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Artigo de periodico
Impact of sepsis on bone marrow mesenchymal stem cells and its implications for hematopoiesis and immunosuppression (2025)
Melo, Thatiana Correa de; Ariga, Suely Kunimi Kubo; Lima, Thais Martins de; Levy, Debora; Bydlowski, Sérgio Paulo ; Soriano, Francisco Garcia
Source: Inflammation Research. Unidade: FM
Subjects: CÉLULAS ESTROMAIS, CÉLULAS DA MEDULA ÓSSEA, HEMATOPOESE, SEPSE
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ABNT
MELO, Thatiana Correa de et al. Impact of sepsis on bone marrow mesenchymal stem cells and its implications for hematopoiesis and immunosuppression. Inflammation Research, v. 74, n. 1, 2025Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/89086. Acesso em: 17 abr. 2026.
APA
Melo, T. C. de, Ariga, S. K. K., Lima, T. M. de, Levy, D., Bydlowski, S. P., & Soriano, F. G. (2025). Impact of sepsis on bone marrow mesenchymal stem cells and its implications for hematopoiesis and immunosuppression. Inflammation Research, 74( 1). doi:10.1007/s00011-025-02083-8
NLM
Melo TC de, Ariga SKK, Lima TM de, Levy D, Bydlowski SP, Soriano FG. Impact of sepsis on bone marrow mesenchymal stem cells and its implications for hematopoiesis and immunosuppression [Internet]. Inflammation Research. 2025 ; 74( 1):[citado 2026 abr. 17 ] Available from: https://observatorio.fm.usp.br/handle/OPI/89086
Vancouver
Melo TC de, Ariga SKK, Lima TM de, Levy D, Bydlowski SP, Soriano FG. Impact of sepsis on bone marrow mesenchymal stem cells and its implications for hematopoiesis and immunosuppression [Internet]. Inflammation Research. 2025 ; 74( 1):[citado 2026 abr. 17 ] Available from: https://observatorio.fm.usp.br/handle/OPI/89086
Artigo de periodico
Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients (2023)
Amaral, Natália Brasil do; Rodrigues, Tamara Silva; Giannini, Marcela Cavichioli; Lopes, Maria Isabel Fernandes; Bonjorno, Letícia Pastorelli; Menezes, Pamella Indira da Silva Oliveira; Dib, Saulo Musse; Gigante, Samara Libich Gusmão; Benatti, Maíra Nilson; Rezek, Uebe C.; Emrich-Filho, Laerte L.; Sousa, Betania A. A.; Almeida, Sérgio Couto Luna de; Luppino-Assad, Rodrigo; Veras, Flávio Protásio ; Schneider, Ayda Henriques; Leiria, Luiz Osório Silveira ; Cunha, Larissa Dias da; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Arruda Neto, Eurico de; Miranda, Carlos Henrique ; Pazin Filho, Antônio ; Martins, Maria Auxiliadora ; Borges, Marcos de Carvalho ; Fonseca, Benedito Antonio Lopes da ; Bollela, Valdes Roberto ; Del-Ben, Cristina Marta ; Cunha, Fernando de Queiroz ; Santana, Rodrigo de Carvalho ; Vilar, Fernando Crivelenti; Zamboni, Dario Simões ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de
Source: Inflammation Research. Unidade: FMRP
Subjects: COVID-19, CITOCINAS, PROTEÍNAS, ALCALOIDES
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ABNT
AMARAL, Natália Brasil do et al. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients. Inflammation Research, v. 72, n. 5, p. 895-899, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-023-01718-y. Acesso em: 17 abr. 2026.
APA
Amaral, N. B. do, Rodrigues, T. S., Giannini, M. C., Lopes, M. I. F., Bonjorno, L. P., Menezes, P. I. da S. O., et al. (2023). Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients. Inflammation Research, 72( 5), 895-899. doi:10.1007/s00011-023-01718-y
NLM
Amaral NB do, Rodrigues TS, Giannini MC, Lopes MIF, Bonjorno LP, Menezes PI da SO, Dib SM, Gigante SLG, Benatti MN, Rezek UC, Emrich-Filho LL, Sousa BAA, Almeida SCL de, Luppino-Assad R, Veras FP, Schneider AH, Leiria LOS, Cunha LD da, Alves Filho JCF, Cunha TM, Arruda Neto E de, Miranda CH, Pazin Filho A, Martins MA, Borges M de C, Fonseca BAL da, Bollela VR, Del-Ben CM, Cunha F de Q, Santana R de C, Vilar FC, Zamboni DS, Louzada Júnior P, Oliveira RDR de. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients [Internet]. Inflammation Research. 2023 ; 72( 5): 895-899.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-023-01718-y
Vancouver
Amaral NB do, Rodrigues TS, Giannini MC, Lopes MIF, Bonjorno LP, Menezes PI da SO, Dib SM, Gigante SLG, Benatti MN, Rezek UC, Emrich-Filho LL, Sousa BAA, Almeida SCL de, Luppino-Assad R, Veras FP, Schneider AH, Leiria LOS, Cunha LD da, Alves Filho JCF, Cunha TM, Arruda Neto E de, Miranda CH, Pazin Filho A, Martins MA, Borges M de C, Fonseca BAL da, Bollela VR, Del-Ben CM, Cunha F de Q, Santana R de C, Vilar FC, Zamboni DS, Louzada Júnior P, Oliveira RDR de. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients [Internet]. Inflammation Research. 2023 ; 72( 5): 895-899.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-023-01718-y
Artigo de periodico
FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility (2023)
Borges, Vanessa de Fátima; Galant, Leticia Selinger; Kanashiro, Alexandre ; Castanheira, Fernanda Vargas e Silva; Monteiro, Valter Vinícius Silva; Duarte, Diego Ângelo; Rodrigues, Filipe Camargo; Silva, Camila Meirelles de Souza; Schneider, Ayda Henriques ; Cebinelli, Guilherme Cesar Martelossi ; Lima, Mikhael Haruo Fernandes de; Viola, João Paulo de Biaso; Cunha, Thiago Mattar ; Costa Neto, Claudio Miguel da ; Alves Filho, José Carlos Farias ; Pupo, André Sampaio; Cunha, Fernando de Queiroz
Source: Inflammation Research. Unidade: FMRP
Subjects: NEUTRÓFILOS, SEPSE, FÁRMACOS IMUNOSSUPRESSORES
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ABNT
BORGES, Vanessa de Fátima et al. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, v. 72, n. 2, p. 203-215, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01669-w. Acesso em: 17 abr. 2026.
