ReP USP - Resultado da busca

Artigo de periodico
Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival (2021)
Vilas-Boas, Eloisa Aparecida ; Carlein, Christopher ; Nalbach, Lisa; Almeida, Davidson Correa de ; Ampofo, Emmanuel ; Carpinelli, Angelo Rafael ; Roma, Leticia Prates ; Ortis, Fernanda
Source: Antioxidants. Unidade: ICB
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, ESTRESSE OXIDATIVO, PERÓXIDO DE HIDROGÊNIO, CITOCINAS, DIABETES MELLITUS, CAMUNDONGOS, ILHOTAS DE LANGERHANS, GLICOSE
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ABNT
VILAS-BOAS, Eloisa Aparecida et al. Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival. Antioxidants, v. 10, n. 8, p. 1-20, 2021Tradução . . Disponível em: https://doi.org/10.3390/antiox10081305. Acesso em: 13 nov. 2024.
APA
Vilas-Boas, E. A., Carlein, C., Nalbach, L., Almeida, D. C. de, Ampofo, E., Carpinelli, A. R., et al. (2021). Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival. Antioxidants, 10( 8), 1-20. doi:10.3390/antiox10081305
NLM
Vilas-Boas EA, Carlein C, Nalbach L, Almeida DC de, Ampofo E, Carpinelli AR, Roma LP, Ortis F. Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival [Internet]. Antioxidants. 2021 ; 10( 8): 1-20.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/antiox10081305
Vancouver
Vilas-Boas EA, Carlein C, Nalbach L, Almeida DC de, Ampofo E, Carpinelli AR, Roma LP, Ortis F. Early cytokine-induced transient NOX2 activity is ER stress-dependent and impacts β-cell function and survival [Internet]. Antioxidants. 2021 ; 10( 8): 1-20.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/antiox10081305
Artigo de periodico
HSPB1 is essential for inducing resistance to proteotoxic stress in beta-cells (2021)
Gomes, Vinícius Maron; Mansano, Rosangela Aparecida Wailemann ; Arini, Gabriel Santos ; Oliveira, Talita Cristina de; Almeida, Daria Raquel Queiroz de ; Santos, Ancély Ferreira dos ; Terra, Letícia Ferreira; Lortz, Stephan ; Labriola, Leticia
Source: Cells. Unidades: IQ, FMRP
Subjects: APOPTOSE, PROTEÍNAS DO CHOQUE TÉRMICO, DIABETES MELLITUS, RETÍCULO ENDOPLASMÁTICO
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GOMES, Vinícius Maron et al. HSPB1 is essential for inducing resistance to proteotoxic stress in beta-cells. Cells, v. 10, n. 9, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10092178. Acesso em: 13 nov. 2024.
APA
Gomes, V. M., Mansano, R. A. W., Arini, G. S., Oliveira, T. C. de, Almeida, D. R. Q. de, Santos, A. F. dos, et al. (2021). HSPB1 is essential for inducing resistance to proteotoxic stress in beta-cells. Cells, 10( 9), 1-16. doi:10.3390/cells10092178
NLM
Gomes VM, Mansano RAW, Arini GS, Oliveira TC de, Almeida DRQ de, Santos AF dos, Terra LF, Lortz S, Labriola L. HSPB1 is essential for inducing resistance to proteotoxic stress in beta-cells [Internet]. Cells. 2021 ; 10( 9): 1-16.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/cells10092178
Vancouver
Gomes VM, Mansano RAW, Arini GS, Oliveira TC de, Almeida DRQ de, Santos AF dos, Terra LF, Lortz S, Labriola L. HSPB1 is essential for inducing resistance to proteotoxic stress in beta-cells [Internet]. Cells. 2021 ; 10( 9): 1-16.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/cells10092178
Artigo de periodico
Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets (2021)
Vilas-Boas, Eloisa Aparecida ; Nalbach, Lisa; Ampofo, Emmanuel; Lucena, Camila Ferraz ; Naudet, Léa ; Morgan, Bruce ; Roma, Leticia Prates ; Ortis, Fernanda ; Carpinelli, Angelo Rafael
Source: Free Radical Biology & Medicine. Unidade: ICB
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, OBESIDADE, DIABETES MELLITUS, HÁBITOS ALIMENTARES, ÁCIDOS GRAXOS, ILHOTAS DE LANGERHANS, ESTRESSE OXIDATIVO, CAMUNDONGOS, INSULINA, HOMEOSTASE
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ABNT
VILAS-BOAS, Eloisa Aparecida et al. Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets. Free Radical Biology & Medicine, v. 162, p. 13 , 2021Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2020.11.023. Acesso em: 13 nov. 2024.
