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Artigo de periodico
Corrigendum to: NCOA3 identified as a new candidate to explain autosomal dominant progressive hearing loss (2022)
Silva, Rodrigo Salazar da ; Dantas, Vitor Lima Goes ; Alves, Leandro Ucela; Batissoco, Ana Carla ; Oiticica, Jeanne ; Lawrence, Elizabeth A ; Kawafi, Abdelwahab ; Yang, Yishi ; Nicastro, Fernanda Stávale; Novaes, Beatriz Caiuby ; Hammond, Chrissy ; Kague, Érika ; Mingroni Netto, Regina Celia
Source: Human Molecular Genetics. Unidades: IB, FM
Subjects: SURDEZ, MUTAÇÃO GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Rodrigo Salazar da et al. Corrigendum to: NCOA3 identified as a new candidate to explain autosomal dominant progressive hearing loss. Human Molecular Genetics, v. 31, n. Ja 2022, 2022Tradução . . Disponível em: https://doi.org/10.1093/hmg/ddab325. Acesso em: 03 nov. 2024.
APA
Silva, R. S. da, Dantas, V. L. G., Alves, L. U., Batissoco, A. C., Oiticica, J., Lawrence, E. A., et al. (2022). Corrigendum to: NCOA3 identified as a new candidate to explain autosomal dominant progressive hearing loss. Human Molecular Genetics, 31( Ja 2022). doi:10.1093/hmg/ddab325
NLM
Silva RS da, Dantas VLG, Alves LU, Batissoco AC, Oiticica J, Lawrence EA, Kawafi A, Yang Y, Nicastro FS, Novaes BC, Hammond C, Kague É, Mingroni Netto RC. Corrigendum to: NCOA3 identified as a new candidate to explain autosomal dominant progressive hearing loss [Internet]. Human Molecular Genetics. 2022 ; 31( Ja 2022):[citado 2024 nov. 03 ] Available from: https://doi.org/10.1093/hmg/ddab325
Vancouver
Silva RS da, Dantas VLG, Alves LU, Batissoco AC, Oiticica J, Lawrence EA, Kawafi A, Yang Y, Nicastro FS, Novaes BC, Hammond C, Kague É, Mingroni Netto RC. Corrigendum to: NCOA3 identified as a new candidate to explain autosomal dominant progressive hearing loss [Internet]. Human Molecular Genetics. 2022 ; 31( Ja 2022):[citado 2024 nov. 03 ] Available from: https://doi.org/10.1093/hmg/ddab325
Artigo de periodico
NCOA3 identified as a new candidate to explain autosomal dominant progressive hearing loss (2020)
Silva, Rodrigo Salazar da ; Dantas, Vitor Lima Goes ; Alves, Leandro Ucela; Batissoco, Ana Carla ; Oiticica, Jeanne ; Lawrence, Elizabeth A ; Kawafi, Abdelwahab ; Yang, Yishi ; Nicastro, Fernanda Stávale; Novaes, Beatriz Caiuby ; Hammond, Chrissy ; Kague, Érika ; Mingroni Netto, Regina Celia
Source: Human Molecular Genetics. Unidades: IB, FM
Subjects: AUDIÇÃO, SURDEZ, MUTAÇÃO GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Rodrigo Salazar da et al. NCOA3 identified as a new candidate to explain autosomal dominant progressive hearing loss. Human Molecular Genetics, v. 29, n. 2, p. 3691–3705, 2020Tradução . . Disponível em: https://doi.org/10.1093/hmg/ddaa240. Acesso em: 03 nov. 2024.
