A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection (2021)
- Authors:
- USP affiliated authors: CONDINO NETO, ANTONIO - ICB ; ZATZ, MAYANA - IB
- Unidades: ICB; IB
- DOI: 10.1038/s41590-021-01030-z
- Subjects: IMUNOLOGIA; INFECÇÕES POR CORONAVIRUS; GENÉTICA MÉDICA; INTERFERON TIPO I; QUIMIOCINAS; PLASMODIUM
- Agências de fomento:
- Financiamento NIH
- Financiamento National Center for Advancing Translational Sciences
- Financiamento NIH Clinical and Translational Science Award
- Financiamento NHGRI
- Financiamento Fisher Center for Alzheimer’s Research Foundation
- Financiamento Meyer Foundation
- Financiamento ANR
- Financiamento Integrative Biology of Emerging Infectious Diseases Laboratory of Excellence
- Financiamento French Foundation for Medical Research/FRM
- Financiamento Fondation du Souffle
- Financiamento Square Foundation
- Financiamento Grandir - Fonds de solidarité pour l’enfance
- Financiamento SCOR Corporate Foundation for Science
- Financiamento Howard Hughes Medical Institute
- Financiamento Rockefeller University
- Financiamento Giles Foundation
- Financiamento Institut National de la Santé et de la Recherche Médicale
- Financiamento European Commission’s Horizon 2020
- Financiamento CURE
- Financiamento TO_AITION
- Financiamento Hellenic Foundation for Research and Innovation
- Financiamento Science Foundation Ireland COVID-19 Program
- Financiamento Regione Lazio
- Financiamento GecoBiomark
- Financiamento CERCA Program/Generalitat de Catalunya
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Nature Immunology
- ISSN: 1529-2916
- Volume/Número/Paginação/Ano: p. 1-6, 2021
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: other-oa
-
ABNT
ANDREAKOS, Evangelos et al. A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection. Nature Immunology, p. 1-6, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41590-021-01030-z. Acesso em: 23 abr. 2024. -
APA
Andreakos, E., Abel, L., Vinh, D. C., Kaja, E., Drolet, B. A., Zhang, Q., et al. (2021). A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection. Nature Immunology, 1-6. doi:10.1038/s41590-021-01030-z -
NLM
Andreakos E, Abel L, Vinh DC, Kaja E, Drolet BA, Zhang Q, O’Farrelly C, Novelli G, Gallego CR, Haerynck F, Prando C, Pujol A, Condino Neto A, Zatz M. A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection [Internet]. Nature Immunology. 2021 ; 1-6.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1038/s41590-021-01030-z -
Vancouver
Andreakos E, Abel L, Vinh DC, Kaja E, Drolet BA, Zhang Q, O’Farrelly C, Novelli G, Gallego CR, Haerynck F, Prando C, Pujol A, Condino Neto A, Zatz M. A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection [Internet]. Nature Immunology. 2021 ; 1-6.[citado 2024 abr. 23 ] Available from: https://doi.org/10.1038/s41590-021-01030-z - Author Correction: a global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection
- The risk of COVID-19 death is much greater and age dependent with type I IFN autoantibodies
- Correction: Rare predicted loss-of-function variants of type I IFN immunity genes are associated with life-threatening COVID-19
- Rare predicted loss-of-function variants of type I IFN immunity genes are associated with life-threatening COVID-19
- Anhidrotic ectodermal dysplasia and T cell immunodeficiency
- BAY 41-2272 aumenta a atividade microbicida in vitro de leucócitos mononucleares humanos
- The role of nuclear factor kappa B (NF-kB) on the NCF1 gene expression
- Essential role of nuclear factor-kappa B for NADPH oxidase activity in normal and anhidrotic ectodermal dysplasia leukocytes
- A single nucleotide polymorphism in the promoter region of the NCF-2 gene
- Essential role of nuclear factor-kappa B for NADPH oxidase activity, CYBB and NCF-1 gene expression in normal and anhidrotic ectodermal dysplasia leukocytes
Informações sobre o DOI: 10.1038/s41590-021-01030-z (Fonte: oaDOI API)
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