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Artigo de periodico
Immune and endocrine alterations at the early stage of inflammatory assemblage in toads after stimulation with heat-killed bacteria (Aeromonas hydrophila) (2024)
Garcia Neto, Patrício G; Titon, Stefanny C. M ; Muxel, Sandra M ; Titon Jr., Braz ; Figueiredo, Aymam C. de ; Floreste, Felipe R ; Lima, Alan S; Assis, Vania R ; Gomes, Fernando R
Source: Comparative Biochemistry and Physiology. Part A: Molecular & Integrative Physiology. Unidades: IB, ICB
Subjects: PATOGENIA ANIMAL, CITOCINAS, IMUNIDADE, HORMÔNIOS
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GARCIA NETO, Patrício G et al. Immune and endocrine alterations at the early stage of inflammatory assemblage in toads after stimulation with heat-killed bacteria (Aeromonas hydrophila). Comparative Biochemistry and Physiology. Part A: Molecular & Integrative Physiology, v. 291, n. art. 111606, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.cbpa.2024.111606. Acesso em: 05 nov. 2024.
APA
Garcia Neto, P. G., Titon, S. C. M., Muxel, S. M., Titon Jr., B., Figueiredo, A. C. de, Floreste, F. R., et al. (2024). Immune and endocrine alterations at the early stage of inflammatory assemblage in toads after stimulation with heat-killed bacteria (Aeromonas hydrophila). Comparative Biochemistry and Physiology. Part A: Molecular & Integrative Physiology, 291( art. 111606). doi:10.1016/j.cbpa.2024.111606
NLM
Garcia Neto PG, Titon SCM, Muxel SM, Titon Jr. B, Figueiredo AC de, Floreste FR, Lima AS, Assis VR, Gomes FR. Immune and endocrine alterations at the early stage of inflammatory assemblage in toads after stimulation with heat-killed bacteria (Aeromonas hydrophila) [Internet]. Comparative Biochemistry and Physiology. Part A: Molecular & Integrative Physiology. 2024 ; 291( art. 111606):[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.cbpa.2024.111606
Vancouver
Garcia Neto PG, Titon SCM, Muxel SM, Titon Jr. B, Figueiredo AC de, Floreste FR, Lima AS, Assis VR, Gomes FR. Immune and endocrine alterations at the early stage of inflammatory assemblage in toads after stimulation with heat-killed bacteria (Aeromonas hydrophila) [Internet]. Comparative Biochemistry and Physiology. Part A: Molecular & Integrative Physiology. 2024 ; 291( art. 111606):[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.cbpa.2024.111606
Artigo de periodico
Moderate-intensity aerobic exercise training improves CD8+ tumor-infiltrating lymphocytes effector function by reducing mitochondrial loss (2024)
Voltarelli, Vanessa Azevedo ; Amano, Mariane Tami ; Tobias, Gabriel Cardial ; Borges, Gabriela Silva ; Paixão, Ailma Oliveira da ; Pereira, Marcelo Gomes ; Camara, Niels Olsen Saraiva ; Caldeira, Waldir; Ribeiro, Alberto de Freitas ; Otterbein, Leo Edmond; Negrão, Carlos Eduardo ; Turner, James Edward ; Brum, Patrícia Chakur ; Camargo, Anamaria Aranha
Source: iScience. Unidades: ICB, IB, EEFE
Subjects: TREINAMENTO FÍSICO, TREINAMENTO AERÓBIO, NEOPLASIAS COLORRETAIS, EXERCÍCIO FÍSICO, CAMUNDONGOS, MITOCÔNDRIAS, CÉLULAS CULTIVADAS DE TUMOR, FÁRMACOS IMUNOSSUPRESSORES, LINFÓCITOS
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VOLTARELLI, Vanessa Azevedo et al. Moderate-intensity aerobic exercise training improves CD8+ tumor-infiltrating lymphocytes effector function by reducing mitochondrial loss. iScience, v. 27, n. 6 , p. 14 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2024.110121. Acesso em: 05 nov. 2024.
APA
Voltarelli, V. A., Amano, M. T., Tobias, G. C., Borges, G. S., Paixão, A. O. da, Pereira, M. G., et al. (2024). Moderate-intensity aerobic exercise training improves CD8+ tumor-infiltrating lymphocytes effector function by reducing mitochondrial loss. iScience, 27( 6 ), 14 . doi:10.1016/j.isci.2024.110121
NLM
Voltarelli VA, Amano MT, Tobias GC, Borges GS, Paixão AO da, Pereira MG, Camara NOS, Caldeira W, Ribeiro A de F, Otterbein LE, Negrão CE, Turner JE, Brum PC, Camargo AA. Moderate-intensity aerobic exercise training improves CD8+ tumor-infiltrating lymphocytes effector function by reducing mitochondrial loss [Internet]. iScience. 2024 ; 27( 6 ): 14 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.isci.2024.110121
Vancouver
Voltarelli VA, Amano MT, Tobias GC, Borges GS, Paixão AO da, Pereira MG, Camara NOS, Caldeira W, Ribeiro A de F, Otterbein LE, Negrão CE, Turner JE, Brum PC, Camargo AA. Moderate-intensity aerobic exercise training improves CD8+ tumor-infiltrating lymphocytes effector function by reducing mitochondrial loss [Internet]. iScience. 2024 ; 27( 6 ): 14 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.isci.2024.110121
Artigo de periodico
Liver vs. spleen: time course of organ-dependent immune gene expression in an LPS-stimulated toad (Rhinella diptycha) (2023)
Floreste, Felipe R; Titon Jr, Braz; Titon, Stefanny C. M; Muxel, Sandra M; Figueiredo, Aymam C. de; Gomes, Fernando Ribeiro ; Assis, Vania R
Source: Comparative Biochemistry and Physiology Part B. Unidades: ICB, IB
Subjects: ANURA, ANFÍBIOS, FISIOLOGIA ANIMAL, SISTEMA IMUNE, CITOCINAS, INFLAMAÇÃO
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FLORESTE, Felipe R et al. Liver vs. spleen: time course of organ-dependent immune gene expression in an LPS-stimulated toad (Rhinella diptycha). Comparative Biochemistry and Physiology Part B, v. 263, n. Ja, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.cbpb.2022.110784. Acesso em: 05 nov. 2024.