APA
Vilas-Boas, E. A., Nalbach, L., Ampofo, E., Lucena, C. F., Naudet, L., Morgan, B., et al. (2021). Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets. Free Radical Biology & Medicine, 162, 13 . doi:10.1016/j.freeradbiomed.2020.11.023
NLM
Vilas-Boas EA, Nalbach L, Ampofo E, Lucena CF, Naudet L, Morgan B, Roma LP, Ortis F, Carpinelli AR. Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets [Internet]. Free Radical Biology & Medicine. 2021 ; 162 13 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2020.11.023
Vancouver
Vilas-Boas EA, Nalbach L, Ampofo E, Lucena CF, Naudet L, Morgan B, Roma LP, Ortis F, Carpinelli AR. Transient NADPH oxidase 2-dependent H2O2 production drives early palmitate-induced lipotoxicity in pancreatic islets [Internet]. Free Radical Biology & Medicine. 2021 ; 162 13 .[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2020.11.023
Artigo de periodico
Regulation of glucose metabolism by MuRF1 and treatment of myopathy in diabetic mice with Small molecules targeting MuRF1 (2021)
Labeit, Siegfried ; Hirner, Stephanie; Bogomolovas, Julijus ; Oliveira, André Cruz de ; Myrzabekova, Moldir ; Moriscot, Anselmo Sigari ; Bowen, Thomas Scott; Adams, Volker
Source: International Journal of Molecular Sciences. Unidade: ICB
Subjects: ANATOMIA, GLICOSE, DIABETES MELLITUS, MÚSCULOS, WESTERN BLOTTING, MÚSCULO ESQUELÉTICO
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ABNT
LABEIT, Siegfried et al. Regulation of glucose metabolism by MuRF1 and treatment of myopathy in diabetic mice with Small molecules targeting MuRF1. International Journal of Molecular Sciences, v. 22, n. 4, p. 14, 2021Tradução . . Disponível em: https://doi.org/10.3390/ijms22042225. Acesso em: 13 nov. 2024.
APA
Labeit, S., Hirner, S., Bogomolovas, J., Oliveira, A. C. de, Myrzabekova, M., Moriscot, A. S., et al. (2021). Regulation of glucose metabolism by MuRF1 and treatment of myopathy in diabetic mice with Small molecules targeting MuRF1. International Journal of Molecular Sciences, 22( 4), 14. doi:10.3390/ijms22042225
NLM
Labeit S, Hirner S, Bogomolovas J, Oliveira AC de, Myrzabekova M, Moriscot AS, Bowen TS, Adams V. Regulation of glucose metabolism by MuRF1 and treatment of myopathy in diabetic mice with Small molecules targeting MuRF1 [Internet]. International Journal of Molecular Sciences. 2021 ; 22( 4): 14.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms22042225
Vancouver
Labeit S, Hirner S, Bogomolovas J, Oliveira AC de, Myrzabekova M, Moriscot AS, Bowen TS, Adams V. Regulation of glucose metabolism by MuRF1 and treatment of myopathy in diabetic mice with Small molecules targeting MuRF1 [Internet]. International Journal of Molecular Sciences. 2021 ; 22( 4): 14.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/ijms22042225
Artigo de periodico
Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress (2021)
Vilas-Boas, Eloisa Aparecida ; Almeida, Davidson Correa ; Roma, Leticia Prates ; Ortis, Fernanda ; Carpinelli, Angelo Rafael
Source: Cells. Unidades: ICB, IQ
Subjects: FISIOLOGIA, BIOLOGIA CELULAR, ESTRESSE OXIDATIVO, ILHOTAS DE LANGERHANS, DIABETES MELLITUS, LIPÍDEOS, MEMBRANA PLASMÁTICA, OBESIDADE, ÁCIDOS GRAXOS, INSULINA, GLICOSE, CÉLULAS MORTAS
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ABNT
VILAS-BOAS, Eloisa Aparecida et al. Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress. Cells, v. 10, n. 12, p. 1-23, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10123328. Acesso em: 13 nov. 2024.
