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Artigo de periodico
Sirtuin 1 regulates the phenotype and functions of dendritic cells through Ido1 pathway in obesity (2024)
Lima, Jean de; Leite, Jefferson Antônio ; Nunes, Bruno Ghirotto ; Silva, Eloisa Martins da ; Menezes-Silva, Luisa ; Hiyane, Meire Ioshie ; Goes, Carolina Purcell ; Coutinho, Luiz Lehmann ; Oliveira, Vinicius de Andrade ; Câmara, Niels Olsen Saraiva
Fonte: Cell Death and Disease. Unidades: ESALQ, ICB, FMRP
Assuntos: CÉLULAS DENDRÍTICAS, CÉLULAS QUE APRESENTAM ANTÍGENOS, FENÓTIPOS, OBESIDADE, PROTEÍNAS
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LIMA, Jean de et al. Sirtuin 1 regulates the phenotype and functions of dendritic cells through Ido1 pathway in obesity. Cell Death and Disease, v. 15, p. 1-17, 2024Tradução . . Disponível em: https://doi.org/10.1038/s41419-024-07125-3. Acesso em: 30 jun. 2025.
APA
Lima, J. de, Leite, J. A., Nunes, B. G., Silva, E. M. da, Menezes-Silva, L., Hiyane, M. I., et al. (2024). Sirtuin 1 regulates the phenotype and functions of dendritic cells through Ido1 pathway in obesity. Cell Death and Disease, 15, 1-17. doi:10.1038/s41419-024-07125-3
NLM
Lima J de, Leite JA, Nunes BG, Silva EM da, Menezes-Silva L, Hiyane MI, Goes CP, Coutinho LL, Oliveira V de A, Câmara NOS. Sirtuin 1 regulates the phenotype and functions of dendritic cells through Ido1 pathway in obesity [Internet]. Cell Death and Disease. 2024 ; 15 1-17.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-024-07125-3
Vancouver
Lima J de, Leite JA, Nunes BG, Silva EM da, Menezes-Silva L, Hiyane MI, Goes CP, Coutinho LL, Oliveira V de A, Câmara NOS. Sirtuin 1 regulates the phenotype and functions of dendritic cells through Ido1 pathway in obesity [Internet]. Cell Death and Disease. 2024 ; 15 1-17.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-024-07125-3
Artigo de periodico
Downregulation of the Rho GTPase pathway abrogates resistance to ionizing radiation in wild-type p53 glioblastoma by suppressing DNA repair mechanisms (2023)
Magalhães, Yuli Thamires ; Boell, Viktor Kalbermatter ; Cardella, Giovanna Duo; Forti, Fabio Luis
Fonte: Cell Death and Disease. Unidade: IQ
Assuntos: NEOPLASIAS CEREBRAIS, RADIAÇÃO IONIZANTE, REPARAÇÃO DE DNA
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MAGALHÃES, Yuli Thamires et al. Downregulation of the Rho GTPase pathway abrogates resistance to ionizing radiation in wild-type p53 glioblastoma by suppressing DNA repair mechanisms. Cell Death and Disease, v. 14, p. 1-16 art. 283, 2023Tradução . . Disponível em: https://doi.org/10.1038/s41419-023-05812-1. Acesso em: 30 jun. 2025.
