ReP USP - Resultado da busca

Artigo de periodico
DNA lesions that block transcription induce the death of Trypanosoma cruzi via ATR activation, which is dependent on the presence of R-loops (2024)
Mendes, Isabela Cecilia ; Bertoldo, Willian dos Reis; Miranda-Junior, Adalberto Sales; Assis, Antônio Vinícius de; Repolês, Bruno Marçal ; Ferreira, Wesley Roger Rodrigues ; Chame, Daniela Ferreira ; Souza, Daniela De Laet ; Pavani, Raphael Souza ; Macedo, Andrea Mara ; Franco, Glória Regina ; Serra, Esteban; Perdomo, Virginia ; Leandro, Giovana da Silva ; Fragoso, Stenio Perdigão ; Elias, Maria Carolina Quartim Barbosa; Machado, Carlos Renato; Menck, Carlos Frederico Martins
Source: DNA Repair. Unidade: ICB
Subjects: MICROBIOLOGIA, DNA POLIMERASES, DANO AO DNA, REPARAÇÃO DE DNA, RADIAÇÃO ULTRAVIOLETA, APOPTOSE, NUCLEOTÍDEOS, TRYPANOSOMA CRUZI
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MENDES, Isabela Cecilia et al. DNA lesions that block transcription induce the death of Trypanosoma cruzi via ATR activation, which is dependent on the presence of R-loops. DNA Repair, v. 141 , p. 19 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.dnarep.2024.103726. Acesso em: 06 nov. 2024.
APA
Mendes, I. C., Bertoldo, W. dos R., Miranda-Junior, A. S., Assis, A. V. de, Repolês, B. M., Ferreira, W. R. R., et al. (2024). DNA lesions that block transcription induce the death of Trypanosoma cruzi via ATR activation, which is dependent on the presence of R-loops. DNA Repair, 141 , 19 . doi:https://doi.org/10.1016/j.dnarep.2024.103726
NLM
Mendes IC, Bertoldo W dos R, Miranda-Junior AS, Assis AV de, Repolês BM, Ferreira WRR, Chame DF, Souza DDL, Pavani RS, Macedo AM, Franco GR, Serra E, Perdomo V, Leandro G da S, Fragoso SP, Elias MCQB, Machado CR, Menck CFM. DNA lesions that block transcription induce the death of Trypanosoma cruzi via ATR activation, which is dependent on the presence of R-loops [Internet]. DNA Repair. 2024 ; 141 19 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.dnarep.2024.103726
Vancouver
Mendes IC, Bertoldo W dos R, Miranda-Junior AS, Assis AV de, Repolês BM, Ferreira WRR, Chame DF, Souza DDL, Pavani RS, Macedo AM, Franco GR, Serra E, Perdomo V, Leandro G da S, Fragoso SP, Elias MCQB, Machado CR, Menck CFM. DNA lesions that block transcription induce the death of Trypanosoma cruzi via ATR activation, which is dependent on the presence of R-loops [Internet]. DNA Repair. 2024 ; 141 19 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.dnarep.2024.103726
Artigo de periodico
Polymerase iota plays a key role during translesion synthesis of UV-induced lesions in the absence of polymerase eta (2024)
Martins, Davi Jardim ; Singh, Jenny Kaur ; Vessoni, Alexandre Teixeira ; Leandro, Giovana da Silva ; Silva, Matheus Molina ; Biard, Denis Serge François ; Quinet, Annabel ; Menck, Carlos Frederico Martins
Source: Photochemistry and Photobiology. Unidade: ICB
Subjects: MICROBIOLOGIA, DANO AO DNA, REPLICAÇÃO DO DNA, REPARAÇÃO DE DNA, DNA POLIMERASES, DNA, FERIMENTOS E LESÕES, FIBROBLASTOS, RADIAÇÃO ULTRAVIOLETA, DOENÇAS GENÉTICAS, NEOPLASIAS CUTÂNEAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARTINS, Davi Jardim et al. Polymerase iota plays a key role during translesion synthesis of UV-induced lesions in the absence of polymerase eta. Photochemistry and Photobiology, v. 100, n. 1, p. 18 , 2024Tradução . . Disponível em: https://doi.org/10.1111/php.13879. Acesso em: 06 nov. 2024.
APA