APA
Borges, V. de F., Galant, L. S., Kanashiro, A., Castanheira, F. V. e S., Monteiro, V. V. S., Duarte, D. Â., et al. (2023). FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility. Inflammation Research, 72( 2), 203-215. doi:10.1007/s00011-022-01669-w
NLM
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Vancouver
Borges V de F, Galant LS, Kanashiro A, Castanheira FV e S, Monteiro VVS, Duarte DÂ, Rodrigues FC, Silva CM de S, Schneider AH, Cebinelli GCM, Lima MHF de, Viola JP de B, Cunha TM, Costa Neto CM da, Alves Filho JCF, Pupo AS, Cunha F de Q. FK506 impairs neutrophil migration that results in increased polymicrobial sepsis susceptibility [Internet]. Inflammation Research. 2023 ; 72( 2): 203-215.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-022-01669-w
Artigo de periodico
The inflammasomes: crosstalk between innate immunity and hematology (2022)
Dutra, Valéria de Freitas ; Leal, Vinicius Nunes Cordeiro ; Pontillo, Alessandra
Source: Inflammation Research. Unidade: ICB
Subjects: IMUNOLOGIA, CITOCINAS, CÉLULAS MORTAS, IMUNIDADE, IMUNOSSUPRESSÃO, DOENÇAS HEMATOLÓGICAS, CÉLULAS CULTIVADAS DE TUMOR, CÉLULAS SANGUÍNEAS, NEOPLASIAS, PROLIFERAÇÃO CELULAR
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ABNT
DUTRA, Valéria de Freitas e LEAL, Vinicius Nunes Cordeiro e PONTILLO, Alessandra. The inflammasomes: crosstalk between innate immunity and hematology. Inflammation Research, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01646-3. Acesso em: 17 abr. 2026.
APA
Dutra, V. de F., Leal, V. N. C., & Pontillo, A. (2022). The inflammasomes: crosstalk between innate immunity and hematology. Inflammation Research, 1-14. doi:10.1007/s00011-022-01646-3
NLM
Dutra V de F, Leal VNC, Pontillo A. The inflammasomes: crosstalk between innate immunity and hematology [Internet]. Inflammation Research. 2022 ; 1-14.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-022-01646-3
Vancouver
Dutra V de F, Leal VNC, Pontillo A. The inflammasomes: crosstalk between innate immunity and hematology [Internet]. Inflammation Research. 2022 ; 1-14.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-022-01646-3
Artigo de periodico
PI3Kγ controls IL-17A expression and attenuates alveolar bone loss in an experimental periodontitis model (2022)
Franchin, Marcelo ; Taira, Thaise Mayumi ; Prado, Douglas da Silva ; Hernandez, Cesar Augusto Speck; Andrade, Fabio Bonifácio de ; Abdalla, Henrique Ballassini ; Napimoga, Marcelo H ; Cunha, Thiago Mattar ; Alves, Sandra Yasuyo Fukada ; Rosalen, Pedro Luiz
Source: Inflammation Research. Unidades: CENA, FMRP, FCFRP, FORP
Subjects: GENGIVA, INTERLEUCINAS, PERIODONTITE, FARMACOTERAPIA, ANIMAIS DE LABORATÓRIO
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ABNT
FRANCHIN, Marcelo et al. PI3Kγ controls IL-17A expression and attenuates alveolar bone loss in an experimental periodontitis model. Inflammation Research, v. 72, p. 107-114, 2022Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01662-3. Acesso em: 17 abr. 2026.
APA
Franchin, M., Taira, T. M., Prado, D. da S., Hernandez, C. A. S., Andrade, F. B. de, Abdalla, H. B., et al. (2022). PI3Kγ controls IL-17A expression and attenuates alveolar bone loss in an experimental periodontitis model. Inflammation Research, 72, 107-114. doi:10.1007/s00011-022-01662-3
NLM
Franchin M, Taira TM, Prado D da S, Hernandez CAS, Andrade FB de, Abdalla HB, Napimoga MH, Cunha TM, Alves SYF, Rosalen PL. PI3Kγ controls IL-17A expression and attenuates alveolar bone loss in an experimental periodontitis model [Internet]. Inflammation Research. 2022 ; 72 107-114.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-022-01662-3
Vancouver
Franchin M, Taira TM, Prado D da S, Hernandez CAS, Andrade FB de, Abdalla HB, Napimoga MH, Cunha TM, Alves SYF, Rosalen PL. PI3Kγ controls IL-17A expression and attenuates alveolar bone loss in an experimental periodontitis model [Internet]. Inflammation Research. 2022 ; 72 107-114.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-022-01662-3
Artigo de periodico
CD14 signaling mediates lung immunopathology and mice mortality induced by Achromobacter xylosoxidans (2022)
Oliveira, Jefferson Elias ; Prado, Morgana Kelly Borges; Souza, Camila Oliveira Silva; Pastore, Marcella Roverato; Ramos, Simone Gusmão; Darini, Ana Lúcia da Costa ; Gardinassi, Luiz Gustavo Araujo ; Faccioli, Lucia Helena
Source: Inflammation Research. Unidades: FMRP, FCFRP
Subjects: INFECÇÕES BACTERIANAS GRAM-NEGATIVAS, LIPOPOLISSACARÍDEOS, PULMÃO, PNEUMONIA, NECROSE
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ABNT
OLIVEIRA, Jefferson Elias et al. CD14 signaling mediates lung immunopathology and mice mortality induced by Achromobacter xylosoxidans. Inflammation Research, v. 71, n. 12, p. 1535–1546, 2022Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01641-8. Acesso em: 17 abr. 2026.