APA
Vilas-Boas, E. A., Almeida, D. C., Roma, L. P., Ortis, F., & Carpinelli, A. R. (2021). Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress. Cells, 10( 12), 1-23. doi:10.3390/cells10123328
NLM
Vilas-Boas EA, Almeida DC, Roma LP, Ortis F, Carpinelli AR. Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress [Internet]. Cells. 2021 ; 10( 12): 1-23.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/cells10123328
Vancouver
Vilas-Boas EA, Almeida DC, Roma LP, Ortis F, Carpinelli AR. Lipotoxicity and β-cell failure in type 2 diabetes: oxidative stress linked to NADPH oxidase and ER stress [Internet]. Cells. 2021 ; 10( 12): 1-23.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3390/cells10123328
Trabalho de evento-resumo periodico
HSPB1 is essential for inducing resistance to proteotoxic stress in beta cells (2020)
Labriola, Leticia ; Gomes, Vinicius M; Wailemann, Rosângela A. M; Almeida, Daria Raquel Queiroz de ; Arini, Gabriel Santos ; Santos, Ancély Ferreira dos ; Terra, Letícia Ferreira; Lortz, S
Source: Diabetologia. Conference titles: Virtual Meeting of the European Association for the Study of Diabetes (EASD). Unidade: IQ
Subjects: APOPTOSE, DIABETES MELLITUS, RETÍCULO ENDOPLASMÁTICO, PROTEÍNAS DO CHOQUE TÉRMICO
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ABNT
LABRIOLA, Leticia et al. HSPB1 is essential for inducing resistance to proteotoxic stress in beta cells. Diabetologia. New York: Instituto de Química, Universidade de São Paulo. . Acesso em: 13 nov. 2024. , 2020
APA
Labriola, L., Gomes, V. M., Wailemann, R. A. M., Almeida, D. R. Q. de, Arini, G. S., Santos, A. F. dos, et al. (2020). HSPB1 is essential for inducing resistance to proteotoxic stress in beta cells. Diabetologia. New York: Instituto de Química, Universidade de São Paulo.
NLM
Labriola L, Gomes VM, Wailemann RAM, Almeida DRQ de, Arini GS, Santos AF dos, Terra LF, Lortz S. HSPB1 is essential for inducing resistance to proteotoxic stress in beta cells. Diabetologia. 2020 ; 63 S184-S185 res. 372.[citado 2024 nov. 13 ]
Vancouver
Labriola L, Gomes VM, Wailemann RAM, Almeida DRQ de, Arini GS, Santos AF dos, Terra LF, Lortz S. HSPB1 is essential for inducing resistance to proteotoxic stress in beta cells. Diabetologia. 2020 ; 63 S184-S185 res. 372.[citado 2024 nov. 13 ]
Artigo de periodico
Macrophage inflammatory state in type 1 diabetes: triggered by NLRP3/iNOS pathway and attenuated by docosahexaenoic acid (DHA) (2020)
Davanso, Mariana Rodrigues ; Crisma, Amanda Rabello ; Braga, Tarcio Teodoro ; Masi, Laureane Nunes ; Amaral, Catia Lira do ; Leal, Vinicius Nunes Cordeiro ; Lima, Dhêmerson Souza de ; Patente, Thiago Andrade ; Giannella, Maria Lucia Correa ; Lauterbach, Mario ; Kolbe, Carl Christian ; Latz, Eicke ; Barbuto, José Alexandre Marzagão ; Câmara, Niels Olsen Saraiva ; Pontillo, Alessandra ; Curi, Rui
Source: Clinical Science (Online): an international journal of translational science and medicine. Unidade: ICB
Subjects: FISIOLOGIA, IMUNOLOGIA, DIABETES MELLITUS, ÁCIDOS GRAXOS, MACRÓFAGOS, INFLAMAÇÃO, DOENÇAS AUTOIMUNES, ILHOTAS DE LANGERHANS, MONÓCITOS, HIPERGLICEMIA, CITOCINAS, ÓXIDO NÍTRICO, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS
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ABNT
DAVANSO, Mariana Rodrigues et al. Macrophage inflammatory state in type 1 diabetes: triggered by NLRP3/iNOS pathway and attenuated by docosahexaenoic acid (DHA). Clinical Science (Online): an international journal of translational science and medicine, p. [29 ], 2020Tradução . . Disponível em: https://doi.org/10.1042/CS20201348. Acesso em: 13 nov. 2024.