APA
Floreste, F. R., Titon Jr, B., Titon, S. C. M., Muxel, S. M., Figueiredo, A. C. de, Gomes, F. R., & Assis, V. R. (2023). Liver vs. spleen: time course of organ-dependent immune gene expression in an LPS-stimulated toad (Rhinella diptycha). Comparative Biochemistry and Physiology Part B, 263( Ja). doi:10.1016/j.cbpb.2022.110784
NLM
Floreste FR, Titon Jr B, Titon SCM, Muxel SM, Figueiredo AC de, Gomes FR, Assis VR. Liver vs. spleen: time course of organ-dependent immune gene expression in an LPS-stimulated toad (Rhinella diptycha) [Internet]. Comparative Biochemistry and Physiology Part B. 2023 ; 263( Ja):[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.cbpb.2022.110784
Vancouver
Floreste FR, Titon Jr B, Titon SCM, Muxel SM, Figueiredo AC de, Gomes FR, Assis VR. Liver vs. spleen: time course of organ-dependent immune gene expression in an LPS-stimulated toad (Rhinella diptycha) [Internet]. Comparative Biochemistry and Physiology Part B. 2023 ; 263( Ja):[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.cbpb.2022.110784
Artigo de periodico
Putrescine supplementation shifts macrophage L-arginine metabolism related-genes reducing Leishmania amazonensis infection (2023)
Zanatta, Jonathan Miguel ; Acuña, Stephanie Maia ; Angelo, Yan de Souza ; Bento, Camilla de Almeida; Muxel, Sandra Marcia ; Peron, Jean Pierre Schatzmann ; Stolf, Beatriz Simonsen
Source: PL o S One. Unidades: ICB, IB
Subjects: IMUNOLOGA, PARASITOLOGIA, LEISHMANIOSE CUTÂNEA, LEISHMANIA, SUPLEMENTOS DIETÉTICOS, MACRÓFAGOS, ARGININA, ÓXIDO NÍTRICO, ANTIPROTOZOÁRIOS, ANIMAIS PARASITOS, EXPRESSÃO GÊNICA
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ZANATTA, Jonathan Miguel et al. Putrescine supplementation shifts macrophage L-arginine metabolism related-genes reducing Leishmania amazonensis infection. PL o S One, v. 3, p. 1-25, 2023Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0283696. Acesso em: 05 nov. 2024.
APA
Zanatta, J. M., Acuña, S. M., Angelo, Y. de S., Bento, C. de A., Muxel, S. M., Peron, J. P. S., & Stolf, B. S. (2023). Putrescine supplementation shifts macrophage L-arginine metabolism related-genes reducing Leishmania amazonensis infection. PL o S One, 3, 1-25. doi:10.1371/journal.pone.0283696
NLM
Zanatta JM, Acuña SM, Angelo Y de S, Bento C de A, Muxel SM, Peron JPS, Stolf BS. Putrescine supplementation shifts macrophage L-arginine metabolism related-genes reducing Leishmania amazonensis infection [Internet]. PL o S One. 2023 ; 3 1-25.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1371/journal.pone.0283696
Vancouver
Zanatta JM, Acuña SM, Angelo Y de S, Bento C de A, Muxel SM, Peron JPS, Stolf BS. Putrescine supplementation shifts macrophage L-arginine metabolism related-genes reducing Leishmania amazonensis infection [Internet]. PL o S One. 2023 ; 3 1-25.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1371/journal.pone.0283696
Artigo de periodico
Comparative transcriptomic analysis of long noncoding RNAs in Leishmania-infected human macrophages (2022)
Fernandes, Juliane C. R; Gonçalves, André N. A; Floeter-Winter, Lucile Maria ; Nakaya, Helder I; Muxel, Sandra M
Source: Frontiers in Genetics. Unidades: IB, ICB, FM, FCF
Subjects: LEISHMANIA, PARASITOLOGIA, EXPRESSÃO GÊNICA, IMUNOLOGIA CELULAR
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FERNANDES, Juliane C. R et al. Comparative transcriptomic analysis of long noncoding RNAs in Leishmania-infected human macrophages. Frontiers in Genetics, v. 13, 2022Tradução . . Disponível em: https://doi.org/10.3389/fgene.2022.1051568. Acesso em: 05 nov. 2024.
APA
Fernandes, J. C. R., Gonçalves, A. N. A., Floeter-Winter, L. M., Nakaya, H. I., & Muxel, S. M. (2022). Comparative transcriptomic analysis of long noncoding RNAs in Leishmania-infected human macrophages. Frontiers in Genetics, 13. doi:10.3389/fgene.2022.1051568
NLM
Fernandes JCR, Gonçalves ANA, Floeter-Winter LM, Nakaya HI, Muxel SM. Comparative transcriptomic analysis of long noncoding RNAs in Leishmania-infected human macrophages [Internet]. Frontiers in Genetics. 2022 ; 13[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fgene.2022.1051568
Vancouver
Fernandes JCR, Gonçalves ANA, Floeter-Winter LM, Nakaya HI, Muxel SM. Comparative transcriptomic analysis of long noncoding RNAs in Leishmania-infected human macrophages [Internet]. Frontiers in Genetics. 2022 ; 13[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fgene.2022.1051568
Artigo de periodico
MS-driven metabolic alterations are recapitulated in iPSC-Derived astrocytes (2022)
Nunes, Bruno Ghirotto ; Oliveira, Danyllo Felipe de ; Cipelli, Marcella ; Basso, Paulo José ; Lima, Jean De ; Breda, Cristiane Naffah de Souza ; Ribeiro, Henrique Caldeira da ; Silva, Camille Cristine Caldeira da ; Sertié, Andréa L ; Oliveira, Antonio Edson Rocha ; Hiyane, Meire Ioshie ; Galdini, Elia G. ; Sussulini, Alessandra ; Oliveira, Enedina Maria Lobato de ; Zatz, Mayana ; Câmara, Niels Olsen Saraiva ; Nakaya, Helder Takashi Imoto ; Kowaltowski, Alicia Juliana
Source: Annals of Neurology. Unidades: ICB, IB, FCF, IQ
Subjects: IMUNOLOGIA, ASTRÓCITOS, CÉLULAS-TRONCO, ÁCIDO GLUTÂMICO, MITOCÔNDRIAS, ESCLEROSE MÚLTIPLA, MICROSCOPIA ELETRÔNICA, CITOMETRIA DE FLUXO, CITOCINAS, EXPRESSÃO GÊNICA
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NUNES, Bruno Ghirotto et al. MS-driven metabolic alterations are recapitulated in iPSC-Derived astrocytes. Annals of Neurology, v. 91, n. 5, p. 652-659, 2022Tradução . . Disponível em: https://doi.org/10.1002/ana.26336. Acesso em: 05 nov. 2024.
APA
Nunes, B. G., Oliveira, D. F. de, Cipelli, M., Basso, P. J., Lima, J. D., Breda, C. N. de S., et al. (2022). MS-driven metabolic alterations are recapitulated in iPSC-Derived astrocytes. Annals of Neurology, 91( 5), 652-659. doi:10.1002/ana.26336
NLM
Nunes BG, Oliveira DF de, Cipelli M, Basso PJ, Lima JD, Breda CN de S, Ribeiro HC da, Silva CCC da, Sertié AL, Oliveira AER, Hiyane MI, Galdini EG, Sussulini A, Oliveira EML de, Zatz M, Câmara NOS, Nakaya HTI, Kowaltowski AJ. MS-driven metabolic alterations are recapitulated in iPSC-Derived astrocytes [Internet]. Annals of Neurology. 2022 ; 91( 5): 652-659.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1002/ana.26336
Vancouver
Nunes BG, Oliveira DF de, Cipelli M, Basso PJ, Lima JD, Breda CN de S, Ribeiro HC da, Silva CCC da, Sertié AL, Oliveira AER, Hiyane MI, Galdini EG, Sussulini A, Oliveira EML de, Zatz M, Câmara NOS, Nakaya HTI, Kowaltowski AJ. MS-driven metabolic alterations are recapitulated in iPSC-Derived astrocytes [Internet]. Annals of Neurology. 2022 ; 91( 5): 652-659.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1002/ana.26336
Artigo de periodico
miR-548d-3p is up-regulated in human visceral leishmaniasis and suppresses parasite growth in macrophages (2022)
Ramos-Sanchez, Eduardo Milton ; Reis, Luiza Campos ; Souza, Marina de Assis; Muxel, Sandra Marcia ; Santos, Kamila Reis ; Lagos, Dimitris; Pereira, Valéria Rêgo Alves; Brito, Maria Edileuza Felinto de; Kaye, Paul Martin; Floeter-Winter, Lucile Maria ; Goto, Hiro
Source: Frontiers in Plant Science. Unidades: ICB, IB, FM, IMT, FMVZ
Subjects: LEISHMANIOSE VISCERAL, EXPRESSÃO GÊNICA, RNA, DOENÇAS PARASITÁRIAS
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RAMOS-SANCHEZ, Eduardo Milton et al. miR-548d-3p is up-regulated in human visceral leishmaniasis and suppresses parasite growth in macrophages. Frontiers in Plant Science, v. 12, 2022Tradução . . Disponível em: https://doi.org/10.3389/fcimb.2022.826039. Acesso em: 05 nov. 2024.