Martins, D. J., Singh, J. K., Vessoni, A. T., Leandro, G. da S., Silva, M. M., Biard, D. S. F., et al. (2024). Polymerase iota plays a key role during translesion synthesis of UV-induced lesions in the absence of polymerase eta. Photochemistry and Photobiology, 100( 1), 18 . doi:10.1111/php.13879
NLM
Martins DJ, Singh JK, Vessoni AT, Leandro G da S, Silva MM, Biard DSF, Quinet A, Menck CFM. Polymerase iota plays a key role during translesion synthesis of UV-induced lesions in the absence of polymerase eta [Internet]. Photochemistry and Photobiology. 2024 ; 100( 1): 18 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.1111/php.13879
Vancouver
Martins DJ, Singh JK, Vessoni AT, Leandro G da S, Silva MM, Biard DSF, Quinet A, Menck CFM. Polymerase iota plays a key role during translesion synthesis of UV-induced lesions in the absence of polymerase eta [Internet]. Photochemistry and Photobiology. 2024 ; 100( 1): 18 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.1111/php.13879
Artigo de periodico
Human translesion DNA polymerases ι and κ mediate tolerance to temozolomide in MGMT-deficient glioblastoma cells (2024)
Latância, Marcela Teatin ; Leandro, Giovana da Silva ; Bastos, André Uchimura ; Moreno, Natália Cestari ; Ariwoola, Abu-Bakr Adetayo ; Martins, Davi Jardim ; Ashton, Nicholas William ; Ribeiro, Victoria Chaves ; Rocha, Clarissa Ribeiro Reily ; Woodgate, Roger ; Hoch, Nicolas Carlos ; Menck, Carlos Frederico Martins
Source: DNA Repair. Unidades: IQ, ICB, BIOTECNOLOGIA
Subjects: MICROBIOLOGIA, NEOPLASIAS CEREBRAIS, GLIOMA, DNA POLIMERASES, QUIMIOTERAPIA, ANTINEOPLÁSICOS, DANO AO DNA, CÉLULAS CULTIVADAS DE TUMOR, CITOMETRIA DE FLUXO, INIBIDORES DE ENZIMAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LATÂNCIA, Marcela Teatin et al. Human translesion DNA polymerases ι and κ mediate tolerance to temozolomide in MGMT-deficient glioblastoma cells. DNA Repair, v. 141, p. 13 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.dnarep.2024.103715. Acesso em: 06 nov. 2024.
APA
Latância, M. T., Leandro, G. da S., Bastos, A. U., Moreno, N. C., Ariwoola, A. -B. A., Martins, D. J., et al. (2024). Human translesion DNA polymerases ι and κ mediate tolerance to temozolomide in MGMT-deficient glioblastoma cells. DNA Repair, 141, 13 . doi:10.1016/j.dnarep.2024.103715
NLM
Latância MT, Leandro G da S, Bastos AU, Moreno NC, Ariwoola A-BA, Martins DJ, Ashton NW, Ribeiro VC, Rocha CRR, Woodgate R, Hoch NC, Menck CFM. Human translesion DNA polymerases ι and κ mediate tolerance to temozolomide in MGMT-deficient glioblastoma cells [Internet]. DNA Repair. 2024 ; 141 13 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.dnarep.2024.103715
Vancouver
Latância MT, Leandro G da S, Bastos AU, Moreno NC, Ariwoola A-BA, Martins DJ, Ashton NW, Ribeiro VC, Rocha CRR, Woodgate R, Hoch NC, Menck CFM. Human translesion DNA polymerases ι and κ mediate tolerance to temozolomide in MGMT-deficient glioblastoma cells [Internet]. DNA Repair. 2024 ; 141 13 .[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.dnarep.2024.103715
Artigo de periodico
Targeting PIP4K2 lipid kinases through Thz-P1-2 reveals vulnerabilities related to mitochondrial homeostasis and autophagy in acute leukemia (2022)
Lima, Keli ; Pereira-Martins Diego A. ; Miranda, Lívia Bassani Lins de ; Coelho-Silva, Juan L. ; Leandro, Giovana da Silva ; Isabel Weinhäuser ; Cavaglieri, Rita de Cássia ; Silva, Wellington F.; Leal, Aline Medeiros; Lange, Ana Paula Alencar de Lima; Velloso, Elvira Deolinda Rodrigues Pereira ; Griessinger, Emmanuel ; Hilberink, Jacobien R. ; Huls, Gerwin A. ; Schuringa, Jan Jacob ; Rego, Eduardo M. ; Machado Neto, João Agostinho
Source: Blood. Unidades: FM, ICB, FMRP