APA
Magalhães, Y. T., Boell, V. K., Cardella, G. D., & Forti, F. L. (2023). Downregulation of the Rho GTPase pathway abrogates resistance to ionizing radiation in wild-type p53 glioblastoma by suppressing DNA repair mechanisms. Cell Death and Disease, 14, 1-16 art. 283. doi:10.1038/s41419-023-05812-1
NLM
Magalhães YT, Boell VK, Cardella GD, Forti FL. Downregulation of the Rho GTPase pathway abrogates resistance to ionizing radiation in wild-type p53 glioblastoma by suppressing DNA repair mechanisms [Internet]. Cell Death and Disease. 2023 ; 14 1-16 art. 283.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-023-05812-1
Vancouver
Magalhães YT, Boell VK, Cardella GD, Forti FL. Downregulation of the Rho GTPase pathway abrogates resistance to ionizing radiation in wild-type p53 glioblastoma by suppressing DNA repair mechanisms [Internet]. Cell Death and Disease. 2023 ; 14 1-16 art. 283.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-023-05812-1
Artigo de periodico
Absence of Bim sensitizes mice to experimental Trypanosoma cruzi infection (2021)
Torres, Marcela Hernandez; Nascimento, Rogério Silva do; Rebouças, Monica Cardozo; Cassado, Alexandra dos Anjos ; Matteucci, Kely Catarine ; Lima, Maria Regina D'Império ; Vasconcelos, José Ronnie C. ; Bortoluci, Karina Ramalho ; Alvarez, José Maria ; Amarante-Mendes, João Gustavo Pessini
Fonte: Cell Death and Disease. Unidade: ICB
Assuntos: IMUNOLOGIA, TRYPANOSOMA CRUZI, CAMUNDONGOS, DOENÇA DE CHAGAS, ÓXIDO NÍTRICO, IMUNIDADE, MACRÓFAGOS
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TORRES, Marcela Hernandez et al. Absence of Bim sensitizes mice to experimental Trypanosoma cruzi infection. Cell Death and Disease, v. 12, p. 1-8, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41419-021-03964-6. Acesso em: 30 jun. 2025.
APA
Torres, M. H., Nascimento, R. S. do, Rebouças, M. C., Cassado, A. dos A., Matteucci, K. C., Lima, M. R. D. 'I., et al. (2021). Absence of Bim sensitizes mice to experimental Trypanosoma cruzi infection. Cell Death and Disease, 12, 1-8. doi:10.1038/s41419-021-03964-6
NLM
Torres MH, Nascimento RS do, Rebouças MC, Cassado A dos A, Matteucci KC, Lima MRD'I, Vasconcelos JRC, Bortoluci KR, Alvarez JM, Amarante-Mendes JGP. Absence of Bim sensitizes mice to experimental Trypanosoma cruzi infection [Internet]. Cell Death and Disease. 2021 ; 12 1-8.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-021-03964-6
Vancouver
Torres MH, Nascimento RS do, Rebouças MC, Cassado A dos A, Matteucci KC, Lima MRD'I, Vasconcelos JRC, Bortoluci KR, Alvarez JM, Amarante-Mendes JGP. Absence of Bim sensitizes mice to experimental Trypanosoma cruzi infection [Internet]. Cell Death and Disease. 2021 ; 12 1-8.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-021-03964-6
Artigo de periodico
Autophagy in metabolic syndrome: breaking the wheel by targeting the renin-angiotensin system (2020)
Menikdiwela, Kalhara R; Ramalingam, Latha; Rasha, Fahmida; Wang, Shu; Dufour, Jannette M; Kalupahana, Nishan S; Sunahara, Karen K. S; Martins, Joilson de Oliveira ; Moussa, Naima Moustaid
Fonte: Cell Death and Disease. Unidade: FCF
Assuntos: METABOLISMO, ANGIOTENSINAS
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MENIKDIWELA, Kalhara R et al. Autophagy in metabolic syndrome: breaking the wheel by targeting the renin-angiotensin system. Cell Death and Disease, v. 11, n. 2, p. 1-17 art. 87, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41419-020-2275-9. Acesso em: 30 jun. 2025.