APA
Ramos-Sanchez, E. M., Reis, L. C., Souza, M. de A., Muxel, S. M., Santos, K. R., Lagos, D., et al. (2022). miR-548d-3p is up-regulated in human visceral leishmaniasis and suppresses parasite growth in macrophages. Frontiers in Plant Science, 12. doi:10.3389/fcimb.2022.826039
NLM
Ramos-Sanchez EM, Reis LC, Souza M de A, Muxel SM, Santos KR, Lagos D, Pereira VRA, Brito MEF de, Kaye PM, Floeter-Winter LM, Goto H. miR-548d-3p is up-regulated in human visceral leishmaniasis and suppresses parasite growth in macrophages [Internet]. Frontiers in Plant Science. 2022 ; 12[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcimb.2022.826039
Vancouver
Ramos-Sanchez EM, Reis LC, Souza M de A, Muxel SM, Santos KR, Lagos D, Pereira VRA, Brito MEF de, Kaye PM, Floeter-Winter LM, Goto H. miR-548d-3p is up-regulated in human visceral leishmaniasis and suppresses parasite growth in macrophages [Internet]. Frontiers in Plant Science. 2022 ; 12[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fcimb.2022.826039
Artigo de periodico
Extreme phenotypes approach to investigate host genetics and COVID-19 outcomes (2021)
Naslavsky, Michel ; Castro, Mateus Vidigal de ; Matos, Larissa do Rêgo Barros; Cória, Vivian Romanholi ; Batista Junior, Pedro Benedito; Razuk, Álvaro; Saldiva, Paulo Hilário Nascimento ; Dolhnikoff, Marisa ; Ferreira, Laire Schidlowski ; Prando, Carolina ; Cunha Neto, Edécio ; Condino Neto, Antonio ; Passos-Bueno, Maria Rita ; Zatz, Mayana
Source: Genetics and Molecular Biology. Unidades: IB, FM, ICB
Subjects: GENÔMICA, IMUNOLOGIA, COVID-19, INFECÇÕES POR VÍRUS DE RNA, INFECÇÕES POR CORONAVIRUS, HOSPEDEIRO IMUNOCOMPROMETIDO
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NASLAVSKY, Michel et al. Extreme phenotypes approach to investigate host genetics and COVID-19 outcomes. Genetics and Molecular Biology, v. 44, n. 1, p. 8 , 2021Tradução . . Disponível em: https://doi.org/10.1590/1678-4685-gmb-2020-0302. Acesso em: 05 nov. 2024.
APA
Naslavsky, M., Castro, M. V. de, Matos, L. do R. B., Cória, V. R., Batista Junior, P. B., Razuk, Á., et al. (2021). Extreme phenotypes approach to investigate host genetics and COVID-19 outcomes. Genetics and Molecular Biology, 44( 1), 8 . doi:10.1590/1678-4685-gmb-2020-0302
NLM
Naslavsky M, Castro MV de, Matos L do RB, Cória VR, Batista Junior PB, Razuk Á, Saldiva PHN, Dolhnikoff M, Ferreira LS, Prando C, Cunha Neto E, Condino Neto A, Passos-Bueno MR, Zatz M. Extreme phenotypes approach to investigate host genetics and COVID-19 outcomes [Internet]. Genetics and Molecular Biology. 2021 ; 44( 1): 8 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1590/1678-4685-gmb-2020-0302
Vancouver
Naslavsky M, Castro MV de, Matos L do RB, Cória VR, Batista Junior PB, Razuk Á, Saldiva PHN, Dolhnikoff M, Ferreira LS, Prando C, Cunha Neto E, Condino Neto A, Passos-Bueno MR, Zatz M. Extreme phenotypes approach to investigate host genetics and COVID-19 outcomes [Internet]. Genetics and Molecular Biology. 2021 ; 44( 1): 8 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1590/1678-4685-gmb-2020-0302
Artigo de periodico
An immunometabolic shift modulates cytotoxic lymphocyte activation during melanoma progression in TRPA1 channel null mice (2021)
Forni, Maria Fernanda Pereira de Araujo Demonte ; Amorocho, Omar Alberto Domínguez ; Assis, Leonardo Vinícius Monteiro de ; Kinker, Gabriela Sarti ; Borges, Maria Nathalia de Carvalho Magalhães Moraes Figueira ; Castrucci, Ana Maria de Lauro ; Câmara, Niels Olsen Saraiva
Source: Frontiers in Oncology. Unidades: IB, ICB
Subjects: IMUNOLOGIA, MELANOMA, MELANOMA ANIMAL, METABOLISMO ANIMAL, LINFÓCITOS, CITOTOXINAS, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS
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FORNI, Maria Fernanda Pereira de Araujo Demonte et al. An immunometabolic shift modulates cytotoxic lymphocyte activation during melanoma progression in TRPA1 channel null mice. Frontiers in Oncology, v. 11, p. 1-14, 2021Tradução . . Disponível em: https://doi.org/10.3389/fonc.2021.667715. Acesso em: 05 nov. 2024.
