Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts (2020)
- Authors:
- USP affiliated authors: FRANTZ, FABIANI GAI - FCFRP ; SILVA, ADELINO SANCHEZ RAMOS DA - EEFERP ; VICENTE, LARISSA GAIOTO DE - FMRP ; PINTO, ANA PAULA - FMRP ; ROVINA, RAFAEL LEMES - EEFERP ; ROCHA, ALISSON LUIZ DA - FMRP
- Unidades: FCFRP; EEFERP; FMRP
- DOI: 10.1016/j.lfs.2019.117107
- Subjects: ECOCARDIOGRAFIA; EXPRESSÃO GÊNICA; BIOINFORMÁTICA; APOPTOSE; EXERCÍCIO FÍSICO; PROTEÍNAS
- Keywords: Echocardiogram; Gene expression; Protein content and phosphorylation; Bioinformatics
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2020
- Source:
- Título: Life Sciences
- ISSN: 0024-3205
- Volume/Número/Paginação/Ano: v. 240, art. 117107, p. 1-9, 2020
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
VICENTE, Larissa Gaioto de et al. Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts. Life Sciences, v. 240, p. 1-9, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2019.117107. Acesso em: 12 fev. 2026. -
APA
Vicente, L. G. de, Pinto, A. P., Muñoz, V. R., Rovina, R. L., Rocha, A. L. da, Gaspar, R. C., et al. (2020). Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts. Life Sciences, 240, 1-9. doi:10.1016/j.lfs.2019.117107 -
NLM
Vicente LG de, Pinto AP, Muñoz VR, Rovina RL, Rocha AL da, Gaspar RC, Silva LECM da, Simabuco FM, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts [Internet]. Life Sciences. 2020 ; 240 1-9.[citado 2026 fev. 12 ] Available from: https://doi.org/10.1016/j.lfs.2019.117107 -
Vancouver
Vicente LG de, Pinto AP, Muñoz VR, Rovina RL, Rocha AL da, Gaspar RC, Silva LECM da, Simabuco FM, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts [Internet]. Life Sciences. 2020 ; 240 1-9.[citado 2026 fev. 12 ] Available from: https://doi.org/10.1016/j.lfs.2019.117107 - Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation
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- Hepatic LC3 II/I ratio is not modulated in exercised mice
- Genetic deletion of IL‐6 increases CK‐MB, a classic cardiac damage marker, and decreases UPRmt genes after exhaustive exercise
- TLR4 deletion increases basal energy expenditure and attenuates heart apoptosis and ER stress but mitigates the training-induced cardiac function and performance improvement
- Excessive treadmill training enhances the insulin signaling pathway and glycogen deposition in mice hearts
- Impact of different physical exercises on the expression of autophagy markers in mice
- Moderate, but not excessive, training attenuates autophagy machinery in metabolic tissues
- One bout of aerobic exercise can enhance the expression of Nr1d1 in oxidative skeletal muscle samples
Informações sobre o DOI: 10.1016/j.lfs.2019.117107 (Fonte: oaDOI API)
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