuronal cells reveals a module of co-expressed genes consistently associated with autism spectrum disorder [Internet]. Molecular Psychiatry. 2021 ; 26 1589–1605.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-020-0669-9
Artigo de periodico
Hyperactivation of P2X7 receptors as a culprit of COVID-19 neuropathology (2021)
Ribeiro, Deidiane Elisa ; Giacomelli, Ágatha Oliveira ; Glaser, Talita ; Sampaio, Vanessa Fernandes Arnaud ; Andrejew, Roberta ; Dieckmann, Luiz; Baranova, Juliana; Lameu, Claudiana ; Ratajczak, Mariusz Z; Ulrich, Henning
Fonte: Molecular Psychiatry. Unidade: IQ
Assuntos: COVID-19, RECEPTORES, SURTOS DE DOENÇAS
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ABNT
RIBEIRO, Deidiane Elisa et al. Hyperactivation of P2X7 receptors as a culprit of COVID-19 neuropathology. Molecular Psychiatry, v. 26, p. 1044–1059, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41380-020-00965-3. Acesso em: 28 jun. 2025.
APA
Ribeiro, D. E., Giacomelli, Á. O., Glaser, T., Sampaio, V. F. A., Andrejew, R., Dieckmann, L., et al. (2021). Hyperactivation of P2X7 receptors as a culprit of COVID-19 neuropathology. Molecular Psychiatry, 26, 1044–1059. doi:10.1038/s41380-020-00965-3
NLM
Ribeiro DE, Giacomelli ÁO, Glaser T, Sampaio VFA, Andrejew R, Dieckmann L, Baranova J, Lameu C, Ratajczak MZ, Ulrich H. Hyperactivation of P2X7 receptors as a culprit of COVID-19 neuropathology [Internet]. Molecular Psychiatry. 2021 ; 26 1044–1059.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-020-00965-3
Vancouver
Ribeiro DE, Giacomelli ÁO, Glaser T, Sampaio VFA, Andrejew R, Dieckmann L, Baranova J, Lameu C, Ratajczak MZ, Ulrich H. Hyperactivation of P2X7 receptors as a culprit of COVID-19 neuropathology [Internet]. Molecular Psychiatry. 2021 ; 26 1044–1059.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-020-00965-3
Artigo de periodico
Cognitive functioning throughout adulthood and illness stages in individuals with psychotic disorders and their unaffected siblings (2021)
Velthorst, Eva; Mollon, Josephine; Murray, Robin M.; Haan, Lieuwe de; Germeys, Inez Myin; Glahn, David C.; Arango, Celso; Ven, Els van der; Forti, Marta Di; Bernardo, Miquel; Guloksuz, Sinan; Delespaul, Philippe; Mezquida, Gisela; Amoretti, Silvia; Bobes, Julio; Saiz, Pilar A.; García-Portilla, María Paz; Santos, José Luís dos; Jiménez-López, Estela; Sanjuan, Julio; Del-Ben, Cristina Marta ; Menezes, Paulo Rossi
Fonte: Molecular Psychiatry. Unidades: FMRP, FM
Assuntos: TRANSTORNOS PSICÓTICOS, TRANSTORNOS COGNITIVOS, ESTUDOS TRANSVERSAIS, FAMÍLIA
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VELTHORST, Eva et al. Cognitive functioning throughout adulthood and illness stages in individuals with psychotic disorders and their unaffected siblings. Molecular Psychiatry, v. 26, n. 8, p. 4529–4543, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41380-020-00969-z. Acesso em: 28 jun. 2025.