APA
Davanso, M. R., Crisma, A. R., Braga, T. T., Masi, L. N., Amaral, C. L. do, Leal, V. N. C., et al. (2020). Macrophage inflammatory state in type 1 diabetes: triggered by NLRP3/iNOS pathway and attenuated by docosahexaenoic acid (DHA). Clinical Science (Online): an international journal of translational science and medicine, [29 ]. doi:10.1042/CS20201348
NLM
Davanso MR, Crisma AR, Braga TT, Masi LN, Amaral CL do, Leal VNC, Lima DS de, Patente TA, Giannella MLC, Lauterbach M, Kolbe CC, Latz E, Barbuto JAM, Câmara NOS, Pontillo A, Curi R. Macrophage inflammatory state in type 1 diabetes: triggered by NLRP3/iNOS pathway and attenuated by docosahexaenoic acid (DHA) [Internet]. Clinical Science (Online): an international journal of translational science and medicine. 2020 ;[29 ].[citado 2024 nov. 13 ] Available from: https://doi.org/10.1042/CS20201348
Vancouver
Davanso MR, Crisma AR, Braga TT, Masi LN, Amaral CL do, Leal VNC, Lima DS de, Patente TA, Giannella MLC, Lauterbach M, Kolbe CC, Latz E, Barbuto JAM, Câmara NOS, Pontillo A, Curi R. Macrophage inflammatory state in type 1 diabetes: triggered by NLRP3/iNOS pathway and attenuated by docosahexaenoic acid (DHA) [Internet]. Clinical Science (Online): an international journal of translational science and medicine. 2020 ;[29 ].[citado 2024 nov. 13 ] Available from: https://doi.org/10.1042/CS20201348
Artigo de periodico
Elevated beta-cell stress levels promote severe diabetes development in mice with MODY4 (2020)
Trojanowski, Bernadette M; Salem, Heba H; Neubauer, Heike; Simon, Eric; Wagner, Martin; Dorajoo, Rajkumar; Boehm, Bernhard O; Labriola, Leticia ; Wirth, Thomas; Baumann, Bernd
Source: Journal of Endocrinology. Unidade: IQ
Subjects: DIABETES MELLITUS, MUTAÇÃO GENÉTICA
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ABNT
TROJANOWSKI, Bernadette M et al. Elevated beta-cell stress levels promote severe diabetes development in mice with MODY4. Journal of Endocrinology, v. 244, n. 2, p. 323-337, 2020Tradução . . Disponível em: https://doi.org/10.1530/JOE-19-0208. Acesso em: 13 nov. 2024.