APA
Menikdiwela, K. R., Ramalingam, L., Rasha, F., Wang, S., Dufour, J. M., Kalupahana, N. S., et al. (2020). Autophagy in metabolic syndrome: breaking the wheel by targeting the renin-angiotensin system. Cell Death and Disease, 11( 2), 1-17 art. 87. doi:10.1038/s41419-020-2275-9
NLM
Menikdiwela KR, Ramalingam L, Rasha F, Wang S, Dufour JM, Kalupahana NS, Sunahara KKS, Martins J de O, Moussa NM. Autophagy in metabolic syndrome: breaking the wheel by targeting the renin-angiotensin system [Internet]. Cell Death and Disease. 2020 ; 11( 2): 1-17 art. 87.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-020-2275-9
Vancouver
Menikdiwela KR, Ramalingam L, Rasha F, Wang S, Dufour JM, Kalupahana NS, Sunahara KKS, Martins J de O, Moussa NM. Autophagy in metabolic syndrome: breaking the wheel by targeting the renin-angiotensin system [Internet]. Cell Death and Disease. 2020 ; 11( 2): 1-17 art. 87.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-020-2275-9
Artigo de periodico
Distinct photo-oxidation-induced cell death pathways lead to selective killing of human breast cancer cells (2020)
Santos, Ancély Ferreira dos ; Inague, Alex ; Arini, Gabriel Santos ; Terra, Letícia Ferreira; Wailemann, Rosângela A. M; Pimentel, André Coppe ; Yoshinaga, Marcos Yukio ; Silva, Ricardo Roberto da; Severino, Divinomar ; Almeida, Daria Raquel Queiroz de ; Gomes, Vinícius M; Bruni-Cardoso, Alexandre ; Terra, Walter Ribeiro ; Miyamoto, Sayuri ; Baptista, Maurício da Silva ; Labriola, Leticia
Fonte: Cell Death and Disease. Unidades: IB, FCFRP, IQ
Assuntos: NEOPLASIAS MAMÁRIAS, TERAPIA FOTODINÂMICA
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ABNT
SANTOS, Ancély Ferreira dos et al. Distinct photo-oxidation-induced cell death pathways lead to selective killing of human breast cancer cells. Cell Death and Disease, v. 11, p. 1-12 art. 1070, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41419-020-03275-2. Acesso em: 30 jun. 2025.
APA
Santos, A. F. dos, Inague, A., Arini, G. S., Terra, L. F., Wailemann, R. A. M., Pimentel, A. C., et al. (2020). Distinct photo-oxidation-induced cell death pathways lead to selective killing of human breast cancer cells. Cell Death and Disease, 11, 1-12 art. 1070. doi:10.1038/s41419-020-03275-2
NLM
Santos AF dos, Inague A, Arini GS, Terra LF, Wailemann RAM, Pimentel AC, Yoshinaga MY, Silva RR da, Severino D, Almeida DRQ de, Gomes VM, Bruni-Cardoso A, Terra WR, Miyamoto S, Baptista M da S, Labriola L. Distinct photo-oxidation-induced cell death pathways lead to selective killing of human breast cancer cells [Internet]. Cell Death and Disease. 2020 ; 11 1-12 art. 1070.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-020-03275-2
Vancouver
Santos AF dos, Inague A, Arini GS, Terra LF, Wailemann RAM, Pimentel AC, Yoshinaga MY, Silva RR da, Severino D, Almeida DRQ de, Gomes VM, Bruni-Cardoso A, Terra WR, Miyamoto S, Baptista M da S, Labriola L. Distinct photo-oxidation-induced cell death pathways lead to selective killing of human breast cancer cells [Internet]. Cell Death and Disease. 2020 ; 11 1-12 art. 1070.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-020-03275-2
Artigo de periodico
ATR mediates cisplatin resistance in 3D-cultured breast cancer cells via translesion DNA synthesis modulation (2019)
Gomes, Luciana Rodrigues; Rocha, Clarissa Ribeiro Reily ; Martins, Davi Jardim ; Fiore, Ana Paula Zen Petisco ; Kinker, Gabriela Sarti ; Bruni-Cardoso, Alexandre ; Menck, Carlos Frederico Martins
Fonte: Cell Death and Disease. Unidades: IQ, ICB, IB
Assuntos: NEOPLASIAS MAMÁRIAS, DNA, CÉLULAS CULTIVADAS DE TUMOR, MATRIZ EXTRACELULAR, ANTINEOPLÁSICOS
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ABNT
GOMES, Luciana Rodrigues et al. ATR mediates cisplatin resistance in 3D-cultured breast cancer cells via translesion DNA synthesis modulation. Cell Death and Disease, v. 10, p. 1-15 art. 459, 2019Tradução . . Disponível em: https://doi.org/10.1038/s41419-019-1689-8. Acesso em: 30 jun. 2025.