APA
Silva, R. S. da, Dantas, V. L. G., Alves, L. U., Batissoco, A. C., Oiticica, J., Lawrence, E. A., et al. (2020). NCOA3 identified as a new candidate to explain autosomal dominant progressive hearing loss. Human Molecular Genetics, 29( 2), 3691–3705. doi:10.1093/hmg/ddaa240
NLM
Silva RS da, Dantas VLG, Alves LU, Batissoco AC, Oiticica J, Lawrence EA, Kawafi A, Yang Y, Nicastro FS, Novaes BC, Hammond C, Kague É, Mingroni Netto RC. NCOA3 identified as a new candidate to explain autosomal dominant progressive hearing loss [Internet]. Human Molecular Genetics. 2020 ;29( 2): 3691–3705.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1093/hmg/ddaa240
Vancouver
Silva RS da, Dantas VLG, Alves LU, Batissoco AC, Oiticica J, Lawrence EA, Kawafi A, Yang Y, Nicastro FS, Novaes BC, Hammond C, Kague É, Mingroni Netto RC. NCOA3 identified as a new candidate to explain autosomal dominant progressive hearing loss [Internet]. Human Molecular Genetics. 2020 ;29( 2): 3691–3705.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1093/hmg/ddaa240
Artigo de periodico
CD105 is regulated by hsa-miR-1287 and its expression is inversely correlated with osteopotential in SHED (2018)
Ishiy, Felipe Augusto André; Fanganiello, Roberto Dalto; Kobayashi, Gerson Shigeru; Kague, Érika; Kuriki, Patrícia Semedo; Passos-Bueno, Maria Rita
Source: Bone. Unidade: IB
Subjects: GENÉTICA MÉDICA, BIOMARCADORES, CÉLULAS-TRONCO, OSTEOGÊNESE, MALFORMAÇÕES, ENGENHARIA TECIDUAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ISHIY, Felipe Augusto André et al. CD105 is regulated by hsa-miR-1287 and its expression is inversely correlated with osteopotential in SHED. Bone, v. 106, p. 112-120, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bone.2017.10.014. Acesso em: 03 nov. 2024.
APA
Ishiy, F. A. A., Fanganiello, R. D., Kobayashi, G. S., Kague, É., Kuriki, P. S., & Passos-Bueno, M. R. (2018). CD105 is regulated by hsa-miR-1287 and its expression is inversely correlated with osteopotential in SHED. Bone, 106, 112-120. doi:10.1016/j.bone.2017.10.014
NLM
Ishiy FAA, Fanganiello RD, Kobayashi GS, Kague É, Kuriki PS, Passos-Bueno MR. CD105 is regulated by hsa-miR-1287 and its expression is inversely correlated with osteopotential in SHED [Internet]. Bone. 2018 ; 106 112-120.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.bone.2017.10.014
Vancouver
Ishiy FAA, Fanganiello RD, Kobayashi GS, Kague É, Kuriki PS, Passos-Bueno MR. CD105 is regulated by hsa-miR-1287 and its expression is inversely correlated with osteopotential in SHED [Internet]. Bone. 2018 ; 106 112-120.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.bone.2017.10.014
Artigo de periodico
Functionally conserved cis-regulatory elements of COL18A1 identified through zebrafish transgenesis (2010)
Kague, Érika; Bessling, Seneca; Lee, Josephine; Hu, Gui; Passos-Bueno, Maria Rita ; Fisher, Shannon
Source: Developmental Biology. Unidade: IB
Assunto: SEQUENCIAMENTO GENÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KAGUE, Érika et al. Functionally conserved cis-regulatory elements of COL18A1 identified through zebrafish transgenesis. Developmental Biology, v. 337, n. 2, p. 496-505, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.ydbio.2009.10.028. Acesso em: 03 nov. 2024.
APA
Kague, É., Bessling, S., Lee, J., Hu, G., Passos-Bueno, M. R., & Fisher, S. (2010). Functionally conserved cis-regulatory elements of COL18A1 identified through zebrafish transgenesis. Developmental Biology, 337( 2), 496-505. doi:10.1016/j.ydbio.2009.10.028
NLM
Kague É, Bessling S, Lee J, Hu G, Passos-Bueno MR, Fisher S. Functionally conserved cis-regulatory elements of COL18A1 identified through zebrafish transgenesis [Internet]. Developmental Biology. 2010 ; 337( 2): 496-505.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.ydbio.2009.10.028
Vancouver
Kague É, Bessling S, Lee J, Hu G, Passos-Bueno MR, Fisher S. Functionally conserved cis-regulatory elements of COL18A1 identified through zebrafish transgenesis [Internet]. Developmental Biology. 2010 ; 337( 2): 496-505.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.ydbio.2009.10.028
Artigo de periodico
Novel pathogenic mutations and skin biopsy analysis in Knobloch syndrome (2009)
Suzuki, Oscar; Kague, Érika; Bagatini, Kelly; Tu, Hongmin; Heljasvaara, Ritva; Carvalhaes, Lorenza; Gava, Elisandra; Oliveira, Gisele de; Godoi, Paulo; Oliva, Glaucius ; Kitten, Gregory; Pihlajaniemi, Taina; Passos-Bueno, Maria Rita
Source: Molecular Vision. Unidades: IFSC, IB
Subjects: MUTAÇÃO GENÉTICA, DIAGNÓSTICO (TÉCNICAS), NEOPLASIAS (CRESCIMENTO E DESENVOLVIMENTO), GENÉTICA (ESTUDO), MOLÉCULA (ANÁLISE), OFTALMOPATIAS, FLUORESCÊNCIA, BIÓPSIA, COLÁGENO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SUZUKI, Oscar et al. Novel pathogenic mutations and skin biopsy analysis in Knobloch syndrome. Molecular Vision, v. 15, n. 82, p. 801-809, 2009Tradução . . Disponível em: http://www.molvis.org/molvis/v15/a82. Acesso em: 03 nov. 2024.
