Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts (2020)
- Authors:
- USP affiliated authors: FRANTZ, FABIANI GAI - FCFRP ; SILVA, ADELINO SANCHEZ RAMOS DA - EEFERP ; VICENTE, LARISSA GAIOTO DE - FMRP ; PINTO, ANA PAULA - FMRP ; ROVINA, RAFAEL LEMES - EEFERP ; ROCHA, ALISSON LUIZ DA - FMRP
- Unidades: FCFRP; EEFERP; FMRP
- DOI: 10.1016/j.lfs.2019.117107
- Subjects: ECOCARDIOGRAFIA; EXPRESSÃO GÊNICA; BIOINFORMÁTICA; APOPTOSE; EXERCÍCIO FÍSICO; PROTEÍNAS
- Keywords: Echocardiogram; Gene expression; Protein content and phosphorylation; Bioinformatics
- Agências de fomento:
- Language: Inglês
- Imprenta:
- Publisher place: Philadelphia
- Date published: 2020
- Source:
- Título: Life Sciences
- ISSN: 0024-3205
- Volume/Número/Paginação/Ano: v. 240, art. 117107, p. 1-9, 2020
- Este periódico é de assinatura
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: hybrid
- Licença: unspecified-oa
-
ABNT
VICENTE, Larissa Gaioto de et al. Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts. Life Sciences, v. 240, p. 1-9, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2019.117107. Acesso em: 04 ago. 2025. -
APA
Vicente, L. G. de, Pinto, A. P., Muñoz, V. R., Rovina, R. L., Rocha, A. L. da, Gaspar, R. C., et al. (2020). Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts. Life Sciences, 240, 1-9. doi:10.1016/j.lfs.2019.117107 -
NLM
Vicente LG de, Pinto AP, Muñoz VR, Rovina RL, Rocha AL da, Gaspar RC, Silva LECM da, Simabuco FM, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts [Internet]. Life Sciences. 2020 ; 240 1-9.[citado 2025 ago. 04 ] Available from: https://doi.org/10.1016/j.lfs.2019.117107 -
Vancouver
Vicente LG de, Pinto AP, Muñoz VR, Rovina RL, Rocha AL da, Gaspar RC, Silva LECM da, Simabuco FM, Frantz FG, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Silva ASR da. Tlr4 participates in the responses of markers of apoptosis, inflammation, and ER stress to different acute exercise intensities in mice hearts [Internet]. Life Sciences. 2020 ; 240 1-9.[citado 2025 ago. 04 ] Available from: https://doi.org/10.1016/j.lfs.2019.117107 - Rapamycin did not prevent the excessive exercise-induced hepatic fat accumulation
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- Impact of different physical exercises on the expression of autophagy markers in mice
- TLR4 deletion increases basal energy expenditure and attenuates heart apoptosis and ER stress but mitigates the training-induced cardiac function and performance improvement
- Excessive treadmill training enhances the insulin signaling pathway and glycogen deposition in mice hearts
- The proinflammatory effects of chronic excessive exercise
- Interleukin-6 ablation does not alter morphofunctional heart characteristics but modulates physiological and inflammatory markers after strenuous exercise
Informações sobre o DOI: 10.1016/j.lfs.2019.117107 (Fonte: oaDOI API)
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