APA
Gomes, L. R., Rocha, C. R. R., Martins, D. J., Fiore, A. P. Z. P., Kinker, G. S., Bruni-Cardoso, A., & Menck, C. F. M. (2019). ATR mediates cisplatin resistance in 3D-cultured breast cancer cells via translesion DNA synthesis modulation. Cell Death and Disease, 10, 1-15 art. 459. doi:10.1038/s41419-019-1689-8
NLM
Gomes LR, Rocha CRR, Martins DJ, Fiore APZP, Kinker GS, Bruni-Cardoso A, Menck CFM. ATR mediates cisplatin resistance in 3D-cultured breast cancer cells via translesion DNA synthesis modulation [Internet]. Cell Death and Disease. 2019 ; 10 1-15 art. 459.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-019-1689-8
Vancouver
Gomes LR, Rocha CRR, Martins DJ, Fiore APZP, Kinker GS, Bruni-Cardoso A, Menck CFM. ATR mediates cisplatin resistance in 3D-cultured breast cancer cells via translesion DNA synthesis modulation [Internet]. Cell Death and Disease. 2019 ; 10 1-15 art. 459.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-019-1689-8
Artigo de periodico
CCR4-dependent reduction in the number and suppressor function of CD4+Foxp3+ cells augments IFN-γ-mediated pulmonary inflammation and aggravates tuberculosis pathogenesis (2019)
Bertolini, Thais B.; Piñeros, Annie R.; Prado, Rafael Q.; Gembre, Ana Flávia; Ramalho, Leandra Naíra Zambelli; Alves Filho, José Carlos Farias; Bonato, Vânia Luiza Deperon
Fonte: Cell Death and Disease. Unidade: FMRP
Assuntos: INFLAMAÇÃO, TUBERCULOSE
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ABNT
BERTOLINI, Thais B. et al. CCR4-dependent reduction in the number and suppressor function of CD4+Foxp3+ cells augments IFN-γ-mediated pulmonary inflammation and aggravates tuberculosis pathogenesis. Cell Death and Disease, v. 10, n. 1, 2019Tradução . . Disponível em: https://doi.org/10.1038/s41419-018-1240-3. Acesso em: 30 jun. 2025.
APA
Bertolini, T. B., Piñeros, A. R., Prado, R. Q., Gembre, A. F., Ramalho, L. N. Z., Alves Filho, J. C. F., & Bonato, V. L. D. (2019). CCR4-dependent reduction in the number and suppressor function of CD4+Foxp3+ cells augments IFN-γ-mediated pulmonary inflammation and aggravates tuberculosis pathogenesis. Cell Death and Disease, 10( 1). doi:10.1038/s41419-018-1240-3
NLM
Bertolini TB, Piñeros AR, Prado RQ, Gembre AF, Ramalho LNZ, Alves Filho JCF, Bonato VLD. CCR4-dependent reduction in the number and suppressor function of CD4+Foxp3+ cells augments IFN-γ-mediated pulmonary inflammation and aggravates tuberculosis pathogenesis [Internet]. Cell Death and Disease. 2019 ; 10( 1):[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-018-1240-3
Vancouver
Bertolini TB, Piñeros AR, Prado RQ, Gembre AF, Ramalho LNZ, Alves Filho JCF, Bonato VLD. CCR4-dependent reduction in the number and suppressor function of CD4+Foxp3+ cells augments IFN-γ-mediated pulmonary inflammation and aggravates tuberculosis pathogenesis [Internet]. Cell Death and Disease. 2019 ; 10( 1):[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-018-1240-3
Artigo de periodico
Inhibition of inflammasome activation by a clinical strain of Klebsiella pneumoniae impairs efferocytosis and leads to bacterial dissemination (2018)
Codo, Ana Campos; Saraiva, Amanda Correia; Santos, Leonardo Lima dos; Visconde, Marina Francisco; Gales, Ana Cristina; Zamboni, Dario Simões; Medeiros, Alexandra Ivo
Fonte: Cell Death and Disease. Unidade: FMRP
Assuntos: BACTÉRIAS GRAM-NEGATIVAS, PNEUMONIA, MACRÓFAGOS
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ABNT
CODO, Ana Campos et al. Inhibition of inflammasome activation by a clinical strain of Klebsiella pneumoniae impairs efferocytosis and leads to bacterial dissemination. Cell Death and Disease, v. 9, n. 12, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41419-018-1214-5. Acesso em: 30 jun. 2025.