APA
Oliveira, J. E., Prado, M. K. B., Souza, C. O. S., Pastore, M. R., Ramos, S. G., Darini, A. L. da C., et al. (2022). CD14 signaling mediates lung immunopathology and mice mortality induced by Achromobacter xylosoxidans. Inflammation Research, 71( 12), 1535–1546. doi:10.1007/s00011-022-01641-8
NLM
Oliveira JE, Prado MKB, Souza COS, Pastore MR, Ramos SG, Darini AL da C, Gardinassi LGA, Faccioli LH. CD14 signaling mediates lung immunopathology and mice mortality induced by Achromobacter xylosoxidans [Internet]. Inflammation Research. 2022 ; 71( 12): 1535–1546.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-022-01641-8
Vancouver
Oliveira JE, Prado MKB, Souza COS, Pastore MR, Ramos SG, Darini AL da C, Gardinassi LGA, Faccioli LH. CD14 signaling mediates lung immunopathology and mice mortality induced by Achromobacter xylosoxidans [Internet]. Inflammation Research. 2022 ; 71( 12): 1535–1546.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-022-01641-8
Artigo de periodico
A common variant close to the “tripwire” linker region of NLRP1 contributes to severe COVID-19 (2022)
Leal, Vinicius Nunes Cordeiro ; Cambui, Raylane Adrielle Gonçalves ; Zupelli, Thiago Gameiro ; Yamada, Suemy Melim ; Oliveira, Leonardo Antonio Teixeira de ; Andrade, Milena Mary de Souza ; Teixeira, Franciane Mouradian Emídio ; Custódio, Ricardo Wesley Alberca ; Silva, Sarah Cristina Gozzi e ; Yendo, Tatiana Mina ; Netto, Lucas Chaves ; Duarte, Alberto José da Silva; Sato, Maria Notomi ; Pontillo, Alessandra
Source: Inflammation Research. Unidades: FM, ICB
Subjects: IMUNOLOGIA, ESTUDOS DE COORTES, VARIAÇÃO GENÉTICA, COVID-19, FATORES DE RISCO, NUCLEOTÍDEOS, CITOCINAS, INFLAMAÇÃO, ENZIMAS PROTEOLÍTICAS
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ABNT
LEAL, Vinicius Nunes Cordeiro et al. A common variant close to the “tripwire” linker region of NLRP1 contributes to severe COVID-19. Inflammation Research, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01670-3. Acesso em: 17 abr. 2026.
APA
Leal, V. N. C., Cambui, R. A. G., Zupelli, T. G., Yamada, S. M., Oliveira, L. A. T. de, Andrade, M. M. de S., et al. (2022). A common variant close to the “tripwire” linker region of NLRP1 contributes to severe COVID-19. Inflammation Research, 1-8. doi:10.1007/s00011-022-01670-3
NLM
Leal VNC, Cambui RAG, Zupelli TG, Yamada SM, Oliveira LAT de, Andrade MM de S, Teixeira FME, Custódio RWA, Silva SCG e, Yendo TM, Netto LC, Duarte AJ da S, Sato MN, Pontillo A. A common variant close to the “tripwire” linker region of NLRP1 contributes to severe COVID-19 [Internet]. Inflammation Research. 2022 ; 1-8.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-022-01670-3
Vancouver
Leal VNC, Cambui RAG, Zupelli TG, Yamada SM, Oliveira LAT de, Andrade MM de S, Teixeira FME, Custódio RWA, Silva SCG e, Yendo TM, Netto LC, Duarte AJ da S, Sato MN, Pontillo A. A common variant close to the “tripwire” linker region of NLRP1 contributes to severe COVID-19 [Internet]. Inflammation Research. 2022 ; 1-8.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-022-01670-3
Artigo de periodico
Notch signaling pathway in infectious diseases: role in the regulation of immune response (2021)
Castro, Ricardo Cardoso; Gonçales, Relber Aguiar; Zambuzi, Fabiana Albani; Frantz, Fabiani Gai
Source: Inflammation Research. Unidades: FCFRP, FMRP
Subjects: DOENÇAS TRANSMISSÍVEIS, RECEPTORES, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
CASTRO, Ricardo Cardoso et al. Notch signaling pathway in infectious diseases: role in the regulation of immune response. Inflammation Research, v. 70, n. 3, p. 261-274, 2021Tradução . . Disponível em: https://doi.org/10.1007/s00011-021-01442-5. Acesso em: 17 abr. 2026.
