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Artigo de periodico
Reduced expression of adherens and gap junction proteins can have a fundamental role in the developmentof heart failure following cardiac hypertrophy in rats (2016)
Santos, Daniele O. dos; Blefari, Valdecir; Prado, Fernanda P.; Silva, Carlos A.; Fazan Júnior, Rubens; Salgado, Hélio César; Ramos, Simone Gusmão; Prado, Cibele Maria
Source: Experimental and Molecular Pathology. Unidade: FMRP
Subjects: HIPERTENSÃO, PROTEÍNAS, INSUFICIÊNCIA CARDÍACA, HIPERTROFIA VENTRICULAR ESQUERDA
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ABNT
SANTOS, Daniele O. dos et al. Reduced expression of adherens and gap junction proteins can have a fundamental role in the developmentof heart failure following cardiac hypertrophy in rats. Experimental and Molecular Pathology, v. 100, n. 1, p. 167- 176, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.yexmp.2015.12.009. Acesso em: 01 nov. 2024.
APA
Santos, D. O. dos, Blefari, V., Prado, F. P., Silva, C. A., Fazan Júnior, R., Salgado, H. C., et al. (2016). Reduced expression of adherens and gap junction proteins can have a fundamental role in the developmentof heart failure following cardiac hypertrophy in rats. Experimental and Molecular Pathology, 100( 1), 167- 176. doi:10.1016/j.yexmp.2015.12.009
NLM
Santos DO dos, Blefari V, Prado FP, Silva CA, Fazan Júnior R, Salgado HC, Ramos SG, Prado CM. Reduced expression of adherens and gap junction proteins can have a fundamental role in the developmentof heart failure following cardiac hypertrophy in rats [Internet]. Experimental and Molecular Pathology. 2016 ; 100( 1): 167- 176.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.yexmp.2015.12.009
Vancouver
Santos DO dos, Blefari V, Prado FP, Silva CA, Fazan Júnior R, Salgado HC, Ramos SG, Prado CM. Reduced expression of adherens and gap junction proteins can have a fundamental role in the developmentof heart failure following cardiac hypertrophy in rats [Internet]. Experimental and Molecular Pathology. 2016 ; 100( 1): 167- 176.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.yexmp.2015.12.009
Trabalho de evento-resumo
Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy (2012)
Rizzi, Elen; Ceron, Carla Speroni; Guimarães, Danielle Aparecida; Prado, Cibele Maria; Rossi, Marcos Antonio; Gerlach, Raquel Fernanda; Tanus-Santos, José Eduardo
Source: Final Program. Conference titles: High Blood Pressure Research Scientific Sessions (HBPR). Unidades: FMRP, FORP
Subjects: ESTRESSE OXIDATIVO, METALOPROTEINASES, HIPERTROFIA VENTRICULAR ESQUERDA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RIZZI, Elen et al. Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy. 2012, Anais.. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2012. . Acesso em: 01 nov. 2024.
APA
Rizzi, E., Ceron, C. S., Guimarães, D. A., Prado, C. M., Rossi, M. A., Gerlach, R. F., & Tanus-Santos, J. E. (2012). Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy. In Final Program. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Rizzi E, Ceron CS, Guimarães DA, Prado CM, Rossi MA, Gerlach RF, Tanus-Santos JE. Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy. Final Program. 2012 ;[citado 2024 nov. 01 ]
Vancouver
Rizzi E, Ceron CS, Guimarães DA, Prado CM, Rossi MA, Gerlach RF, Tanus-Santos JE. Imbalance between MMP-2 and TIMP-4 associated with oxidative stress contributes to temporal renovascular hypertension-induced cardiac hypertrophy. Final Program. 2012 ;[citado 2024 nov. 01 ]
Artigo de periodico
Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension (2010)
Rizzi, Élen; Castro, Michele M.; Prado, Cibele M.; Silva, Carlos A.; Fazan Junior, Rubens; Rossi, Marcos Antonio; Tanus-Santos, José Eduardo; Gerlach, Raquel Fernanda
Source: Journal of Cardiac Failure. Unidades: FMRP, FORP
Subjects: HIPERTROFIA VENTRICULAR ESQUERDA, HIPERTENSÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RIZZI, Élen et al. Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension. Journal of Cardiac Failure, v. 16, n. 7, p. 599-608, 2010Tradução . . Acesso em: 01 nov. 2024.
APA
Rizzi, É., Castro, M. M., Prado, C. M., Silva, C. A., Fazan Junior, R., Rossi, M. A., et al. (2010). Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension. Journal of Cardiac Failure, 16( 7), 599-608.
NLM
Rizzi É, Castro MM, Prado CM, Silva CA, Fazan Junior R, Rossi MA, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension. Journal of Cardiac Failure. 2010 ; 16( 7): 599-608.[citado 2024 nov. 01 ]
Vancouver
Rizzi É, Castro MM, Prado CM, Silva CA, Fazan Junior R, Rossi MA, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension. Journal of Cardiac Failure. 2010 ; 16( 7): 599-608.[citado 2024 nov. 01 ]
Trabalho de evento-resumo
Molecular mechanisms involved in the dual blockade of the renin angiotensin system (RAS) on the left ventricular remodeling in renal hypertensive rats (2K-1C) (2010)
Corrêa, J. W. N; Callera, G. E.; Yogi, A.; He, Y.; Araújo, A. V.; Vercesi, Juliana Aparecida; Riul, Maria Elena; Prado, C. M.; Rossi, Marcos Antonio; Touyz, R. M.; Bendhack, Lusiane Maria
Source: Programa. Conference titles: Congresso Brasileiro de Farmacologia e Terapêutica Experimental. Unidades: FCFRP, FMRP
Subjects: HIPERTROFIA VENTRICULAR ESQUERDA, SISTEMA RENINA-ANGIOTENSINA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CORRÊA, J. W. N et al. Molecular mechanisms involved in the dual blockade of the renin angiotensin system (RAS) on the left ventricular remodeling in renal hypertensive rats (2K-1C). 2010, Anais.. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo, 2010. . Acesso em: 01 nov. 2024.
APA
Corrêa, J. W. N., Callera, G. E., Yogi, A., He, Y., Araújo, A. V., Vercesi, J. A., et al. (2010). Molecular mechanisms involved in the dual blockade of the renin angiotensin system (RAS) on the left ventricular remodeling in renal hypertensive rats (2K-1C). In Programa. Ribeirão Preto: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo.
NLM
Corrêa JWN, Callera GE, Yogi A, He Y, Araújo AV, Vercesi JA, Riul ME, Prado CM, Rossi MA, Touyz RM, Bendhack LM. Molecular mechanisms involved in the dual blockade of the renin angiotensin system (RAS) on the left ventricular remodeling in renal hypertensive rats (2K-1C). Programa. 2010 ;[citado 2024 nov. 01 ]
Vancouver
Corrêa JWN, Callera GE, Yogi A, He Y, Araújo AV, Vercesi JA, Riul ME, Prado CM, Rossi MA, Touyz RM, Bendhack LM. Molecular mechanisms involved in the dual blockade of the renin angiotensin system (RAS) on the left ventricular remodeling in renal hypertensive rats (2K-1C). Programa. 2010 ;[citado 2024 nov. 01 ]

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