APA
Codo, A. C., Saraiva, A. C., Santos, L. L. dos, Visconde, M. F., Gales, A. C., Zamboni, D. S., & Medeiros, A. I. (2018). Inhibition of inflammasome activation by a clinical strain of Klebsiella pneumoniae impairs efferocytosis and leads to bacterial dissemination. Cell Death and Disease, 9( 12). doi:10.1038/s41419-018-1214-5
NLM
Codo AC, Saraiva AC, Santos LL dos, Visconde MF, Gales AC, Zamboni DS, Medeiros AI. Inhibition of inflammasome activation by a clinical strain of Klebsiella pneumoniae impairs efferocytosis and leads to bacterial dissemination [Internet]. Cell Death and Disease. 2018 ; 9( 12):[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-018-1214-5
Vancouver
Codo AC, Saraiva AC, Santos LL dos, Visconde MF, Gales AC, Zamboni DS, Medeiros AI. Inhibition of inflammasome activation by a clinical strain of Klebsiella pneumoniae impairs efferocytosis and leads to bacterial dissemination [Internet]. Cell Death and Disease. 2018 ; 9( 12):[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-018-1214-5
Artigo de periodico
TGFβ1 signaling sustains aryl hydrocarbon receptor (AHR) expression and restrains the pathogenic potential of TH17 cells by an AHR-independent mechanism (2018)
Lima, Kalil Alves de; Donate, Paula Barbim; Talbot, Jhimmy; Davoli-Ferreira, Marcela; Peres, Raphael Sanches; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz
Fonte: Cell Death and Disease. Unidade: FMRP
Assuntos: HIDROCARBONETOS, RECEPTORES, MARCADOR MOLECULAR
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ABNT
LIMA, Kalil Alves de et al. TGFβ1 signaling sustains aryl hydrocarbon receptor (AHR) expression and restrains the pathogenic potential of TH17 cells by an AHR-independent mechanism. Cell Death and Disease, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41419-018-1107-7. Acesso em: 30 jun. 2025.