Subjects: FARMACOLOGIA, LEUCEMIA MIELOIDE AGUDA, NEOPLASIAS POR TIPO HISTOLÓGICO, AMINOÁCIDOS, PROTEÍNAS QUINASES, DANO AO DNA, MITOCÔNDRIAS, HOMEOSTASE, LISOSSOMOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Keli et al. Targeting PIP4K2 lipid kinases through Thz-P1-2 reveals vulnerabilities related to mitochondrial homeostasis and autophagy in acute leukemia. Blood, v. 140, p. 5965–5966, 2022Tradução . . Disponível em: https://doi.org/10.1182/blood-2022-165172. Acesso em: 06 nov. 2024.
APA
Lima, K., Pereira-Martins Diego A.,, Miranda, L. B. L. de, Coelho-Silva, J. L., Leandro, G. da S., Isabel Weinhäuser,, et al. (2022). Targeting PIP4K2 lipid kinases through Thz-P1-2 reveals vulnerabilities related to mitochondrial homeostasis and autophagy in acute leukemia. Blood, 140, 5965–5966. doi:10.1182/blood-2022-165172
NLM
Lima K, Pereira-Martins Diego A., Miranda LBL de, Coelho-Silva JL, Leandro G da S, Isabel Weinhäuser, Cavaglieri R de C, Silva WF, Leal AM, Lange APA de L, Velloso EDRP, Griessinger E, Hilberink JR, Huls GA, Schuringa JJ, Rego EM, Machado Neto JA. Targeting PIP4K2 lipid kinases through Thz-P1-2 reveals vulnerabilities related to mitochondrial homeostasis and autophagy in acute leukemia [Internet]. Blood. 2022 ; 140 5965–5966.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1182/blood-2022-165172
Vancouver
Lima K, Pereira-Martins Diego A., Miranda LBL de, Coelho-Silva JL, Leandro G da S, Isabel Weinhäuser, Cavaglieri R de C, Silva WF, Leal AM, Lange APA de L, Velloso EDRP, Griessinger E, Hilberink JR, Huls GA, Schuringa JJ, Rego EM, Machado Neto JA. Targeting PIP4K2 lipid kinases through Thz-P1-2 reveals vulnerabilities related to mitochondrial homeostasis and autophagy in acute leukemia [Internet]. Blood. 2022 ; 140 5965–5966.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1182/blood-2022-165172
Artigo de periodico
The PIP4K2 inhibitor THZ-P1-2 exhibits antileukemia activity by disruption of mitochondrial homeostasis and autophagy (2022)
Lima, Keli Cristina de ; Martins, Diego Antonio Pereira ; Miranda, Lívia Bassani Lins De ; Silva, Juan Luiz Coelho ; Leandro, Giovana da Silva ; Weinhäuser, Isabel ; Cavaglieri, Rita de Cássia; Leal, Aline de Medeiros; Silva, Wellington Fernandes da; Lange, Ana Paula Alencar de Lima; Velloso, Elvira Deolinda Rodrigues Pereira ; Rego, Eduardo Magalhães ; Griessinger, Emmanuel ; Hilberink, Jacobien R. ; Ammatuna, Emanuele ; Huls, Gerwin ; Schuringa, Jan Jacob ; Machado Neto, João Agostinho
Source: Blood Cancer Journal. Unidades: FM, ICB, FMRP
Subjects: FARMACOLOGIA, LEUCEMIA MIELOIDE AGUDA, HOMEOSTASE, APOPTOSE, MITOCÔNDRIAS, ESPECTROMETRIA DE MASSAS, CROMATOGRAFIA LÍQUIDA, CÉLULAS-TRONCO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Keli Cristina de et al. The PIP4K2 inhibitor THZ-P1-2 exhibits antileukemia activity by disruption of mitochondrial homeostasis and autophagy. Blood Cancer Journal, v. 12, p. 1-11, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41408-022-00747-w. Acesso em: 06 nov. 2024.
APA
Lima, K. C. de, Martins, D. A. P., Miranda, L. B. L. D., Silva, J. L. C., Leandro, G. da S., Weinhäuser, I., et al. (2022). The PIP4K2 inhibitor THZ-P1-2 exhibits antileukemia activity by disruption of mitochondrial homeostasis and autophagy. Blood Cancer Journal, 12, 1-11. doi:10.1038/s41408-022-00747-w
NLM
Lima KC de, Martins DAP, Miranda LBLD, Silva JLC, Leandro G da S, Weinhäuser I, Cavaglieri R de C, Leal A de M, Silva WF da, Lange APA de L, Velloso EDRP, Rego EM, Griessinger E, Hilberink JR, Ammatuna E, Huls G, Schuringa JJ, Machado Neto JA. The PIP4K2 inhibitor THZ-P1-2 exhibits antileukemia activity by disruption of mitochondrial homeostasis and autophagy [Internet]. Blood Cancer Journal. 2022 ; 12 1-11.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1038/s41408-022-00747-w