APA
Forni, M. F. P. de A. D., Amorocho, O. A. D., Assis, L. V. M. de, Kinker, G. S., Borges, M. N. de C. M. M. F., Castrucci, A. M. de L., & Câmara, N. O. S. (2021). An immunometabolic shift modulates cytotoxic lymphocyte activation during melanoma progression in TRPA1 channel null mice. Frontiers in Oncology, 11, 1-14. doi:10.3389/fonc.2021.667715
NLM
Forni MFP de AD, Amorocho OAD, Assis LVM de, Kinker GS, Borges MN de CMMF, Castrucci AM de L, Câmara NOS. An immunometabolic shift modulates cytotoxic lymphocyte activation during melanoma progression in TRPA1 channel null mice [Internet]. Frontiers in Oncology. 2021 ; 11 1-14.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fonc.2021.667715
Vancouver
Forni MFP de AD, Amorocho OAD, Assis LVM de, Kinker GS, Borges MN de CMMF, Castrucci AM de L, Câmara NOS. An immunometabolic shift modulates cytotoxic lymphocyte activation during melanoma progression in TRPA1 channel null mice [Internet]. Frontiers in Oncology. 2021 ; 11 1-14.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3389/fonc.2021.667715
Artigo de periodico-carta/editorial
Author Correction: a global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection (2021)
Andreakos, Evangelos ; Abel, Laurent ; Vinh, Donald C. ; Kaja, Elzbieta ; Drolet, Beth A. ; Zhang, Qian ; O\2019Farrelly, Cliona ; Novelli, Giuseppe ; Gallego, Carlos Rodríguez ; Haerynck, Filomeen ; Prando, Carolina ; Pujol, Aurora ; Condino Neto, Antonio ; Su, Helen C. ; Casanova, Jean-Laurent ; Spaan, András N. ; Zatz, Mayana
Source: Nature Immunology. Unidades: ICB, IB
Subjects: IMUNOLOGIA, INFECÇÕES POR CORONAVIRUS, CORONAVIRUS, GENÉTICA MÉDICA
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ANDREAKOS, Evangelos et al. Author Correction: a global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection. Nature Immunology. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1038/s41590-021-01096-9. Acesso em: 05 nov. 2024. , 2021
APA
Andreakos, E., Abel, L., Vinh, D. C., Kaja, E., Drolet, B. A., Zhang, Q., et al. (2021). Author Correction: a global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection. Nature Immunology. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1038/s41590-021-01096-9
NLM
Andreakos E, Abel L, Vinh DC, Kaja E, Drolet BA, Zhang Q, O\2019Farrelly C, Novelli G, Gallego CR, Haerynck F, Prando C, Pujol A, Condino Neto A, Su HC, Casanova J-L, Spaan AN, Zatz M. Author Correction: a global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection [Internet]. Nature Immunology. 2021 ;1 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1038/s41590-021-01096-9
Vancouver
Andreakos E, Abel L, Vinh DC, Kaja E, Drolet BA, Zhang Q, O\2019Farrelly C, Novelli G, Gallego CR, Haerynck F, Prando C, Pujol A, Condino Neto A, Su HC, Casanova J-L, Spaan AN, Zatz M. Author Correction: a global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection [Internet]. Nature Immunology. 2021 ;1 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1038/s41590-021-01096-9
Artigo de periodico
A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection (2021)
Andreakos, Evangelos ; Abel, Laurent ; Vinh, Donald C. ; Kaja, Elzbieta ; Drolet, Beth A. ; Zhang, Qian ; O\2019Farrelly, Cliona ; Novelli, Giuseppe ; Gallego, Carlos Rodríguez ; Haerynck, Filomeen ; Prando, Carolina ; Pujol, Aurora ; Condino Neto, Antonio ; Zatz, Mayana
Source: Nature Immunology. Unidades: ICB, IB
Subjects: IMUNOLOGIA, INFECÇÕES POR CORONAVIRUS, GENÉTICA MÉDICA, INTERFERON TIPO I, QUIMIOCINAS, PLASMODIUM
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANDREAKOS, Evangelos et al. A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection. Nature Immunology, p. 1-6, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41590-021-01030-z. Acesso em: 05 nov. 2024.
APA
Andreakos, E., Abel, L., Vinh, D. C., Kaja, E., Drolet, B. A., Zhang, Q., et al. (2021). A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection. Nature Immunology, 1-6. doi:10.1038/s41590-021-01030-z
NLM
Andreakos E, Abel L, Vinh DC, Kaja E, Drolet BA, Zhang Q, O\2019Farrelly C, Novelli G, Gallego CR, Haerynck F, Prando C, Pujol A, Condino Neto A, Zatz M. A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection [Internet]. Nature Immunology. 2021 ; 1-6.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1038/s41590-021-01030-z
Vancouver
Andreakos E, Abel L, Vinh DC, Kaja E, Drolet BA, Zhang Q, O\2019Farrelly C, Novelli G, Gallego CR, Haerynck F, Prando C, Pujol A, Condino Neto A, Zatz M. A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection [Internet]. Nature Immunology. 2021 ; 1-6.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1038/s41590-021-01030-z
Artigo de periodico
Autoantibodies neutralizing type I IFNs are present in ~4% of uninfected individuals over 70 years old and account for ~20% of COVID-19 deaths (2021)
Bastard, Paul ; Gervais, Adrian ; Voyer, Tom Le ; Rosain, Jérémie ; Philippot, Quentin; Manry, Jérémy ; Michailidis, Eleftherios ; Hoffmann, Hans-Heinrich ; Eto, Shohei; Garcia-Prat, Marina; Bizien, Lucy; Parra-Martínez, Alba; Yang, Rui; Liis, Haljasmägi; Migaud, Mélanie; Särekannu, Karita; Maslovskaja, Julia; Prost, Nicolas de; Tandjaoui-Lambiotte, Yacine; Luyt , Charles-Edouard; Amador-Borrero, Blanca; Alexandre, Gaudet; Poissy, Julien; Morel, Pascal; Richard, Pascale; Cognasse, Fabrice; Troya, Jesus; Trouillet-Assant, Sophie; Belot, Alexandre; Saker, Kahina; Garçon, Pierre; Rivière, Jacques G; Lagier, Jean-Christophe; Gentile, Stéphanie; Rosen, Lindsey B; Shaw, Elana; Morio, Tomohiro; Tanaka, Junko; Dalmau, David; Tharaux, Pierre-Louis; Sene, Damien; Stepanian, Alain; Megarbane, Bruno; Triantafyllia, Vasiliki; Fekkar, Arnaud; Heath, James R; Franco, José Luis; Anaya, Juan-Manuel; Solé-Violán, Jordi; Imberti, Luisa; Biondi, Andrea; Bonfanti, Paolo; Castagnoli, Riccardo; Delmonte, Ottavia M; Zhang, Yu; Snow, Andrew L; Holland, Steven M; Biggs, Catherine M; Moncada-Vélez, Marcela; Arias, Andrés Augusto; Lorenzo, Lazaro; Boucherit, Soraya; Coulibaly, Boubacar; Anglicheau, Dany; Planas, Anna