APA
Lima, K. A. de, Donate, P. B., Talbot, J., Davoli-Ferreira, M., Peres, R. S., Cunha, T. M., et al. (2018). TGFβ1 signaling sustains aryl hydrocarbon receptor (AHR) expression and restrains the pathogenic potential of TH17 cells by an AHR-independent mechanism. Cell Death and Disease, 9. doi:10.1038/s41419-018-1107-7
NLM
Lima KA de, Donate PB, Talbot J, Davoli-Ferreira M, Peres RS, Cunha TM, Alves Filho JCF, Cunha F de Q. TGFβ1 signaling sustains aryl hydrocarbon receptor (AHR) expression and restrains the pathogenic potential of TH17 cells by an AHR-independent mechanism [Internet]. Cell Death and Disease. 2018 ; 9[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-018-1107-7
Vancouver
Lima KA de, Donate PB, Talbot J, Davoli-Ferreira M, Peres RS, Cunha TM, Alves Filho JCF, Cunha F de Q. TGFβ1 signaling sustains aryl hydrocarbon receptor (AHR) expression and restrains the pathogenic potential of TH17 cells by an AHR-independent mechanism [Internet]. Cell Death and Disease. 2018 ; 9[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/s41419-018-1107-7
Artigo de periodico
Cnbp ameliorates Treacher Collins Syndrome craniofacial anomalies through a pathway that involves redox-responsive genes (2016)
Peralta, Mauro S Porcel de; Mouguelar, Valeria S; Sdrigotti, María Antonella; Ishiy, Felipe A. A; Fanganiello, Roberto D; Passos-Bueno, Maria Rita ; Coux, Gabriela; Calcaterra, Nora
Fonte: Cell Death and Disease. Unidade: IB
Assuntos: DOENÇAS CONGÊNITAS, ANOMALIA CRANIOFACIAL, FISSURA PALATINA, MUTAÇÃO GENÉTICA, EXPRESSÃO GÊNICA, ANTIOXIDANTES
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ABNT
PERALTA, Mauro S Porcel de et al. Cnbp ameliorates Treacher Collins Syndrome craniofacial anomalies through a pathway that involves redox-responsive genes. Cell Death and Disease, v. 7, p. 1-11, 2016Tradução . . Disponível em: https://doi.org/10.1038/cddis.2016.299. Acesso em: 30 jun. 2025.
APA
Peralta, M. S. P. de, Mouguelar, V. S., Sdrigotti, M. A., Ishiy, F. A. A., Fanganiello, R. D., Passos-Bueno, M. R., et al. (2016). Cnbp ameliorates Treacher Collins Syndrome craniofacial anomalies through a pathway that involves redox-responsive genes. Cell Death and Disease, 7, 1-11. doi:10.1038/cddis.2016.299
NLM
Peralta MSP de, Mouguelar VS, Sdrigotti MA, Ishiy FAA, Fanganiello RD, Passos-Bueno MR, Coux G, Calcaterra N. Cnbp ameliorates Treacher Collins Syndrome craniofacial anomalies through a pathway that involves redox-responsive genes [Internet]. Cell Death and Disease. 2016 ; 7 1-11.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/cddis.2016.299
Vancouver
Peralta MSP de, Mouguelar VS, Sdrigotti MA, Ishiy FAA, Fanganiello RD, Passos-Bueno MR, Coux G, Calcaterra N. Cnbp ameliorates Treacher Collins Syndrome craniofacial anomalies through a pathway that involves redox-responsive genes [Internet]. Cell Death and Disease. 2016 ; 7 1-11.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/cddis.2016.299
Artigo de periodico
A myosin-Va tail fragment sequesters dynein light chains leading to apoptosis in melanoma cells (2013)
Izidoro-Toledo, T. C.; Borges, A. C.; Araújo, D. D.; Mazzi, D. P. S. Leitão; Nascimento Júnior, F. O.; Sousa, J. F.; Alves, C. P.; Paiva, A. P. B.; Trindade, D. M.; Patussi, E. V.; Peixoto, P. M.; Kinnaly, K. W.; Espreáfico, Enilza Maria
Fonte: Cell Death and Disease. Unidade: FMRP
Assuntos: APOPTOSE, FIBROBLASTOS, REGULAÇÃO GÊNICA, MELANOMA, NEOPLASIAS CUTÂNEAS
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ABNT
IZIDORO-TOLEDO, T. C. et al. A myosin-Va tail fragment sequesters dynein light chains leading to apoptosis in melanoma cells. Cell Death and Disease, v. 4, 2013Tradução . . Disponível em: https://doi.org/10.1038/cddis.2013.45. Acesso em: 30 jun. 2025.