Vancouver
Lima KC de, Martins DAP, Miranda LBLD, Silva JLC, Leandro G da S, Weinhäuser I, Cavaglieri R de C, Leal A de M, Silva WF da, Lange APA de L, Velloso EDRP, Rego EM, Griessinger E, Hilberink JR, Ammatuna E, Huls G, Schuringa JJ, Machado Neto JA. The PIP4K2 inhibitor THZ-P1-2 exhibits antileukemia activity by disruption of mitochondrial homeostasis and autophagy [Internet]. Blood Cancer Journal. 2022 ; 12 1-11.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1038/s41408-022-00747-w
Artigo de periodico
DNA polymerase eta protects human cells against DNA damage induced by the tumor chemotherapeutic temozolomide (2022)
Latancia, Marcela Teatin ; Moreno, Natália Cestari ; Leandro, Giovana da Silva ; Ribeiro, Victoria Chaves ; Souza, Izadora de; Vieira, William Kleber Martins; Bastos, André Uchimura ; Hoch, Nicolas Carlos ; Rocha, Clarissa Ribeiro Reily ; Menck, Carlos Frederico Martins
Source: Mutation Research - Genetic Toxicology and Environmental Mutagenesis. Unidades: IQ, ICB, BIOTECNOLOGIA
Subjects: MICROBIOLOGIA, GLIOMA, NEOPLASIAS CEREBRAIS, DNA POLIMERASES, DANO AO DNA, ANTINEOPLÁSICOS, CÉLULAS CULTIVADAS DE TUMOR, MUTAGÊNESE, MELANOMA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LATANCIA, Marcela Teatin et al. DNA polymerase eta protects human cells against DNA damage induced by the tumor chemotherapeutic temozolomide. Mutation Research - Genetic Toxicology and Environmental Mutagenesis, v. 878, p. 1-11, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.mrgentox.2022.503498. Acesso em: 06 nov. 2024.
APA
Latancia, M. T., Moreno, N. C., Leandro, G. da S., Ribeiro, V. C., Souza, I. de, Vieira, W. K. M., et al. (2022). DNA polymerase eta protects human cells against DNA damage induced by the tumor chemotherapeutic temozolomide. Mutation Research - Genetic Toxicology and Environmental Mutagenesis, 878, 1-11. doi:10.1016/j.mrgentox.2022.503498
NLM
Latancia MT, Moreno NC, Leandro G da S, Ribeiro VC, Souza I de, Vieira WKM, Bastos AU, Hoch NC, Rocha CRR, Menck CFM. DNA polymerase eta protects human cells against DNA damage induced by the tumor chemotherapeutic temozolomide [Internet]. Mutation Research - Genetic Toxicology and Environmental Mutagenesis. 2022 ; 878 1-11.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.mrgentox.2022.503498
Vancouver
Latancia MT, Moreno NC, Leandro G da S, Ribeiro VC, Souza I de, Vieira WKM, Bastos AU, Hoch NC, Rocha CRR, Menck CFM. DNA polymerase eta protects human cells against DNA damage induced by the tumor chemotherapeutic temozolomide [Internet]. Mutation Research - Genetic Toxicology and Environmental Mutagenesis. 2022 ; 878 1-11.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.mrgentox.2022.503498
Parte de monografia/livro
Useful protocols to study DNA damage (2022)
Leandro, Giovana da Silva ; Latância, Marcela Teatin ; Quintero-Ruiz, Nathalia ; Menck, Carlos Frederico Martins
Source: Epigenetics and DNA Damage. Unidades: ICB, BIOTECNOLOGIA
Subjects: EPIGÊNESE GENÉTICA, DANO AO DNA, IMUNOFLUORESCÊNCIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEANDRO, Giovana da Silva et al. Useful protocols to study DNA damage. Epigenetics and DNA Damage. Tradução . Amsterdan: Elsevier, 2022. . Disponível em: https://doi.org/10.1016/B978-0-323-91081-1.00003-0. Acesso em: 06 nov. 2024.
APA