M; Haerynck, Filomeen; Duvlis, Sotirija; Nussbaum, Robert L; Ozcelik, Tayfun; Keles, Sevgi; Bousfiha, Ahmed A; ElBakkouri, Jalila; Ramirez-Santana, Carolina; Paul, Stéphane; Pan-Hammarström, Qiang; Hammarström, Lennart; Dupont, Annabelle; Kurolap, Alina; Metz, Christine N; Aiuti, Alessandro; Casari, Giorgio; Lampasona, Vito; Ciceri, Fabio; Barreiros, Lucila A; Dominguez-Garrido, Elena; Vidigal, Mateus; Zatz, Mayana ; Beek, Diederik van de; Sahanic, Sabina; Tancevski, Ivan; Stepanovskyy, Yurii; Boyarchuk, Oksana; Nukui, Yoko; Tsumura, Miyuki; Vidaur, Loreto; Tangye, Stuart G; Burrel, Sonia; Duffy, Darragh; Quintana-Murci, Lluis; Klocperk, Adam; Kann, Nelli Y; Shcherbina, Anna; Lau, Yu-Lung; Leung, Daniel; Coulongeat, Matthieu; Marlet, Julien; Koning, Rutger; Reyes, Luis Felipe; Chauvineau-Grenier, Angélique; Vene, Fabienne; Monneret, Guillaume; Nussenzweig, Michel C; Arrestier, Romain; Boudhabhay, Idris; Baris-Feldman, Hagit; Hagin, David; Wauters, Joost; Meyts, Isabelle; Dyer, Adam H; Kennelly, Sean P; Bourke, Nollaig M; Halwani, Rabih; Sharif-Askari, Narjes Saheb; Dorgham, Karim; Sallette, Jérome; Sedkaoui, Souad Mehlal; AlKhater, Suzan; Rigo-Bonnin, Raúl; Morandeira, Francisco; Roussel, Lucie; Vinh, Donald C; Ostrowski, Sisse Rye; Condino Neto, Antonio ; Prando, Carolina ; Bondarenko, Anastasiia; Spaan, András N; Gilardin, Laurent; Fellay, Jacques; Lyonnet, Stanislas; Bilguvar, Kaya; Lifton, Richard P; Mane, Shrikant; Anderson, Mark S; Boisson, Bertrand; Béziat, Vivien; Zhang, Shen-Ying; Andreakos, Evangelos; Hermine, Olivier; Pujol, Aurora; Peterson, Pärt; Mogensen, Trine; H Rowen, Lee; Mond, James; Debette, Stéphanie; Lamballerie, Xavier de; Duval, Xavier; Mentré, France; Zins, Marie; Soler-Palacin, Pere; Colobran, Roger; Gorochov, Guy; Solanich, Xavier; Susen, Sophie; Martinez-Picado, Javier; Raoult, Didier; Vasse, Marc; Gregersen, Peter K; Piemonti, Lorenzo; Rodríguez-Gallego, Carlos; Notarangelo, Luigi D; Su, Helen C; Kisand, Kai; Okada, Satoshi; Puel, Anne; Jouanguy, Emmanuelle; Rice, Charles M; Tiberghien, Pierre; Zhang, Qian; Cobat, Aurélie; Abel, Laurent; Casanova, Jean-Laurent
Source: Science Immunology. Unidades: IB, ICB
Subjects: COVID-19, INFECÇÕES POR CORONAVIRUS, ANTICORPOS, IMUNOLOGIA, HOMENS
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ABNT
BASTARD, Paul et al. Autoantibodies neutralizing type I IFNs are present in ~4% of uninfected individuals over 70 years old and account for ~20% of COVID-19 deaths. Science Immunology, v. 6, n. 62, p. 1-30, 2021Tradução . . Disponível em: https://doi.org/10.1126/sciimmunol.abl4340. Acesso em: 05 nov. 2024.
APA
Bastard, P., Gervais, A., Voyer, T. L., Rosain, J., Philippot, Q., Manry, J., et al. (2021). Autoantibodies neutralizing type I IFNs are present in ~4% of uninfected individuals over 70 years old and account for ~20% of COVID-19 deaths. Science Immunology, 6( 62), 1-30. doi:10.1126/sciimmunol.abl4340
NLM
Bastard P, Gervais A, Voyer TL, Rosain J, Philippot Q, Manry J, Michailidis E, Hoffmann H-H, Eto S, Garcia-Prat M, Bizien L, Parra-Martínez A, Yang R, Liis H, Migaud M, Särekannu K, Maslovskaja J, Prost N de, Tandjaoui-Lambiotte Y, Luyt C-E, Amador-Borrero B, Alexandre G, Poissy J, Morel P, Richard P, Cognasse F, Troya J, Trouillet-Assant S, Belot A, Saker K, Garçon P, Rivière JG, Lagier J-C, Gentile S, Rosen LB, Shaw E, Morio T, Tanaka J, Dalmau D, Tharaux P-L, Sene D, Stepanian A, Megarbane B, Triantafyllia V, Fekkar A, Heath JR, Franco JL, Anaya J-M, Solé-Violán J, Imberti L, Biondi A, Bonfanti P, Castagnoli R, Delmonte OM, Zhang Y, Snow AL, Holland SM, Biggs CM, Moncada-Vélez M, Arias AA, Lorenzo L, Boucherit S, Coulibaly B, Anglicheau D, Planas AM, Haerynck F, Duvlis S, Nussbaum RL, Ozcelik T, Keles S, Bousfiha AA, ElBakkouri J, Ramirez-Santana C, Paul S, Pan-Hammarström Q, Hammarström L, Dupont A, Kurolap A, Metz CN, Aiuti A, Casari G, Lampasona V, Ciceri F, Barreiros LA, Dominguez-Garrido E, Vidigal M, Zatz M, Beek D van de, Sahanic S, Tancevski I, Stepanovskyy Y, Boyarchuk O, Nukui Y, Tsumura M, Vidaur L, Tangye SG, Burrel S, Duffy D, Quintana-Murci L, Klocperk A, Kann NY, Shcherbina A, Lau Y-L, Leung D, Coulongeat M, Marlet J, Koning R, Reyes LF, Chauvineau-Grenier A, Vene F, Monneret G, Nussenzweig MC, Arrestier R, Boudhabhay I, Baris-Feldman H, Hagin D, Wauters J, Meyts I, Dyer AH, Kennelly SP, Bourke NM, Halwani R, Sharif-Askari NS, Dorgham K, Sallette J, Sedkaoui SM, AlKhater S, Rigo-Bonnin R, Morandeira F, Roussel L, Vinh DC, Ostrowski SR, Condino Neto A, Prando C, Bondarenko A, Spaan AN, Gilardin L, Fellay J, Lyonnet S, Bilguvar K, Lifton RP, Mane S, Anderson MS, Boisson B, Béziat V, Zhang S-Y, Andreakos E, Hermine O, Pujol A, Peterson P, Mogensen T, H Rowen L, Mond J, Debette S, Lamballerie X de, Duval X, Mentré F, Zins M, Soler-Palacin P, Colobran R, Gorochov G, Solanich X, Susen S, Martinez-Picado J, Raoult D, Vasse M, Gregersen PK, Piemonti L, Rodríguez-Gallego C, Notarangelo LD, Su HC, Kisand K, Okada S, Puel A, Jouanguy E, Rice CM, Tiberghien P, Zhang Q, Cobat A, Abel L, Casanova J-L. Autoantibodies neutralizing type I IFNs are present in ~4% of uninfected individuals over 70 years old and account for ~20% of COVID-19 deaths [Internet]. Science Immunology. 2021 ; 6( 62): 1-30.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1126/sciimmunol.abl4340
Vancouver
