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Filtros : "BIOINFORMÁTICA" "Pinto, Ana Paula" "Rocha, Alisson Luiz da" Limpar

Filtros

USP affiliated authors
- frantz, fabiani gai (2)
- pinto, ana paula (2)
- rocha, alisson luiz da (2)
- rovina, rafael lemes (2)
- silva, adelino sanchez ramos da (2)

Artigo de periodico
TLR4 deletion increases basal energy expenditure and attenuates heart apoptosis and ER stress but mitigates the training-induced cardiac function and performance improvement (2021)
Vicente, Larissa Gaioto de; Muñoz, Vitor Rosetto; Pinto, Ana Paula ; Rovina, Rafael Lemes ; Rocha, Alisson Luiz da ; Marafon, Bruno Brieda ; Tavares, Maria Eduarda de Almeida; Teixeira, Giovana Rampazzo; Ferrari, Gustavo Duarte; Alberici, Luciane Carla ; Frantz, Fabiani Gai ; Simabuco, Fernando Moreira; Ropelle, Eduardo Rochete; Moura, Leandro Pereira de; Cintra, Dennys Esper; Pauli, José Rodrigo; Silva, Adelino Sanchez Ramos da
Source: Life Sciences. Unidades: FCFRP, EEFERP, FMRP
Subjects: BIOINFORMÁTICA, COMPUTAÇÃO APLICADA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VICENTE, Larissa Gaioto de et al. TLR4 deletion increases basal energy expenditure and attenuates heart apoptosis and ER stress but mitigates the training-induced cardiac function and performance improvement. Life Sciences, v. 285, p. 1-10, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.119988. Acesso em: 02 nov. 2024.
APA
Vicente, L. G. de, Muñoz, V. R., Pinto, A. P., Rovina, R. L., Rocha, A. L. da, Marafon, B. B., et al. (2021). TLR4 deletion increases basal energy expenditure and attenuates heart apoptosis and ER stress but mitigates the training-induced cardiac function and performance improvement. Life Sciences, 285, 1-10. doi:10.1016/j.lfs.2021.119988
NLM
Vicente LG de, Muñoz VR, Pinto AP, Rovina RL, Rocha AL da, Marafon BB, Tavares ME de A, Teixeira GR, Ferrari GD, Alberici LC, Frantz FG, Simabuco FM, Ropelle ER, Moura LP de, Cintra DE, Pauli JR, Silva ASR da. TLR4 deletion increases basal energy expenditure and attenuates heart apoptosis and ER stress but mitigates the training-induced cardiac function and performance improvement [Internet]. Life Sciences. 2021 ; 285 1-10.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.lfs.2021.119988
Vancouver
Vicente LG de, Muñoz VR, Pinto AP, Rovina RL, Rocha AL da, Marafon BB, Tavares ME de A, Teixeira GR, Ferrari GD, Alberici LC, Frantz FG, Simabuco FM, Ropelle ER, Moura LP de, Cintra DE, Pauli JR, Silva ASR da. TLR4 deletion increases basal energy expenditure and attenuates heart apoptosis and ER stress but mitigates the training-induced cardiac function and performance improvement [Internet]. Life Sciences. 2021 ; 285 1-10.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.lfs.2021.119988
Artigo de periodico
Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts (2020)
Vicente, Larissa Gaioto de; Pinto, Ana Paula; Muñoz, Vitor R.; Rovina, Rafael Lemes ; Rocha, Alisson Luiz da ; Gaspar, Rafael C.; Silva, Lilian E. C. M. da; Simabuco, Fernando M.; Frantz, Fabiani Gai ; Pauli, José R.; Moura, Leandro P. de; Cintra, Dennys E.; Ropelle, Eduardo R.; Silva, Adelino Sanchez Ramos da
Source: Life Sciences. Unidades: FCFRP, EEFERP, FMRP
Subjects: ECOCARDIOGRAFIA, EXPRESSÃO GÊNICA, BIOINFORMÁTICA, APOPTOSE, EXERCÍCIO FÍSICO, PROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VICENTE, Larissa Gaioto de et al. Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts. Life Sciences, v. 240, p. 1-9, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2019.117107. Acesso em: 02 nov. 2024.
APA
Vicente, L. G. de, Pinto, A. P., Muñoz, V. R., Rovina, R. L., Rocha, A. L. da, Gaspar, R. C., et al. (2020). Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts. Life Sciences, 240, 1-9. doi:10.1016/j.lfs.2019.117107
NLM
Vicente LG de, Pinto AP, Muñoz VR, Rovina RL, Rocha AL da, Gaspar RC, Silva LECM da, Simabuco FM, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts [Internet]. Life Sciences. 2020 ; 240 1-9.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.lfs.2019.117107
Vancouver
Vicente LG de, Pinto AP, Muñoz VR, Rovina RL, Rocha AL da, Gaspar RC, Silva LECM da, Simabuco FM, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts [Internet]. Life Sciences. 2020 ; 240 1-9.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.lfs.2019.117107

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