APA
Velthorst, E., Mollon, J., Murray, R. M., Haan, L. de, Germeys, I. M., Glahn, D. C., et al. (2021). Cognitive functioning throughout adulthood and illness stages in individuals with psychotic disorders and their unaffected siblings. Molecular Psychiatry, 26( 8), 4529–4543. doi:10.1038/s41380-020-00969-z
NLM
Velthorst E, Mollon J, Murray RM, Haan L de, Germeys IM, Glahn DC, Arango C, Ven E van der, Forti MD, Bernardo M, Guloksuz S, Delespaul P, Mezquida G, Amoretti S, Bobes J, Saiz PA, García-Portilla MP, Santos JL dos, Jiménez-López E, Sanjuan J, Del-Ben CM, Menezes PR. Cognitive functioning throughout adulthood and illness stages in individuals with psychotic disorders and their unaffected siblings [Internet]. Molecular Psychiatry. 2021 ; 26( 8): 4529–4543.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-020-00969-z
Vancouver
Velthorst E, Mollon J, Murray RM, Haan L de, Germeys IM, Glahn DC, Arango C, Ven E van der, Forti MD, Bernardo M, Guloksuz S, Delespaul P, Mezquida G, Amoretti S, Bobes J, Saiz PA, García-Portilla MP, Santos JL dos, Jiménez-López E, Sanjuan J, Del-Ben CM, Menezes PR. Cognitive functioning throughout adulthood and illness stages in individuals with psychotic disorders and their unaffected siblings [Internet]. Molecular Psychiatry. 2021 ; 26( 8): 4529–4543.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-020-00969-z
Artigo de periodico
Purinergic modulation of pathways associated to suicidal behavior (2020)
Gonçalves, Maria Carolina Bittencourt; Velloso, Juliana Correa; Naaldijk, Yahaira; Cheffer, Arquimedes; Ulrich, Henning
Fonte: Molecular Psychiatry. Unidade: IQ
Assuntos: SUICÍDIO, NEUROBIOLOGIA
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ABNT
GONÇALVES, Maria Carolina Bittencourt et al. Purinergic modulation of pathways associated to suicidal behavior. Molecular Psychiatry, v. 25, p. 514-516, 2020Tradução . . Acesso em: 28 jun. 2025.
APA
Gonçalves, M. C. B., Velloso, J. C., Naaldijk, Y., Cheffer, A., & Ulrich, H. (2020). Purinergic modulation of pathways associated to suicidal behavior. Molecular Psychiatry, 25, 514-516.
NLM
Gonçalves MCB, Velloso JC, Naaldijk Y, Cheffer A, Ulrich H. Purinergic modulation of pathways associated to suicidal behavior. Molecular Psychiatry. 2020 ; 25 514-516.[citado 2025 jun. 28 ]
Vancouver
Gonçalves MCB, Velloso JC, Naaldijk Y, Cheffer A, Ulrich H. Purinergic modulation of pathways associated to suicidal behavior. Molecular Psychiatry. 2020 ; 25 514-516.[citado 2025 jun. 28 ]
Artigo de periodico
The pathophysiological impact of stress on the dopamine system is dependent on the state of the critical period of vulnerability (2020)
Gomes, Felipe Villela ; Zhu, Xiyu; Grace, Anthony A.
Fonte: Molecular Psychiatry. Unidade: FMRP
Assuntos: MODELOS ANIMAIS, DEPRESSÃO, ESQUIZOFRENIA, NEUROCIÊNCIAS, ESTRESSE
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ABNT
GOMES, Felipe Villela e ZHU, Xiyu e GRACE, Anthony A. The pathophysiological impact of stress on the dopamine system is dependent on the state of the critical period of vulnerability. Molecular Psychiatry, v. 25, n. 12, p. 3278-3291, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41380-019-0514-1. Acesso em: 28 jun. 2025.
APA
Gomes, F. V., Zhu, X., & Grace, A. A. (2020). The pathophysiological impact of stress on the dopamine system is dependent on the state of the critical period of vulnerability. Molecular Psychiatry, 25( 12), 3278-3291. doi:10.1038/s41380-019-0514-1
NLM
Gomes FV, Zhu X, Grace AA. The pathophysiological impact of stress on the dopamine system is dependent on the state of the critical period of vulnerability [Internet]. Molecular Psychiatry. 2020 ; 25( 12): 3278-3291.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-019-0514-1
Vancouver
Gomes FV, Zhu X, Grace AA. The pathophysiological impact of stress on the dopamine system is dependent on the state of the critical period of vulnerability [Internet]. Molecular Psychiatry. 2020 ; 25( 12): 3278-3291.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-019-0514-1
Artigo de periodico
The role of co-neurotransmitters in sleep and wake regulation (2019)
Oh, Jun ; Petersen, Cathrine ; Walsh, Christine M.; Bittencourt, Jackson Cioni ; Neylan, Thomas C. ; Grinberg, Lea T.