Bastard P, Gervais A, Voyer TL, Rosain J, Philippot Q, Manry J, Michailidis E, Hoffmann H-H, Eto S, Garcia-Prat M, Bizien L, Parra-Martínez A, Yang R, Liis H, Migaud M, Särekannu K, Maslovskaja J, Prost N de, Tandjaoui-Lambiotte Y, Luyt C-E, Amador-Borrero B, Alexandre G, Poissy J, Morel P, Richard P, Cognasse F, Troya J, Trouillet-Assant S, Belot A, Saker K, Garçon P, Rivière JG, Lagier J-C, Gentile S, Rosen LB, Shaw E, Morio T, Tanaka J, Dalmau D, Tharaux P-L, Sene D, Stepanian A, Megarbane B, Triantafyllia V, Fekkar A, Heath JR, Franco JL, Anaya J-M, Solé-Violán J, Imberti L, Biondi A, Bonfanti P, Castagnoli R, Delmonte OM, Zhang Y, Snow AL, Holland SM, Biggs CM, Moncada-Vélez M, Arias AA, Lorenzo L, Boucherit S, Coulibaly B, Anglicheau D, Planas AM, Haerynck F, Duvlis S, Nussbaum RL, Ozcelik T, Keles S, Bousfiha AA, ElBakkouri J, Ramirez-Santana C, Paul S, Pan-Hammarström Q, Hammarström L, Dupont A, Kurolap A, Metz CN, Aiuti A, Casari G, Lampasona V, Ciceri F, Barreiros LA, Dominguez-Garrido E, Vidigal M, Zatz M, Beek D van de, Sahanic S, Tancevski I, Stepanovskyy Y, Boyarchuk O, Nukui Y, Tsumura M, Vidaur L, Tangye SG, Burrel S, Duffy D, Quintana-Murci L, Klocperk A, Kann NY, Shcherbina A, Lau Y-L, Leung D, Coulongeat M, Marlet J, Koning R, Reyes LF, Chauvineau-Grenier A, Vene F, Monneret G, Nussenzweig MC, Arrestier R, Boudhabhay I, Baris-Feldman H, Hagin D, Wauters J, Meyts I, Dyer AH, Kennelly SP, Bourke NM, Halwani R, Sharif-Askari NS, Dorgham K, Sallette J, Sedkaoui SM, AlKhater S, Rigo-Bonnin R, Morandeira F, Roussel L, Vinh DC, Ostrowski SR, Condino Neto A, Prando C, Bondarenko A, Spaan AN, Gilardin L, Fellay J, Lyonnet S, Bilguvar K, Lifton RP, Mane S, Anderson MS, Boisson B, Béziat V, Zhang S-Y, Andreakos E, Hermine O, Pujol A, Peterson P, Mogensen T, H Rowen L, Mond J, Debette S, Lamballerie X de, Duval X, Mentré F, Zins M, Soler-Palacin P, Colobran R, Gorochov G, Solanich X, Susen S, Martinez-Picado J, Raoult D, Vasse M, Gregersen PK, Piemonti L, Rodríguez-Gallego C, Notarangelo LD, Su HC, Kisand K, Okada S, Puel A, Jouanguy E, Rice CM, Tiberghien P, Zhang Q, Cobat A, Abel L, Casanova J-L. Autoantibodies neutralizing type I IFNs are present in ~4% of uninfected individuals over 70 years old and account for ~20% of COVID-19 deaths [Internet]. Science Immunology. 2021 ; 6( 62): 1-30.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1126/sciimmunol.abl4340
Artigo de periodico
Host genetics contributes to the effectiveness of dendritic cell-based HIV immunotherapy (2018)
Reis, Edione Cristina dos; Silva, Lais Teodoro da; Silva, Wanessa Cardoso da; Rios, Alexandre; Duarte, Alberto José da Silva; Sumida, Telma Miyuki Oshiro; Crovella, Sergio; Pontillo, Alessandra
Source: Human vaccines & immunotherapeutics. Unidades: FM, ICB, IB
Subjects: HIV, IMUNOTERAPIA, IMUNOGENÉTICA, CÉLULAS DENDRÍTICAS, POLIMORFISMO, IMUNIZAÇÃO
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ABNT
REIS, Edione Cristina dos et al. Host genetics contributes to the effectiveness of dendritic cell-based HIV immunotherapy. Human vaccines & immunotherapeutics, v. 14, n. 8, p. 1995-2002, 2018Tradução . . Disponível em: https://doi.org/10.1080/21645515.2018.1463942. Acesso em: 05 nov. 2024.
APA
Reis, E. C. dos, Silva, L. T. da, Silva, W. C. da, Rios, A., Duarte, A. J. da S., Sumida, T. M. O., et al. (2018). Host genetics contributes to the effectiveness of dendritic cell-based HIV immunotherapy. Human vaccines & immunotherapeutics, 14( 8), 1995-2002. doi:10.1080/21645515.2018.1463942
NLM
Reis EC dos, Silva LT da, Silva WC da, Rios A, Duarte AJ da S, Sumida TMO, Crovella S, Pontillo A. Host genetics contributes to the effectiveness of dendritic cell-based HIV immunotherapy [Internet]. Human vaccines & immunotherapeutics. 2018 ; 14( 8): 1995-2002.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1080/21645515.2018.1463942
Vancouver
Reis EC dos, Silva LT da, Silva WC da, Rios A, Duarte AJ da S, Sumida TMO, Crovella S, Pontillo A. Host genetics contributes to the effectiveness of dendritic cell-based HIV immunotherapy [Internet]. Human vaccines & immunotherapeutics. 2018 ; 14( 8): 1995-2002.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1080/21645515.2018.1463942
Artigo de jornal-dep/entr
Não se faz ciência sem responsabilidade e comprometimento. [Entrevista] (2017)
Peron, Jean Pierre Schatzmann ; Torres, Tatiana Teixeira
Source: Jornal da USP. Unidades: ICB, IB
Subjects: DIVULGAÇÃO CIENTÍFICA, CIÊNCIA, PESQUISA CIENTÍFICA
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PERON, Jean Pierre Schatzmann e TORRES, Tatiana Teixeira. Não se faz ciência sem responsabilidade e comprometimento. [Entrevista]. Jornal da USP. São Paulo: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: http://jornal.usp.br/atualidades/nao-se-faz-ciencia-sem-responsabilidade-e-comprometimento/. Acesso em: 05 nov. 2024. , 2017
APA
Peron, J. P. S., & Torres, T. T. (2017). Não se faz ciência sem responsabilidade e comprometimento. [Entrevista]. Jornal da USP. São Paulo: Instituto de Ciências Biomédicas, Universidade de São Paulo. Recuperado de http://jornal.usp.br/atualidades/nao-se-faz-ciencia-sem-responsabilidade-e-comprometimento/
NLM
Peron JPS, Torres TT. Não se faz ciência sem responsabilidade e comprometimento. [Entrevista] [Internet]. Jornal da USP. 2017 ;[citado 2024 nov. 05 ] Available from: http://jornal.usp.br/atualidades/nao-se-faz-ciencia-sem-responsabilidade-e-comprometimento/
Vancouver
Peron JPS, Torres TT. Não se faz ciência sem responsabilidade e comprometimento. [Entrevista] [Internet]. Jornal da USP. 2017 ;[citado 2024 nov. 05 ] Available from: http://jornal.usp.br/atualidades/nao-se-faz-ciencia-sem-responsabilidade-e-comprometimento/
Artigo de periodico
Immunoglobulin therapy ameliorates the phenotype and increases lifespan in the severely affected dystrophin-utrophin double knockout mice (2017)
Nunes, Bruno Ghirotto ; Loures, Flavio Vieira ; Bueno, Heloisa Maria Siqueira ; Cangussu, Erica Baroni; Goulart, Ernesto ; Coatti, Giuliana Castello ; Caldini, EliaTamaso Espin Garcia ; Condino Neto, Antonio ; Zatz, Mayana
Source: European Journal of Human Genetics. Unidades: FM, ICB, IB
Subjects: IMUNOLOGIA, SISTEMA IMUNE, IMUNOGLOBULINAS, DOENÇAS AUTOIMUNES, CAMUNDONGOS, FENÓTIPOS, DISTROFIA MUSCULAR, MUTAÇÃO GENÉTICA, CITOCINAS, SECREÇÃO, CÉLULAS DENDRÍTICAS, LINFÓCITOS
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NUNES, Bruno Ghirotto et al. Immunoglobulin therapy ameliorates the phenotype and increases lifespan in the severely affected dystrophin-utrophin double knockout mice. European Journal of Human Genetics, v. 25, n. 12, p. 1388-1396, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41431-017-0017-y. Acesso em: 05 nov. 2024.