APA
Trojanowski, B. M., Salem, H. H., Neubauer, H., Simon, E., Wagner, M., Dorajoo, R., et al. (2020). Elevated beta-cell stress levels promote severe diabetes development in mice with MODY4. Journal of Endocrinology, 244( 2), 323-337. doi:10.1530/JOE-19-0208
NLM
Trojanowski BM, Salem HH, Neubauer H, Simon E, Wagner M, Dorajoo R, Boehm BO, Labriola L, Wirth T, Baumann B. Elevated beta-cell stress levels promote severe diabetes development in mice with MODY4 [Internet]. Journal of Endocrinology. 2020 ; 244( 2): 323-337.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1530/JOE-19-0208
Vancouver
Trojanowski BM, Salem HH, Neubauer H, Simon E, Wagner M, Dorajoo R, Boehm BO, Labriola L, Wirth T, Baumann B. Elevated beta-cell stress levels promote severe diabetes development in mice with MODY4 [Internet]. Journal of Endocrinology. 2020 ; 244( 2): 323-337.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1530/JOE-19-0208
Artigo de periodico
Heat shock protein B1 is a key mediator of prolactin-induced beta-cell cytoprotection against oxidative stress (2019)
Terra, Letícia F.; Wailemann, Rosângela A. M; Santos, Ancély Ferreira dos ; Gomes, Vinícius M.; Silva, Railmara Pereira da; Laporte, Anna; Meotti, Flavia Carla ; Terra, Walter Ribeiro ; Palmisano, Giuseppe ; Lortz, Stephan; Labriola, Leticia
Source: Free Radical Biology and Medicine. Unidades: IQ, ICB
Subjects: ESTRESSE OXIDATIVO, DIABETES MELLITUS, PARASITOLOGIA, PROLACTINA, ESTRESSE OXIDATIVO, PROTEÍNAS, ILHOTAS DE LANGERHANS, APOPTOSE, PROTEÍNAS DO CHOQUE TÉRMICO, CITOCINAS, ESPECTROMETRIA DE MASSAS, ANTIOXIDANTES
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ABNT
TERRA, Letícia F. et al. Heat shock protein B1 is a key mediator of prolactin-induced beta-cell cytoprotection against oxidative stress. Free Radical Biology and Medicine, v. 134, p. 394-405, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.freeradbiomed.2019.01.023. Acesso em: 13 nov. 2024.
APA
Terra, L. F., Wailemann, R. A. M., Santos, A. F. dos, Gomes, V. M., Silva, R. P. da, Laporte, A., et al. (2019). Heat shock protein B1 is a key mediator of prolactin-induced beta-cell cytoprotection against oxidative stress. Free Radical Biology and Medicine, 134, 394-405. doi:10.1016/j.freeradbiomed.2019.01.023
NLM
Terra LF, Wailemann RAM, Santos AF dos, Gomes VM, Silva RP da, Laporte A, Meotti FC, Terra WR, Palmisano G, Lortz S, Labriola L. Heat shock protein B1 is a key mediator of prolactin-induced beta-cell cytoprotection against oxidative stress [Internet]. Free Radical Biology and Medicine. 2019 ; 134 394-405.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2019.01.023
Vancouver
Terra LF, Wailemann RAM, Santos AF dos, Gomes VM, Silva RP da, Laporte A, Meotti FC, Terra WR, Palmisano G, Lortz S, Labriola L. Heat shock protein B1 is a key mediator of prolactin-induced beta-cell cytoprotection against oxidative stress [Internet]. Free Radical Biology and Medicine. 2019 ; 134 394-405.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.freeradbiomed.2019.01.023
Trabalho de evento
Síntese da Rosiglitazona em fluxo em microrreator capilar (2018)
Silva, Renan Rodrigues de Oliveira da; Merfels, C. A; Palma, Mauri Sérgio Alves
Source: Anais. Conference titles: Congresso Brasileiro de Engenharia Química/COBEQ. Unidade: FCF
Subjects: DIABETES MELLITUS, SOLVENTE
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ABNT
SILVA, Renan Rodrigues de Oliveira da e MERFELS, C. A e PALMA, Mauri Sérgio Alves. Síntese da Rosiglitazona em fluxo em microrreator capilar. 2018, Anais.. São Paulo: Associação Brasileira de Engenharia Química/ABEQ, 2018. . Acesso em: 13 nov. 2024.
APA
Silva, R. R. de O. da, Merfels, C. A., & Palma, M. S. A. (2018). Síntese da Rosiglitazona em fluxo em microrreator capilar. In Anais. São Paulo: Associação Brasileira de Engenharia Química/ABEQ.