Fonte: Molecular Psychiatry. Unidade: ICB
Assuntos: ANATOMIA, CÉREBRO, TRONCO CEREBRAL, DIENCÉFALO, HIPOTÁLAMO, NEUROTRANSMISSORES, SONO, VIGÍLIA
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ABNT
OH, Jun et al. The role of co-neurotransmitters in sleep and wake regulation. Molecular Psychiatry, v. 24, p. 1284–1295, 2019Tradução . . Disponível em: https://doi.org/10.1038/s41380-018-0291-2. Acesso em: 28 jun. 2025.
APA
Oh, J., Petersen, C., Walsh, C. M., Bittencourt, J. C., Neylan, T. C., & Grinberg, L. T. (2019). The role of co-neurotransmitters in sleep and wake regulation. Molecular Psychiatry, 24, 1284–1295. doi:10.1038/s41380-018-0291-2
NLM
Oh J, Petersen C, Walsh CM, Bittencourt JC, Neylan TC, Grinberg LT. The role of co-neurotransmitters in sleep and wake regulation [Internet]. Molecular Psychiatry. 2019 ; 24 1284–1295.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-018-0291-2
Vancouver
Oh J, Petersen C, Walsh CM, Bittencourt JC, Neylan TC, Grinberg LT. The role of co-neurotransmitters in sleep and wake regulation [Internet]. Molecular Psychiatry. 2019 ; 24 1284–1295.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/s41380-018-0291-2
Artigo de periodico
Purinergic system in psychiatric diseases (2018)
Cheffer, Arquimedes; Castillo, Ana Regina Geciauskas Lage; Velloso, juliana da Cruz Corrêa; Gonçalves, M. C. B; Naaldijk, Yahaira; Nascimento, Isis Cristina do; Burnstock, Geoffrey; Ulrich, Henning
Fonte: Molecular Psychiatry. Unidade: IQ
Assuntos: TRANSTORNOS MENTAIS, ADENOSINA TRIFOSFATO
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ABNT
CHEFFER, Arquimedes et al. Purinergic system in psychiatric diseases. Molecular Psychiatry, v. 23, n. 1, p. 94-106, 2018Tradução . . Disponível em: https://doi.org/10.1038/mp.2017.188. Acesso em: 28 jun. 2025.
APA
Cheffer, A., Castillo, A. R. G. L., Velloso, juliana da C. C., Gonçalves, M. C. B., Naaldijk, Y., Nascimento, I. C. do, et al. (2018). Purinergic system in psychiatric diseases. Molecular Psychiatry, 23( 1), 94-106. doi:10.1038/mp.2017.188
NLM
Cheffer A, Castillo ARGL, Velloso juliana da CC, Gonçalves MCB, Naaldijk Y, Nascimento IC do, Burnstock G, Ulrich H. Purinergic system in psychiatric diseases [Internet]. Molecular Psychiatry. 2018 ; 23( 1): 94-106.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2017.188
Vancouver
Cheffer A, Castillo ARGL, Velloso juliana da CC, Gonçalves MCB, Naaldijk Y, Nascimento IC do, Burnstock G, Ulrich H. Purinergic system in psychiatric diseases [Internet]. Molecular Psychiatry. 2018 ; 23( 1): 94-106.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2017.188
Artigo de periodico
A homozygous loss-of-function mutation in inositol monophosphatase 1 (IMPA1) causes severe intellectual disability (2016)
Figueiredo, T; Melo, US ; Pessoa, ALS; Nóbrega, P. R; Kitajima, JP; Rusch, H; Vaz, F; Lucato, LT; Zatz, Mayana ; Kok, F ; Santos, S
Fonte: Molecular Psychiatry. Unidades: IB, FM
Assuntos: PESSOAS COM DEFICIÊNCIA INTELECTUAL, MUTAÇÃO GENÉTICA, QUOCIENTE DE INTELIGÊNCIA, GENÉTICA, NEUROPSIQUIATRIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FIGUEIREDO, T et al. A homozygous loss-of-function mutation in inositol monophosphatase 1 (IMPA1) causes severe intellectual disability. Molecular Psychiatry, v. 21, p. 1125-1129, 2016Tradução . . Disponível em: https://doi.org/10.1038/mp.2015.150. Acesso em: 28 jun. 2025.
