Fructose 1,6-bisphosphate inhibits osteoclastogenesis by attenuating RANKL-induced NF-κB/NFATc-1 (2019)
- Authors:
- USP affiliated authors: CUNHA, FERNANDO DE QUEIROZ - FMRP ; ALVES FILHO, JOSÉ CARLOS FARIAS - FMRP ; ALVES, SANDRA YASUYO FUKADA - FCFRP ; VIACAVA, PAULA RAMOS - FMRP
- Unidades: FMRP; FCFRP
- DOI: 10.1007/s00011-019-01228-w
- Subjects: REMODELAÇÃO ÓSSEA; OSTEOCLASTO; FRUTOSE
- Keywords: Bone remodeling; Osteoclasts; Fructose 1,6-bisphosphate
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Source:
- Título: Inflammation Research
- ISSN: 1023-3830
- Volume/Número/Paginação/Ano: v. 68, n. 5, p. 415-421, 2019
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
BUITRAGO, Liseth Yamile Wilches et al. Fructose 1,6-bisphosphate inhibits osteoclastogenesis by attenuating RANKL-induced NF-κB/NFATc-1. Inflammation Research, v. 68, n. 5, p. 415-421, 2019Tradução . . Disponível em: https://doi.org/10.1007/s00011-019-01228-w. Acesso em: 28 dez. 2025. -
APA
Buitrago, L. Y. W., Viacava, P. R., Cunha, F. de Q., Alves Filho, J. C. F., & Fukada, S. Y. (2019). Fructose 1,6-bisphosphate inhibits osteoclastogenesis by attenuating RANKL-induced NF-κB/NFATc-1. Inflammation Research, 68( 5), 415-421. doi:10.1007/s00011-019-01228-w -
NLM
Buitrago LYW, Viacava PR, Cunha F de Q, Alves Filho JCF, Fukada SY. Fructose 1,6-bisphosphate inhibits osteoclastogenesis by attenuating RANKL-induced NF-κB/NFATc-1 [Internet]. Inflammation Research. 2019 ; 68( 5): 415-421.[citado 2025 dez. 28 ] Available from: https://doi.org/10.1007/s00011-019-01228-w -
Vancouver
Buitrago LYW, Viacava PR, Cunha F de Q, Alves Filho JCF, Fukada SY. Fructose 1,6-bisphosphate inhibits osteoclastogenesis by attenuating RANKL-induced NF-κB/NFATc-1 [Internet]. Inflammation Research. 2019 ; 68( 5): 415-421.[citado 2025 dez. 28 ] Available from: https://doi.org/10.1007/s00011-019-01228-w - A O-GlcNAcilação promove a osteoclastogênese e potencializa a reabsorção óssea in vitro e em modelo de lesão periapical
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Informações sobre o DOI: 10.1007/s00011-019-01228-w (Fonte: oaDOI API)
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