APA
Castro, R. C., Gonçales, R. A., Zambuzi, F. A., & Frantz, F. G. (2021). Notch signaling pathway in infectious diseases: role in the regulation of immune response. Inflammation Research, 70( 3), 261-274. doi:10.1007/s00011-021-01442-5
NLM
Castro RC, Gonçales RA, Zambuzi FA, Frantz FG. Notch signaling pathway in infectious diseases: role in the regulation of immune response [Internet]. Inflammation Research. 2021 ; 70( 3): 261-274.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-021-01442-5
Vancouver
Castro RC, Gonçales RA, Zambuzi FA, Frantz FG. Notch signaling pathway in infectious diseases: role in the regulation of immune response [Internet]. Inflammation Research. 2021 ; 70( 3): 261-274.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-021-01442-5
Artigo de periodico
Immunomodulatory activity of hyaluronidase is associated with metabolic adaptations during acute inflammation (2020)
Pereira, Priscilla Aparecida Tártari; Bitencourt, Claudia Silva; Reis, Mouzarllem Barros dos ; Frantz, Fabiani Gai ; Sorgi, Carlos Arterio ; Souza, Camila O. S.; Silva, Célio Lopes ; Gardinassi, Luiz Gustavo Araujo ; Faccioli, Lucia Helena
Source: Inflammation Research. Unidades: FCFRP, FMRP, FFCLRP
Subjects: ENZIMAS, INFLAMAÇÃO, METABOLÔMICA
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ABNT
PEREIRA, Priscilla Aparecida Tártari et al. Immunomodulatory activity of hyaluronidase is associated with metabolic adaptations during acute inflammation. Inflammation Research, v. 69, n. 1, p. 105-113, 2020Tradução . . Disponível em: https://doi.org/10.1007/s00011-019-01297-x. Acesso em: 17 abr. 2026.
APA
Pereira, P. A. T., Bitencourt, C. S., Reis, M. B. dos, Frantz, F. G., Sorgi, C. A., Souza, C. O. S., et al. (2020). Immunomodulatory activity of hyaluronidase is associated with metabolic adaptations during acute inflammation. Inflammation Research, 69( 1), 105-113. doi:10.1007/s00011-019-01297-x
NLM
Pereira PAT, Bitencourt CS, Reis MB dos, Frantz FG, Sorgi CA, Souza COS, Silva CL, Gardinassi LGA, Faccioli LH. Immunomodulatory activity of hyaluronidase is associated with metabolic adaptations during acute inflammation [Internet]. Inflammation Research. 2020 ; 69( 1): 105-113.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-019-01297-x
Vancouver
Pereira PAT, Bitencourt CS, Reis MB dos, Frantz FG, Sorgi CA, Souza COS, Silva CL, Gardinassi LGA, Faccioli LH. Immunomodulatory activity of hyaluronidase is associated with metabolic adaptations during acute inflammation [Internet]. Inflammation Research. 2020 ; 69( 1): 105-113.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-019-01297-x
Artigo de periodico
Thirty days after anterior cruciate ligament transection is sufficient to induce signs of knee osteoarthritis in rats: pain, functional impairment, and synovial inflammation (2020)
Barbosa, Germanna Medeiros; Cunha, Jonathan Emanuel; Russo, Thiago L.; Cunha, Thiago Mattar; Castro, Paula Aiello Tomé de Souza; Oliveira, Francisco Fábio Bezerra; Cunha, Fernando de Queiroz ; Ramalho, Fernando Silva ; Salvini, Tânia Fátima
Source: Inflammation Research. Unidade: FMRP
Subjects: INFLAMAÇÃO, FERIMENTOS E LESÕES, JOELHO, DOENÇAS MUSCULOSQUELÉTICAS, SINOVITE ANIMAL, OSTEOARTRITE DO JOELHO, ARTICULAÇÃO DO JOELHO
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ABNT
BARBOSA, Germanna Medeiros et al. Thirty days after anterior cruciate ligament transection is sufficient to induce signs of knee osteoarthritis in rats: pain, functional impairment, and synovial inflammation. Inflammation Research, v. 69, n. 3, p. 279-288, 2020Tradução . . Disponível em: https://doi.org/10.1007/s00011-020-01317-1. Acesso em: 17 abr. 2026.
APA
Barbosa, G. M., Cunha, J. E., Russo, T. L., Cunha, T. M., Castro, P. A. T. de S., Oliveira, F. F. B., et al. (2020). Thirty days after anterior cruciate ligament transection is sufficient to induce signs of knee osteoarthritis in rats: pain, functional impairment, and synovial inflammation. Inflammation Research, 69( 3), 279-288. doi:10.1007/s00011-020-01317-1
NLM
Barbosa GM, Cunha JE, Russo TL, Cunha TM, Castro PAT de S, Oliveira FFB, Cunha F de Q, Ramalho FS, Salvini TF. Thirty days after anterior cruciate ligament transection is sufficient to induce signs of knee osteoarthritis in rats: pain, functional impairment, and synovial inflammation [Internet]. Inflammation Research. 2020 ; 69( 3): 279-288.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-020-01317-1
Vancouver
Barbosa GM, Cunha JE, Russo TL, Cunha TM, Castro PAT de S, Oliveira FFB, Cunha F de Q, Ramalho FS, Salvini TF. Thirty days after anterior cruciate ligament transection is sufficient to induce signs of knee osteoarthritis in rats: pain, functional impairment, and synovial inflammation [Internet]. Inflammation Research. 2020 ; 69( 3): 279-288.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-020-01317-1
Artigo de periodico
Methyl gallate attenuates inflammation induced by Toll-like receptor ligands by inhibiting MAPK and NF-Κb signaling pathways (2020)
Correa, Luana Barbosa; Seito, Leonardo Noboru; Manchope, Marilia F.; Verri Júnior, Waldiceu Aparecido; Cunha, Thiago Mattar; Henriques, Maria G.; Rosas, Elaine Cruz
Source: Inflammation Research. Unidade: FMRP
Subjects: ANTI-INFLAMATÓRIOS NÃO ESTEROIDES, USOS TERAPÊUTICOS, CITOCINAS, INFLAMAÇÃO, FARMACOTERAPIA, PROTEÍNAS QUINASES, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
CORREA, Luana Barbosa et al. Methyl gallate attenuates inflammation induced by Toll-like receptor ligands by inhibiting MAPK and NF-Κb signaling pathways. Inflammation Research, v. 69, n. 12, p. 1257-1270, 2020Tradução . . Disponível em: https://doi.org/10.1007/s00011-020-01407-0. Acesso em: 17 abr. 2026.