APA
Figueiredo, T., Melo, U. S., Pessoa, A. L. S., Nóbrega, P. R., Kitajima, J. P., Rusch, H., et al. (2016). A homozygous loss-of-function mutation in inositol monophosphatase 1 (IMPA1) causes severe intellectual disability. Molecular Psychiatry, 21, 1125-1129. doi:10.1038/mp.2015.150
NLM
Figueiredo T, Melo US, Pessoa ALS, Nóbrega PR, Kitajima JP, Rusch H, Vaz F, Lucato LT, Zatz M, Kok F, Santos S. A homozygous loss-of-function mutation in inositol monophosphatase 1 (IMPA1) causes severe intellectual disability [Internet]. Molecular Psychiatry. 2016 ; 21 1125-1129.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2015.150
Vancouver
Figueiredo T, Melo US, Pessoa ALS, Nóbrega PR, Kitajima JP, Rusch H, Vaz F, Lucato LT, Zatz M, Kok F, Santos S. A homozygous loss-of-function mutation in inositol monophosphatase 1 (IMPA1) causes severe intellectual disability [Internet]. Molecular Psychiatry. 2016 ; 21 1125-1129.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2015.150
Artigo de periodico
Altered proliferation and networks in neural cells derived from idiopathic autistic individuals (2016)
Marchetto, Maria Carolina Nasser; Belinson, H.; Tian, Y.; Freitas, Beatriz C. G; Fu, C.; Vadodaria, Krishna C; Beltrão-Braga, Patrícia ; Trujillo, Cleber Augusto; Mendes, Ana Paula Diniz; Padmanabhan, Krishnan; Nunez, Y.; Ou, J.; Ghosh, H.; Wright, Rebecca; Brennand, Kristen J.; Pierce, Karen L.; Eichenfield, Lawrence; Pramparo, Tiziano; Eyler, Lisa T; Barnes, Cynthia Carter; Courchesne, Eric; Geschwind, Daniel H; Gage, Fred H.; Wynshaw-Boris, Anthony; Muotri, Alysson Renato
Fonte: Molecular Psychiatry. Unidade: EACH
Assuntos: AUTISMO, NEURÔNIOS, CÉLULAS-TRONCO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARCHETTO, Maria Carolina Nasser et al. Altered proliferation and networks in neural cells derived from idiopathic autistic individuals. Molecular Psychiatry, p. 1-16, 2016Tradução . . Disponível em: https://doi.org/10.1038/mp.2016.95. Acesso em: 28 jun. 2025.