Leandro, G. da S., Latância, M. T., Quintero-Ruiz, N., & Menck, C. F. M. (2022). Useful protocols to study DNA damage. In Epigenetics and DNA Damage. Amsterdan: Elsevier. doi:10.1016/B978-0-323-91081-1.00003-0
NLM
Leandro G da S, Latância MT, Quintero-Ruiz N, Menck CFM. Useful protocols to study DNA damage [Internet]. In: Epigenetics and DNA Damage. Amsterdan: Elsevier; 2022. [citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/B978-0-323-91081-1.00003-0
Vancouver
Leandro G da S, Latância MT, Quintero-Ruiz N, Menck CFM. Useful protocols to study DNA damage [Internet]. In: Epigenetics and DNA Damage. Amsterdan: Elsevier; 2022. [citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/B978-0-323-91081-1.00003-0
Artigo de periodico
Transcription blockage by DNA damage in nucleotide excision repair-related neurological dysfunctions (2021)
Kajitani, Gustavo Satoru; Nascimento, Livia Luz de Souza ; Neves, Maira Rodrigues de Camargo ; Leandro, Giovana da Silva ; Garcia, Camila Carrião Machado ; Menck, Carlos Frederico Martins
Source: Seminars in Cell and Developmental Biology. Unidades: ICB, BIOINFORMÁTICA
Subjects: MICROBIOLOGIA, DANO AO DNA, DEGENERAÇÃO NEURAL, NUCLEOTÍDEOS, DOENÇAS GENÉTICAS, METABOLISMO CELULAR, FENÓTIPOS, ENVELHECIMENTO CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KAJITANI, Gustavo Satoru et al. Transcription blockage by DNA damage in nucleotide excision repair-related neurological dysfunctions. Seminars in Cell and Developmental Biology, v. 114, p. 20-35, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.semcdb.2020.10.009. Acesso em: 06 nov. 2024.
APA
Kajitani, G. S., Nascimento, L. L. de S., Neves, M. R. de C., Leandro, G. da S., Garcia, C. C. M., & Menck, C. F. M. (2021). Transcription blockage by DNA damage in nucleotide excision repair-related neurological dysfunctions. Seminars in Cell and Developmental Biology, 114, 20-35. doi:10.1016/j.semcdb.2020.10.009
NLM
Kajitani GS, Nascimento LL de S, Neves MR de C, Leandro G da S, Garcia CCM, Menck CFM. Transcription blockage by DNA damage in nucleotide excision repair-related neurological dysfunctions [Internet]. Seminars in Cell and Developmental Biology. 2021 ; 114 20-35.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.semcdb.2020.10.009
Vancouver
Kajitani GS, Nascimento LL de S, Neves MR de C, Leandro G da S, Garcia CCM, Menck CFM. Transcription blockage by DNA damage in nucleotide excision repair-related neurological dysfunctions [Internet]. Seminars in Cell and Developmental Biology. 2021 ; 114 20-35.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.semcdb.2020.10.009
Artigo de periodico
Changes in Expression Profiles Revealed by Transcriptomic Analysis in Peripheral Blood Mononuclear Cells of Alzheimer's Disease Patients (2018)
Leandro, Giovana da Silva ; Evangelista, Adriane Feijó; Lobo, Rômulo Rebouças; Xavier, Danilo Jordão; Moriguti, Júlio César; Sakamoto-Hojo, Elza Tiemi
Source: Journal of Alzheimer's Disease. Unidades: FMRP, FFCLRP
Subjects: DOENÇA DE ALZHEIMER, EXPRESSÃO GÊNICA, CÉLULAS SANGUÍNEAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEANDRO, Giovana da Silva et al. Changes in Expression Profiles Revealed by Transcriptomic Analysis in Peripheral Blood Mononuclear Cells of Alzheimer's Disease Patients. Journal of Alzheimer's Disease, v. 66, n. 4, p. 1483-1495, 2018Tradução . . Disponível em: https://doi.org/10.3233/jad-170205. Acesso em: 06 nov. 2024.
APA
Leandro, G. da S., Evangelista, A. F., Lobo, R. R., Xavier, D. J., Moriguti, J. C., & Sakamoto-Hojo, E. T. (2018). Changes in Expression Profiles Revealed by Transcriptomic Analysis in Peripheral Blood Mononuclear Cells of Alzheimer's Disease Patients. Journal of Alzheimer's Disease, 66( 4), 1483-1495. doi:10.3233/jad-170205