APA
Nunes, B. G., Loures, F. V., Bueno, H. M. S., Cangussu, E. B., Goulart, E., Coatti, G. C., et al. (2017). Immunoglobulin therapy ameliorates the phenotype and increases lifespan in the severely affected dystrophin-utrophin double knockout mice. European Journal of Human Genetics, 25( 12), 1388-1396. doi:10.1038/s41431-017-0017-y
NLM
Nunes BG, Loures FV, Bueno HMS, Cangussu EB, Goulart E, Coatti GC, Caldini ETEG, Condino Neto A, Zatz M. Immunoglobulin therapy ameliorates the phenotype and increases lifespan in the severely affected dystrophin-utrophin double knockout mice [Internet]. European Journal of Human Genetics. 2017 ; 25( 12): 1388-1396.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1038/s41431-017-0017-y
Vancouver
Nunes BG, Loures FV, Bueno HMS, Cangussu EB, Goulart E, Coatti GC, Caldini ETEG, Condino Neto A, Zatz M. Immunoglobulin therapy ameliorates the phenotype and increases lifespan in the severely affected dystrophin-utrophin double knockout mice [Internet]. European Journal of Human Genetics. 2017 ; 25( 12): 1388-1396.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1038/s41431-017-0017-y
Artigo de periodico
Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity (2017)
Schimke, Lena F; Hibbard, James; Martinez-Barricarte, Ruben; Khan, Taj Ali; Cavalcante, Ricardo de Souza; Oliveira-Junior, Edgar Borges de; França, Tábata Takahashi; Iqbal, Asif; Yamamoto, Guilherme; Arslanian, Christina; Feriotti, Claudia; Costa, Tânia Alves; Bustamante, Jacinta; Boisson-Dupuis, Stéphanie; Casanova, Jean-Laurent; Barbuto, José Alexandre Marzagão ; Zatz, Mayana ; Mendes, Rinaldo Poncio; Calich, Vera Lúcia Garcia; Ochs, Hans D; Torgerson, Troy R; Cabral-Marques, Otavio; Condino Neto, Antonio
Source: Journal of Infectious Diseases. Unidades: ICB, IB
Subjects: PARACOCCIDIOIDOMICOSE, IMUNOLOGIA, MUTAÇÃO GENÉTICA, PREDISPOSIÇÃO GENÉTICA PARA DOENÇA, INFECÇÕES BACTERIANAS E MICOSES
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SCHIMKE, Lena F et al. Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity. Journal of Infectious Diseases, v. 216, n. 12, p. 1623-1634, 2017Tradução . . Disponível em: https://doi.org/10.1093/infdis/jix522. Acesso em: 05 nov. 2024.
APA
Schimke, L. F., Hibbard, J., Martinez-Barricarte, R., Khan, T. A., Cavalcante, R. de S., Oliveira-Junior, E. B. de, et al. (2017). Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity. Journal of Infectious Diseases, 216( 12), 1623-1634. doi:10.1093/infdis/jix522
NLM
Schimke LF, Hibbard J, Martinez-Barricarte R, Khan TA, Cavalcante R de S, Oliveira-Junior EB de, França TT, Iqbal A, Yamamoto G, Arslanian C, Feriotti C, Costa TA, Bustamante J, Boisson-Dupuis S, Casanova J-L, Barbuto JAM, Zatz M, Mendes RP, Calich VLG, Ochs HD, Torgerson TR, Cabral-Marques O, Condino Neto A. Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity [Internet]. Journal of Infectious Diseases. 2017 ; 216( 12): 1623-1634.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1093/infdis/jix522
Vancouver
Schimke LF, Hibbard J, Martinez-Barricarte R, Khan TA, Cavalcante R de S, Oliveira-Junior EB de, França TT, Iqbal A, Yamamoto G, Arslanian C, Feriotti C, Costa TA, Bustamante J, Boisson-Dupuis S, Casanova J-L, Barbuto JAM, Zatz M, Mendes RP, Calich VLG, Ochs HD, Torgerson TR, Cabral-Marques O, Condino Neto A. Paracoccidioidomycosis Associated With a Heterozygous STAT4 Mutation and Impaired IFN-γ Immunity [Internet]. Journal of Infectious Diseases. 2017 ; 216( 12): 1623-1634.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1093/infdis/jix522
Trabalho de evento-resumo
Caloric restriction promotes remodeling as a thermoregulatory mechanism (2016)
Forni, Maria Fernanda; Peloggia, Julia; Braga, Tárcio Teodoro; Chinchilla, Jesus Eduardo Ortega; Navas, Carlos ; Câmara, Niels Olsen Saraiva ; Kowaltowski, Alicia Juliana
Source: Abstracts. Conference titles: Annual Meeting of the Brazilian Society for Biochemistry and Molecular Biology (SBBq). Unidades: IB, ICB, IQ
Subjects: DIETA, CÉLULAS-TRONCO
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ABNT
FORNI, Maria Fernanda et al. Caloric restriction promotes remodeling as a thermoregulatory mechanism. 2016, Anais.. Natal: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2016. . Acesso em: 05 nov. 2024.
APA
Forni, M. F., Peloggia, J., Braga, T. T., Chinchilla, J. E. O., Navas, C., Câmara, N. O. S., & Kowaltowski, A. J. (2016). Caloric restriction promotes remodeling as a thermoregulatory mechanism. In Abstracts. Natal: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq).
NLM
Forni MF, Peloggia J, Braga TT, Chinchilla JEO, Navas C, Câmara NOS, Kowaltowski AJ. Caloric restriction promotes remodeling as a thermoregulatory mechanism. Abstracts. 2016 ;[citado 2024 nov. 05 ]
Vancouver
Forni MF, Peloggia J, Braga TT, Chinchilla JEO, Navas C, Câmara NOS, Kowaltowski AJ. Caloric restriction promotes remodeling as a thermoregulatory mechanism. Abstracts. 2016 ;[citado 2024 nov. 05 ]
Artigo de periodico
Human tubal-derived mesenchymal stromal cells associated with Low level laser therapy significantly reduces cigarette smoke–induced COPD in C57BL/6 mice (2015)
Peron, Jean Pierre Schatzmann ; Brito, Auriléia Aparecida de; Pelatti, Mayra; Brandao, Wesley Nogueira; Vitoretti, Luana Beatriz; Greiffo, Flávia Regina; Silveira, Elaine Cristina da; Oliveira-Junior, Manuel Carneiro; Maluf, Mariangela; Evangelista, Lucila; Halpern, Silvio; Nisenbaum, Marcelo Gil; Perin, Paulo; Czeresnia, Carlos Eduardo; Câmara, Niels Olsen Saraiva ; Aimbire, Flavio; Vieira, Rodolfo de Paula; Zatz, Mayana ; Oliveira, Ana Paula Ligeiro de
Source: PLOS ONE. Unidades: ICB, IB
Subjects: DOENÇA PULMONAR OBSTRUTIVA CRÔNICA, DOENÇAS INFECCIOSAS, CÉLULAS-TRONCO, FUMO, TERAPÊUTICA, IMUNOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PERON, Jean Pierre Schatzmann et al. Human tubal-derived mesenchymal stromal cells associated with Low level laser therapy significantly reduces cigarette smoke–induced COPD in C57BL/6 mice. PLOS ONE, v. 10, n. 8, p. e0136942, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0136942. Acesso em: 05 nov. 2024.