APA
Marchetto, M. C. N., Belinson, H., Tian, Y., Freitas, B. C. G., Fu, C., Vadodaria, K. C., et al. (2016). Altered proliferation and networks in neural cells derived from idiopathic autistic individuals. Molecular Psychiatry, 1-16. doi:10.1038/mp.2016.95
NLM
Marchetto MCN, Belinson H, Tian Y, Freitas BCG, Fu C, Vadodaria KC, Beltrão-Braga P, Trujillo CA, Mendes APD, Padmanabhan K, Nunez Y, Ou J, Ghosh H, Wright R, Brennand KJ, Pierce KL, Eichenfield L, Pramparo T, Eyler LT, Barnes CC, Courchesne E, Geschwind DH, Gage FH, Wynshaw-Boris A, Muotri AR. Altered proliferation and networks in neural cells derived from idiopathic autistic individuals [Internet]. Molecular Psychiatry. 2016 ; 1-16.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2016.95
Vancouver
Marchetto MCN, Belinson H, Tian Y, Freitas BCG, Fu C, Vadodaria KC, Beltrão-Braga P, Trujillo CA, Mendes APD, Padmanabhan K, Nunez Y, Ou J, Ghosh H, Wright R, Brennand KJ, Pierce KL, Eichenfield L, Pramparo T, Eyler LT, Barnes CC, Courchesne E, Geschwind DH, Gage FH, Wynshaw-Boris A, Muotri AR. Altered proliferation and networks in neural cells derived from idiopathic autistic individuals [Internet]. Molecular Psychiatry. 2016 ; 1-16.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2016.95
Artigo de periodico-carta/editorial
Altered mTORC1 signaling in multipotent stem cells from nearly 25% of patients with nonsyndromic autism spectrum disorders [Letter to the Editor] (2015)
Suzuki, A. M.; Griesi-Oliveira, K.; Machado, C. de Oliveira Freitas; Vadasz, E.; Zachi, E. C.; Passos-Bueno, Maria Rita ; Sertie, A. L.
Fonte: Molecular Psychiatry. Unidade: IB
Assuntos: AUTISMO, DOENÇAS GENÉTICAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SUZUKI, A. M. et al. Altered mTORC1 signaling in multipotent stem cells from nearly 25% of patients with nonsyndromic autism spectrum disorders [Letter to the Editor]. Molecular Psychiatry. London: Instituto de Biociências, Universidade de São Paulo. Disponível em: https://doi.org/10.1038/mp.2014.175. Acesso em: 28 jun. 2025. , 2015
APA
Suzuki, A. M., Griesi-Oliveira, K., Machado, C. de O. F., Vadasz, E., Zachi, E. C., Passos-Bueno, M. R., & Sertie, A. L. (2015). Altered mTORC1 signaling in multipotent stem cells from nearly 25% of patients with nonsyndromic autism spectrum disorders [Letter to the Editor]. Molecular Psychiatry. London: Instituto de Biociências, Universidade de São Paulo. doi:10.1038/mp.2014.175
NLM
Suzuki AM, Griesi-Oliveira K, Machado C de OF, Vadasz E, Zachi EC, Passos-Bueno MR, Sertie AL. Altered mTORC1 signaling in multipotent stem cells from nearly 25% of patients with nonsyndromic autism spectrum disorders [Letter to the Editor] [Internet]. Molecular Psychiatry. 2015 ;( Ja 2015.): 1-2.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2014.175
Vancouver
Suzuki AM, Griesi-Oliveira K, Machado C de OF, Vadasz E, Zachi EC, Passos-Bueno MR, Sertie AL. Altered mTORC1 signaling in multipotent stem cells from nearly 25% of patients with nonsyndromic autism spectrum disorders [Letter to the Editor] [Internet]. Molecular Psychiatry. 2015 ;( Ja 2015.): 1-2.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2014.175
Artigo de periodico
Neurodevelopmental and neuropsychiatric disorders represent an interconnected molecular system (2014)
Cristino, A. S.; Williams, S. M.; Hawi, Z.; An, J. Y.; Bellgrove, M. A.; Schwartz, C. E.; Costa, Luciano da Fontoura ; Claudianos, C.
Fonte: Molecular Psychiatry. Unidade: IFSC
Assuntos: NEUROPSIQUIATRIA, TRANSTORNOS MENTAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CRISTINO, A. S. et al. Neurodevelopmental and neuropsychiatric disorders represent an interconnected molecular system. Molecular Psychiatry, v. 19, p. 294-301, 2014Tradução . . Disponível em: https://doi.org/10.1038/mp.2013.16. Acesso em: 28 jun. 2025.