APA
Suzuki, O., Kague, É., Bagatini, K., Tu, H., Heljasvaara, R., Carvalhaes, L., et al. (2009). Novel pathogenic mutations and skin biopsy analysis in Knobloch syndrome. Molecular Vision, 15( 82), 801-809. Recuperado de http://www.molvis.org/molvis/v15/a82
NLM
Suzuki O, Kague É, Bagatini K, Tu H, Heljasvaara R, Carvalhaes L, Gava E, Oliveira G de, Godoi P, Oliva G, Kitten G, Pihlajaniemi T, Passos-Bueno MR. Novel pathogenic mutations and skin biopsy analysis in Knobloch syndrome [Internet]. Molecular Vision. 2009 ; 15( 82): 801-809.[citado 2024 nov. 03 ] Available from: http://www.molvis.org/molvis/v15/a82
Vancouver
Suzuki O, Kague É, Bagatini K, Tu H, Heljasvaara R, Carvalhaes L, Gava E, Oliveira G de, Godoi P, Oliva G, Kitten G, Pihlajaniemi T, Passos-Bueno MR. Novel pathogenic mutations and skin biopsy analysis in Knobloch syndrome [Internet]. Molecular Vision. 2009 ; 15( 82): 801-809.[citado 2024 nov. 03 ] Available from: http://www.molvis.org/molvis/v15/a82
Artigo de periodico
COL18A1 is highly expressed during human adipocyte differentiation and the SNP c.1136C > T in its "frizzled" motif is associated with obesity in diabetes type 2 patients (2008)
Errera, Flávia Imbroisi Valle; Canani, Luís Henrique; Yeh, Erika; Kague, Érika; Armelin-Correa, Lucia Maria; Suzuki, Oscar Takeo; Tschiedel, Balduíno; Silva, Maria Elizabeth Rossi da; Sertié, Andréa Laurato; Passos-Bueno, Maria Rita
Source: Anais da Academia Brasileira de Ciências. Unidade: IB
Subjects: COLÁGENO, OBESIDADE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ERRERA, Flávia Imbroisi Valle et al. COL18A1 is highly expressed during human adipocyte differentiation and the SNP c.1136C > T in its "frizzled" motif is associated with obesity in diabetes type 2 patients. Anais da Academia Brasileira de Ciências, v. 80, n. 1, p. 167-177, 2008Tradução . . Disponível em: https://doi.org/10.1590/s0001-37652008000100012. Acesso em: 03 nov. 2024.
APA
Errera, F. I. V., Canani, L. H., Yeh, E., Kague, É., Armelin-Correa, L. M., Suzuki, O. T., et al. (2008). COL18A1 is highly expressed during human adipocyte differentiation and the SNP c.1136C > T in its "frizzled" motif is associated with obesity in diabetes type 2 patients. Anais da Academia Brasileira de Ciências, 80( 1), 167-177. doi:10.1590/s0001-37652008000100012
NLM
Errera FIV, Canani LH, Yeh E, Kague É, Armelin-Correa LM, Suzuki OT, Tschiedel B, Silva MER da, Sertié AL, Passos-Bueno MR. COL18A1 is highly expressed during human adipocyte differentiation and the SNP c.1136C > T in its "frizzled" motif is associated with obesity in diabetes type 2 patients [Internet]. Anais da Academia Brasileira de Ciências. 2008 ; 80( 1): 167-177.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1590/s0001-37652008000100012
Vancouver
Errera FIV, Canani LH, Yeh E, Kague É, Armelin-Correa LM, Suzuki OT, Tschiedel B, Silva MER da, Sertié AL, Passos-Bueno MR. COL18A1 is highly expressed during human adipocyte differentiation and the SNP c.1136C > T in its "frizzled" motif is associated with obesity in diabetes type 2 patients [Internet]. Anais da Academia Brasileira de Ciências. 2008 ; 80( 1): 167-177.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1590/s0001-37652008000100012

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