APA
Correa, L. B., Seito, L. N., Manchope, M. F., Verri Júnior, W. A., Cunha, T. M., Henriques, M. G., & Rosas, E. C. (2020). Methyl gallate attenuates inflammation induced by Toll-like receptor ligands by inhibiting MAPK and NF-Κb signaling pathways. Inflammation Research, 69( 12), 1257-1270. doi:10.1007/s00011-020-01407-0
NLM
Correa LB, Seito LN, Manchope MF, Verri Júnior WA, Cunha TM, Henriques MG, Rosas EC. Methyl gallate attenuates inflammation induced by Toll-like receptor ligands by inhibiting MAPK and NF-Κb signaling pathways [Internet]. Inflammation Research. 2020 ; 69( 12): 1257-1270.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-020-01407-0
Vancouver
Correa LB, Seito LN, Manchope MF, Verri Júnior WA, Cunha TM, Henriques MG, Rosas EC. Methyl gallate attenuates inflammation induced by Toll-like receptor ligands by inhibiting MAPK and NF-Κb signaling pathways [Internet]. Inflammation Research. 2020 ; 69( 12): 1257-1270.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-020-01407-0
Artigo de periodico
IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis (2020)
Fattori, Victor; Staurengo-Ferrari, Larissa; Zaninelli, Tiago H.; Casagrande, Rúbia; Oliveira, Renê Donizeti Ribeiro de; Louzada Júnior, Paulo; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias ; Teixeira, Mauro Martins; Cunha, Fernando de Queiroz ; Amaral, Flávio Almeida; Verri Júnior, Waldiceu Aparecido
Source: Inflammation Research. Unidade: FMRP
Subjects: ARTRITE, INFLAMAÇÃO, INTERLEUCINA 1, MACRÓFAGOS, DOR, PEROXIDASE, MEMBRANA SINOVIAL, ÁCIDO ÚRICO
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ABNT
FATTORI, Victor et al. IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis. Inflammation Research, v. 69, n. 12, p. 1271-1282, 2020Tradução . . Disponível em: https://doi.org/10.1007/s00011-020-01399-x. Acesso em: 17 abr. 2026.
APA
Fattori, V., Staurengo-Ferrari, L., Zaninelli, T. H., Casagrande, R., Oliveira, R. D. R. de, Louzada Júnior, P., et al. (2020). IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis. Inflammation Research, 69( 12), 1271-1282. doi:10.1007/s00011-020-01399-x
NLM
Fattori V, Staurengo-Ferrari L, Zaninelli TH, Casagrande R, Oliveira RDR de, Louzada Júnior P, Cunha TM, Alves Filho JCF, Teixeira MM, Cunha F de Q, Amaral FA, Verri Júnior WA. IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis [Internet]. Inflammation Research. 2020 ; 69( 12): 1271-1282.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-020-01399-x
Vancouver
Fattori V, Staurengo-Ferrari L, Zaninelli TH, Casagrande R, Oliveira RDR de, Louzada Júnior P, Cunha TM, Alves Filho JCF, Teixeira MM, Cunha F de Q, Amaral FA, Verri Júnior WA. IL-33 enhances macrophage release of IL-1β and promotes pain and inflammation in gouty arthritis [Internet]. Inflammation Research. 2020 ; 69( 12): 1271-1282.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-020-01399-x
Artigo de periodico
Fructose 1,6-bisphosphate inhibits osteoclastogenesis by attenuating RANKL-induced NF-κB/NFATc-1 (2019)
Buitrago, Liseth Yamile Wilches; Viacava, Paula Ramos; Cunha, Fernando de Queiroz ; Alves Filho, José Carlos Farias ; Fukada, Sandra Yasuyo
Source: Inflammation Research. Unidades: FMRP, FCFRP
Subjects: REMODELAÇÃO ÓSSEA, OSTEOCLASTO, FRUTOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BUITRAGO, Liseth Yamile Wilches et al. Fructose 1,6-bisphosphate inhibits osteoclastogenesis by attenuating RANKL-induced NF-κB/NFATc-1. Inflammation Research, v. 68, n. 5, p. 415-421, 2019Tradução . . Disponível em: https://doi.org/10.1007/s00011-019-01228-w. Acesso em: 17 abr. 2026.