APA
Peron, J. P. S., Brito, A. A. de, Pelatti, M., Brandao, W. N., Vitoretti, L. B., Greiffo, F. R., et al. (2015). Human tubal-derived mesenchymal stromal cells associated with Low level laser therapy significantly reduces cigarette smoke–induced COPD in C57BL/6 mice. PLOS ONE, 10( 8), e0136942. doi:10.1371/journal.pone.0136942
NLM
Peron JPS, Brito AA de, Pelatti M, Brandao WN, Vitoretti LB, Greiffo FR, Silveira EC da, Oliveira-Junior MC, Maluf M, Evangelista L, Halpern S, Nisenbaum MG, Perin P, Czeresnia CE, Câmara NOS, Aimbire F, Vieira R de P, Zatz M, Oliveira APL de. Human tubal-derived mesenchymal stromal cells associated with Low level laser therapy significantly reduces cigarette smoke–induced COPD in C57BL/6 mice [Internet]. PLOS ONE. 2015 ; 10( 8): e0136942.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1371/journal.pone.0136942
Vancouver
Peron JPS, Brito AA de, Pelatti M, Brandao WN, Vitoretti LB, Greiffo FR, Silveira EC da, Oliveira-Junior MC, Maluf M, Evangelista L, Halpern S, Nisenbaum MG, Perin P, Czeresnia CE, Câmara NOS, Aimbire F, Vieira R de P, Zatz M, Oliveira APL de. Human tubal-derived mesenchymal stromal cells associated with Low level laser therapy significantly reduces cigarette smoke–induced COPD in C57BL/6 mice [Internet]. PLOS ONE. 2015 ; 10( 8): e0136942.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1371/journal.pone.0136942
Artigo de periodico
Simvastatin ameliorates experimental autoimmune encephalomyelitis by inhibiting Th1/Th17 response and cellular infiltration (2015)
Oliveira, Daniel May de; Oliveira, Enedina Maria Lobato de; Ferrari, Merari de Fátima Ramires ; Semedo, Patricia; Hiyane, Meire Ioshie; Cenedeze, Marcos Antonio; Pacheco-Silva, Alvaro; Câmara, Niels Olsen Saraiva ; Peron, Jean Pierre Schatzmann
Source: Inflammopharmacology. Unidades: IB, ICB
Subjects: DOENÇAS AUTOIMUNES, SISTEMA NERVOSO CENTRAL, ESCLEROSE MÚLTIPLA, INFLAMAÇÃO, ENCEFALOMIELITE ANIMAL, PROLIFERAÇÃO CELULAR, FÁRMACOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Daniel May de et al. Simvastatin ameliorates experimental autoimmune encephalomyelitis by inhibiting Th1/Th17 response and cellular infiltration. Inflammopharmacology, v. 23, n. 6, p. 343-354, 2015Tradução . . Disponível em: https://doi.org/10.1007/s10787-015-0252-1. Acesso em: 05 nov. 2024.
APA
Oliveira, D. M. de, Oliveira, E. M. L. de, Ferrari, M. de F. R., Semedo, P., Hiyane, M. I., Cenedeze, M. A., et al. (2015). Simvastatin ameliorates experimental autoimmune encephalomyelitis by inhibiting Th1/Th17 response and cellular infiltration. Inflammopharmacology, 23( 6), 343-354. doi:10.1007/s10787-015-0252-1
NLM
Oliveira DM de, Oliveira EML de, Ferrari M de FR, Semedo P, Hiyane MI, Cenedeze MA, Pacheco-Silva A, Câmara NOS, Peron JPS. Simvastatin ameliorates experimental autoimmune encephalomyelitis by inhibiting Th1/Th17 response and cellular infiltration [Internet]. Inflammopharmacology. 2015 ; 23( 6): 343-354.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1007/s10787-015-0252-1
Vancouver
Oliveira DM de, Oliveira EML de, Ferrari M de FR, Semedo P, Hiyane MI, Cenedeze MA, Pacheco-Silva A, Câmara NOS, Peron JPS. Simvastatin ameliorates experimental autoimmune encephalomyelitis by inhibiting Th1/Th17 response and cellular infiltration [Internet]. Inflammopharmacology. 2015 ; 23( 6): 343-354.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1007/s10787-015-0252-1
Artigo de periodico
Human endometrial-derived mesenchymal stem cells suppress inflammation in the central nervous system of EAE mice (2012)
Peron, Jean Pierre Schatzmann ; Jazedje, T; Brandão, W. N; Perin, P M; Maluf, M; Evangelista, L P; Halpern, S; Nisenbaum, M. G; Czeresnia, C E; Zatz, Mayana ; Câmara, Niels Olsen Saraiva ; Rizzo, L. V
Source: Stem Cell Reviews and Reports. Unidades: IB, ICB
Subjects: CÉLULAS-TRONCO, SISTEMA NERVOSO CENTRAL, INFLAMAÇÃO, RATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PERON, Jean Pierre Schatzmann et al. Human endometrial-derived mesenchymal stem cells suppress inflammation in the central nervous system of EAE mice. Stem Cell Reviews and Reports, 2012Tradução . . Disponível em: https://doi.org/10.1007/s12015-011-9338-3. Acesso em: 05 nov. 2024.
APA
Peron, J. P. S., Jazedje, T., Brandão, W. N., Perin, P. M., Maluf, M., Evangelista, L. P., et al. (2012). Human endometrial-derived mesenchymal stem cells suppress inflammation in the central nervous system of EAE mice. Stem Cell Reviews and Reports. doi:10.1007/s12015-011-9338-3
NLM
Peron JPS, Jazedje T, Brandão WN, Perin PM, Maluf M, Evangelista LP, Halpern S, Nisenbaum MG, Czeresnia CE, Zatz M, Câmara NOS, Rizzo LV. Human endometrial-derived mesenchymal stem cells suppress inflammation in the central nervous system of EAE mice [Internet]. Stem Cell Reviews and Reports. 2012 ;[citado 2024 nov. 05 ] Available from: https://doi.org/10.1007/s12015-011-9338-3
Vancouver
Peron JPS, Jazedje T, Brandão WN, Perin PM, Maluf M, Evangelista LP, Halpern S, Nisenbaum MG, Czeresnia CE, Zatz M, Câmara NOS, Rizzo LV. Human endometrial-derived mesenchymal stem cells suppress inflammation in the central nervous system of EAE mice [Internet]. Stem Cell Reviews and Reports. 2012 ;[citado 2024 nov. 05 ] Available from: https://doi.org/10.1007/s12015-011-9338-3

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