APA
Cristino, A. S., Williams, S. M., Hawi, Z., An, J. Y., Bellgrove, M. A., Schwartz, C. E., et al. (2014). Neurodevelopmental and neuropsychiatric disorders represent an interconnected molecular system. Molecular Psychiatry, 19, 294-301. doi:10.1038/mp.2013.16
NLM
Cristino AS, Williams SM, Hawi Z, An JY, Bellgrove MA, Schwartz CE, Costa L da F, Claudianos C. Neurodevelopmental and neuropsychiatric disorders represent an interconnected molecular system [Internet]. Molecular Psychiatry. 2014 ; 19 294-301.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2013.16
Vancouver
Cristino AS, Williams SM, Hawi Z, An JY, Bellgrove MA, Schwartz CE, Costa L da F, Claudianos C. Neurodevelopmental and neuropsychiatric disorders represent an interconnected molecular system [Internet]. Molecular Psychiatry. 2014 ; 19 294-301.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2013.16
Artigo de periodico
Modeling non-syndromic autism and the impact of TRPC6 disruption in human neurons (2014)
Griesi-Oliveira, K; Acab, A; Gupta, A. R; Sunaga, D. Y; Chailangkarn, T; Nicol, X; Walker, M. F; Murdoch, J. D; Sanders, S J; Fernandez, T. V; Ji, W; Lifton, R. P; Vadsz, E; Dietrich, A; Pradhan, D; Song, H; Ming, G-I; Gu, X; Haddad, G; Marchetto, M C N; Spitzer, N; Passos-Bueno, Maria Rita ; State, M. W; Muotri, A R
Fonte: Molecular Psychiatry. Unidade: IB
Assuntos: AUTISMO, DOENÇAS GENÉTICAS, CÉLULAS-TRONCO, FENÓTIPOS, TRANSTORNOS MENTAIS, TRANSTORNOS NEUROPSICOLÓGICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GRIESI-OLIVEIRA, K et al. Modeling non-syndromic autism and the impact of TRPC6 disruption in human neurons. Molecular Psychiatry, v. No 2014, p. 1-16, 2014Tradução . . Disponível em: https://doi.org/10.1038/mp.2014.141. Acesso em: 28 jun. 2025.
APA
Griesi-Oliveira, K., Acab, A., Gupta, A. R., Sunaga, D. Y., Chailangkarn, T., Nicol, X., et al. (2014). Modeling non-syndromic autism and the impact of TRPC6 disruption in human neurons. Molecular Psychiatry, No 2014, 1-16. doi:10.1038/mp.2014.141
NLM
Griesi-Oliveira K, Acab A, Gupta AR, Sunaga DY, Chailangkarn T, Nicol X, Walker MF, Murdoch JD, Sanders SJ, Fernandez TV, Ji W, Lifton RP, Vadsz E, Dietrich A, Pradhan D, Song H, Ming G-I, Gu X, Haddad G, Marchetto MCN, Spitzer N, Passos-Bueno MR, State MW, Muotri AR. Modeling non-syndromic autism and the impact of TRPC6 disruption in human neurons [Internet]. Molecular Psychiatry. 2014 ; No 2014 1-16.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2014.141
Vancouver
Griesi-Oliveira K, Acab A, Gupta AR, Sunaga DY, Chailangkarn T, Nicol X, Walker MF, Murdoch JD, Sanders SJ, Fernandez TV, Ji W, Lifton RP, Vadsz E, Dietrich A, Pradhan D, Song H, Ming G-I, Gu X, Haddad G, Marchetto MCN, Spitzer N, Passos-Bueno MR, State MW, Muotri AR. Modeling non-syndromic autism and the impact of TRPC6 disruption in human neurons [Internet]. Molecular Psychiatry. 2014 ; No 2014 1-16.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2014.141
Artigo de periodico
Mechanisms of initiation and reversal of drug-seeking behavior induced by prenatal exposure to glucocorticoids (2012)
Rodrigues, A J; Leão, Pedro Puech; Pêgo, J M; Cardona, D; Carvalho, M M; Oliveira, M; Costa, B M; Carvalho, A F; Morgado, P; Araújo, D; Palha, A; Almeida, O F X; Sousa, N
Fonte: Molecular Psychiatry. Unidade: FM
Assuntos: ESTRESSE, EXPOSIÇÃO AMBIENTAL, ALCALOIDES, FETO (EFEITOS DE DROGAS), CÉLULAS DENDRÍTICAS, RECEPTORES DE DOPAMINA, CÉREBRO (ESTRUTURA), VULNERABILIDADE, COMPORTAMENTO SOCIAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRIGUES, A J et al. Mechanisms of initiation and reversal of drug-seeking behavior induced by prenatal exposure to glucocorticoids. Molecular Psychiatry, v. 17, n. 12, p. 1295-1305, 2012Tradução . . Disponível em: https://doi.org/10.1038/mp.2011.126. Acesso em: 28 jun. 2025.