APA
Buitrago, L. Y. W., Viacava, P. R., Cunha, F. de Q., Alves Filho, J. C. F., & Fukada, S. Y. (2019). Fructose 1,6-bisphosphate inhibits osteoclastogenesis by attenuating RANKL-induced NF-κB/NFATc-1. Inflammation Research, 68( 5), 415-421. doi:10.1007/s00011-019-01228-w
NLM
Buitrago LYW, Viacava PR, Cunha F de Q, Alves Filho JCF, Fukada SY. Fructose 1,6-bisphosphate inhibits osteoclastogenesis by attenuating RANKL-induced NF-κB/NFATc-1 [Internet]. Inflammation Research. 2019 ; 68( 5): 415-421.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-019-01228-w
Vancouver
Buitrago LYW, Viacava PR, Cunha F de Q, Alves Filho JCF, Fukada SY. Fructose 1,6-bisphosphate inhibits osteoclastogenesis by attenuating RANKL-induced NF-κB/NFATc-1 [Internet]. Inflammation Research. 2019 ; 68( 5): 415-421.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-019-01228-w
Artigo de periodico
Immunomodulating action of the 3-phenylcoumarin derivative 6,7-dihydroxy-3-[3',4'-methylenedioxyphenyl]-coumarin in neutrophils from patients with rheumatoid arthritis and in rats with acute joint inflammation (2019)
Albiero, Lucinéia Reuse; Andrade, Micássio Fernandes de ; Marchi, Larissa F. ; Landi-Librandi, Ana Paula; Figueiredo-Rinhel, Andréa Silva Garcia de; Carvalho, Camila Andressa; Kabeya, Luciana Mariko ; Oliveira, Renê Donizete Ribeiro de; Azzolini, Ana Elisa Caleiro Seixas ; Pupo, Monica Tallarico ; Emery, Flavio da Silva ; Lucisano-Valim, Yara Maria
Source: Inflammation Research. Unidades: FCFRP, FMRP
Subjects: QUIMIOTAXIA, INFLAMAÇÃO, LIPOSSOMOS, NEUTRÓFILOS, ARTRITE REUMATOIDE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALBIERO, Lucinéia Reuse et al. Immunomodulating action of the 3-phenylcoumarin derivative 6,7-dihydroxy-3-[3',4'-methylenedioxyphenyl]-coumarin in neutrophils from patients with rheumatoid arthritis and in rats with acute joint inflammation. Inflammation Research, v. 69, n. 1, p. 115-130, 2019Tradução . . Disponível em: https://doi.org/10.1007/s00011-019-01298-w. Acesso em: 17 abr. 2026.
APA
Albiero, L. R., Andrade, M. F. de, Marchi, L. F., Landi-Librandi, A. P., Figueiredo-Rinhel, A. S. G. de, Carvalho, C. A., et al. (2019). Immunomodulating action of the 3-phenylcoumarin derivative 6,7-dihydroxy-3-[3',4'-methylenedioxyphenyl]-coumarin in neutrophils from patients with rheumatoid arthritis and in rats with acute joint inflammation. Inflammation Research, 69( 1), 115-130. doi:10.1007/s00011-019-01298-w
NLM
Albiero LR, Andrade MF de, Marchi LF, Landi-Librandi AP, Figueiredo-Rinhel ASG de, Carvalho CA, Kabeya LM, Oliveira RDR de, Azzolini AECS, Pupo MT, Emery F da S, Lucisano-Valim YM. Immunomodulating action of the 3-phenylcoumarin derivative 6,7-dihydroxy-3-[3',4'-methylenedioxyphenyl]-coumarin in neutrophils from patients with rheumatoid arthritis and in rats with acute joint inflammation [Internet]. Inflammation Research. 2019 ; 69( 1): 115-130.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-019-01298-w
Vancouver
Albiero LR, Andrade MF de, Marchi LF, Landi-Librandi AP, Figueiredo-Rinhel ASG de, Carvalho CA, Kabeya LM, Oliveira RDR de, Azzolini AECS, Pupo MT, Emery F da S, Lucisano-Valim YM. Immunomodulating action of the 3-phenylcoumarin derivative 6,7-dihydroxy-3-[3',4'-methylenedioxyphenyl]-coumarin in neutrophils from patients with rheumatoid arthritis and in rats with acute joint inflammation [Internet]. Inflammation Research. 2019 ; 69( 1): 115-130.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-019-01298-w
Artigo de periodico
Human neutrophils are targets to paracoccin, a lectin expressed by Paracoccidioides brasiliensis (2018)
Ricci-Azevedo, R.; Gonçalves, R. A.; Roque-Barreira, Maria Cristina; Girard, D.
Source: Inflammation Research. Unidade: FMRP
Subjects: APOPTOSE, ADJUVANTES IMUNOLÓGICOS, NEUTRÓFILOS, ESPÉCIES REATIVAS DE OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RICCI-AZEVEDO, R. et al. Human neutrophils are targets to paracoccin, a lectin expressed by Paracoccidioides brasiliensis. Inflammation Research, v. 67, n. 1, p. 31-41, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00011-017-1093-8. Acesso em: 17 abr. 2026.