NLM
Silva RR de O da, Merfels CA, Palma MSA. Síntese da Rosiglitazona em fluxo em microrreator capilar. Anais. 2018 ;[citado 2024 nov. 13 ]
Vancouver
Silva RR de O da, Merfels CA, Palma MSA. Síntese da Rosiglitazona em fluxo em microrreator capilar. Anais. 2018 ;[citado 2024 nov. 13 ]
Artigo de periodico
Foveal thickeness reduction after anti-vascular endothelial growth factor treatment in chronic diabetic macular edema (2017)
Willmann, Gabriel; Nepomuceno, Antonio Brunno; Messias, Katharina; Barroso, Leticia; Scott, Ingrid U.; Messias, André Márcio Vieira; Jorge, Rodrigo
Source: International Journal of Ophthalmology. Unidade: FMRP
Subjects: DIABETES MELLITUS, EDEMA MACULAR CISTOIDE, TOMOGRAFIA, DOENÇAS RETINIANAS
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ABNT
WILLMANN, Gabriel et al. Foveal thickeness reduction after anti-vascular endothelial growth factor treatment in chronic diabetic macular edema. International Journal of Ophthalmology, v. 10, n. 5, p. 760-764, 2017Tradução . . Disponível em: https://doi.org/10.18240/ijo.2017.05.17. Acesso em: 13 nov. 2024.
APA
Willmann, G., Nepomuceno, A. B., Messias, K., Barroso, L., Scott, I. U., Messias, A. M. V., & Jorge, R. (2017). Foveal thickeness reduction after anti-vascular endothelial growth factor treatment in chronic diabetic macular edema. International Journal of Ophthalmology, 10( 5), 760-764. doi:10.18240/ijo.2017.05.17
NLM
Willmann G, Nepomuceno AB, Messias K, Barroso L, Scott IU, Messias AMV, Jorge R. Foveal thickeness reduction after anti-vascular endothelial growth factor treatment in chronic diabetic macular edema [Internet]. International Journal of Ophthalmology. 2017 ; 10( 5): 760-764.[citado 2024 nov. 13 ] Available from: https://doi.org/10.18240/ijo.2017.05.17
Vancouver
Willmann G, Nepomuceno AB, Messias K, Barroso L, Scott IU, Messias AMV, Jorge R. Foveal thickeness reduction after anti-vascular endothelial growth factor treatment in chronic diabetic macular edema [Internet]. International Journal of Ophthalmology. 2017 ; 10( 5): 760-764.[citado 2024 nov. 13 ] Available from: https://doi.org/10.18240/ijo.2017.05.17
Trabalho de evento-resumo
Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to type 1 diabetes onset (2015)
Ribeiro, Frederico; Françozo, Marcela Cristina Santiago; Zamboni, Dario Simões; Ramos, Simone Gusmão; Câmara, Niels Olsen Saraiva ; Silva, João Santana da; Sartori, Daniela
Source: Abstracts. Conference titles: INNATE IMMUNITY 2015. Unidades: FMRP, ICB
Subjects: DIABETES MELLITUS, INTESTINOS, BACTÉRIAS, PROTEÍNAS
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ABNT
RIBEIRO, Frederico et al. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to type 1 diabetes onset . 2015, Anais.. Guarujá: SBI, 2015. Disponível em: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I. Acesso em: 13 nov. 2024.
APA
Ribeiro, F., Françozo, M. C. S., Zamboni, D. S., Ramos, S. G., Câmara, N. O. S., Silva, J. S. da, & Sartori, D. (2015). Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to type 1 diabetes onset . In Abstracts. Guarujá: SBI. Recuperado de https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I
NLM
Ribeiro F, Françozo MCS, Zamboni DS, Ramos SG, Câmara NOS, Silva JS da, Sartori D. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to type 1 diabetes onset [Internet]. Abstracts. 2015 ;[citado 2024 nov. 13 ] Available from: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I
Vancouver
Ribeiro F, Françozo MCS, Zamboni DS, Ramos SG, Câmara NOS, Silva JS da, Sartori D. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to type 1 diabetes onset [Internet]. Abstracts. 2015 ;[citado 2024 nov. 13 ] Available from: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I

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