NLM
Leandro G da S, Evangelista AF, Lobo RR, Xavier DJ, Moriguti JC, Sakamoto-Hojo ET. Changes in Expression Profiles Revealed by Transcriptomic Analysis in Peripheral Blood Mononuclear Cells of Alzheimer's Disease Patients [Internet]. Journal of Alzheimer's Disease. 2018 ; 66( 4): 1483-1495.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3233/jad-170205
Vancouver
Leandro G da S, Evangelista AF, Lobo RR, Xavier DJ, Moriguti JC, Sakamoto-Hojo ET. Changes in Expression Profiles Revealed by Transcriptomic Analysis in Peripheral Blood Mononuclear Cells of Alzheimer's Disease Patients [Internet]. Journal of Alzheimer's Disease. 2018 ; 66( 4): 1483-1495.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3233/jad-170205
Artigo de periodico
Autophagy roles in the modulation of DNA repair pathways (2017)
Gomes, Luciana Rodrigues; Menck, Carlos Frederico Martins ; Leandro, Giovana da Silva
Source: International Journal of Molecular Sciences. Unidade: ICB
Subjects: MICROBIOLOGIA, REPARAÇÃO DE DNA, CÉLULAS MORTAS, DNA RECOMBINANTE, NUCLEOTÍDEOS, HOMEOSTASE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GOMES, Luciana Rodrigues e MENCK, Carlos Frederico Martins e LEANDRO, Giovana da Silva. Autophagy roles in the modulation of DNA repair pathways. International Journal of Molecular Sciences, v. 18, n. 11, p. 1-21, 2017Tradução . . Disponível em: https://doi.org/10.3390/ijms18112351. Acesso em: 06 nov. 2024.
APA
Gomes, L. R., Menck, C. F. M., & Leandro, G. da S. (2017). Autophagy roles in the modulation of DNA repair pathways. International Journal of Molecular Sciences, 18( 11), 1-21. doi:10.3390/ijms18112351
NLM
Gomes LR, Menck CFM, Leandro G da S. Autophagy roles in the modulation of DNA repair pathways [Internet]. International Journal of Molecular Sciences. 2017 ; 18( 11): 1-21.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3390/ijms18112351
Vancouver
Gomes LR, Menck CFM, Leandro G da S. Autophagy roles in the modulation of DNA repair pathways [Internet]. International Journal of Molecular Sciences. 2017 ; 18( 11): 1-21.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3390/ijms18112351
Trabalho de evento-resumo
Analysis of the genotoxic potential of metformin in human fibroblasts (2007)
Leandro, Giovana da Silva; Xavier, D. J.; Sakamoto-Hojo, Elza Tiemi
Source: Resumos. Conference titles: Meeting in Fundamental Aspects of DNA repair and Mutagenese. Unidade: FFCLRP
Subjects: FIBROBLASTOS, DNA (REPARAÇÃO)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEANDRO, Giovana da Silva e XAVIER, D. J. e SAKAMOTO-HOJO, Elza Tiemi. Analysis of the genotoxic potential of metformin in human fibroblasts. 2007, Anais.. São Paulo: USP, 2007. . Acesso em: 06 nov. 2024.
APA
Leandro, G. da S., Xavier, D. J., & Sakamoto-Hojo, E. T. (2007). Analysis of the genotoxic potential of metformin in human fibroblasts. In Resumos. São Paulo: USP.
NLM
Leandro G da S, Xavier DJ, Sakamoto-Hojo ET. Analysis of the genotoxic potential of metformin in human fibroblasts. Resumos. 2007 ;[citado 2024 nov. 06 ]
Vancouver
Leandro G da S, Xavier DJ, Sakamoto-Hojo ET. Analysis of the genotoxic potential of metformin in human fibroblasts. Resumos. 2007 ;[citado 2024 nov. 06 ]

USP Schools

ReP

Filtros

USP Schools

Departament

Authors

USP affiliated authors

Date published

Subjects

Source title

Funding Agencies

Indexado em

Non normalized affiliation

Countries of institutions of collaboration