APA
Ricci-Azevedo, R., Gonçalves, R. A., Roque-Barreira, M. C., & Girard, D. (2018). Human neutrophils are targets to paracoccin, a lectin expressed by Paracoccidioides brasiliensis. Inflammation Research, 67( 1), 31-41. doi:10.1007/s00011-017-1093-8
NLM
Ricci-Azevedo R, Gonçalves RA, Roque-Barreira MC, Girard D. Human neutrophils are targets to paracoccin, a lectin expressed by Paracoccidioides brasiliensis [Internet]. Inflammation Research. 2018 ; 67( 1): 31-41.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-017-1093-8
Vancouver
Ricci-Azevedo R, Gonçalves RA, Roque-Barreira MC, Girard D. Human neutrophils are targets to paracoccin, a lectin expressed by Paracoccidioides brasiliensis [Internet]. Inflammation Research. 2018 ; 67( 1): 31-41.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-017-1093-8
Artigo de periodico
Polymorphisms and expression of inflammasome genes are associated with the development and severity of rheumatoid arthritis in Brazilian patients (2018)
Addobbati, Catarina; Cruz, Heidi Lacerda Alves da; Adelino, José Eduardo; Ramos, Amanda Luíze Melo Tavares; Fragoso, Thiago Sotero; Domingues, Alexandre; Duarte, Ângela Luzia Branco Pinto; Oliveira, Renê Donizeti Ribeiro; Louzada Júnior, Paulo ; Donadi, Eduardo Antônio ; Pontillo, Alessandra ; Silva, Jaqueline de Azevêdo; Crovella, Sergio; Garcia, Paula Sandrin
Source: Inflammation Research. Unidades: FMRP, ICB
Subjects: AUTOIMUNIDADE, ARTRITE REUMATOIDE, MONÓCITOS, EXPRESSÃO GÊNICA, POLIMORFISMO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ADDOBBATI, Catarina et al. Polymorphisms and expression of inflammasome genes are associated with the development and severity of rheumatoid arthritis in Brazilian patients. Inflammation Research, v. 67, n. 3, p. 255-264, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00011-017-1119-2. Acesso em: 17 abr. 2026.
APA
Addobbati, C., Cruz, H. L. A. da, Adelino, J. E., Ramos, A. L. M. T., Fragoso, T. S., Domingues, A., et al. (2018). Polymorphisms and expression of inflammasome genes are associated with the development and severity of rheumatoid arthritis in Brazilian patients. Inflammation Research, 67( 3), 255-264. doi:10.1007/s00011-017-1119-2
NLM
Addobbati C, Cruz HLA da, Adelino JE, Ramos ALMT, Fragoso TS, Domingues A, Duarte ÂLBP, Oliveira RDR, Louzada Júnior P, Donadi EA, Pontillo A, Silva J de A, Crovella S, Garcia PS. Polymorphisms and expression of inflammasome genes are associated with the development and severity of rheumatoid arthritis in Brazilian patients [Internet]. Inflammation Research. 2018 ; 67( 3): 255-264.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-017-1119-2
Vancouver
Addobbati C, Cruz HLA da, Adelino JE, Ramos ALMT, Fragoso TS, Domingues A, Duarte ÂLBP, Oliveira RDR, Louzada Júnior P, Donadi EA, Pontillo A, Silva J de A, Crovella S, Garcia PS. Polymorphisms and expression of inflammasome genes are associated with the development and severity of rheumatoid arthritis in Brazilian patients [Internet]. Inflammation Research. 2018 ; 67( 3): 255-264.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-017-1119-2
Artigo de periodico
The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88 (2018)
Sônego, Fabiane; Castanheira, Fernanda V. S.; Horta, Catarina V.; Kanashiro, Alexandre; Czaikoski, Paula G.; Zamboni, Dario Simões; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz
Source: Inflammation Research. Unidade: FMRP
Subjects: PNEUMONIA, NEUTRÓFILOS, QUIMIOCINAS, BACTÉRIAS GRAM-NEGATIVAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SÔNEGO, Fabiane et al. The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88. Inflammation Research, v. 67, n. 5, p. 435-443, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00011-018-1135-x. Acesso em: 17 abr. 2026.
APA
Sônego, F., Castanheira, F. V. S., Horta, C. V., Kanashiro, A., Czaikoski, P. G., Zamboni, D. S., et al. (2018). The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88. Inflammation Research, 67( 5), 435-443. doi:10.1007/s00011-018-1135-x
NLM
Sônego F, Castanheira FVS, Horta CV, Kanashiro A, Czaikoski PG, Zamboni DS, Alves Filho JCF, Cunha F de Q. The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88 [Internet]. Inflammation Research. 2018 ; 67( 5): 435-443.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-018-1135-x
Vancouver
Sônego F, Castanheira FVS, Horta CV, Kanashiro A, Czaikoski PG, Zamboni DS, Alves Filho JCF, Cunha F de Q. The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88 [Internet]. Inflammation Research. 2018 ; 67( 5): 435-443.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-018-1135-x
Artigo de periodico
Naringenin mitigates titanium dioxide (TiO2)-induced chronic arthritis in mice: role of oxidative stress, cytokines, and NFκB (2018)
Manchope, Marília F.; Artero, Nayara A.; Fattori, Victor; Mizokami, Sandra S.; Pitol, Dimitrius Leonardo; Issa, João Paulo Mardegan; Fukada, Sandra Yasuyo; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Casagrande, Rubia; Verri Júnior, Waldiceu A.
Source: Inflammation Research. Unidades: FORP, FCFRP, FMRP
Subjects: ARTRITE, ARTROPLASTIA, FLAVONOIDES, DOR, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MANCHOPE, Marília F. et al. Naringenin mitigates titanium dioxide (TiO2)-induced chronic arthritis in mice: role of oxidative stress, cytokines, and NFκB. Inflammation Research, v. 67, n. 11-12, p. 997-1012, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00011-018-1195-y. Acesso em: 17 abr. 2026.