APA
Izidoro-Toledo, T. C., Borges, A. C., Araújo, D. D., Mazzi, D. P. S. L., Nascimento Júnior, F. O., Sousa, J. F., et al. (2013). A myosin-Va tail fragment sequesters dynein light chains leading to apoptosis in melanoma cells. Cell Death and Disease, 4. doi:10.1038/cddis.2013.45
NLM
Izidoro-Toledo TC, Borges AC, Araújo DD, Mazzi DPSL, Nascimento Júnior FO, Sousa JF, Alves CP, Paiva APB, Trindade DM, Patussi EV, Peixoto PM, Kinnaly KW, Espreáfico EM. A myosin-Va tail fragment sequesters dynein light chains leading to apoptosis in melanoma cells [Internet]. Cell Death and Disease. 2013 ; 4[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/cddis.2013.45
Vancouver
Izidoro-Toledo TC, Borges AC, Araújo DD, Mazzi DPSL, Nascimento Júnior FO, Sousa JF, Alves CP, Paiva APB, Trindade DM, Patussi EV, Peixoto PM, Kinnaly KW, Espreáfico EM. A myosin-Va tail fragment sequesters dynein light chains leading to apoptosis in melanoma cells [Internet]. Cell Death and Disease. 2013 ; 4[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/cddis.2013.45
Artigo de periodico
Temporal blastemal cell gene expression analysis in the kidney reveals new Wnt and related signaling pathway genes to be essential for Wilms' tumor onset (2011)
Maschietto, M; Trapé, A. P; Piccoli, F. S; Ricca, T. I; Dias, A. A. M; Coudry, R. A; Galante, P. A; Torres, C; Fahhan, L; Lourenço, S; Grundy, P. E; Camargo, B de; Souza, S de; Neves, Eduardo Jordão; Soares, F A; Brentani, Helena Paula; Carraro, D M
Fonte: Cell Death and Disease. Unidades: FM, IME
Assuntos: EXPRESSÃO GÊNICA, BLASTOCISTO, GENES (ANÁLISE), RIM, NEFROPATIAS, DIFERENCIAÇÃO CELULAR, MARCADOR MOLECULAR, REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
MASCHIETTO, M et al. Temporal blastemal cell gene expression analysis in the kidney reveals new Wnt and related signaling pathway genes to be essential for Wilms' tumor onset. Cell Death and Disease, v. 2, p. e224, 2011Tradução . . Disponível em: https://doi.org/10.1038/cddis.2011.105. Acesso em: 30 jun. 2025.
APA
Maschietto, M., Trapé, A. P., Piccoli, F. S., Ricca, T. I., Dias, A. A. M., Coudry, R. A., et al. (2011). Temporal blastemal cell gene expression analysis in the kidney reveals new Wnt and related signaling pathway genes to be essential for Wilms' tumor onset. Cell Death and Disease, 2, e224. doi:10.1038/cddis.2011.105
NLM
Maschietto M, Trapé AP, Piccoli FS, Ricca TI, Dias AAM, Coudry RA, Galante PA, Torres C, Fahhan L, Lourenço S, Grundy PE, Camargo B de, Souza S de, Neves EJ, Soares FA, Brentani HP, Carraro DM. Temporal blastemal cell gene expression analysis in the kidney reveals new Wnt and related signaling pathway genes to be essential for Wilms' tumor onset [Internet]. Cell Death and Disease. 2011 ; 2 e224.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/cddis.2011.105
Vancouver
Maschietto M, Trapé AP, Piccoli FS, Ricca TI, Dias AAM, Coudry RA, Galante PA, Torres C, Fahhan L, Lourenço S, Grundy PE, Camargo B de, Souza S de, Neves EJ, Soares FA, Brentani HP, Carraro DM. Temporal blastemal cell gene expression analysis in the kidney reveals new Wnt and related signaling pathway genes to be essential for Wilms' tumor onset [Internet]. Cell Death and Disease. 2011 ; 2 e224.[citado 2025 jun. 30 ] Available from: https://doi.org/10.1038/cddis.2011.105

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