APA
Rodrigues, A. J., Leão, P. P., Pêgo, J. M., Cardona, D., Carvalho, M. M., Oliveira, M., et al. (2012). Mechanisms of initiation and reversal of drug-seeking behavior induced by prenatal exposure to glucocorticoids. Molecular Psychiatry, 17( 12), 1295-1305. doi:10.1038/mp.2011.126
NLM
Rodrigues AJ, Leão PP, Pêgo JM, Cardona D, Carvalho MM, Oliveira M, Costa BM, Carvalho AF, Morgado P, Araújo D, Palha A, Almeida OFX, Sousa N. Mechanisms of initiation and reversal of drug-seeking behavior induced by prenatal exposure to glucocorticoids [Internet]. Molecular Psychiatry. 2012 ; 17( 12): 1295-1305.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2011.126
Vancouver
Rodrigues AJ, Leão PP, Pêgo JM, Cardona D, Carvalho MM, Oliveira M, Costa BM, Carvalho AF, Morgado P, Araújo D, Palha A, Almeida OFX, Sousa N. Mechanisms of initiation and reversal of drug-seeking behavior induced by prenatal exposure to glucocorticoids [Internet]. Molecular Psychiatry. 2012 ; 17( 12): 1295-1305.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2011.126
Artigo de periodico
Expression of 'alfa'-synuclein is increased in the hippocampus of rats with high levels of innate anxiety (2009)
Chiavegatto, Silvana ; Izidio, G. S.; Mendes-Lana, A.; Aneas, I.; Freitas, T. A.; Torrão, Andréa da Silva ; Conceição, I. M.; Britto, Luiz Roberto Giorgetti de ; Ramos, A.
Fonte: Molecular Psychiatry. Unidade: ICB
Assuntos: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHIAVEGATTO, Silvana et al. Expression of 'alfa'-synuclein is increased in the hippocampus of rats with high levels of innate anxiety. Molecular Psychiatry, v. 14, n. 9, p. 894-905, 2009Tradução . . Disponível em: https://doi.org/10.1038/mp.2008.43. Acesso em: 28 jun. 2025.
APA
Chiavegatto, S., Izidio, G. S., Mendes-Lana, A., Aneas, I., Freitas, T. A., Torrão, A. da S., et al. (2009). Expression of 'alfa'-synuclein is increased in the hippocampus of rats with high levels of innate anxiety. Molecular Psychiatry, 14( 9), 894-905. doi:10.1038/mp.2008.43
NLM
Chiavegatto S, Izidio GS, Mendes-Lana A, Aneas I, Freitas TA, Torrão A da S, Conceição IM, Britto LRG de, Ramos A. Expression of 'alfa'-synuclein is increased in the hippocampus of rats with high levels of innate anxiety [Internet]. Molecular Psychiatry. 2009 ; 14( 9): 894-905.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2008.43
Vancouver
Chiavegatto S, Izidio GS, Mendes-Lana A, Aneas I, Freitas TA, Torrão A da S, Conceição IM, Britto LRG de, Ramos A. Expression of 'alfa'-synuclein is increased in the hippocampus of rats with high levels of innate anxiety [Internet]. Molecular Psychiatry. 2009 ; 14( 9): 894-905.[citado 2025 jun. 28 ] Available from: https://doi.org/10.1038/mp.2008.43

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