APA
Manchope, M. F., Artero, N. A., Fattori, V., Mizokami, S. S., Pitol, D. L., Issa, J. P. M., et al. (2018). Naringenin mitigates titanium dioxide (TiO2)-induced chronic arthritis in mice: role of oxidative stress, cytokines, and NFκB. Inflammation Research, 67( 11-12), 997-1012. doi:10.1007/s00011-018-1195-y
NLM
Manchope MF, Artero NA, Fattori V, Mizokami SS, Pitol DL, Issa JPM, Fukada SY, Cunha TM, Alves Filho JCF, Cunha F de Q, Casagrande R, Verri Júnior WA. Naringenin mitigates titanium dioxide (TiO2)-induced chronic arthritis in mice: role of oxidative stress, cytokines, and NFκB [Internet]. Inflammation Research. 2018 ; 67( 11-12): 997-1012.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-018-1195-y
Vancouver
Manchope MF, Artero NA, Fattori V, Mizokami SS, Pitol DL, Issa JPM, Fukada SY, Cunha TM, Alves Filho JCF, Cunha F de Q, Casagrande R, Verri Júnior WA. Naringenin mitigates titanium dioxide (TiO2)-induced chronic arthritis in mice: role of oxidative stress, cytokines, and NFκB [Internet]. Inflammation Research. 2018 ; 67( 11-12): 997-1012.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-018-1195-y
Artigo de periodico
Probucol attenuates overt pain-like behavior and carrageenan-induced inflammatory hyperalgesia and leukocyte recruitment by inhibiting NF-кB activation and cytokine production without antioxidant effects (2017)
Zucoloto, Amanda Z.; Manchope, Marília F.; Staurengo-Ferrari, Larissa; Alves Filho, José Carlos Farias; Cunha, Thiago Mattar; Antunes, Maísa Mota; Menezes, Gustavo B.; Cunha, Fernando de Queiroz; Casagrande, Rúbia; Verri Júnior, Waldiceu Aparecido
Source: Inflammation Research. Unidade: FMRP
Subjects: ANALGÉSICOS, ANTI-INFLAMATÓRIOS, LEUCÓCITOS, DOR, CAVIDADE PERITONEAL, ESTIMULAÇÃO FÍSICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ZUCOLOTO, Amanda Z. et al. Probucol attenuates overt pain-like behavior and carrageenan-induced inflammatory hyperalgesia and leukocyte recruitment by inhibiting NF-кB activation and cytokine production without antioxidant effects. Inflammation Research, v. 66, n. 7, p. 591-602, 2017Tradução . . Disponível em: https://doi.org/10.1007/s00011-017-1040-8. Acesso em: 17 abr. 2026.
APA
Zucoloto, A. Z., Manchope, M. F., Staurengo-Ferrari, L., Alves Filho, J. C. F., Cunha, T. M., Antunes, M. M., et al. (2017). Probucol attenuates overt pain-like behavior and carrageenan-induced inflammatory hyperalgesia and leukocyte recruitment by inhibiting NF-кB activation and cytokine production without antioxidant effects. Inflammation Research, 66( 7), 591-602. doi:10.1007/s00011-017-1040-8
NLM
Zucoloto AZ, Manchope MF, Staurengo-Ferrari L, Alves Filho JCF, Cunha TM, Antunes MM, Menezes GB, Cunha F de Q, Casagrande R, Verri Júnior WA. Probucol attenuates overt pain-like behavior and carrageenan-induced inflammatory hyperalgesia and leukocyte recruitment by inhibiting NF-кB activation and cytokine production without antioxidant effects [Internet]. Inflammation Research. 2017 ; 66( 7): 591-602.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-017-1040-8
Vancouver
Zucoloto AZ, Manchope MF, Staurengo-Ferrari L, Alves Filho JCF, Cunha TM, Antunes MM, Menezes GB, Cunha F de Q, Casagrande R, Verri Júnior WA. Probucol attenuates overt pain-like behavior and carrageenan-induced inflammatory hyperalgesia and leukocyte recruitment by inhibiting NF-кB activation and cytokine production without antioxidant effects [Internet]. Inflammation Research. 2017 ; 66( 7): 591-602.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-017-1040-8
Artigo de periodico
Hyaluronidase decreases neutrophils infiltration to the inflammatory site (2016)
Fronza, Marcio; Muhr, Cornélia; Silveira, Denise Sayuri Calheiros da; Sorgi, Carlos Arterio; Rodrigues, Stephen Fernandes de Paula ; Farsky, Sandra Helena Poliselli ; Silva, Francisco Wanderley Garcia de Paula e; Merfort, Irmgard; Faccioli, Lucia Helena
Source: Inflammation Research. Unidades: FCFRP, FCF, FORP
Subjects: NEUTRÓFILOS, INFLAMAÇÃO, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FRONZA, Marcio et al. Hyaluronidase decreases neutrophils infiltration to the inflammatory site. Inflammation Research, p. 1-10, 2016Tradução . . Disponível em: https://doi.org/10.1007/s00011-016-0935-0. Acesso em: 17 abr. 2026.
APA
Fronza, M., Muhr, C., Silveira, D. S. C. da, Sorgi, C. A., Rodrigues, S. F. de P., Farsky, S. H. P., et al. (2016). Hyaluronidase decreases neutrophils infiltration to the inflammatory site. Inflammation Research, 1-10. doi:10.1007/s00011-016-0935-0
NLM
Fronza M, Muhr C, Silveira DSC da, Sorgi CA, Rodrigues SF de P, Farsky SHP, Silva FWG de P e, Merfort I, Faccioli LH. Hyaluronidase decreases neutrophils infiltration to the inflammatory site [Internet]. Inflammation Research. 2016 ; 1-10.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-016-0935-0
Vancouver
Fronza M, Muhr C, Silveira DSC da, Sorgi CA, Rodrigues SF de P, Farsky SHP, Silva FWG de P e, Merfort I, Faccioli LH. Hyaluronidase decreases neutrophils infiltration to the inflammatory site [Internet]. Inflammation Research. 2016 ; 1-10.[citado 2026 abr. 17 ] Available from: https://doi.org/10.1007/s00011-016-0935-0

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